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1                       Our findings thus link cytokinetic abnormalities to a hereditary cancer syndrom
2  the PRD is required for ALIX to function in cytokinetic abscission and retroviral budding, but not i
3 ncreases this postmitotic process and delays cytokinetic abscission by keeping the abscission checkpo
4 the absence of ESCRT function in C. elegans, cytokinetic abscission is delayed but can be completed,
5                              We propose that cytokinetic abscission is driven by an ESCRT-III fission
6 ate quantitative imaging of ESCRT-III during cytokinetic abscission with biophysical properties of ES
7                                       During cytokinetic abscission, the endosomal sorting complex re
8                                              Cytokinetic abscission, the final stage of cell division
9 ry plays an evolutionarily conserved role in cytokinetic abscission, the final step of cell division
10  between postmitotic genome surveillance and cytokinetic abscission.
11 , HIV budding, nuclear envelope closure, and cytokinetic abscission.
12 est a computational model for ESCRT-mediated cytokinetic abscission.
13 ays critical roles in retroviral budding and cytokinetic abscission.
14 iotemporal control of the ESCRT machinery of cytokinetic abscission.
15 n maturation of NSC midbodies, which mediate cytokinetic abscission.
16 cal separation between daughter cells during cytokinetic abscission.
17 ctility and formin-dependent assembly of the cytokinetic actin contractile ring.
18                                              Cytokinetic actin ring (CAR) formation in Schizosaccharo
19 e IQGAP-related protein Iqg1 (Cyk1) promotes cytokinetic actin ring formation and is required for cyt
20           mto2Delta cells fail to anchor the cytokinetic actin ring in the medial region of the cell
21 and the EMTOC are critical for anchoring the cytokinetic actin ring to the medial region of the cell
22 pparatus determine the position at which the cytokinetic actomyosin array forms, but the molecular me
23 uctures, such as actin stress fibers and the cytokinetic actomyosin contractile ring.
24 g protein, Rho1/RhoA plays a central role in cytokinetic actomyosin ring (CAR) assembly and cytokines
25 atids are separated to opposite sides of the cytokinetic actomyosin ring (CAR).
26 s regulatory proteins play a central role in cytokinetic actomyosin ring assembly and cytokinesis.
27                            In budding yeast, cytokinetic actomyosin ring contraction and membrane ing
28 that did not constrict with actomyosin ring (cytokinetic actomyosin ring) invagination; instead, it s
29  chain that is an essential component of the cytokinetic actomyosin ring.
30 m a three-component system that co-ordinates cytokinetic and cell wall homeostatic processes.
31 aneous gcrA/ccrM disruption ameliorating the cytokinetic and growth defect of DeltagcrA cells.
32       Animal cells decide where to build the cytokinetic apparatus by sensing the position of the mit
33                            However, once the cytokinetic apparatus expands into a ring the MAP become
34                         In normal cells, the cytokinetic apparatus forms in a region of lower cortica
35 l by which the central spindle organizes the cytokinetic apparatus is premised on an antiparallel arr
36                                          The cytokinetic apparatus of bacteria is initially formed by
37    In plant cells, microtubules (MTs) in the cytokinetic apparatus phragmoplast exhibit an antiparall
38    We conclude that microtubules specify the cytokinetic apparatus via a dynamic zone of local RhoA a
39 lp1 out of the nucleolus, 2) maintaining the cytokinetic apparatus, and 3) halting the cell cycle unt
40               The current model of the plant cytokinetic apparatus, describing it as being composed o
41 is in plant cells depends on guidance of the cytokinetic apparatus, the phragmoplast, to a cortical "
42 cortex is contacted by the outwardly growing cytokinetic apparatus.
43 otic spindle and cleavage of the cell by the cytokinetic apparatus.
44 e our model to estimate the duration of post-cytokinetic attachment between a S.cerevisiae mother and
45 e specificity is unclear but could involve a cytokinetic birth scar that marks the newborn pole as th
46 h genes resulted in lethality and a complete cytokinetic block, suggesting overlapping function.
47  two-pronged recruitment of ESCRT-III to the cytokinetic bridge and implicates ALIX in abscission che
48 c spindle function and the resolution of the cytokinetic bridge because its depletion resulted in spi
49 h KV mitotic cells strategically place their cytokinetic bridges at the rosette center, where Rab11-a
50 t KV-destined cells remain interconnected by cytokinetic bridges that position at the rosette's cente
51                                  We explored cytokinetic calcium transients in the fission yeast Schi
52 tomyosin ring generates force to ingress the cytokinetic cleavage furrow in animal cells, yet its fil
53     We suggest that chromatin trapped in the cytokinetic cleavage furrow is the more likely reason fo
54                                          The cytokinetic cleavage furrow is typically positioned symm
55 not localize to the interphase cortex or the cytokinetic cleavage furrow, whereas a 500-residue regio
56 on promotes filament reorientation along the cytokinetic cleavage furrow, which might have implicatio
57 calizes to the interphase cortex but not the cytokinetic cleavage furrow.
58 omyces pombe proteins that contribute to the cytokinetic contractile ring accumulate during interphas
59    Our findings reveal how components of the cytokinetic contractile ring are reemployed during inter
60  proteins to test the popular model that the cytokinetic contractile ring assembles from a single myo
61 s centralspindlin activates ECT-2 to promote cytokinetic contractile ring formation, we show that the
62  that fission yeast assemble and constrict a cytokinetic contractile ring in a precisely timed, seque
63 assembly of cortical nodes that generate the cytokinetic contractile ring in fission yeast.
64                                Assembly of a cytokinetic contractile ring is a form of cell polarizat
65 o-dependent proteins control assembly of the cytokinetic contractile ring, yet it remains unclear how
66 in patches, polarizing actin cables, and the cytokinetic contractile ring.
67 2) and other proteins that condense into the cytokinetic contractile ring.
68 ivation, Rng3p colocalizes with Myo2p in the cytokinetic contractile ring.
69          Only Dia3 strongly localized at the cytokinetic contractile ring.
70   Formins polymerize actin filaments for the cytokinetic contractile ring.
71 gether and mature into the precursors of the cytokinetic contractile ring.
72  most advanced models of the dynamics of the cytokinetic contractile ring.
73 of the proteins in the functional units of a cytokinetic contractile ring.
74 processive elongation of actin filaments for cytokinetic contractile rings and other cellular structu
75 n yeast lacking Aip1 are viable and assemble cytokinetic contractile rings normally, but rings in the
76  regulators of the MEN, failed to remedy the cytokinetic defect of these mutants, indicating that Cdc
77 ween the connected cell bodies, indicating a cytokinetic defect.
78 er-associated mutations results in increased cytokinetic defects but has no effect on BRCA2-dependent
79 d midzone formation can be restored, and the cytokinetic defects can be rescued in Kif4 esiRNA-treate
80                                        These cytokinetic defects correlate with cardiomyocyte ineffic
81 in cell plate formation are seedling lethal, cytokinetic defects in et2 predominantly occur in flower
82 use attenuation of MOR signaling rescued the cytokinetic defects of SIN mutants and allowed weak SIN
83 erations in cell wall formation, and similar cytokinetic defects were sporadically observed in other
84 terol biosynthesis enzyme, also lead to weak cytokinetic defects, primarily in the flowers.
85 e formation of multipolar spindle arrays and cytokinetic defects.
86                                          The cytokinetic division ring of Escherichia coli comprises
87 -to-pole oscillation to help ensure that the cytokinetic division septum forms only at the mid-cell p
88              An extreme example of this post-cytokinetic DNA segregation occurs during spore formatio
89 f metazoans, and the third gives rise to the cytokinetic dynamins of amoebozoans and plants and to ch
90 demonstrate that bcl-XS can have substantial cytokinetic effects under circumstances that produce rel
91 e we use cellularization, the first complete cytokinetic event in Drosophila embryos, to show that cl
92 he Drosophila embryo undergoes a large-scale cytokinetic event that packages thousands of syncytial n
93 20 and SPG20 mRNAs enabled the enrichment of cytokinetic events (CDC20(high)SPG20(high)).
94 me segregation, the mitotic spindle controls cytokinetic events at the cell envelope.
95 ormed a Z-ring at a time in development when cytokinetic events normally have ceased.
96 tly regulated to ensure that it occurs after cytokinetic events such as chromosome segregation.
97 1 is also required for the highly asymmetric cytokinetic events that extrude the two polar bodies dur
98 at recruit specific proteins and orchestrate cytokinetic events, such as sister nuclei being kept apa
99                     Similarly, the essential cytokinetic factor anillin, which functions at the cell
100 ated spindles with a diffuse distribution of cytokinetic factors.
101 of MICAL3 leads to an increased frequency of cytokinetic failure and a delayed abscission.
102                                Additionally, cytokinetic failure at meiosis II gives rise to bi-nucle
103                                 The frequent cytokinetic failure caused by loss of MAP65-3 was not re
104 cally, NMIIB-deficient spermatocytes exhibit cytokinetic failure in meiosis I, resulting in bi-nuclea
105 mentin mutant in T24 cultured cells leads to cytokinetic failure, resulting in binucleation (multinuc
106 n, slower gravitropic response in roots, and cytokinetic failure.
107              Here, we identify the essential cytokinetic formin Cdc12 as a key CR substrate of SIN ki
108 N-triggered oligomeric switch that modulates cytokinetic formin function, revealing a novel mechanism
109 rcumferential ring structures that flank the cytokinetic FtsZ ring and appear to be associated with d
110 ter membrane depends on the formation of the cytokinetic FtsZ ring at midcell.
111 a formin Cdc12 recruitment, defining a novel cytokinetic function for an F-BAR domain.
112 per execution of cytoskeletal remodeling and cytokinetic functions.
113  localizations of the CPC are coupled to its cytokinetic functions.
114 s are required for proper positioning of the cytokinetic furrow [1] [2], the role of pre-anaphase mic
115  to sense micron-scale contours, such as the cytokinetic furrow and base of neuronal branches.
116 ere is micron-scale curvature, including the cytokinetic furrow and the base of cell protrusions.
117                                          The cytokinetic furrow arises from spatial and temporal regu
118                                          The cytokinetic furrow cleaves the cell by ingressing from b
119 ors at the spindle pole body help coordinate cytokinetic furrow formation in fission yeast.
120  gross defects in chromosome segregation and cytokinetic furrow ingression.
121         Chiral morphogenesis is timed by the cytokinetic furrow of a neighbor of the sister pair, pro
122 ionship between microtubule organization and cytokinetic furrow position.
123 nd GEF-H1, LARG depletion does not result in cytokinetic furrow regression nor does it affect interna
124    Loss of Nm23-H1 in diploid cells leads to cytokinetic furrow regression, followed by cytokinesis f
125            In animal cells, formation of the cytokinetic furrow requires activation of the GTPase Rho
126  disjunction, anaphase B, and formation of a cytokinetic furrow, which split the spindle.
127  of the spindle apparatus and ultimately the cytokinetic furrow.
128 idzone coordinately directs formation of the cytokinetic furrow.
129 cortex and, thus, blocking initiation of the cytokinetic furrow.
130 targeting, is required for elongation of the cytokinetic furrow.
131 yosin can both determine the position of the cytokinetic furrow.
132 vity in mammalian cells and demonstrate that cytokinetic furrowing is primarily regulated at the leve
133 yosin II in the cytokinetic ring, and faster cytokinetic furrowing, following depletion of GCK-1 or C
134 ity drives many cell shape changes including cytokinetic furrowing.
135  which occurs during anaphase B and prior to cytokinetic furrowing.
136  nonmotor cross-linkers affects the speed of cytokinetic furrowing.
137  contractility is responsible for asymmetric cytokinetic furrowing.
138 broader region of ingressing membrane during cytokinetic furrowing.
139 ion of RhoA activity, leading to assembly of cytokinetic furrows that partially ingress.
140  thought to provide the ingression force for cytokinetic furrows, but the role of membrane traffickin
141 tion of binucleated cells by stabilizing the cytokinetic intercellular bridge (ICB).
142 division proteins, ultimately assembling the cytokinetic machine that splits the cell.
143  protein initiates assembly of the bacterial cytokinetic machinery by polymerizing into a ring struct
144 ins are likely to be a common feature of the cytokinetic machinery in bacteria.
145  evidence indicates that ZapA is part of the cytokinetic machinery of the cell and acts by promoting
146 estricts growth to cell ends and targets the cytokinetic machinery to the middle of the cell.
147 ndomembrane vacuoles, mislocalization of the cytokinetic machinery, and extensive cortical membrane b
148 ocalization, microtubule biogenesis, and the cytokinetic machinery, as well as a substantial uncoupli
149 ules and without canonical components of the cytokinetic machinery.
150 until chromosomes have been cleared from the cytokinetic machinery.
151     These PRC1-mediated modifications to the cytokinetic mechanism may be related to the specializati
152 essful cytokinesis in budding yeast, but new cytokinetic mechanisms can evolve through genetic change
153 suggests a direct role for these proteins in cytokinetic membrane abscission.
154 ed precise co-localization to interphase and cytokinetic microtubule arrays.
155  ESCRT-III polymerization at the edge of the cytokinetic midbody structure, located at the center of
156 ulating these events, as it localizes to the cytokinetic midbody.
157 all stubs observed upon drug treatment or in cytokinetic mutants.
158 ition to localizing at the spindle poles and cytokinetic neck filaments, Cdc5 induces and localizes t
159 ddition to localization at spindle poles and cytokinetic neck filaments, Plk induces and localizes to
160 y of Cdc5p to spindle pole bodies (SPBs) and cytokinetic neck-filaments.
161 dies concluded that cells without Mid1p lack cytokinetic nodes and assemble rings unreliably from myo
162 -II, Rng2p, and Cdc15p to nodes and to place cytokinetic nodes around the cell equator.
163                              Myo2 appears in cytokinetic nodes around the equator 10 min before spind
164                                          How cytokinetic nodes assemble, whether the order of assembl
165 eral kinases appear early in G2, mature into cytokinetic nodes by adding anillin Mid1p, myosin-II, fo
166                                  Assembly of cytokinetic nodes requires Mid1p, which recruits IQGAP-r
167 n Deltamid1 cells that Cdc12p accumulates in cytokinetic nodes scattered in the cortex and produces a
168                              During mitosis, cytokinetic nodes with Mid1p and all of the type 2 node
169 g and de novo assembly of the plant-specific cytokinetic organelle, the cell plate, which develops ac
170  complex activation and a polo-box-dependent cytokinetic pathway.
171 n inhibition of Cyk2/Hof1- and Myo1-mediated cytokinetic pathways.
172 ediated lethality could not be attributed to cytokinetic perturbations, nor did ara-CTP formation or
173 nd other drugs, we showed that exit from the cytokinetic phase of the cell cycle depends on ubiquitin
174  either cyk3 or hof1 alone results in a mild cytokinetic phenotype [5-7], but deletion of both genes
175         Depletion of MITD1 causes a distinct cytokinetic phenotype consistent with destabilization of
176                Following nuclear division, a cytokinetic phragmoplast forms between the daughter nucl
177 itosis, and relocates to the recently formed cytokinetic plane, where it establishes a fully polarize
178                          Microtubules direct cytokinetic polarization via the central spindle and ast
179          If this interaction is compromised, cytokinetic precursors are asymmetrically distributed in
180 network (SIN) induces Cdr2 dissociation from cytokinetic precursors at this stage [12-14].
181 iates and whether this step is important for cytokinetic progression has been unknown.
182 sion are mediated in part by the actin-like, cytokinetic protein FtsA.
183  formation of higher-order assemblies of the cytokinetic protein in vitro.
184 id-cell was dependent on the presence on the cytokinetic protein, FtsZ.
185                Our data demonstrate that the cytokinetic proteins epithelial cell transforming 2 and
186 as a mechanical scaffold that recruits other cytokinetic proteins to establish functional divisomes.
187  Chk1 in mitotic mammalian cells resulted in cytokinetic regression and binucleation, increased chrom
188        We observed that Aurora B, a critical cytokinetic regulator and a recently identified Chk1 sub
189 s-Galbraith and colleagues report that a key cytokinetic regulator in fission yeast, Cdc15, is phosph
190 factor ECT2, an orthologue of the Drosophila cytokinetic regulator Pebble, providing a direct means f
191 otes the equatorial recruitment of important cytokinetic regulators.
192 s approach identifies Aip1, Ede1 and Inn1 as cytokinetic regulators.
193 eviously utilized cell division sites called cytokinetic remnants (CRMs).
194                                              Cytokinetic RhoA activity zones are common to four echin
195  We propose that the most active form of the cytokinetic RhoGEF involves complex formation between EC
196 ide using a contractile apparatus called the cytokinetic ring (CR) that associates dynamically with t
197 g and constricting a medial actomyosin-based cytokinetic ring (CR).
198 FtsA are recruited independently to the FtsZ cytokinetic ring (Z ring) and are essential for cell div
199                  Gef1 localizes first to the cytokinetic ring and promotes timely constriction, where
200 ount of FtsZ available for assembly into the cytokinetic ring and with it cell size.
201                Assembly of proteins into the cytokinetic ring appears to occur in a hierarchial order
202 tile ring assembly in vivo.The fission yeast cytokinetic ring assembles by Search-Capture-Pull-Releas
203  migrates to one side of the cell before the cytokinetic ring assembles on the opposite cortex.
204 ization, the small GTPase Rho and formins in cytokinetic ring assembly.
205 l player in the cytoskeletal family, forms a cytokinetic ring at mid-cell, and recruits the division
206            The bacterial GTPase FtsZ forms a cytokinetic ring at midcell, recruits the division machi
207  that the amidases require activation at the cytokinetic ring by proteins with LytM domains, of which
208                                        Thus, cytokinetic ring closure is promoted by moderate levels
209                 Here we report how two novel cytokinetic ring components, GCK-1 (germinal center kina
210                                       Before cytokinetic ring constriction, Cdc42 activation, is Gef1
211               In addition, the inhibition of cytokinetic ring contraction can be reversed by exposure
212 us reveal a key role for amidase activity in cytokinetic ring contraction.
213 cles through contraction and relaxation, the cytokinetic ring disassembles during contraction through
214 es of FtsZ, a bacterial protein that forms a cytokinetic ring during cell division, are essential for
215 zone from late anaphase and localizes to the cytokinetic ring during cytokinesis.
216  proposed to arise from stabilization of the cytokinetic ring during incomplete cytokinesis [1].
217 B, but not delta N592, were localized to the cytokinetic ring during mitosis, indicating that, in ver
218 mbly and in maintaining the integrity of the cytokinetic ring during the early stages of division.
219 nt tubulin homolog, FtsZ, for assembling the cytokinetic ring essential for cell division, but are ot
220 ation may help bridge two existing models of cytokinetic ring formation.
221                                          The cytokinetic ring generates tensile force that drives cel
222                EzrA also concentrates at the cytokinetic ring in an FtsZ-dependent manner, although i
223  to filaments in interphase cells and to the cytokinetic ring in dividing cells.
224 he localization of Tol-Pal components to the cytokinetic ring in Escherichia coli has led to the prop
225 n-related protein Mid1 does not position the cytokinetic ring in the fission yeast Schizosaccharomyce
226 esis is the assembly of a stable but dynamic cytokinetic ring made up of the essential tubulin homolo
227 earlier than normal, shortening the stage of cytokinetic ring maturation by 50%.
228   Pom1 restricts to the cell middle cortical cytokinetic ring precursor nodes organized by the SAD-li
229 is high, which ensures proper positioning of cytokinetic ring precursors at the cell geometrical cent
230 s, first promotes the medial localization of cytokinetic ring precursors organized by the SAD kinase
231 tly disrupt the assembly or stability of the cytokinetic ring protein FtsZ, nor does it affect the re
232  tubulins, is a GTPase that assembles into a cytokinetic ring structure essential for cell division i
233 its bacterial homologue FtsZ establishes the cytokinetic ring that constricts during cell division.
234 -like FtsZ GTPase into a membrane-associated cytokinetic ring that defines the division plane in bact
235 ess fibers (SFs), muscle sarcomeres, and the cytokinetic ring to both generate and sense mechanical f
236 orescent phalloidin into the medium, and the cytokinetic ring was disrupted after injection of the my
237 ordinates assembly and placement of the FtsZ cytokinetic ring with bipolar localization of the newly
238 multinucleate cells, failure to maintain the cytokinetic ring, and compromised SPB association of the
239 to perform two functions: stabilize the FtsZ cytokinetic ring, and facilitate septal peptidoglycan sy
240 vity, anillin and nonmuscle myosin II in the cytokinetic ring, and faster cytokinetic furrowing, foll
241 ssential for ensuring proper assembly of the cytokinetic ring, and its deletion leads to mis-localiza
242     Cytokinesis in bacteria is mediated by a cytokinetic ring, termed the Z ring, which forms a scaff
243  recruited by active RhoA and anillin to the cytokinetic ring, where they in turn limit RhoA activity
244 pombe, cytokinesis also involves a conserved cytokinetic ring, which has been generally assumed to pr
245 ents of the tubulin-like protein FtsZ into a cytokinetic ring, which then constricts.
246 mation, maintenance, and constriction of the cytokinetic ring.
247 nd concentrated at the cell poles and/or the cytokinetic ring.
248  of downstream proteins to form a functional cytokinetic ring.
249 overned by conserved subcomplexes within the cytokinetic ring.
250 tsW to prevent the final constriction of the cytokinetic ring.
251 ling their activation to the assembly of the cytokinetic ring.
252 at is required for the dynamic nature of the cytokinetic ring.
253 has been shown to inhibit contraction of the cytokinetic ring.
254 ytoplasmic actin cables, and the actin-based cytokinetic ring.
255  II may be necessary for localization to the cytokinetic ring.
256 nchoring scheme that generate tension in the cytokinetic ring.
257 ynamics in the Caenorhabditis elegans zygote cytokinetic ring.
258  cell division and final constriction of the cytokinetic ring.
259 age of cell wall material synthesized by the cytokinetic ring.
260 AR protein Cdc15, a central component of the cytokinetic ring.
261  may be in scaffolding, not positioning, the cytokinetic ring.
262 dge stability by anillins, key regulators of cytokinetic rings and cytoplasmic bridges [1, 4-7].
263 assembly of diverse actin structures such as cytokinetic rings and filopodia.
264 adoxically, proteins that promote closure of cytokinetic rings are enriched on stably open intercellu
265 structures such as filopodia, stress fibers, cytokinetic rings, and focal adhesions.
266 ity of myo2-E1-Sup1 cells depend on the late cytokinetic S. pombe myosin II isoform, Myp2p, a non-ess
267 nvestigate the F-BAR domain of the essential cytokinetic scaffold, Schizosaccharomyces pombe Cdc15, d
268           In the presence of dicentrics, the cytokinetic septa pinch the nucleus, suggesting that dic
269 he cell, ensuring midcell positioning of the cytokinetic septum.
270 represents a key step in the delivery of the cytokinetic signal to the cortex.
271 ses, the cell assembles essentially the same cytokinetic signaling ensemble-opposed astral microtubul
272 e cell cycle in a ring that marks the future cytokinetic site.
273 e polar and new static complexes form at pre-cytokinetic sites, ensuring positioning at the new pole
274                               These inactive cytokinetic SNARE complexes were already assembled at th
275                   FtsZ is part of a mid-cell cytokinetic structure termed the Z-ring that recruits a
276 es have noted important distinctions between cytokinetic structures in dividing cells and muscle sarc
277 cates from the pericentrosomal region to key cytokinetic structures including the cleavage furrow, an
278 transitional hourglass that pre-patterns two cytokinetic structures-a septin double ring and an actom
279 nderlying the assembly and regulation of the cytokinetic structures.
280 subcellular locations and induce or organize cytokinetic structures.
281 tes the DivJ kinase and directly acts on the cytokinetic tubulin, FtsZ, to tune cytokinesis with the
282 ial tubulin homolog required to assemble the cytokinetic Z ring and recruit the components of the div
283 n Escherichia coli, precise placement of the cytokinetic Z ring at midcell requires the concerted act
284 lis involves a switch in the location of the cytokinetic Z ring from midcell to the pole.
285                                          The cytokinetic Z ring is required for bacterial cell divisi
286 gulatory system that limits formation of the cytokinetic Z ring to midcell by preventing its formatio
287  local negative control over assembly of the cytokinetic Z ring to prevent potential cutting of the c
288 n-like FtsZ protein, helping to assemble the cytokinetic Z ring, anchor it to the cytoplasmic membran
289 inD is involved in spatial regulation of the cytokinetic Z ring, and ParAs are involved in chromosome
290 ght about by a change in the location of the cytokinetic Z ring, which is composed of the tubulin-lik
291 ation required for spatial regulation of the cytokinetic Z ring.
292 , forming a complex that can destabilize the cytokinetic Z ring.
293 erizes in a GTP-dependent manner to form the cytokinetic Z ring.
294 ted by FtsZ, which polymerizes to create the cytokinetic Z ring.
295                         The formation of the cytokinetic Z-ring by the tubulin homologue FtsZ is regu
296  GTPase FtsZ assemble at midcell to form the cytokinetic Z-ring, which coordinates peptidoglycan (PG)
297 protein FtsZ assemble at midcell to form the cytokinetic Z-ring.
298 e tubulin-like GTPase, FtsZ, which forms the cytokinetic Z-ring.
299                        This process requires cytokinetic Z-rings formed by the bacterial tubulin homo
300 symmetric division involves the formation of cytokinetic Z-rings near both poles of the developing ce

 
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