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1 companied by the enlargement of the adjacent cutaneous nerve.
2 ent excitability properties of the activated cutaneous nerves.
3 iators, rather than through its receptors on cutaneous nerves.
4 e chronically denervated by resecting dorsal cutaneous nerves.
5 lineages such as adipocytes, melanocytes and cutaneous nerves.
6 ase (HRP) after transection of the posterior cutaneous nerve and inferior gluteal nerve, and found th
7 se transcription-PCR was performed on normal cutaneous nerve and ventral root and on graft preparatio
8 icant increase in substance P content of the cutaneous nerves and an accompanying increase in itching
9  of physical contact between melanocytes and cutaneous nerves and for the first time strongly suggest
10  this study confirm the presence of VAChT in cutaneous nerves and in both epidermal melanocytes and k
11 estinal peptide (VIP) has also been found in cutaneous nerves and mRNA, for the VIP receptor vasoacti
12 t ectoderm is necessary for the formation of cutaneous nerves, and for the normal growth and guidance
13 th the observed projections of dorsal roots, cutaneous nerves, and individual axons.
14 sion of adhesion molecules, proliferation of cutaneous nerves, and upregulation of neuropeptides.
15 levels, axons projecting along an individual cutaneous nerve are found together in bundles that are s
16 , axons normally destined for the 'deprived' cutaneous nerve are not segregated appropriately within
17 ot spots" for tumor formation, and implicate cutaneous nerves as mediators of tumorigenesis.
18 of Golgi cells were evoked by stimulation of cutaneous nerves at stimulus intensities that activated
19                                           14 cutaneous nerve biopsies revealed loss of myelinated ner
20 nerve group block (PNGB) and lateral femoral cutaneous nerve block (LFCNB) on block range and analges
21                         Stimulation of other cutaneous nerve branches serving the dorsum of the ipsil
22 ed vigorously by denervated and reinnervated cutaneous nerve but minimally by ventral root.
23 g that application of ProTx-II to desheathed cutaneous nerves completely blocked the C-fiber compound
24   Electrical stimulation to segmental dorsal cutaneous nerves (DCNs) activates a nociceptive sensorim
25          Stimulation of rat segmental dorsal cutaneous nerves (DCNs) evokes the nociceptive intersegm
26  cells, these changes do not account for the cutaneous nerve deficit.
27                                              Cutaneous nerve endings are assumed to change due to der
28   Thus, calcitonin gene-related peptide from cutaneous nerve endings plays a key role in the local im
29 hat UVR, by causing the release of CGRP from cutaneous nerve endings, triggers mast cell release of T
30 iation (UVR) causes CGRP to be released from cutaneous nerve endings, we examined whether CGRP partic
31 n in some patients is caused by the anterior cutaneous nerve entrapment syndrome (ACNES).
32                                   Similarly, cutaneous nerves established a "normal" somatotopic map
33                                              Cutaneous nerves extend throughout the dermis and epider
34 y delivered IL-31 induced an increase in the cutaneous nerve fiber density in lesional skin in vivo.
35 idermal Langerhans' cell (LC) frequency; and cutaneous nerve fiber expression of protein gene product
36 tinib, a significantly greater volume of the cutaneous nerve fibers (67.3%) in the plantar skin was p
37 l-derived TNF, can promote the elongation of cutaneous nerve fibers during contact hypersensitivity i
38 ntrolled skin cooling to specifically target cutaneous nerve fibers has the potential to be useful fo
39 ate a neuroprotective effect of Foretinib on cutaneous nerve fibers in experimental diabetic neuropat
40 l-specific T cells are in close proximity to cutaneous nerve fibers in vivo.
41  capsaicin produced morphological changes in cutaneous nerve fibers that would account for its analge
42 terminate in the epidermis, as well as other cutaneous nerve fibers.
43 fects on the sciatic nerve and on downstream cutaneous nerve fibers.
44 to be facilitated by cytokines that activate cutaneous nerve fibers; however, the molecular component
45           This study was performed to assess cutaneous nerve fibre loss in conjunction with temperatu
46      We find there is a critical period when cutaneous nerve formation requires target ectoderm.
47 4 reversed the effect of ectoderm removal on cutaneous nerve formation, but did not act as a chemoatt
48 e ectoderm, work in concert to ensure proper cutaneous nerve formation.
49 ix remodeling, and processes associated with cutaneous nerve function.
50  precisely, in these cases the medial dorsal cutaneous nerve got injured during the fascial opening o
51 tally, bundles of axons projecting along one cutaneous nerve gradually join one another, becoming inc
52  first report demonstrating the induction of cutaneous nerve growth factor by sensory nerve-derived n
53 ve growth factor; however, the regulation of cutaneous nerve growth factor production still remains t
54 the distribution of the lateral antebrachial cutaneous nerve in six extremities (2.6%).
55 ated by light microscopic techniques in fine cutaneous nerves in naive animals.
56 nces for the maintenance and regeneration of cutaneous nerves in normal skin and during inflammation
57  To characterize the regenerative pattern of cutaneous nerves in simian immunodeficiency virus (SIV)-
58 or the initial formation of one of the major cutaneous nerves in the embryonic chick limb.
59 tors were 61%, 43% and 48%, respectively, in cutaneous nerves in the inflamed paw compared to 48%, 22
60                                              Cutaneous nerves innervating dorsal skin of KC-Tie2 anim
61 of calcitonin gene-related protein (CGRP), a cutaneous nerve neuropeptide, on NO production in human
62 e selective cutaneous C-fiber deficit in the cutaneous nerves of naked mole-rats is unlikely to be du
63  upregulation of NGF receptor (NGF-R) in the cutaneous nerves of psoriatic plaques.
64 on deletion, more trkC+ afferents project in cutaneous nerves on the deleted side of NT3-treated embr
65 ither from increased collateral formation in cutaneous nerve or from increased collateral pruning in
66 n between sensory and motor nerve, grafts of cutaneous nerve or ventral root were denervated, reinner
67 on of somatic afferents in posterior femoral cutaneous nerve (PFCN) could modulate the micturition re
68 ntify regenerating neurons demonstrated that cutaneous nerve preferentially supported cutaneous axon
69 ought to develop and validate a standardized cutaneous nerve regeneration model and to define the rat
70 s of reinnervation occur after injury to the cutaneous nerves: regenerative growth of the injured ner
71  Graded electrical stimulation of muscle and cutaneous nerves revealed that long-lasting depressions
72 ns that ultimately projected along different cutaneous nerves showed increased intermixing in the spi
73 , obturator nerve trunk, and lateral femoral cutaneous nerve simultaneously.
74 o chronic incomplete SCI, using non-invasive cutaneous nerve stimulation.
75 re significantly more abundant in denervated cutaneous nerve than in denervated ventral root, but tha
76     Motoneurons maintain more collaterals in cutaneous nerve than in muscle nerve, even without muscl
77 a is a mononeuropathy of the lateral femoral cutaneous nerve that can lead to significant disability
78 eptide, neuropeptides known to be present in cutaneous nerves, to examine their possible functions in
79 cr4, initiates blood vessel remodeling along cutaneous nerve trajectories to establish the proper pat
80  tungsten microelectrode was inserted into a cutaneous nerve, usually the superficial peroneal close
81              The femoral and lateral femoral cutaneous nerves were consistently covered when S-FICB-H
82 normally; that is, some sensory axons formed cutaneous nerves while other sensory axons projected to
83 e instead misdirected along an inappropriate cutaneous nerve, while others have seemingly failed to r