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1  inhibitors of tTG (monodansylcadaverine and cystamine).
2 more than a hundred times more potently than cystamine.
3 ium ionophore and prevented by the inhibitor cystamine.
4  formation is inhibited by the TG inhibitor, cystamine.
5  density, cysteamine (0.55%) was better than cystamine (0.55%), and the viscous Cystadrops (0.55%) wa
6 NA complex via a disulfide bond, 2'-deoxy-6-(cystamine)-2-aminopurine (d6Cys2AP) was synthesized and
7 H induces perihematomal tTG upregulation and cystamine, a tTG inhibitor, reduces ICH-induced brain sw
8                               Cysteamine and cystamine also augmented basal intracellular cysteamine
9                                              Cystamine also decreased HIV-1 protein expression in CBM
10                  We previously reported that cystamine and a metabolic cystine precursor inactivate P
11 arent KI = 1.1 mM), and it is inactivated by cystamine and buthionine sulfoximine.
12 d monocyte/macrophage cultures revealed that cystamine and cysteamine possess significant antiviral p
13 y state percentage yields of cysteamine from cystamine and pantethine during the transit through OHSC
14 ubstrate specificity, and inhibition by GSH, cystamine, and transition state analog sulfoximines.
15 in the pathogenesis of HD, and they identify cystamine as a potential therapeutic strategy for treati
16 sized via degradation of a N,N'-bis(acryloyl)cystamine (BAC) cross-linked core out of a non-degradabl
17  degradable crosslinker N, N'-bis (acryloyl) cystamine (BAC), sulfated 2-acrylamido-2-methyl-1-propan
18 fferent amounts of poly(triethylenetetramine/cystamine bisacrylamide) (p(TETA/CBA) and its pegylated
19 lated counterpart, poly(triethylenetetramine/cystamine bisacrylamide)- poly(ethylene glycol) (p(TETA/
20       The bioreducible cationic polymer poly(cystamine bisacrylamide-diamino hexane) (p(CBA-DAH)) was
21 led onto a gold quartz crystal electrode via cystamine bond.
22 hydroxyproline, methylhistidine, tryptophan, cystamine, carnitine, and trimethylamine were lower.
23 noxalin-1-one (ODQ), methylene blue (M-BLU), cystamine (CYS)), L-ARG had only excitatory effects, whe
24 cyclase inhibitors methylene blue (M-BLU) or cystamine (CYS), NO donors had only excitatory effects,
25 oxadiazolo[4,3, -a]quinoxalin-1-one (ODQ) or cystamine (CYS); (ii) the adenylate cyclase inhibitors n
26          We therefore studied the actions of cystamine, cysteamine and several reference thiol agents
27                                  Compared to cystamine, cysteamine showed benefits in terms of crysta
28  We found here that the reversible inhibitor cystamine decreases the enzymatic activity of TGase from
29 ontrast, the reduced forms of disulfiram and cystamine, diethyl dithiocarbamate and cysteamine, respe
30 armacological treatment with mithramycin and cystamine down-regulates ESET gene expression and reduce
31  after the appearance of abnormal movements, cystamine extended survival, reduced associated tremor a
32                                              Cystamine given intraperitoneally entered brain where it
33  with the thiol cysteamine and its disulfide cystamine have demonstrated dramatic cytoprotection in e
34 s with net charges, oxidized glutathione and cystamine, however, the apparent Bronsted coefficients w
35 s with other GCL, buthionine sulfoximine and cystamine inactivate the Arabidopsis enzyme but with ina
36 spectrometric analysis of peptide digests of cystamine-inactivated, carbamidomethylated PKCepsilon de
37                   These studies suggest that cystamine inactivates the enzyme by blocking substrate a
38  gamma-GCS renders the enzyme insensitive to cystamine inactivation without significantly affecting t
39 glutaminase inhibitors dansyl-cadaverine and cystamine, indicating that apoptosis of Mphi is dependen
40                                Additionally, cystamine inhibited caspase-3 activity to the same exten
41                                      In situ cystamine inhibited in a concentration-dependent manner
42  or an antisense for tTG, demonstrating that cystamine inhibits caspase activity independently of any
43                   In this study we show that cystamine inhibits recombinant active caspase-3 in a con
44                                              Cystamine injection decreased TGase activity in HPT with
45           Similarly to the mammalian enzyme, cystamine is a time-dependent, irreversible inhibitor of
46                        At low concentrations cystamine is an uncompetitive inhibitor of caspase-3 act
47 t has been suggested by several studies that cystamine labels an active site Cys residue essential fo
48       Thus, these observations indicate that cystamine may have the potential to limit HIV-1 replicat
49          However, it has been suggested that cystamine may inhibit other thiol-dependent enzymes in a
50              These findings demonstrate that cystamine may prolong neuronal survival and delay the on
51                               The IC(50) for cystamine-mediated inhibition of caspase-3 activity in v
52                  In conclusion, we show that cystamine-mediated TGase inhibition directly releases HP
53                            Interestingly the cystamine-mediated TGase inhibition reduced aggressive b
54 ly and efficiently leads to cysteamine (half-cystamine) modification of a single site on Gbeta, likel
55 f silver underpotentially deposited onto the cystamine modified-Au-electrode surface is used as the r
56                    This inhibitory effect of cystamine occurred with all five HIV-1 strains (both lab
57                    The inhibitory effects of cystamine on HIV-1 did not appear to be caused by toxici
58                               The effects of cystamine on the human immunodeficiency virus (HIV-1) ex
59 fter ICH and the effects of a tTG inhibitor, cystamine, on ICH-induced brain edema and neurological d
60 ng the enzymes reported to be inactivated by cystamine, only one Cys residue is invariant (Cys-319 in
61 traperitoneally with either a tTG inhibitor, cystamine, or vehicle.
62 s approach we have tracked the metabolism of cystamine, pantethine and CoA in the extracellular space
63                      Finally, treatment with cystamine resulted in a robust increase in the levels of
64 mice with the pan-transglutaminase inhibitor cystamine resulted in significantly diminished CIA patho
65 D mice, using the transglutaminase inhibitor cystamine, significantly extended survival, improved bod
66                                              Cystamine suppressed HIV-1 expression in CBMDM and lymph
67 and [3H]-thymidine incorporation at doses of cystamine that inhibit the virus.
68               A literature search identified cystamine, thiamine disulfide, and disulfiram as compoun
69                              The addition of cystamine to cultures of HIV-1 chronically infected CBMD
70 stration of the TGase competitive inhibitor, cystamine, to transgenic mice expressing exon 1 of hunti
71 topathic effect (CPE), respectively, whereas cystamine-treated cultures lacked the giant syncytia or
72                                              Cystamine treatment attenuated ICH-induced brain swellin
73                                Unexpectedly, cystamine treatment increased transcription of one of th
74                                              Cystamine treatment normalized transglutaminase and GGEL
75 fected CBMDM or lymphocyte cultures (without cystamine treatment) demonstrated giant syncytium format
76 h with 5.7 at % Br that further reacted with cystamine under microwave irradiation, resulting in a pr
77            Reductive alkylation of bis-tosyl-cystamine was integrated into the synthetic route, elimi
78 profibrotic pantothenic acid and pro-oxidant cystamine, we tested this pathway in the pathophysiology
79                  Oxidized dithiothreitol and cystamine were reduced to a lesser degree.
80                  Similarly administration of cystamine, which can inhibit transglutaminase, prolonged