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1 ngulate cortex (possibly as a consequence of cortical spreading depression).
2 centage of neurons that showed activation by cortical spreading depression.
3 nisms such as an increased susceptibility to cortical spreading depression.
4 sels in the trigemino-vascular system and in cortical spreading depression.
5 cortical neuronal excitability that leads to cortical spreading depression.
6 na, including hearing, vision, epilepsy, and cortical spreading depression.
7 or the first time it has been shown that the cortical spreading depression activated the trigeminovas
8 gene, was increased without the presence of cortical spreading depression after LEI-106 administrati
9 an cause neurovascular dysfunction and evoke cortical spreading depression, an event that is widely t
10 e significance to migraine of events such as cortical spreading depression and activation of the trig
11 e the typical hypoperfusion responses during cortical spreading depression and alpha-chloralose anaes
12 g evidence for the occurrence of spontaneous cortical spreading depression and peri-infarct depolariz
13 under isoflurane anesthesia showed that both cortical spreading depression and terminal anoxic depola
14 that these gain-of-function effects lead to cortical spreading depression, aura, and potentially mig
15 shold (central) trigeminovascular neurons by cortical spreading depression, but not their activation
16 se results are the first to demonstrate that cortical spreading depression can be blocked in vivo usi
20 blood flow (rCBF) changes that occur during cortical spreading depression (CSD) are considered to be
21 dentified, although, animal models highlight cortical spreading depression (CSD) as a potential candi
22 onal imaging has confirmed the importance of cortical spreading depression (CSD) as the pathophysiolo
24 to detect and characterize complex waves of cortical spreading depression (CSD) evoked with KCL plac
30 during intensive care and were classified as cortical spreading depression (CSD) if they took place i
31 ssing channelrhodopsin to trigger and record cortical spreading depression (CSD) in freely behaving s
47 thought to be the key factor for generating cortical spreading depression (CSD), a pathological mech
49 ession and, if so, the threshold for evoking cortical spreading depression (CSD), a process sharing c
50 n, insufficient to cause infarcts, triggered cortical spreading depression (CSD), a propagating slow
51 2Q mutation exhibit increased propensity for cortical spreading depression (CSD), a propagating wave
54 injected mice, we show that a single wave of cortical spreading depression (CSD), an animal model of
55 nsient neurological deficits associated with cortical spreading depression (CSD), preceding headache
56 V 2.1 channels and are highly susceptible to cortical spreading depression (CSD), the electrophysiolo
59 ocytes during somatosensory stimulations and cortical spreading depression (CSD), the putative mechan
60 ation is optical triggering and recording of cortical spreading depression (CSD), the slowly propagat
62 rpose of this study was to determine whether cortical spreading depression (CSD)--an event believed t
63 geal nociceptors, it significantly reduces a cortical spreading depression (CSD)-induced early respon
64 spontaneous and evoked activity in naive and cortical spreading depression (CSD)-sensitized trigemino
66 nal groups (with [n=7] or without [n=7] HH), cortical spreading depressions (CSD) were elicited to as
67 a2-Na/K ATPase knockout triggers spontaneous cortical spreading depression events that are associated
69 we tested the effect of fremanezumab on the cortical spreading depression-evoked activation of mecha
70 ., seizure) that is evident in human (unlike cortical spreading depression) gives rise to specific an
71 Spreading depolarization, usually termed cortical spreading depression has been proposed as the p
72 g multiwavelength optical intrinsic imaging, cortical spreading depression has been shown to have a t
74 aine aura and headache by demonstrating that cortical spreading depression, implicated in migraine vi
75 to focal cerebral ischaemia, and blocking of cortical spreading depression improved stroke outcome in
76 uncture wound, global cerebral ischemia, and cortical spreading depression in C57BL6 mice; 1 day afte
77 unique to each headache disorder, including cortical spreading depression in migraine, rhythmicity o
80 netic stimulation blocked the propagation of cortical spreading depression in two of eight animals.
82 ral activity in reducing the total number of cortical spreading depressions induced by potassium chlo
83 derlying mechanism for the susceptibility of cortical spreading depression initiation in migraine dis
90 ammation, the central autonomic response and cortical spreading depression might drive cerebrovascula
91 des demonstrated significant activity in the cortical spreading depression model of migraine as we re
95 observed spontaneous, recurring episodes of cortical spreading depression (SD) as early as 20 min po
99 emale hormone levels in these mice modulated cortical spreading depression susceptibility in much the
100 or R192Q mutation are highly susceptible to cortical spreading depression, the electrophysiological
104 ephalic cat cortices, when administered post-cortical spreading depression, transcranial magnetic sti
105 ascular function, one during the propagating cortical spreading depression wave and a second much lon
106 The direct current shift associated with the cortical spreading depression wave was accompanied by ma
107 potassium concentration increased during the cortical spreading depression wave, but recovered and re
110 bited both mechanical and chemically-induced cortical spreading depression when administered immediat
111 study bypasses controversies associated with cortical spreading depression, which is less readily obs
112 excitability, and enhanced susceptibility to cortical spreading depression, which is the electrophysi
113 l, mild, and transient ischaemia can trigger cortical spreading depression without an enduring tissue