ライフサイエンスコーパス: cytokine profile

1 ithout affecting responding T cell number or cytokine profile.

2 Th-2; ie IL-4, IL-13) to Th-1 (ie IFN-gamma) cytokine profile.

3 skewing of the immune response toward a Th2 cytokine profile.

4 osite to the elevated donor pro-inflammatory cytokine profile.

5 l activation threshold and broadened the Th1 cytokine profile.

6 f the TH2 response with a shift toward a TH1 cytokine profile.

7 -cell function and secrete a proinflammatory cytokine profile.

8 their immune response is biased toward a TH2 cytokine profile.

9 sts potentially controls the decidual T cell cytokine profile.

10 ractions with immune cells and the resultant cytokine profile.

11 sinophilia, local IgE formation, and a T(H)2 cytokine profile.

12 entifies Th17 and Tc17 cells with a peculiar cytokine profile.

13 infection was not associated with a specific cytokine profile.

14 ivariate ANOVA were used to analyze the T(H) cytokine profile.

15 e-derived dendritic cells with a tolerogenic cytokine profile.

16 on was determined from surface phenotype and cytokine profile.

17 TLR4 allele-specific DNA sequences with the cytokine profile.

18 iated with higher probability of a favorable cytokine profile.

19 ymphodepletion without achieving a favorable cytokine profile.

20 sociated with HSV-2 infection and a distinct cytokine profile.

21 the myotube surface area as well as gene and cytokine profiles.

22 ized by their cell surface markers and their cytokine profiles.

23 re generally rare and heterogeneous in their cytokine profiles.

24 tract microbiota signatures, metabolite, and cytokine profiles.

25 d as instructors of subsequent CD4(+) T cell cytokine profiles.

26 rtially activated populations with different cytokine profiles.

27 e response as judged by antibody isotype and cytokine profiles.

28 iferation and include regulation of effector cytokine profiles.

29 d T lymphocytes, contributed to these innate cytokine profiles.

30 culture in IL-1beta and IL-2 modified NK-22 cytokine profiles.

31 according to their transcription factor and cytokine profiles.

32 tent viral blips, were associated with these cytokine profiles.

33 l surface markers, transcription factors and cytokine profiles.

34 signature is supported by flow cytometry and cytokine profiling.

35 d efficacy, CD22 CAR T-cell persistence, and cytokine profiling.

36 coring of the inflammation in tissues and by cytokine profiling.

37 was analyzed histopathologically and through cytokine profiling.

38 Cytokine profiles 72 hours after injury are consistent w

39 n serum LDH levels above normal, a favorable cytokine profile after lymphodepletion was associated wi

40 hildren with ASD and a more activated T cell cytokine profile after phytohemagglutinin stimulation we

41 lammatory response with an anti-inflammatory cytokine profile, an extracellular matrix rich in type I

42 Cytokine-profiling analyses revealed that heparin affect

43 Cytokine profile analysis during EAU revealed MC5r and A

44 y weight assessment, histologic scoring, and cytokine profile analysis.

45 Young patients with CHB have a Th1-cell cytokine profile and a partial profile of T-cell exhaust

46 amma on AML blasts generated an inflammatory cytokine profile and activated NK cells.

47 that have a broad specificity and favorable cytokine profile and are suitable for adoptive T-cell th

48 (LOX) in the aortic wall, improved systemic cytokine profile and attenuated adipose inflammation.

49 , in poor healing associated with an altered cytokine profile and fewer alternatively activated macro

50 phenotype associated with a proinflammatory cytokine profile and impaired antifungal activity of pol

51 ccompanied by an exaggerated proinflammatory cytokine profile and increased STAT3 signaling.

52 ed NKT cell subset was then analyzed for its cytokine profile and its suppressive in vitro and in viv

53 h HDM and cockroach induced a type 2/type 17 cytokine profile and mixed granulocytic inflammation in

54 Pro-inflammatory cytokine profile and multiple markers of oxidative stres

55 ficient mice had an exacerbated inflammatory cytokine profile and showed enhanced CCR2-mediated macro

56 es, have a predominant T helper type-2 (Th2) cytokine profile and significantly elevated plasma level

57 2A, T(reg) cells altered their metabolic and cytokine profile and were unable to suppress effector im

58 rrent literature regarding advances in serum cytokine profiles and associated challenges preventing t

59 clinical data and disease severity indices, cytokine profiles and C-reactive protein (CRP) concentra

60 We compared bacteria- and fungi-induced cytokine profiles and found that most cytokine responses

61 xpression of PLZF, T-bet, and RORgammat, and cytokine profiles and found that, although monoclonal iN

62 uman Treg cells changed their phenotypes and cytokine profiles and had potent suppressive function.

63 sferred T lymphocytes exhibited Th1 and Th17 cytokine profiles and minimal Th2.

64 have evaluated the BAL cellular composition, cytokine profiles and protein constituents in lung trans

65 Cytokine profiles and staining with flow cytometry were

66 The cytokine profiles and STAT signaling of these tetramer-p

67 The comparison of the inflammatory response, cytokine profiles and Th-1, Th-2 and Th-17 cells numbers

68 Cytokine profiling and examination of peripheral blood s

69 Human cytokine profiling and murine models of muscle inflammat

70 ession, BAL and tissue inflammatory cell and cytokine profile, and apoptosis were assessed.

71 nts with Chagas disease, determine the serum cytokine profile, and evaluate the potential association

72 ide GC Tfh cells by gene expression profile, cytokine profile, and functional properties.

73 analyzed their association with viral load, cytokine profile, and severity.

74 hallenged wild-type mice maintain a distinct cytokine profile, and, unlike eosinophils isolated from

75 cluding oxygen and vasopressor requirements, cytokine profiles, and C-reactive protein (CRP) levels p

76 Work in humans suggests different cytokine profiles, and different treatment strategies fo

77 e pattern of cell surface marker expression, cytokine profiles, and gene expression, suggesting that

78 iatic epidermal phenotype and characteristic cytokine profiles, and responded to various classes of p

79 y, next-generation RNA-sequencing, multiplex cytokine profiling, and phospho-signaling analyses.

80 r bacterial counts, histological evaluation, cytokine profiles, antibody level and binding activity d

81 ), bronchoalveolar fluid (BALF) cellular and cytokine profile, antigen-specific IgE and IgG1, and lun

82 Distinct airway cytokine profiles are present in current smokers and nev

83 h-RC viruses exhibit a distinct inflammatory cytokine profile as well as significantly elevated T-cel

84 differences exist in associated magnitude or cytokine profiles as a function of disease severity.

85 Using a cytokine-profiling assay, we show that granulocyte-macro

86 xhibit an IL-4(hi)IFN-gamma(lo)TNF-alpha(hi) cytokine profile associated with a high capacity to sust

87 eutrophil phenotype and an anti-inflammatory cytokine profile associated with heightened LPS levels.

88 ematurity, reduced fetal weight, altered the cytokine profile at the maternal-fetal interface, and in

89 We demonstrate that peripheral cytokine profiles at birth are associated with ASD later

90 y recruit and activate DCs with Th1-dominant cytokine profiles at the injection site in vivo.

91 These preliminary cytokine profiling-based observations provide a possible

92 ared acute HCV-specific T-cell responses and cytokine profiles between 20 HIV/HCV-coinfected and 20 H

93 d that CD1d(hi)CD5(+) B cells altered T cell cytokine profile but did not directly inhibit T cell pro

94 havioral effects and tissue-specific altered cytokine profile, but absence of glial activation in spi

95 rmal in terms of both phenotype and effector cytokine profile, but they exhibited a significantly red

96 eages with distinct transcription factor and cytokine profiles, but the mechanisms underlying such fa

97 metry using HLA class II tetramers and their cytokine profile by intracellular staining.

98 to harvest jejunal tissue for histology and cytokine profiles by ELISA.

99 ng that IgG immune complexes elicit distinct cytokine profiles by human myeloid immune cells, which a

100 Sputum cytokine profiling can determine distinct and overlappin

101 To study the systemic cytokine profile characteristic of Strongyloides infecti

102 e p35(-/-) mice also demonstrated a distinct cytokine profile characterized by a shift from a T-helpe

103 gh-affinity CD4(+) T cells showed an altered cytokine profile characterized by the production of prot

104 ophoblast cultures induced a proinflammatory cytokine profile, characterized by elevated levels of se

105 ls from obese mice produce a proinflammatory cytokine profile compared with B cells from lean mice.

106 ity lymphodepletion and achieved a favorable cytokine profile compared with those who received the sa

107 stinct T-cell receptor usage and genomic and cytokine profiles compared with the classical type I NKT

108 , but displayed a cell-surface phenotype and cytokine profile consistent with memory precursors, rais

109 -Rapa cell recipients had a balanced Th2/Th1 cytokine profile, conversion of mixed chimerism toward f

110 Seminal cytokine profiles correlated with transmission or non-tr

111 Seminal cytokine profiles correlated with transmission or nontra

112 onstrate this concept by determining whether cytokine profiles could discriminate RA patients accordi

113 Among 48 vaginal cytokines profiled, cytokines associated with HPV infect

114 ith chronic liver disease, but comprehensive cytokine profiling data across different clinical charac

115 te dehydrogenase (LDH) level and a favorable cytokine profile, defined as serum day 0 monocyte chemoa

116 functions that included antigenic response, cytokine profile, diabetogenicity, and suppressive funct

117 nemia, cytopenias, hypofibrinogenemia, and a cytokine profile diagnostic for HLH.

118 Cytokine profiles did not distinguish cirrhotic HBV pati

119 vident across all tissues examined, the iNKT cytokine profile differed more by tissue of origin than

120 Among HCC and non-HCC patients, cytokine profiles differed between HBV and HCV patients

121 ion in cytokine responses, yet the resulting cytokine profile discriminated only between survivors an

122 an altered surface phenotype and acquired a cytokine profile distinct from that of equivalent cells

123 Blood cytokine profiles distinguish PAH immune phenotypes with

124 eriodontitis (CP) patients to assess whether cytokine profiles distinguish patients with RA and patie

125 m VRs, displaying an altered phenotype and a cytokine profile dominated by cytokines IL-2, tumor necr

126 We measured viral loads and cytokine profile during patients' acute infections.

127 uggest that induction of Tregs and change in cytokine profiles during helminthic therapies were respo

128 An 11-cytokine profile effectively differentiated patients wit

129 hat the suppression of arthritis and the Th2 cytokine profile elicited by A9 is dependent on the pres

130 Intriguingely, as predicted from their cytokine profile, endogenous biTregs displayed additiona

131 The low IL-12p40 and high IL-6 cytokine profile expressed by BRD509(pSBRT7)-stimulated

132 oll-like receptor-8 (TLR8) evokes a distinct cytokine profile favoring the generation of Type 1 helpe

133 s of the protein and a more balanced Th1/Th2 cytokine profile from antigen-specific T cells.

134 lity of the lead entity to induce a pro-Th17 cytokine profile from primary human peripheral blood mon

135 ements involved the serological response and cytokine profile from reactivated splenocytes, plethysmo

136 mal RNA, metagenomic, metatranscriptomic and cytokine profiles from 45 preterm and 90 term birth cont

137 e the effect of EMD and its fractions on the cytokine profiles from human gingival fibroblasts in vit

138 was to examine the molecular mechanisms and cytokine profiles generated following beta-glucan stimul

139 We investigated their phenotype, cytokine profile, generation, and pathological relevance

140 Cytokine profiling identified elevated signatures of inf

141 These "T(FH)13" cells have an unusual cytokine profile (IL-13(hi)IL-4(hi)IL-5(hi)IL-21(lo)) an

142 ownregulation on human DC surface phenotype, cytokine profile, immunogenicity (using IDO activity as

143 tory syndromes result in the same pathogenic cytokine profile, implying that a personalized approach

144 eron-gamma by re-expressing CD8 and a type 1 cytokine profile in association with partial Cd8a demeth

145 We assessed the Th2 cytokine profile in bile samples and brush cytology samp

146 We initially examined the cytokine profile in cultured human AML compared with AML

147 This comprehensive cytokine profile in HIES patients reveals distinctive bi

148 is associated with survival and a serum Th1 cytokine profile in HIV-infected individuals.

149 oorly controlled T2D may differ from the GCF cytokine profile in medically healthy individuals with p

150 otch and TLR stimulation results in a unique cytokine profile in mouse bone-marrow derived DCs charac

151 ort the hypothesis of an endogenously poised cytokine profile in neonates and suggest a link between

152 The GCF cytokine profile in patients with and without T2D seems

153 zed that the gingival crevicular fluid (GCF) cytokine profile in patients with periodontitis with poo

154 ontrast, the gut mucosa presented an anergic cytokine profile in relation to ANXA1 expression.

155 The cytokine profile in seminal fluid, but not in peripheral

156 The cytokine profile in seminal, but not in the peripheral b

157 NSPT) on the gingival crevicular fluid (GCF) cytokine profile in sites with standardized periodontal

158 The cytokine profile in unstimulated whole saliva (UWS) of p

159 HIV acquisition and relate these to vaginal cytokine profiles in an observational cohort of women at

160 id (CSF) cultures, inflammatory markers, and cytokine profiles in ART-naive patients with AIDS who di

161 Thus, age-related cytokine profiles in chronic rhinosinusitis (CRS) need t

162 s trial compares the clinical parameters and cytokine profiles in gingival crevicular fluid of patien

163 by house dust mites, pulmonary function and cytokine profiles in Htr4-null mice differed little from

164 ormal semen quality and changes in chemokine-cytokine profiles in infertile men.

165 r different inflammatory cell population and cytokine profiles in lesional cutaneous lupus erythemato

166 play a role in the bedside identification of cytokine profiles in patients with sepsis.

167 ombocytopenia and vascular leak with altered cytokine profiles in plasma are features of severe dengu

168 thod, we demonstrate that HIEs have distinct cytokine profiles in response to pro-inflammatory stimul

169 in MR1(+/+) Valpha19i-Tg mice had disparate cytokine profiles in response to RL Ag.

170 portion of Atp6v0a2) and secreted chemokine-cytokine profiles in seminal fluid were measured.

171 IgG reactivity to common target antigens and cytokine profiles in sputum samples were examined.

172 The aim of this study was to compare cytokine profiles in stimulated primary mononuclear cell

173 The aim was to review the cytokine profiles in the GCF of patients with periodonti

174 sbiosis of the gut microbiota would suppress cytokine profiles in the host, thereby leading to change

175 nally, we observed pronounced differences in cytokine profiles in the livers of mutant-infected mice,

176 iated with modifications in the cellular and cytokine profiles in the lung.

177 this study was to classify these same plasma cytokine profiles in the three achalasia subtypes.

178 We sought to investigate SC IL-1 cytokine profiles in the uninvolved skin of controls and

179 Differences between cytokine profiles in transmitters versus non-transmitter

180 Differences between cytokine profiles in transmitters versus nontransmitters

181 subphenotypes are associated with consistent cytokine profiles in two distinct cohorts of infected pa

182 Cytokine profiling in locally inflamed DRG showed increa

183 We conducted cytokine profiling in tumor, lymph nodes, and serum of a

184 metabolism and reversed the proinflammatory cytokine profile, in part due to the restoration of Lin2

185 (IL)-10 production in vitro, distinct to the cytokine profile induced by Toll-like receptor ligation.

186 ders had markedly different changes in serum cytokine profiles induced by duvelisib.

187 mune polarization and whether post-treatment cytokine profiles influence reinfection status.

188 Most importantly, it skewed the cytokine profiles into antitumor one in the tumor microe

189 On cytokine profiling, LDA increased placental growth facto

190 This high iron content alters macrophage cytokine profiles, leads to higher arginase level and ac

191 ifying the risk of an asthma exacerbation by cytokine profile may aid the targeting of personalized t

192 Here, we sought to determine whether plasma cytokine profiles may provide further information into t

193 es and resolution were characterized through cytokine profiling, microscopy, and quantitation of epit

194 ombination of proinflammatory and regulatory cytokine profiles, natural killer cells showed a predomi

195 Inflammatory cell and cytokine profile (nuclear factor-kappaB, IL-6, tumor nec

196 late with the ex vivo MAIT hyperinflammatory cytokine profile observed in older adults.

197 We aimed to describe the cytokine profile observed in the duodenal mucosa of pati

198 but was not associated with the prototypical cytokine profile observed in women with intra-amniotic i

199 The cytokine profile observed suggests a skew towards inappr

200 The inflammatory CSF cytokine profiles observed at time of IRIS can distingui

201 a source of C3a, and its uptake altered the cytokine profile of activated CD4+ T cells.

202 ite antigens and pollen allergens and/or the cytokine profile of allergic individuals.

203 g that expression of TL by IEC modulates the cytokine profile of CD4(+) T cells favoring IL-17 produc

204 pletely understood.Objectives: To define the cytokine profile of COVID-19 and to identify evidence of

205 ich were also correlated with changes in the cytokine profile of crewmembers.

206 t neonatal TBI study demonstrated a reversed cytokine profile of downregulation.

207 The cytokine profile of gammadelta T cells is hard-wired alr

208 n this article, we sought to investigate the cytokine profile of IFN-gamma-activated keratinocytes, a

209 The phenotype and cytokine profile of IL-17(+)Foxp3(+)CD4(+) T cells was o

210 Further examination of the cytokine profile of iNKT-keratinocyte cocultures showed

211 We studied the effect of HLA-G5 on the cytokine profile of purified human macrophages and Ag-sp

212 ve response was evaluated by determining the cytokine profile of the draining lymph node (LN) cells o

213 e IL-6R activity might serve to maintain the cytokine profile of the Th cell infiltrate.

214 subsets which mirror the transcriptional and cytokine profile of their T cell subset counterparts.

215 ppressor cells, and changes in the chemokine/cytokine profile of tumors.

216 We examined serum cytokine profiles of 411 patients with HCC (n = 102: 32%

217 ogen-specific changes after 1 year of ART in cytokine profiles of CD4 T-cell responses that were asso

218 We examined the cytokine profiles of human PBMCs and found that subsets

219 We aimed to examine the cytokine profiles of individuals sensitized to eight com

220 We assessed cytokine profiles of liver tissues using a multiplexed a

221 V populations, potential differences in milk cytokine profiles of natural and nonnatural SIV hosts we

222 rikingly, there were also differences in the cytokine profiles of the chronically infected patients r

223 Sickness behavior, temperature, and cytokine profiles of the pregnant monkeys confirmed a st

224 Sputum cellular and cytokine profiles of the validation subgroups were simil

225 Cytokine profiling of GEM-treated tumor cells identifies

226 Extensive cytokine profiling of sputum revealed a TH2-dominated mi

227 Cytokine profiling of supernatants from noncontact cocul

228 city associated with a raised serum Th1 type cytokine profile on transfer into preconditioned mice.

229 , plasma aspartate aminotransferase, hepatic cytokine profile, oxidative stress, and terminal deoxynu

230 analysis of cytokine responses and compared cytokine profiles, pathologies, and macrophage (Mac) pol

231 s) were originally classified based on their cytokine profiles, placing natural killer (NK) cells and

232 A T-helper (Th) 2 cell cytokine profile predominates in liver tissues from thes

233 ished HIV-1 infection and alterations of the cytokine profile produced by brain pericytes.

234 he maternal fetal interface by modifying the cytokine profile produced by the stromal/decidual cells.

235 anti-Ly6C mAb modulated the Ly6Chi monocyte cytokine profile, reduced monocyte recruitment to the sp

236 Histological examination and cytokine profiling reveal that intramuscular (i.m.) admi

237 Factor and cluster analyses of the sputum cytokine profiles revealed 3 biological clusters: cluste

238 Multiplex cytokine profiling revealed production of an array of pr

239 Cytokine profiling revealed that ODNs promote polarizati

240 T(3) reversed many of the lung chemokine and cytokine profiles seen in response to VILI, demonstratin

241 d upon activation produce a pro-inflammatory cytokine profile similar to blood monocyte-derived macro

242 as associated with higher IL-17A and IL-6, a cytokine profile similar to other autoimmune diseases.

243 with TNF-alpha after toxin altered the renal cytokine profile so that the expression of proinflammato

244 This occurred with a modification of the cytokine profile, such as an increase in IFN-gamma and a

245 th or without HCC, have distinctly different cytokine profiles, suggesting potential differences in d

246 y analyzing the cell morphology, LPS-induced cytokine profile, surface marker expression, and phagocy

247 lper T cell 1 (Th1) to helper T cell 2 (Th2) cytokine profile switch.

248 fficacious and, based on its proinflammatory cytokine profile, targeted treatment option in PRP.

249 CACs expressed a broader cytokine profile than CSCs, with 3 cytokines in common.

250 and cultured T cell clones did not express a cytokine profile that indicated immune-dampening propert

251 matopoietic, proinflammatory, and pro-atopic cytokine profile that may aid in treatment.

252 sing hESC-DCs also induced a proinflammatory cytokine profile that may favor the T cell priming.

253 tive signs on the day of blood drawing had a cytokine profile that was similar to that of non-AMD ind

254 isplay a dominant TH1 signature and atypical cytokine profiles that link to allergic status.

255 In patients with PMF who were studied by cytokine profiling, the prognostic value of an increased

256 ough T cell subsets are routinely defined by cytokine profiles, there may be important subdivisions b

257 n DCs accelerated maturation and shifted the cytokine profile, thereby favoring the differentiation o

258 Based on their unique surface markers and cytokine profiles, these cells were confirmed as group 3

259 FLG) is associated with an increased SC IL-1 cytokine profile; this profile is also seen in a murine

260 matory genes and dampens the proinflammatory cytokine profile through PI3K-mediated downregulation of

261 latent herpesviruses can directly alter host cytokine profiles through viral expression of cytokine-l

262 ction, murine mphis elicited an inflammatory cytokine profile (TNF-alpha >> TGF-beta + IL-10) and low

263 and therapy was shown to alter diabetogenic cytokine profile, to diminish T-cell effector frequency

264 patients with SR asthma by directing the SR cytokine profile toward a more SS immune phenotype, sugg

265 ed with the myeloid subpopulation and T cell cytokine profiles underlying mutual activation between b

266 Based on their cytokine profile upon exposure to pathogenic stimuli, hu

267 e supernatant (plasma) harvested to evaluate cytokine profiles using Enzyme-Linked Immunosorbent Assa

268 We monitored the cytokine profile (using enzyme-linked immunosorbent assa

269 ociated with ASD later in childhood and that cytokine profiles vary depending on ASD severity.

270 Cytokine profiles, viral cycle thresholds (Cts), blood s

271 The anti-C1q-induced inflammatory cytokine profile was accompanied by a downregulation of

272 An inflammatory mucosal and systemic cytokine profile was characterized by expression of IL-1

273 activated CD4(+)-T cells with a Th1 and Th17 cytokine profile was increased in cMy-mOVA-OT-II mice af

274 The same cytokine profile was observed in ex vivo culture of cerv

275 altered T cell functional potential; T cell cytokine profile was preserved.

276 sal TH1 to TH2 or T-cytotoxic 1 (TC1) to TC2 cytokine profiles was not evident.

277 Cytokine profiling was conducted by using a multiplex ar

278 ctivation, animal survival, lung injury, and cytokine profile were assessed.

279 ntitumor efficacy, whereas T cell number and cytokine profile were not.

280 TRIF-signaling pathways and their associated cytokine profiles were altered in the absence of CD14 in

281 Lamina propria dendritic cell phenotype and cytokine profiles were assessed by flow cytometric analy

282 intestinal histology and systemic and local cytokine profiles were compared between the treated and

283 Cytokine profiles were evaluated by ELISA.

284 Cell supernatant cytokine profiles were evaluated by multiplex assays.

285 Cytokine profiles were evaluated by proteome microarray

286 rkers than those without ACLF; (2) different cytokine profiles were identified according to the type

287 ent to lesions, degree of tissue damage, and cytokine profiles were measured.

288 croglial activation, astrogliosis, and brain cytokine profiles were not altered by SARM1 deficiency.

289 miRNA-mediated changes in cytokine profiles were observed at transcriptional and/o

290 Cytokine profiles were obtained 20 min after the first s

291 Plasma pro- and antiinflammatory cytokine profiles were performed by enzyme-linked immuno

292 nohistochemical testing, flow cytometry, and cytokine profiling were performed on samples from the pa

293 sting, flow cytometry, and transcriptome and cytokine profiling were performed.

294 Genome-wide microarrays and cytokine profiling were used to interrogate the impact o

295 CD91 interaction on APCs stimulates a unique cytokine profile, which dictates priming of specific Th

296 ance of apoptotic cells and alters the serum cytokine profile, which in turn provokes the generation

297 g) NC mice showed a T helper type-1-dominant cytokine profile, which may account for the expansion of

298 ular mechanisms were associated with a mixed cytokine profile with a tendency toward increased levels

299 Alterations in immune cytokine profiles with aging show significant gender spe

300 on resulted in a significant reversal of the cytokine profile, with increased levels of proinflammato