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1 on of beta2-adrenergic receptor alters cAMP (cyclic adenosine 3',5'-monophosphate) and triggers PKA (
2 , enabling a comparison of the mechanisms of cyclic adenosine 3',5'-monophosphate biosynthesis.
3                  Regulation of intracellular cyclic adenosine 3 ',5 '-monophosphate (cAMP) is integra
4                                              Cyclic adenosine 3', 5'-monophosphate (cAMP) is a ubiqui
5                An elevation of intracellular cyclic adenosine 3', 5'-monophosphate (cAMP) is known to
6                               I-LTD requires cyclic adenosine 3', 5'-monophosphate (cAMP)-dependent p
7  4B (PDE4B) inactivates the second messenger cyclic adenosine 3',5' monophosphate (cAMP) and abrogate
8  of all SCF deletion constructs treated with cyclic adenosine 3',5'-monophosphate (cAMP) and forskoli
9  the hypothesis that increased intracellular cyclic adenosine 3',5'-monophosphate (cAMP) could inhibi
10                                    Increased cyclic adenosine 3',5'-monophosphate (cAMP) has been pre
11              Experiments were performed with cyclic adenosine 3',5'-monophosphate (cAMP) in the pipet
12                                              Cyclic adenosine 3',5'-monophosphate (cAMP) is a key sec
13                                              Cyclic adenosine 3',5'-monophosphate (cAMP) is a recogni
14 iocyte proliferation and secretin-stimulated cyclic adenosine 3',5'-monophosphate (cAMP) levels.
15  secretin receptor (SR) gene expression, and cyclic adenosine 3',5'-monophosphate (cAMP) levels.
16 or (SR) gene expression and secretin-induced cyclic adenosine 3',5'-monophosphate (cAMP) synthesis an
17 ellular development is organized by periodic cyclic adenosine 3',5'-monophosphate (cAMP) waves, which
18 tes generate a pulse of the chemoattractant, cyclic adenosine 3',5'-monophosphate (cAMP), every 6 min
19 PDE3A inhibition generates compartmentalized cyclic adenosine 3',5'-monophosphate (cAMP), which furth
20                                              Cyclic adenosine 3',5'-monophosphate (cAMP)-elevating ag
21 ical level, CC2D1A transduces signals to the cyclic adenosine 3',5'-monophosphate (cAMP)-protein kina
22 ocess induced by bicarbonate and mediated by cyclic adenosine 3',5'-monophosphate (cAMP).
23 feron-y production in CD8(+) T cells through cyclic adenosine 3',5'-monophosphate (cAMP)/cAMP respons
24 ctivation of parallel fibres (PFs) induces a cyclic adenosine 3'5'-monophosphate (cAMP) and protein k
25 tion in alpha1A-transfected cells, inhibited cyclic adenosine 3'5'-monophosphate (cAMP) formation in
26  PKC agonists induced mkp1 mRNA levels, only cyclic adenosine 3':5'-monophosphate (cAMP)-PKA inhibiti
27                                              Cyclic adenosine 3, 5-monophosphate (cAMP) levels were m
28    We have examined the cytotoxic effects of cyclic adenosine-3', 5'-monophosphate (cAMP) derivatives
29 ace of intact cells revealed the presence of cyclic adenosine 3':5'-monophosphate-(cAMP) dependent pr
30                   Sutherland, who discovered cyclic adenosine 3'-5'-monophosphate (cyclic AMP) and op
31 cting with secretin receptor (SR) activating cyclic adenosine 3',5'-monophosphate/cystic fibrosis tra
32            Similarly, in mammalian cells the cyclic adenosine 3',5'-monophosphate-dependent protein k
33                      Agonists that stimulate cyclic adenosine 3',5'-monophosphate production led to a
34  receptor activation to adenylyl cyclase and cyclic adenosine 3'-5' monophosphate production and the
35 tion of A2B receptors recruits the canonical cyclic adenosine 3',5'-monophosphate-protein kinase A si
36                                          The cyclic adenosine 3',5'-monophosphate receptor protein (C
37 e of protein kinase A (PKA) and dopamine and cyclic adenosine 3',5' monophosphate-regulated phosphopr
38 ian transcription factors-nuclear receptors, cyclic adenosine 3',5'-monophosphate-regulated enhancer
39 in kinase, mitogen-activated protein kinase, cyclic adenosine 3',5'-monophosphate response element bi
40 modulin-dependent protein kinase (CaMK), and cyclic adenosine 3',5'-monophosphate-responsive element-
41 or CaMK kinase in mediating mGluR5-dependent cyclic adenosine 3',5'-monophosphate-responsive element-
42 2+)/calmodulin-dependent protein kinase, and cyclic adenosine 3',5'-monophosphate-responsive element-
43 te after bile duct ligation by activation of cyclic adenosine 3', 5'-monophosphate signaling.
44                                          The cyclic adenosine 3',5'-monophosphate signalling pathway
45 ts result in the sustained activation of the cyclic adenosine 3',5'-monophosphate system in specific
46                                The activated cyclic adenosine 3',5'-monophosphate system leads to the