1 nfiltration with characteristic perivascular cuffing.

2 ) brains exhibited a scarcity of lymphocytic cuffing and displayed reduced numbers of infiltrating le

3 re associated with perivascular inflammatory cuffing and parenchymal microglial activation but preced

4 ent perivascular/peribronchiolar lymphocytic cuffing and well-formed granulomas with few fungal eleme

5 fluid eosinophilia, peribronchiolar cellular cuffing, and Ig subclass switching, with increase in OVA

6                                   Thus, hand-cuffing depends on a highly ordered oligomerization of p

7 and bronchiolar hyperplasia and perivascular cuffing in ferret lung tissue, as seen previously in inf

8 ealed CD3-positive T lymphocyte perivascular cuffing in the brain.

9 aive mice caused meningitis and perivascular cuffing not only in the brain parenchyma but also in the

10  with minimal demyelination and perivascular cuffing of inflammatory cells.

11 his deficiency was associated with increased cuffing of T cells around the vessels in the lungs of th

12 lly administered HMB attenuated perivascular cuffing, preserved the integrity of the blood-brain barr

13 s, gliosis, angiopathy with mononuclear cell cuffing, progressive calcification affecting basal gangl

14  local interaction at the 0 (ionic) layer by cuffing Sx1A and Sb2.

15 ed a local interaction at the ionic layer by cuffing syntaxin 1A and synaptobrevin 2, similar to the

16                    Melanoma cells were found cuffing the outside of vessels around the tumor.

17              Neuropathic pain was induced by cuffing the right sciatic nerve of C57BL/6J mice.

18  acute phase of disease whereas perivascular cuffing was the predominant feature during chronic disea

19 ytes and macrophages as well as perivascular cuffing were seen early in the infection.