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1  claudin 3, 4, and 7 and the luminal marker, cytokeratin 18.
2 8, but in the presence of actin, AVP cleaved cytokeratin 18.
3 cells exhibited caspase-specific cleavage of cytokeratin 18.
4 ith a monoclonal antibody specific for human cytokeratin 18.
5 pase-3, and immunohistochemical staining for cytokeratin 18.
6                            For AVP to cleave cytokeratin 18, a cellular cofactor was shown to be requ
7 skeletal filaments: vimentin, cytokeratin 8, cytokeratin 18, actin, and hair type II basic keratin.
8                                Serum cleaved cytokeratin 18 and caspase-3/7 decreased significantly.
9 ormal unperturbed RPE are immunoreactive for cytokeratin 18 and negative for cytokeratin 19, vimentin
10 ch as increased expression of E-Cadherin and cytokeratin-18 and decreased expression of Snail.
11 2-derived peptide, alanine aminotransferase, Cytokeratin-18 and homeostasis model of insulin resistan
12                                  Colonies of cytokeratin-18 and human albumin-expressing cells were p
13 ation of fibrosis-associated markers such as cytokeratins 18 and 19 and annexin 2, as determined both
14 e positive for prostate-specific antigen and cytokeratins 18 and 19 and negative for keratin 15.
15                                       Type I cytokeratins 18 and 19 present in these cells specifical
16 tested M65 and M30 (circulating fragments of cytokeratin-18) and their respective fraction carried by
17 , hepatocyte nuclear factor 4alpha, albumin, cytokeratin 18, and cytochrome P450 3A.
18 epithelial phenotype, expressing E-cadherin, cytokeratin 18, and desmin.
19 nced steatohepatitis fibrosis (MASEF) score, cytokeratin 18, and pro-C3.
20 es in HeLa cells revealed AVP localized with cytokeratin 18, and this was followed by destruction of
21 ll as the epithelial markers pancytokeratin, cytokeratin-18, and occludin, but not mesenchymal (CD44,
22        The proteinase alone could not cleave cytokeratin 18, but in the presence of actin, AVP cleave
23 rum transaminases were normal in TASH, total cytokeratin 18, but not the caspase-cleaved fragment, wa
24 ppearance of E1A protein and the cleavage of cytokeratin 18 by AVP.
25 tro, actin was a cofactor in the cleavage of cytokeratin 18 by AVP.
26         Levels of M30, a cleavage product of cytokeratin-18 caspase, are significantly increased in s
27 e aminotransferase (ALT) and caspase-cleaved cytokeratin 18 (CK-18 caspase).
28    We have recently demonstrated that plasma cytokeratin 18 (CK-18) fragment levels correlate with th
29              Associations of liver injury or cytokeratin 18 (CK-18) levels with each chemical group a
30 say (ELISA), which detects a caspase-cleaved cytokeratin-18 (CK-18) fragment and thereby apoptotic ce
31 mined by alanine aminotransaminase [ALT] and cytokeratin-18 [CK-18]).
32 necrosis in serum was quantified using serum cytokeratin 18 (CK18) M30 and M65 enzyme-linked immunoso
33 reased expression of HDAC1 target genes Bax, cytokeratin 18 (CK18), and p21(WAF1/CIP1), whereas maspi
34 nsulin, aspartate aminotransferase (AST) and cytokeratin 18 (CK18).
35          At 48 h, liver histology, ALT, AST, cytokeratin-18 (CK18), microRNA-122 (miR-122), and trypt
36       Apoptosis was monitored by determining cytokeratin 18 cleavage and caspase-3 activation.
37 vities, and immunohistochemical staining for cytokeratin 18 cleavage products indicated no activation
38 ated with increased apoptosis (caspase 3 and cytokeratin 18 cleavage) in excised tumors.
39 ropeptide Y receptor Y1, p21 WAF-1, p55 PIK, cytokeratin 18 (cloned twice), fructose-1,6-biphosphatas
40 caspase-dependent apoptosis (caspase-cleaved cytokeratin-18) compared to control; caspase-dependent a
41 cells express cytokeratins 5 and 14, but not cytokeratin 18, consistent with a basal epithelial cell
42 itative Insulin-Sensitivity Check Index, and cytokeratin-18 correlated with NASH.
43 rly reactive RPE (7 days in culture) express cytokeratin 18, cytokeratin 19, and vimentin.
44 he cells accumulated lipid droplets, and the cytokeratin 18 cytoskeleton was reorganized.
45 brils with active caspase-3, caspase-cleaved cytokeratin-18, death-effector-domain containing DNA-bin
46 amplification loop to facilitate cleavage of cytokeratin-18, disruption of the cytoskeleton and the e
47 of differentiation as shown by expression of cytokeratin 18, E-cadherin, thyroglobulin, TTF-1 and Pax
48              Immunocytochemistry staining of cytokeratin-18 (epithelial marker)/vimentin (mesenchymal
49 lesterol and adiponectin concentrations, and cytokeratin-18 fragment elevation.
50                                              Cytokeratin-18 fragment levels independently predicted N
51 rum levels of M30 and M65 antigen (the total cytokeratin-18 fragment, a marker of apoptosis and necro
52 sured with transient elastography, and serum cytokeratin-18 fragment.Measurements and Main Results: A
53 3 +/- 1.5 vs. 1.7 +/- 1.4; P = 0.004), serum cytokeratin-18 fragments (283 vs. 404 U/L; P < 0.001) an
54                             Plasma levels of cytokeratin-18 fragments are reliable noninvasive marker
55 labeling-positive nuclei and accumulation of cytokeratin-18 fragments in the liver, was independent o
56                                       Plasma cytokeratin-18 fragments were markedly increased in pati
57                          Caspase-3-generated cytokeratin-18 fragments were measured in situ via immun
58  cytokines, markers of hepatocyte apoptosis (cytokeratin-18 fragments), and hepatic fibrogenesis (hya
59 ed low-density lipoproteins, adipokines, and cytokeratin-18 fragments, and an oral glucose tolerance
60                                              Cytokeratin-18 fragments, controlled attenuation paramet
61 ment of plasma lipoproteins, adipokines, and cytokeratin-18 fragments.
62 ment of plasma lipoproteins, adipokines, and cytokeratin-18 fragments.
63 fferentially expressed genes: cytokeratin 8, cytokeratin 18, Hsp27 and GPCR -Br.
64 4% as quantified in the lung interstitium by cytokeratin 18 immunostaining.
65    That protein is tentatively identified as cytokeratin 18 in a companion paper.
66 of lactate dehydrogenase and caspase-cleaved cytokeratin 18 in the perfused medium.
67 inotransferase (AST), bilirubin, bile acids, cytokeratin-18, insulin, estimated glomerular filtration
68 helial cell markers pan-cytokeratin (Pan-K), cytokeratin-18 (K-18), and occludin.
69 -122), glutamate dehydrogenase (GLDH), total cytokeratin 18 (K18), caspase cleaved K18, glutathione S
70 erotypic complexes of cytokeratin 8 (K8) and cytokeratin 18 (K18).
71 rting enzyme 2 (ACE2) receptor driven by the cytokeratin-18 (K18) gene promoter (K18-hACE2) as a mode
72 w that by measuring a circulating biomarker, cytokeratin-18 (K18), accurate detection of drug-induced
73 alpha, ERbeta, cytokeratin 14 (basal cells), cytokeratin 18 (luminal cells), and dorsolateral protein
74  enterocyte proliferation and migration, (2) cytokeratin-18 M30-immunohistochemistry to compare level
75                    Total and caspase-cleaved cytokeratin-18 (M65 and M30) measured at admission and s
76 strogen, and progesterone receptor-positive, cytokeratin 18-positive (ER(+)PR(+)CK18(+)) subtype, and
77                                    ALT, AST, cytokeratin 18, pro-C3, and MASEF score decreased signif
78 zyme-linked immunosorbent assays for various cytokeratin 18 products (eg, M65, cell death, M30, and a
79 sin-converting enzyme 2 (hACE2) by the human cytokeratin 18 promoter (K18 hACE2) represent a suscepti
80      Notably, mice expressing hDPP4 with the cytokeratin 18 promoter developed progressive, uniformly
81 trol of the surfactant protein C promoter or cytokeratin 18 promoter that are susceptible to infectio
82 ling-positive cells, caspase-3 activity, and cytokeratin-18 staining in the liver.
83                               Elevated total cytokeratin 18 suggested the presence of necrotic cell d
84 umin, alpha-fetoprotein, cytochrome P4502E1, cytokeratin-18, type-1 collagen, transforming growth fac
85  cirrhosis contained hepatocyte-derived MPs (cytokeratin-18(+)), whereas plasma from controls did not
86 re used to identify five proteins, including cytokeratin 18, which is the product of the most highly
87  markers of colonic epithelial cells such as cytokeratin 18, zonula occludens-1, mucins-1 and -2, ant