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1 C- and non-BCC-infected patients to minimize cross infection.
2 more than 40% without increasing the risk of cross infection.
3 gy with respect to ribotype distribution and cross-infection.
4 g a multi-type Lotka-Volterra framework with cross-infection.
5 lides or aminoglycosides, further indicating cross-infection.
6 hould be instituted with measures to prevent cross-infection.
7 r genomes, but we also find evidence of rare cross-infection.
8  to C difficile infection, but who can cause cross-infection.
9 cia complex frequently occurs as a result of cross infection among individuals with cystic fibrosis.
10 cile as a possibly significant problem, with cross-infection and a distinct ribotype distribution, in
11 tive surveillance for Pseudomonas aeruginosa cross-infection at a large regional adult cystic fibrosi
12  eradicate biofilm and eliminate the risk of cross infection between patients.
13  Phylogenetic analyses revealed evidence for cross infection between the resting and activated CD4+ T
14 sign and ventilation) to limit P. aeruginosa cross-infection between patients with cystic fibrosis.
15 viremic individuals and provide evidence for cross-infection between these 2 cellular compartments.
16 vides a good approximation when the level of cross-infection between vector species is very small.
17 y interventions, such as early treatment and cross-infection control, might restrict the spread of ex
18  related viruses, raising the possibility of cross-infection despite protective isolation.
19                                   In a large cross-infection experiment involving four species of Dro
20 ients from non-colonised patients to prevent cross-infection has been recommended, there is little ev
21 he practice of strict hygiene, P. aeruginosa cross-infection has continued.
22 ryngitis in a boarding school (serotype M5), cross-infection in a hospital burn unit (serotype M76),
23 prevent medical device-related infection and cross-infection in the hospital would yield benefit with
24 inetobacter baumannii are highly clonal, and cross-infection investigations can be difficult.
25    Otherwise, particularly when the level of cross-infection is high, the two-host, two-vector formul
26 ommended, there is little evidence that such cross-infection is widespread.
27 early with respect to the first variant, the cross-infection level does not impact the detection time
28 ts with cystic fibrosis despite conventional cross-infection measures.
29 i21 coevolve and diversify to form elaborate cross-infection networks.
30 s and/or emergence of barriers to successful cross-infection occurs rapidly.
31    This limited human epidemic resulted from cross-infection of prairie dogs by imported African rode
32                                   Reports of cross-infection of shared cystic-fibrosis-specific P. ae
33                                              Cross-infections of SIVagm.sab92018 and SIVagm.ver644 in
34  time, its infectiousness advantage and, its cross-infection on the novel variant's detection time, a
35 s following passive immunization, sequential cross-infection, or vaccination with inactivated virus.
36 ht the importance of global surveillance and cross-infection prevention in averting the emergence of
37 l technologies have several drawbacks (i.e., cross-infection risk, filtration efficiency improvements
38  staff-to-staff infections, student-to-staff cross infections, student-to-student infections, and env
39                                              Cross-infection studies of 81,926 host-phage pairs delin
40  tuberculosis from four presumed episodes of cross-infection were examined.
41                We identified two clusters of cross-infection with indistinguishable isolates of ribot