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1  its ability to adhere to endothelial cells (cytoadherence).
2 gulates PfEMP1 expression and the parasite's cytoadherence.
3 hat contains kahrp and pfemp3 causes reduced cytoadherence.
4  expression of surface proteins required for cytoadherence.
5 lag3 genes previously assumed to function in cytoadherence.
6 etory (E/S) products can facilitate in vitro cytoadherence.
7 d them for their ability to mediate in vitro cytoadherence.
8 ositively charged protamine sulfate promoted cytoadherence.
9                        Nutrient acquisition, cytoadherence and antigenic variation are among the key
10  expression of parasite proteins involved in cytoadherence and can reveal antigens associated with cl
11 e system, is protective against T. vaginalis cytoadherence and cytolysis of host cells.
12 acute stress in erythrocytes, which enhances cytoadherence and hemolysis.
13 he glycan contacts, underscoring its role in cytoadherence and in antigenic variation in malaria.
14 ronemal proteins (MICs) are key mediators of cytoadherence and invasion for Toxoplasma gondii.
15     Clinical interventions aimed at reducing cytoadherence and microvascular inflammation may improve
16                                              Cytoadherence and sequestration of erythrocytes containi
17 to study the components involved in B. bovis cytoadherence and sequestration.
18  A large family of variant proteins mediates cytoadherence and their binding specificity determines p
19 ns, differentiation, cell cycle progression, cytoadherence, and both stimulation and evasion of host
20 strated relationships among knob expression, cytoadherence, and infectivity.
21                           Both rosetting and cytoadherence are mediated by the parasite-derived IE su
22                                  An in vitro cytoadherence assay was used to investigate the presence
23                                              Cytoadherence assays demonstrated specific binding to hu
24 rapeutics for cerebral, placental, and other cytoadherence-associated malaria illnesses.
25 AHRP and PfEMP3, play important roles in the cytoadherence by mediating the clustering of PfEMP1 in r
26 irculation times, possibly related to higher cytoadherence capacity.
27  shown to assist the development of virulent cytoadherence characteristics.
28 histidine-rich protein (KAHRP), an essential cytoadherence component.
29 domains) specifically inhibited and reversed cytoadherence down to low concentrations (<10 mug/ml of
30 ylated proteins, including those involved in cytoadherence, drug resistance, signaling, development,
31                                Rosetting and cytoadherence have been widely studied as separate entit
32  on the erythrocyte membrane is critical for cytoadherence, however the molecular mechanisms behind t
33  with a link between antigenic variation and cytoadherence in B. bovis and suggest that the VESA1 Ag
34 udies of cytokine activation and erythrocyte cytoadherence in babesiosis and malaria have exploited t
35     This conundrum is further complicated as cytoadherence in the microvasculature is still a matter
36  oral anthelminthic drug levamisole inhibits cytoadherence in vitro and reduces sequestration of late
37 ein-1-mediated phenomena appears to diminish cytoadherence in vivo and to protect against disease in
38 at the erythrocyte membrane is necessary for cytoadherence in vivo, our findings have implications fo
39 otection in AS children by further weakening cytoadherence interactions.
40             We have also shown that in vitro cytoadherence is a surrogate for the formation of antifi
41 ated that loss of EPCR and TM at sites of IE cytoadherence is detectible in nonfatal CM.
42 locking methods, we found that M. pneumoniae cytoadherence is important for the induction of cytokine
43  heparinase had no significant inhibition on cytoadherence, it is unlikely that sulfated glycoconjuga
44 The attendant increase in cell stiffness and cytoadherence leads to sequestration of infected RBCs in
45 ne protein-1 (PfEMP-1), the parasite's major cytoadherence ligand and virulence factor on the erythro
46 al cell-surface display of the main variable cytoadherence ligand, PfEMP-1 (P. falciparum erythrocyte
47      The first gene characterizing the clag (cytoadherence linked asexual gene) family of Plasmodium
48 s study has shown that the parasite protein, cytoadherence-linked asexual gene 9 (CLAG9), is also ess
49  first report demonstrating up-regulation by cytoadherence of a plasminogen-binding alpha-enolase in
50 s to visualise the steps leading to vascular cytoadherence of erythrocytes infected with the human pa
51                                              Cytoadherence of HB3EC-6 to human microvascular endothel
52 brane protein 1 (PfEMP1), is responsible for cytoadherence of infected cells to host endothelial rece
53 f Plasmodium falciparum malaria is caused by cytoadherence of infected erythrocytes, which promotes p
54 responsible for both antigenic variation and cytoadherence of infected erythrocytes.
55 has important implications for understanding cytoadherence of infected red blood cells and potentiall
56                                   Therefore, cytoadherence of IT/R29 IE is distinct from rosetting, w
57 ytes by Plasmodium falciparum merozoites and cytoadherence of parasitized erythrocytes (PRBC) to endo
58 ble to inhibit CSA-, CD36-, and TSP-mediated cytoadherence of PE.
59 e invasion of erythrocytes by merozoites and cytoadherence of PRBC to endothelial cells by increasing
60 ponse and host spleen clearance by mediating cytoadherence of the iRBC to the endothelial wall, but t
61                         We have compared the cytoadherence properties of parasitized AS and AA erythr
62 y changes in the permeability, rigidity, and cytoadherence properties of the host erythrocyte.
63 rocyte membrane protein 1 (PfEMP1) family of cytoadherence receptors.
64  that the spiral and dense coat organize the cytoadherence structures in the knob, and anchor them in
65 tope may be targeted to disrupt the parasite cytoadherence system.
66 s restored knob expression and CD36-mediated cytoadherence, thereby showing that the human environmen
67 nd/or erythrocyte sequestration by increased cytoadherence to endothelium.
68  RBC and the sudden transition from firm RBC cytoadherence to flipping on the endothelial surface.
69 the host cell surface leading to ablation of cytoadherence to host receptors.
70 the more biologically relevant phenomenon of cytoadherence to PBMC, can result in considerable enhanc
71 malaria parasite P. falciparum, and mediates cytoadherence to the host vascular endothelium.
72 s that have the ability to block erythrocyte cytoadherence to the PvDBP.
73 deformability and they also exhibit enhanced cytoadherence to vascular endothelium and other healthy
74                   To determine their role in cytoadherence, two Plasmodium falciparum transgenic line
75                       Knobs are required for cytoadherence under flow conditions, and they contain bo
76                                              Cytoadherence was inhibited by heparin and by treatment
77  demonstrated that variant genes involved in cytoadherence were dependent on the spleen for their exp
78 ghest inhibitory effect on both invasion and cytoadherence, whereas the positively charged protamine