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1 interaction of pathogen strains, mediated by cross immunity.
2                 Pertussis vaccine may induce cross-immunity.
3  otherwise identical strains compete through cross-immunity.
4  repeatedly interpreted as inconsistent with cross-immunity.
5 tions expected in the presence or absence of cross-immunity.
6  We suggest that this may be an indicator of cross-immunity.
7 e been insufficient to identify even intense cross-immunity.
8 strains into a single parameter representing cross-immunity.
9 distinct serotypes, resulting in inefficient cross-immunity.
10  genotypes, determining the degree of strain cross-immunity.
11 er of antigenic types that interact via host cross-immunity.
12 n be modeled through coinfection or complete cross-immunity.
13 se lpf phase-off variants were able to evade cross-immunity.
14 ction ynamics, presumably via heterosubtypic cross-immunity.
15 s disempowering the hypothesis of protective cross-immunity.
16 formulation, assuming polarized immunity and cross-immunity act to reduced transmission probability.
17                    These data suggested that cross-immunity against LP fimbrial proteins cannot be ev
18 still propagate in environments with reduced cross-immunity among different strain groups, even after
19 tructed to evaluate how different degrees of cross-immunity among M. tuberculosis groups could affect
20                            Assuming complete cross-immunity among strains, namely, recovery from any
21 f immune-mediated interactions, arising from cross-immunity and antibody-dependent enhancement, betwe
22 ns unclear how such ecologic competition via cross-immunity and antigenic mutations that allow immune
23 rogenitor are most successful under moderate cross-immunity and frequent re-infections, and (3) the i
24 sk of re-infection, thus showing the role of cross-immunity and its association with protection.
25 er replacement depends on the specificity of cross-immunity and on the underlying pathogen mutation r
26 the pathogen population requires long-ranged cross-immunity and sufficiently large population sizes.
27 idence that T cells are responsible for this cross-immunity and that cross-stimulation of T cells als
28 ibility associated to each influenza strain, cross-immunity and the timing of the onset of the second
29 998, we are able to estimate the strength of cross-immunity between each virus-pairs, the timing and
30                                              Cross-immunity between HPV types is consistent with epid
31                                The degree of cross-immunity between human papillomavirus (HPV) types
32                                  The lack of cross-immunity between O-antigen serotypes appears to co
33 mplified in locations where there is reduced cross-immunity between originating strain groups.
34 cent epidemiological modelling suggests that cross-immunity between RSV, HMPV and human parainfluenza
35  lpf phase variation is a mechanism to evade cross-immunity between Salmonella serotypes, thereby all
36  phase variation may be a mechanism to evade cross-immunity between Salmonella serotypes.
37 ariation instead may be a mechanism to evade cross-immunity between Salmonella serotypes.
38 ation of LP fimbriae is a mechanism to evade cross-immunity between serotypes Enteritidis and Typhimu
39 gen of B. parapertussis confers asymmetrical cross-immunity between the causative agents of whooping
40  effective reproduction number or short-term cross-immunity between these viruses can explain the dyn
41 e core loci (DCL) proteins is complicated by cross-immunity between variants.
42                                 We find that cross-immunity can produce odds ratios of infection comp
43 type but only temporary partial immunity, or cross immunity (CI), to others.
44  Increasing population density or decreasing cross-immunity could not fully explain the observed patt
45                                     Although cross-immunity diminishes as further mutations accumulat
46 used estimates of seasonality, immunity, and cross-immunity for human coronavirus OC43 (HCoV-OC43) an
47 munity in all types/subtypes, with strongest cross-immunity from A(H1N1) against A(H3N2).
48  longitudinal survey to determine the effect cross-immunity has on the prevalence of multiple infecti
49             We show that depending on strain cross-immunity, heterogeneity of the host population, an
50 he interaction of viral replication rate and cross-immunity imprint host population immunity, which i
51                          We find evidence of cross-immunity in all types/subtypes, with strongest cro
52 and we address the role of host mobility and cross-immunity in shaping possible dominance/co-dominanc
53 ssibility, and antigenic changes that reduce cross-immunity induced by previous infections or vaccina
54  structure (e.g. particular host behaviours, cross-immunity, interspecific competition) could be affe
55 aphy and, crucially, a short-lived period of cross-immunity is sufficient.
56                            Variations in the cross-immunity level induce a transition between presenc
57 d wave can occur whenever R 01 < 1.5 or when cross-immunity levels are less than 0.58 for our estimat
58 se this information to explore the impact of cross-immunity levels on the dynamics of the second wave
59                                              Cross-immunity may cause significant type replacement fo
60                          Endemic coronavirus cross-immunity may play a role in protection against sev
61 se domains act as NAD-depleting toxins, with cross-immunity observed between non-cognate toxin-immuni
62 investigate the effects of recombination and cross-immunity on the invasion of new strains.
63 ination, strain competition mediated through cross-immunity structures the parasite population into a
64 suggest that mixed species infections elicit cross-immunity that can modulate pathogenicity and disea
65 ree modes to discuss the relationships among cross-immunity, the basic reproductive rates of each str
66      The functional relevance of preexisting cross-immunity to severe acute respiratory syndrome coro
67  of two strains of influenza interacting via cross-immunity to simulate two temporal waves of influen
68 y mixed populations, our model confirms that cross-immunity to strains sharing alleles at antigenic l
69 turation patterns, anamnestic responses, and cross-immunity to the common-cold coronaviruses.
70 ccinology approaches could enhance intrinsic cross-immunity to these paramyxoviruses and approaches t
71 nt epitopes typically increases the range of cross-immunity values in which chaotic strain dynamics a
72 period of time have shown how the effects of cross-immunity vary with the time between exposures and
73 rain disease transmission model with perfect cross immunity where environmental transmission is broad