ライフサイエンスコーパス: cystitis

1 ymptomatic bacteriuria or bladder infection (cystitis).

2 efractory bladder pain syndrome/interstitial cystitis.

3 paring antimicrobial for acute uncomplicated cystitis.

4 GBS may suppress bladder inflammation during cystitis.

5 biotic-exposed sister with chronic recurrent cystitis.

6 nce of painful bladder syndrome/interstitial cystitis.

7 K, and ERK1/2 in the inflamed bladder during cystitis.

8 r than being attributed to radiation-induced cystitis.

9 ry bladder and in bladder hypertrophy during cystitis.

10 RG with intermediate and chronic CYP-induced cystitis.

11 ed to prevent ifosfamide-induced hemorrhagic cystitis.

12 )-IR was also increased (P < or = 0.01) with cystitis.

13 ders such as overactive bladder syndrome and cystitis.

14 AI contributes to UPEC pathogenesis in acute cystitis.

15 scade in a well-characterized mouse model of cystitis.

16 cord injury, bladder outlet obstruction, or cystitis.

17 diseases, e.g., irritable bowel syndrome or cystitis.

18 al for UPEC virulence in the murine model of cystitis.

19 nd is a potential biomarker for interstitial cystitis.

20 tion of p-CREB-IR cells expressed p-Trk with cystitis.

21 nic (10 days) cyclophosphamide (CYP)-induced cystitis.

22 YP injection and was maintained with chronic cystitis.

23 cells in the L4-L5 DRG was not altered with cystitis.

24 fold; P < or = 0.05) after acute and chronic cystitis.

25 e resistance in pyelonephritis as well as in cystitis.

26 in L1 DRG with chronic (10 days) CYP-induced cystitis.

27 ed urinary tract plasticity with CYP-induced cystitis.

28 Trk-IR with acute and/or chronic CYP-induced cystitis.

29 nic (10 days) cyclophosphamide (CYP)-induced cystitis.

30 sis, interstitial nephritis, and hemorrhagic cystitis.

31 omarkers and shed light into the etiology of cystitis.

32 lexia in the clinical syndrome, interstitial cystitis.

33 ed infections of the urinary tract including cystitis.

34 single most prevalent clonal group in acute cystitis.

35 tance P and neurokinin-1 (NK-1) receptors in cystitis.

36 ly evaluate the importance of substance P in cystitis.

37 onic model of cyclophosphamide (CYP)-induced cystitis.

38 ladder function observed in animal models of cystitis.

39 the bacterial properties necessary to cause cystitis.

40 amide to reduce the incidence of hemorrhagic cystitis.

41 ive pathological changes, including ketamine cystitis.

42 utic target for the treatment of hemorrhagic cystitis.

43 lH significantly protected mice from chronic cystitis.

44 ry tract infections, clinically described as cystitis.

45 icroenvironment characteristic of high-titer cystitis.

46 emia and was not associated with hemorrhagic cystitis.

47 on UPEC virulence in an acute mouse model of cystitis.

48 ot significantly correlated with hemorrhagic cystitis.

49 requency and decreased bladder capacity with cystitis.

50 g is an important pathway mediating ketamine cystitis.

51 A) strains isolated from patients with acute cystitis.

52 acellular bacterial communities during acute cystitis.

53 d cyclooxygenase (COX)-2 in ketamine-induced cystitis.

54 herichia coli (UPEC) is the leading cause of cystitis.

55 ild-type mice in a well-established model of cystitis.

56 riate first-line therapies for uncomplicated cystitis.

57 ssociated with increasing bacteriuria during cystitis.

58 athogens have complicated treatment of acute cystitis.

59 complications included transient hemorrhagic cystitis (1 patient), vaginal bleeding (2 patients), gas

60 re: diarrhea (53%), fever (21%), hemorrhagic cystitis (12%), and pneumonitis (11%).

61 age, 6.4% had moderate or severe hemorrhagic cystitis, 2.8% had pulmonary hemorrhage, and 2% had intr

62 of patients with diagnosis of acute urinary cystitis, 294 patients whose urine cultures were positiv

63 mon infections were urinary tract infection (cystitis) (34.9% vs 25.2%), cytomegalovirus viremia (17.

64 syndromes of acute pyelonephritis (79%) and cystitis (82%) than in those associated with catheter-as

65 Among women with uncomplicated cystitis, a 3-day regimen of cefpodoxime compared with c

66 n the United States suffer from interstitial cystitis, a chronic painful urinary bladder disorder cha

67 oxazaphosphorines can result in hemorrhagic cystitis, a constellation of complications caused by acr

68 reemerge to cause some episodes of recurrent cystitis, a familiar clinical scenario in otherwise heal

69 premenopausal women with acute uncomplicated cystitis accurately showed evidence of bladder E. coli b

70 o increased (P < or = 0.05) with CYP-induced cystitis (acute, intermediate, and chronic).

71 ) is associated with symptomatic hemorrhagic cystitis after hematopoietic cell transplantation (HCT).

72 adder, urinary incontinence and interstitial cystitis (also known as bladder pain syndrome)(4,6,7).

73 L2: 8-fold; P < or = 0.05) only with chronic cystitis, although it increased in the L6-S1 DRG with CY

74 ecimens [of which 39 were from patients with cystitis and 40 were from patients with pyelonephritis],

75 l antibiotic use in women with uncomplicated cystitis and 42.6% Escherichia coli fluoroquinolone resi

76 Women with acute cystitis and a history of UTI within the previous year s

77 idence of participation of NK-1 receptors in cystitis and a mandatory participation of these receptor

78 Cystitis and acute pyelonephritis, infection of the blad

79 yoma-BK virus is associated with hemorrhagic cystitis and also with polyomavirus nephropathy (PVN).

80 ptococcus agalactiae causes both symptomatic cystitis and asymptomatic bacteriuria (ABU); however, gr

81 ary tract abnormalities, whereas in cases of cystitis and asymptomatic bacteriuria the focus should b

82 Cyclophosphamide may cause hemorrhagic cystitis and eventually bladder urothelial cancer.

83 In contrast, cystitis and fecal isolates differed minimally.

84 such conditions as renal colic, interstitial cystitis and inflammatory bowel disease by purinergic ag

85 onization by the parent strain caused severe cystitis and interstitial nephritis as determined by his

86 Cystitis and invasive high-grade urothelial carcinoma sa

87 on the predictability of the agents causing cystitis and knowledge of their antimicrobial susceptibi

88 f clonal and pathotypic similarities between cystitis and NBM isolates of E. coli O18:K1:H7.

89 commonality between O18:K1:H7 isolates from cystitis and NBM suggests common pathogenetic mechanisms

90 atypia in the setting of an acute or chronic cystitis and nonmuscle invasive-type CIS.

91 ection, control and SCI rats developed acute cystitis and pyelitis without acute differences in histo

92 They respond to both cystitis and pyelonephritis by delivering bacteriostatic

93 athogens: 2 probes were more prevalent among cystitis and pyelonephritis isolates, 2 among pyelonephr

94 n receptor as a UPEC virulence factor during cystitis and pyelonephritis, a fitness factor during bac

95 hogens causing community-acquired UTIs, both cystitis and pyelonephritis, is increasing.

96 severe advanced kidney pathologies, such as cystitis and pyelonephritis, which are associated with a

97 ated in clinical trials for the treatment of cystitis and pyelonephritis.

98 at 30 mg/kg of body weight twice a day cured cystitis and renal infection in noncatheterized mice.

99 is isolate used to study the pathogenesis of cystitis and to develop a FimH (type 1 fimbrial adhesin)

100 syndrome and most environmentally similar to cystitis and urethritis, all of which are inflammatory d

101 rier have been described in certain forms of cystitis and were hypothesized to contribute to irritati

102 , significant (P < or = 0.015) predictors of cystitis and/or pyelonephritis (versus fecal) included a

103 al groups were significantly associated with cystitis and/or pyelonephritis.

104 upper UTI (acute pyelonephritis), lower UTI (cystitis), and asymptomatic bacteriuria, is useful for n

105 sted), 50 (81%) had symptoms consistent with cystitis, and 12 (19%) had symptoms of pyelonephritis.

106 riuria, 45 complicated UTI, 38 uncomplicated cystitis, and 77 pyelonephritis).

107 Infection progressed to cystitis, and a biofilmlike covering was observed over t

108 tis, pneumonia, gastroenteritis, hemorrhagic cystitis, and keratoconjunctivitis.

109 Irritable bowel syndrome, interstitial cystitis, and other chronic pelvic pain (CPP) disorders

110 to gastrointestinal hemorrhage, hemorrhagic cystitis, and pulmonary hemorrhage.

111 ase, acquired hemolytic anemia, interstitial cystitis, and Sjogren's syndrome had higher prevalence r

112 rature on bladder pain syndrome/interstitial cystitis, and the implications for their patients.

113 lar disorder, tension headache, interstitial cystitis, and the postconcussion syndrome.

114 efractory bladder pain syndrome/interstitial cystitis appears promising, larger-scale studies with ad

115 f postinvasion events in the pathogenesis of cystitis are largely undetermined.

116 tags remained in mice that developed chronic cystitis, arguing that during the acute stages of infect

117 ervoirs (QIRs) to the development of chronic cystitis as defined by persistent bacteriuria.

118 This report describes two episodes of acute cystitis associated with "mixed flora" in an elderly mal

119 ence genotype that included papG allele III (cystitis-associated P fimbrial adhesin), sfaS (S fimbria

120 d was derived from the same (meningitis- and cystitis-associated) subclone of E. coli O18:K1:H7 as th

121 Among the 147 participants without cystitis (asymptomatic), 58 (40%) had any viremia.

122 ssified as causing acute cystitis, recurrent cystitis, asymptomatic bacteriuria, or pyelonephritis co

123 A/OT-I mice spontaneously develop autoimmune cystitis at 10 wk of age.

124 and inflamed (cyclophosphamide (CYP)-induced cystitis) bladder urothelium and their contribution to l

125 scopic urinalysis for hematuria (hemorrhagic cystitis, bladder cancer), ECG (anthracycline-related co

126 f the micturition reflex in diseases such as cystitis, bladder/sphincter dyssynergia following spinal

127 Interstitial cystitis/bladder pain syndrome (IC) is associated with s

128 Patients with interstitial cystitis/bladder pain syndrome (IC/BPS) suffer from chro

129 l pelvic pain disorders such as interstitial cystitis/bladder pain syndrome (IC/BPS).

130 bated symptoms in patients with interstitial cystitis/bladder pain syndrome (IC/BPS).

131 s/chronic pelvic pain syndrome, interstitial cystitis/bladder pain syndrome and chronic orchialgia ar

132 rome (UCPPS), which encompasses interstitial cystitis/bladder pain syndrome and chronic prostatitis/c

133 hronic pelvic pain syndrome and interstitial cystitis/bladder pain syndrome.

134 Conditions covered include acute cystitis (both uncomplicated and complicated), catheter-

135 genic GBS isolated from a patient with acute cystitis bound to human T24 bladder uroepithelial cells

136 ted from cells of patients with interstitial cystitis but not controls.

137 cutaneous hypersensitivity in the context of cystitis, but had no effect in uninjured, naive mice.

138 herichia coli (UPEC) in the initial stage of cystitis, but the bacterial determinants of postinvasion

139 Normal responses following induction of cystitis by cyclophosphamide were also observed in both

140 sses neutrophil migration early in bacterial cystitis by eliciting an IDO-mediated increase in local

141 ated with E. coli but appear to rarely cause cystitis by themselves.

142 ine model of urinary tract infections (UTI), cystitis by uropathogenic Escherichia coli (UPEC) occurs

143 (+/-SD) days of antibiotic therapy for ASB, cystitis, CA-UTI and pyelonephritis were 10.0 (4.5), 11.

144 regulation of p-CREB in DRG, suggesting that cystitis can reveal an altered CREB phosphorylation that

145 Most notably, they are found in up to 50% of cystitis cases in children and 30% of pyelonephritis in

146 g organ hyperactivity and pain in a model of cystitis caused by adenoviral-mediated expression of cla

147 ibodies against CNF1 and/or Hly would reduce cystitis caused by CP9.

148 new bladder pain syndrome, ketamine-induced cystitis, characterized by chronic inflammation and urot

149 The dog's cystitis clone was the most extensively recovered clone.

150 aged 18 to 55 years with acute uncomplicated cystitis comparing ciprofloxacin (n = 150) with cefpodox

151 -37), were infected transurethrally with the cystitis-derived uropathogenic Escherichia coli (UPEC) s

152 rthermore, in women with acute uncomplicated cystitis, empirical therapy without a pretherapy urine c

153 ients treated for BKV-associated hemorrhagic cystitis experienced complete resolution of gross hematu

154 rs of recurrence and/or persistence in acute cystitis, extended virulence genotypes were compared wit

155 The O18:K1:H7 cystitis, fecal, and NBM isolates were clonally derived.

156 were reported including vomiting, diarrhoea, cystitis, gastroenteritis, and bleeding.

157 ave been elucidated, and the murine model of cystitis has generated a new paradigm by which acute and

158 ary tract infections (UTIs) and interstitial cystitis have been studied utilizing 16S rRNA rapid next

159 ctory bladder pain syndrome and interstitial cystitis have emerged.

160 ts, are associated not only with hemorrhagic cystitis (HC) but also with hepatitis, conjunctivitis, a

161 iation of BK plasma viremia with hemorrhagic cystitis (HC) in hematopoietic cell transplant (HCT) rec

162 Hemorrhagic cystitis (HC) remains a common complication of allogenei

163 otype O18:K1:H7, taken from women with acute cystitis, healthy control patients, and infants with neo

164 y protein, can ameliorate ifosfamide-related cystitis; however, the mechanisms underlying this urotox

165 This study on interstitial cystitis (IC) aims to identify a unique urine metabolomi

166 atory bladder disorders such as interstitial cystitis (IC) deserve attention since a major problem of

167 Interstitial cystitis (IC) is a debilitating disease that has been ad

168 d in the urine of patients with interstitial cystitis (IC)-a chronic, painful bladder disease of unkn

169 ithelial cells of patients with interstitial cystitis (IC).

170 dder problems (possibly urinary interstitial cystitis [IC]), thyroid disorders, chronic headaches/mig

171 The analysis of 236 episodes of cystitis in 226 women yielded 202 paired specimens of mi

172 antoin, or fosfomycin is indicated for acute cystitis in adult women.

173 in renal transplant patients and hemorrhagic cystitis in bone marrow transplant patients.

174 in renal transplant patients or hemorrhagic cystitis in bone marrow transplant recipients.

175 Acute uropathogenic Escherichia coli (UPEC) cystitis in C57BL/6 and C3H/HeN males recapitulated the

176 ly responsible for the signs and symptoms of cystitis in humans infected with toxigenic UPEC.

177 i type 1 pili may have utility in preventing cystitis in humans.

178 unctionally to reveal marked exacerbation of cystitis in IL-10-deficient mice.

179 ome of uropathogenic Escherichia coli (UPEC) cystitis in mice using genome-wide expression profiling

180 e inflammatory signaling of ketamine-induced cystitis in rat urinary bladder.

181 As with cystitis in reproductive-age women, sexual behaviors and

182 acute (24 h) and chronic (8 day) CYP-induced cystitis in the rat, B1R mRNA was detected throughout th

183 under the name Elmiron for the treatment of cystitis in the United States.

184 Acute uncomplicated cystitis in women can be diagnosed without an office vis

185 NA-seq) analyses of RNA from E. coli causing cystitis in women that iron acquisition systems, includi

186 es for the management of acute uncomplicated cystitis in women that recommend empirical therapy in pr

187 choice for treatment of acute uncomplicated cystitis in women.

188 ng empirical therapy for acute uncomplicated cystitis in women.

189 Virulence genes, group B2, and having caused cystitis (in the mother or dog) corresponded to coloniza

190 38-51% of co-trimoxazole resistance in acute cystitis), including ten (34%) of 29 isolates that were

191 CYP-induced cystitis increased (P < or = 0.001) p75(NTR) expression

192 These results demonstrated that CYP-induced cystitis increases the expression and phosphorylation of

193 elevated urinary IL-10 in patients with UPEC cystitis, indicating a role for IL-10 in the innate resp

194 in micturition reflex pathways after chronic cystitis induced by cyclophosphamide (CYP).

195 zing NGF antibody also subsequently reversed cystitis-induced increases in bladder weight.

196 hanges in sensory pathways may contribute to cystitis-induced pain and hyperactivity of the bladder.

197 use the neutralizing NGF antibody attenuated cystitis-induced type I collagen up-regulation in the in

198 Interstitial cystitis is a common disease among women and is frequent

199 Interstitial cystitis is a complex inflammatory condition of the blad

200 Bladder pain syndrome/interstitial cystitis is an important and poorly understood disorder.

201 Hemorrhagic cystitis is an inflammatory and ulcerative bladder condi

202 Cystitis is caused by a relatively small number of bacte

203 and urothelial ATP release with CYP-induced cystitis is decreased with TGF-beta inhibition.

204 The cause of acute uncomplicated cystitis is determined on the basis of cultures of voide

205 standing of the pathogenesis of interstitial cystitis is emerging and this will hopefully lead to the

206 h we view bladder pain syndrome/interstitial cystitis is evolving, as is apparent in the literature t

207 The pathophysiology of interstitial cystitis is incompletely understood, although altered ep

208 II(UTI89) during the establishment of acute cystitis is maintenance of wild-type leuX, and not PAI I

209 Acute cystitis is one of the most commonly encountered bacteri

210 tutive expression of type 1 fimbriae in UPEC cystitis isolate F11 and the laboratory strain E. coli K

211 ed mutants of uropathogenic Escherichia coli cystitis isolate F11 were used to assess the role of the

212 eys were challenged with 1 mL of 108 E. coli cystitis isolate NU14.

213 The pathogenesis of the K. pneumoniae cystitis isolate TOP52 was compared to that of the uropa

214 isolate UTI89 and the Klebsiella pneumoniae cystitis isolate TOP52.

215 Escherichia coli NU14, a cystitis isolate used to study the pathogenesis of cysti

216 virulence of two reference UPEC strains, the cystitis isolate UTI89 and the urosepsis isolate CFT073.

217 migration by purified YbcL(UTI), encoded by cystitis isolate UTI89, required the presence of a uroep

218 studies showed that the effects of the UTI89 cystitis isolate were fimH dependent.

219 e factors (VFs) among the 35 sought than did cystitis isolates (n=23).

220 The cystitis isolates and archetypal NBM isolates RS218 and

221 brial adhesin gene) predominates among human cystitis isolates and confers an adherence phenotype res

222 ng pyelonephritis isolates only, and 3 among cystitis isolates only than among fecal isolates.

223 Cystitis isolates present in the urine of mice during th

224 revealed distinct signatures between ASB and cystitis isolates, including in the purine pathway (prev

225 Compared with fecal and cystitis isolates, pyelonephritis isolates exhibited a g

226 In general, cystitis isolates, unlike pyelonephritis isolates, were

227 om seven UPEC (three pyelonephritis and four cystitis) isolates and three fecal/commensal strains, in

228 iator of bladder dysfunction and pain during cystitis, it is presently unclear if it is also importan

229 Ketamine-induced ulcerative cystitis (KIC) initially damaged the bladder mucosa and

230 tiates bladder pain and, in ketamine-induced cystitis, loss of urothelium from large areas of the bla

231 isplayed a striking predilection for chronic cystitis, manifesting as persistent bacteriuria, high-ti

232 Changes in PACAP expression after cystitis may play a role in altered visceral sensation (

233 Alterations in GAP-43-IR following chronic cystitis may suggest a reorganization of bladder afferen

234 er titers and formed more IBCs in the murine cystitis model than wild type.

235 In a murine cystitis model, UTI89 surA::kan was unable to persist in

236 richia coli (UPEC) isolate UTI89 in a murine cystitis model.

237 ivity has not been delineated in a dedicated cystitis model.

238 both an in vitro model and an in vivo murine cystitis model.

239 States with acute pyelonephritis (n = 170), cystitis (n = 83), or no infection (fecal; n = 76).

240 Cystitis, nausea, vomiting, and diarrhea were also obser

241 Episodes of urinary tract infection (cystitis) occurred more often in the denosumab than in t

242 increased in the L6-S1 DRG with CYP-induced cystitis of acute (2-3-fold; P < or = 0.05) and chronic

243 t in the L2, L4, and L5 DRG with CYP-induced cystitis of acute and chronic duration compared with con

244 The effects of acute cystitis on distal colonic sensory thresholds to CRD and

245 ts who experienced grades 2 to 3 hemorrhagic cystitis, only 1 of whom had received continuous bladder

246 No hemorrhagic cystitis or bone marrow suppression was observed.

247 ntiating Escherichia coli strains that cause cystitis or pyelonephritis from fecal E. coli remain inc

248 ion of patients (20% vs. 25%) with recurrent cystitis or pyelonephritis with the same ESBL-producing

249 tion of patients (20% vs 25%) with recurrent cystitis or pyelonephritis with the same ESBL-producing

250 id clinical isolates from cases of pediatric cystitis or pyelonephritis, adult pyelonephritis or uros

251 infections, which can present clinically as cystitis or pyelonephritis.

252 ands may have increased risks of hemorrhagic cystitis or urothelial cancer from persistent and ectopi

253 After chronic cystitis, PACAP immunoreactivity in cells in the L1, L2,

254 After chronic, CYP-induced cystitis, PACAP immunoreactivity increased dramatically

255 ive bladder syndrome (OABS) and interstitial cystitis/painful bladder syndrome (IC/PBS) are debilitat

256 ycopeptide urinary biomarker of interstitial cystitis/painful bladder syndrome (IC/PBS), a chronic co

257 Functional disorders (i.e. interstitial cystitis/painful bladder syndrome and irritable bowel sy

258 ical symptoms characteristic of interstitial cystitis/painful bladder syndrome, which manifests due t

259 secreted by bladder cells from interstitial cystitis patients and is a potent inhibitor of both norm

260 Cav1.2 in smooth muscle mimics the ketamine cystitis phenotype.

261 lly relevant subgroups within the "recurrent cystitis" population.

262 a (range, 0-1.0 x 10 copies/mL), hemorrhagic cystitis (present/absent), and data on kidney function.

263 vances in both in vitro and animal models of cystitis promise to provide insights into the bacterial

264 from 18 to 49 years of age with symptoms of cystitis provided specimens of midstream urine, after wh

265 c human clinical isolates from patients with cystitis, pyelonephritis, bacteremia, or meningitis, inc

266 ine of women and classified as causing acute cystitis, recurrent cystitis, asymptomatic bacteriuria,

267 ppropriate diagnostic tests for interstitial cystitis remain uncertain.

268 gnificantly among uropathogens causing acute cystitis, resistance to nitrofurantoin and ciprofloxacin

269 en expression in the inflamed bladder during cystitis revealed that Akt, JNK, and ERK1/2 activities w

270 We report that during cystitis, shedding of infected bladder epithelial cells

271 voiding abnormalities presenting with acute cystitis should be treated similarly to women without di

272 ted here that cyclophosphamide (CYP)-induced cystitis significantly increased the production of type

273 pared the impact of ybtPQ on a model E. coli cystitis strain during in vitro culture and experimental

274 ce, an attribute not exhibited by a clinical cystitis strain.

275 There was no significant correlation between cystitis strains (n = 20) and the pic determinant.

276 The cystitis strains also suppressed epithelial cytokine res

277 The cystitis strains induced significantly less IL-6 than di

278 onclude that, in this in vitro model system, cystitis strains of UPEC have genes encoding factors tha

279 sion of type 1 fimbriae is more critical for cystitis strains than for pyelonephritis strains in the

280 nses of bladder epithelial cell lines to the cystitis strains UTI89 and NU14.

281 on, a higher number of IBCs precedes chronic cystitis than precedes QIR formation.

282 nized mice also exhibited significantly less cystitis than sham-immunized mice.

283 t voiding symptoms and pain seen in forms of cystitis that coexist with urothelial barrier dysfunctio

284 t only resulted in the development of strong cystitis that persisted significantly longer than in mic

285 t study we used established animal models of cystitis to determine the time course of bladder inflamm

286 may be lower among women with uncomplicated cystitis treated with TMP-SMX when the infecting pathoge

287 In a murine model of cystitis, type 1 pili-mediated bladder epithelial invasi

288 infection was developed using the prototypic cystitis UPEC isolate NU14 harboring a plasmid encoding

289 During the establishment of bacterial cystitis, UPEC suppresses innate responses via multiple

290 During murine cystitis, uropathogenic E. coli (UPEC) utilizes type 1 p

291 During acute cystitis, uropathogenic Escherichia coli (UPEC) induce b

292 During cystitis, uropathogenic Escherichia coli (UPEC) subvert

293 rain 807, isolated from a patient with acute cystitis, using metabolic arrays of >2,500 substrates an

294 al titers nor a reduction in the severity of cystitis versus that of sham-immunized mice.

295 in vitro and in bladder tissue during murine cystitis via a noncanonical, interferon-independent path

296 olonization of the urine or kidney; however, cystitis was reduced significantly in mice treated with

297 strains in the urine of a patient with acute cystitis, where half of the isolates carried a glycine-t

298 provided a fitness advantage during chronic cystitis, which is manifested as persistent bacteriuria,

299 nal cohort study, the outpatients with acute cystitis with isolation of E. coli in their urine cultur

300 -beta signalling in cyclophosphamide-induced cystitis with TbetaR-1 inhibition decreased afferent ner