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6 enic factor CCBE1 or VEGFR3 function, appear cyanotic and die shortly after birth due to failure of l
9 cally stable and without a hemoglobinopathy, cyanotic cardiac condition, or severe hypoxemia, the int
10 justed rate ratio, 95% confidence interval): cyanotic CHD (6.44, 3.95-10.50), endocardial cushion def
11 ailed prenatal or early newborn detection of cyanotic CHD (CCHD) can have disastrous consequences.
12 and tetralogy of Fallot, two common forms of cyanotic CHD lesions, as well as dilated and hypertrophi
13 ngitudinal observational study, fetuses with cyanotic CHD underwent late gestational fetal cardiovasc
23 is did not influence outcome in infants, but cyanotic children showed worse reperfusion injury and cl
28 Between 1988 and 1995, 162 patients with cyanotic congenital heart disease (mean age 37 years, ra
29 g nonlinear mixed effects model, presence of cyanotic congenital heart disease (odds ratio, 7.35; p <
30 igned infants 92 days of age or younger with cyanotic congenital heart disease and a systemic-to-pulm
31 erebrovascular events in adult patients with cyanotic congenital heart disease and to evaluate any co
35 he recommendation that routine screening for cyanotic congenital heart disease be added to the panel
36 and BNP are markedly elevated in adults with cyanotic congenital heart disease despite reduced body w
37 ascular events are a serious complication of cyanotic congenital heart disease in infants and childre
40 Chronic hypoxia (CH) present in infants with cyanotic congenital heart disease may be responsible for
43 Tetralogy of Fallot (TOF) is the most common cyanotic congenital heart disease, and sudden cardiac de
44 ble increase in basal coronary blood flow in cyanotic congenital heart disease, flow reserve remains
45 iteria were trainees intubating infants with cyanotic congenital heart disease, known or suspected di
51 Nkx2-5(Deltaenh/Deltaenh) mice recapitulated cyanotic conotruncal defects seen in patients with NKX2-
52 who undergo heart surgery have a congenital cyanotic defect in which the heart is chronically perfus
53 ho undergo cardiac surgery have a congenital cyanotic defect where the heart is chronically perfused
58 Tetralogy of Fallot (TOF)-the most common cyanotic heart defect in newborns-has evidence of multip
60 Lack of past cardiac surgery (p = 0.04), cyanotic heart disease (p = 0.03), and early postoperati
61 ffered to patients with a variety of complex cyanotic heart disease at younger ages, and has resulted
62 ry syncytial virus infection, or presence of cyanotic heart disease or residual right-to-left intraca
64 DS criteria more than 24 h before screening, cyanotic heart disease, active perinatal lung disease, a
65 Fallot, transposition of the great arteries, cyanotic heart disease, Ebstein anomaly, and Fontan circ
66 diopathic pulmonary hypertension, congenital cyanotic heart disease, morbid obesity associated with s
72 phometric or hemodynamic differences between cyanotic infants with critical PS and asymptomatic infan
79 here was no difference in oxytocin levels in cyanotic patients compared with control subjects (P=0.49
84 tion and hematocrit significantly greater in cyanotic patients than in control subjects (82+/-6 versu
88 hest potential for vascular injury: younger, cyanotic patients with longer pump times and longer post
90 ein kinases (MAP kinases) in 15 infants with cyanotic (SaO2<85%) or acyanotic (SaO2>95%) heart defect
91 ent animals and found that these mice become cyanotic shortly before death because of lung maturation
92 ed generalized swelling of the third finger, cyanotic skin, and fingernail splitting on the second fi
93 Caesarean section at embryonic day 18.5 were cyanotic, suffered from respiratory distress, and failed