ライフサイエンスコーパス: cytolysis

1 ial cells (i.e. apoptosis, barrier function, cytolysis).

2 ut do not support full activation or sustain cytolysis.

3 the interface center were closely related to cytolysis.

4 g of tumors that are usually resistant to NK cytolysis.

5 umans, are highly susceptible to PVL-induced cytolysis.

6 ot mediate resistance to CD8 T cell-mediated cytolysis.

7 sceptible to perforin-dependent CTL-mediated cytolysis.

8 lls occurs through TAA-specific CTL-mediated cytolysis.

9 omal destabilization correlated closely with cytolysis.

10 usceptibility of tumor cells to CTL-mediated cytolysis.

11 satory manner dominated by perforin-mediated cytolysis.

12 fic IL-10-positive CD8(+) T cells suppressed cytolysis.

13 in vitro blocks tumor-induced defective TIL cytolysis.

14 d-binding or oligomerization surface reduced cytolysis.

15 ied and typically results in virally induced cytolysis.

16 cell-derived IFN-gamma via perforin-mediated cytolysis.

17 T-cell alloresponses, and were targets of NK cytolysis.

18 ntrations, far below those required to cause cytolysis.

19 nt destruction of cancer cells through rapid cytolysis.

20 the cytoskeletal rearrangements required for cytolysis.

21 oliferation, cytokine secretion, and natural cytolysis.

22 assayed by anti-NKG2D Ab-mediated redirected cytolysis.

23 a-herpesvirus CMV through perforin-dependent cytolysis.

24 FasL signaling also contributed to enhanced cytolysis.

25 uptake and dilated blebbing coincident with cytolysis.

26 9 demonstrated both IFN- gamma secretion and cytolysis.

27 cidate the molecular mechanism of eosinophil cytolysis.

28 function, including their ability to mediate cytolysis.

29 ity of normal platelets to protect them from cytolysis.

30 s and protected them from leukocyte-mediated cytolysis.

31 ue to reduced adhesion rather than increased cytolysis.

32 is factor alpha (TNF-alpha)- and Fas-induced cytolysis.

33 itro and in vivo, inducing proliferation and cytolysis.

34 1,3-Gal] inhibited both antibody binding and cytolysis.

35 M-CSF but prevented adhesion, spreading, and cytolysis.

36 e tumor by IFN-gamma production and specific cytolysis.

37 ivity to anthrax lethal toxin (LeTx)-induced cytolysis.

38 ss perforin, a molecule that is required for cytolysis.

39 ion to its defect in pore formation-mediated cytolysis.

40 kely reflecting eosinophil degranulation via cytolysis.

41 d-defective Lm is largely independent of CTL cytolysis.

42 members capable of mediating CPE binding and cytolysis.

43 ting cytoplasmic vacuolization and, finally, cytolysis.

44 activated ras are also susceptible to viral cytolysis.

45 functions including cytokine production and cytolysis.

46 centration induced passive degranulation via cytolysis.

47 e absolutely resistant to leukotoxin-induced cytolysis.

48 ells without the direct contact required for cytolysis.

49 ct of CTLA4-Ig and rapamycin on DSA-mediated cytolysis.

50 MAPK or autophagy did not affect PLO-induced cytolysis.

51 mpetent and capable of mediating/redirecting cytolysis.

52 functions, including IFN-gamma secretion and cytolysis.

53 ting cytokines and through contact-dependent cytolysis.

54 ion to ligand-expressing targets and enhance cytolysis.

55 is threshold can be reached without inducing cytolysis.

56 s, all strains show strict contact-dependent cytolysis.

57 cells also significantly impairs TCR-induced cytolysis.

58 vated NK cells susceptible to NKp80-mediated cytolysis.

59 notion that NETs can form during nonspecific cytolysis.

60 Listeria involved specialized utilization of cytolysis.

61 improved polyfunctionality and strong tumor cytolysis.

62 and promote NK cell-mediated recognition and cytolysis.

63 MTOC polarization and defective target cell cytolysis.

64 ced activity in the patient population (K562 cytolysis, 19% +/- 21% SD versus 40% +/- 17%) (P < .001)

65 lymphocytes (TIL) are severely deficient in cytolysis, a defect that may permit tumor escape from im

66 treated human NK cells exhibit reduced tumor cytolysis and abrogated perforin polarization to the imm

67 alpha was mainly responsible for enhanced NK cytolysis and also important for CD69 up-regulation, whe

68 s that degrade basement membranes and induce cytolysis and apoptosis of the cellular elements of the

69 s linked to the ability of betaH/C to induce cytolysis and apoptosis of the phagocytes.

70 of basal keratinocytes leading to epidermal cytolysis and blistering.

71 ectors of the innate immune response through cytolysis and bridge to the adaptive immune response thr

72 on of cell surface P2X7 receptors results in cytolysis and cell death of macrophages.

73 noma cell susceptibility to NK-cell-mediated cytolysis and cisplatin-induced apoptosis.

74 elanoma cell sensitivity to NK-cell-mediated cytolysis and cisplatin-induced apoptosis.

75 -dependent proliferation and specific target cytolysis and cytokine production were retained after al

76 This induces effector functions such as cytolysis and cytokine release.

77 IV-specific CD8+ T cells were discordant for cytolysis and cytokine secretion, notably IFN-gamma, whe

78 -6/CFP-10 deletion mutant all showed reduced cytolysis and cytotoxicity to macrophages and significan

79 phage lysis could play a role in LT-mediated cytolysis and discovered that a potent P2X7 antagonist,

80 but are defective in pore formation-mediated cytolysis and egress from mammalian and protozoan cells,

81 ular replication and pore formation-mediated cytolysis and egress from mammalian cells.

82 ion but defective in pore formation-mediated cytolysis and egress from protozoan and mammalian cells.

83 lecules resulted in increased serum-mediated cytolysis and eliminated the costimulatory blockade.

84 es primary breast carcinomas to CPE-mediated cytolysis and emphasizes the potential of CPE in breast

85 MCPT4]), BECs underwent caspase-1-associated cytolysis and exfoliation.

86 The contributions of perforin-mediated cytolysis and gamma interferon (IFN-gamma) secretion by

87 -positive CD8+ T cells resulted in increased cytolysis and IL-2, but not IFN-gamma, production by bot

88 tead leads to changes in genes implicated in cytolysis and NK cell function.

89 t KSR1 is required for efficient NK-mediated cytolysis and polarization of cytolytic granules.

90 type CTL, gld-CTL efficiently mediated tumor cytolysis and produced comparable amounts of IFN-gamma,

91 inflammation in mice can involve eosinophil cytolysis and release of cell-free granules.

92 d have redundant effector functions, such as cytolysis and release of potent antimycobacterial cytoki

93 T cells from normal human donors that induce cytolysis and secrete copious IFN-gamma in response to s

94 gh two contact-dependent effector functions: cytolysis and secretion of antiviral cytokines.

95 ersus control cells, thus establishing viral cytolysis and spread as the cause of the observed cell k

96 ith these pathways would modulate eosinophil cytolysis and subsequent eosinophil-driven tissue damage

97 0 and p865 CTLs as shown by peptide-specific cytolysis and tetramer staining, indicating that hTERT i

98 our knowledge of the mechanisms of host cell cytolysis and the egress of intracellular pathogens is s

99 of microtubule (MT) arrays was necessary for cytolysis and was accompanied by changes in MT dynamics

100 Expression of CD107a (a marker of cytolysis) and production of IFN-gamma and macrophage in

101 lls (suppressor cytokines, IL-2 consumption, cytolysis) and those that primarily target antigen-prese

102 ization is known to enhance NK cell-mediated cytolysis, and a potential mechanism for Crry-mediated i

103 rometastases, an increase in target-specific cytolysis, and an increase in survival correlated with d

104 er cell- and cytotoxic T-lymphocyte-mediated cytolysis, and CD4 T-cell alloproliferation.

105 functions including cytokine production and cytolysis, and differentiate into long-lived memory cell

106 irulence in animal models, mediates cellular cytolysis, and inhibits IL-12 production by mononuclear

107 es, we assessed the efficiency of infection, cytolysis, and replication of green fluorescent protein

108 ibility to natural killer (NK) cell-mediated cytolysis, and sensitivity to apoptosis were assessed.

109 Antibody blocking, redirected cytolysis, and small interfering RNA (siRNA) studies usi

110 are abnormal, with greater than 80% showing cytolysis, and therefore that evaluation by means of lig

111 al responses including IFN-gamma production, cytolysis, and tumor homing, suggesting that NK cells wi

112 Perforin-dependent cytolysis appeared to be the major cytolytic mechanism;

113 ic hepatitis C, but higher levels of NK cell cytolysis are associated with less liver fibrosis.

114 esis of antiviral beta-chemokines has joined cytolysis as a potential mechanism for the control of HI

115 protected stromal cells against PLO-induced cytolysis as determined by 3-(4,5-dimethylthiazol-2-yl)-

116 Ia-restricted CTLs, however, were capable of cytolysis as measured by redirected killing.

117 r target cell deposition during the in vitro cytolysis assay.

118 Unlike cytolysis assays with macrophages, E6 expression did not

119 In additional cytolysis assays, UL18-mediated protection was also evid

120 lization signal of VP4 were required for (i) cytolysis associated with prolonged expression; (ii) nuc

121 We confirmed cytolysis at antigen levels similar to those on primary

122 rment, however, was restricted to LT-induced cytolysis, because proteasome inhibitors did not block c

123 (i.e., proliferation, IFN-gamma production, cytolysis) between adult and old memory T cells.

124 o eliminate virally infected cells by direct cytolysis but may also contribute to pulmonary inflammat

125 tly via IFN-gamma production or directly via cytolysis, but evidence for either mechanism is largely

126 elper (Th)1 and Th2 cytokines and to mediate cytolysis, but it is unclear how these contrasting funct

127 o created to compare their susceptibility to cytolysis by diabetogenic CD8(+) T-cells in vitro.

128 Nucleated cells resist MAC-mediated cytolysis by expression of inhibitors that block MAC ass

129 nsertion of the D4 loops is required for the cytolysis by ILY.

130 nd VP3(101-120)) sensitized target cells for cytolysis by infiltrating T cells or splenic T cells fro

131 , this viral peptide inhibits HLA-E-mediated cytolysis by natural killer cells.

132 primary human immune cells in vitro against cytolysis by PVL and alpha-toxin and hence may serve as

133 ion levels suggesting that the resistance to cytolysis by rSUM149 cells was not related to MHC class

134 Our data demonstrate that the hemolysis and cytolysis by S. aureus were noticeably augmented when S.

135 rations was relatively resistant in vitro to cytolysis by sensitized T cells, whether the eyecups wer

136 This 9-mer serves to direct cytolysis by T cell lines, whereas a related 10-mer (E7(

137 ) cytolytic NK cells (P = .02), overall K562 cytolysis by unfractionated peripheral blood mononuclear

138 NK cell cytolysis can be induced directly through diverse recept

139 partially dependent on complement-dependent cytolysis (CDC), in which the immune system surveys for

140 ve CD8(+) T cells led to higher HIV-specific cytolysis compared with the removal of Nef-specific IL-1

141 Levels of adherence and cytolysis correlate for weakly adherent/cytolytic strain

142 results identified no global differences in cytolysis, degranulation, interferon-gamma production, o

143 Our data imply that cytolysis does not correlate with MEK1 cleavage, and thi

144 iltrating lymphocytes (TIL) are defective in cytolysis due to tumor-induced inhibition of proximal TC

145 n vitro resulted in rapid and dose-dependent cytolysis exclusively in breast cancer cells, correlatin

146 ith CPE resulted in rapid and dose-dependent cytolysis exclusively in cells that expressed CLDN 3 and

147 tial susceptibility of murine macrophages to cytolysis following in vitro exposure to LT, was identif

148 avidity is indicated by the independence of cytolysis from CD8/MHC class I interaction.

149 epithelial cells require a specific cue for cytolysis from recruited sentinel inflammatory cells bef

150 f this polarization and their importance for cytolysis have not been resolved.

151 achment was found to be necessary to trigger cytolysis; however, this threshold can be reached withou

152 cancer cells susceptible to NK cell-mediated cytolysis if expressed at sufficiently high levels.

153 , potassium, and magnesium in the absence of cytolysis, implicating these ion movements in the toxin'

154 and demonstrated efficient cancer-selective cytolysis in a variety of tumor cell lines, including HT

155 oguanidine (MNNG), both cell lines underwent cytolysis in a very similar manner, suggesting the prese

156 (2)) as a central molecule in NKG2D-mediated cytolysis in CTLs.

157 advances the current view of the role of CTL cytolysis in immunity to intracellular pathogens.

158 activates the NLRP3 inflammasome and causes cytolysis in mononuclear phagocytes.

159 e evaluated the effects of ITK deficiency on cytolysis in murine CTLs deficient in ITK, and both huma

160 rmine the contribution of perforin-dependent cytolysis in protective immunity to LM.

161 necrosis factor receptor 1 (TNFR1)-mediated cytolysis in regulating T-cell homeostasis.

162 sensitivity to natural killer cell-mediated cytolysis in vitro and rejection in vivo.

163 M plays a role in perforin/granzyme-mediated cytolysis in vitro.

164 l or stromal cells from trophozoite-mediated cytolysis in vitro.

165 ons display predominantly perforin-dependent cytolysis in vitro.

166 IL-4 and IL-5 and display perforin-dependent cytolysis in vitro.

167 s was safe and induced T-cell activation and cytolysis, including HIV-1-infected cells, in a subset o

168 Interestingly, triggering cytolysis is associated with an induction of autophagy i

169 We found that CTL cytolysis is critical for protective immunity to Lm capa

170 The mechanism for LT-induced cytolysis is currently unknown.

171 Cytolysis is largely Fas dependent and results from very

172 ayed-type hypersensitivity and that cellular cytolysis is the root cause of the necrosis.

173 emonstrate that an important function of CTL cytolysis is to counter the microbial virulence strategy

174 g a new method of detecting NK cell-mediated cytolysis, it was observed that NK cells efficiently lys

175 cytes and astrocytes of the CNS, it produces cytolysis, leading to formation of demyelinated lesions

176 Cytolysis levels displayed even greater variability, fro

177 ypothesized that immunological approaches to cytolysis may be used to overcome drug resistance.

178 wer than or equal to those that cause direct cytolysis, may alter the phenotype of target tissue by u

179 Consequently, PCC were highly resistant to cytolysis mediated by freshly isolated NK cells.

180 Rather than inducing cytolysis, membrane attack complexes upregulated inflamm

181 anemia, abdominal tenderness, severe hepatic cytolysis, metabolic acidosis, and hemodynamic dysfuncti

182 ysis of GalT(+) target cells, with extensive cytolysis observed consistently at serum IgM titers of >

183 free or clustered, indicate that eosinophil cytolysis occurs in vivo, but the mechanisms and consequ

184 TCR-dependent recognition leading to direct cytolysis of aminobisphosphonate-sensitized osteoclast o

185 8F4 mediated complement-dependent cytolysis of AML blasts and Lin(-)CD34(+)CD38(-) leukemi

186 y, ImmTAV-Env redirection of T cells induced cytolysis of antigen-positive HCC cells and cells infect

187 o-GM1 antiserum in vivo and NK-cell-mediated cytolysis of B16LS9 melanoma cells in vitro.

188 ylation of the cytosolic domain of CD18, and cytolysis of bovine leukocytes.

189 Importantly, we observed that the cytolysis of bystander cells and early infected cells in

190 mine whether CD95-L was sufficient to induce cytolysis of CD40-activated CLL cells, we used Chinese h

191 uction but increased perforin expression and cytolysis of cell line K562 targets.

192 cribes antigen-specific CD8+ T cell-mediated cytolysis of cognate antigen-expressing melanoma cells i

193 P-deficient NK cells that prevents efficient cytolysis of complex targets.

194 receptors is crucial for NK recognition and cytolysis of complex targets.

195 The capacity of aPA and MIP-133 to induce cytolysis of corneal epithelial cells was tested in vitr

196 o phosphoantigens and tumor cells, prevented cytolysis of Daudi B cells, and reduced cytokine product

197 bility to induce Ab and complement-dependent cytolysis of DENV-infected cells as well as to block the

198 sized to provide a first line of defence via cytolysis of dysregulated intestinal epithelial cells (I

199 , from no detectable cytolysis to 80% or 90% cytolysis of Ect1 and BPH-1, respectively.

200 ither inducing apoptosis or mediating direct cytolysis of effector T cells.

201 also did not correlate with adherence to or cytolysis of either male (BPH-1)- or female (Ect1)-deriv

202 HF are not the direct result of EBOV-induced cytolysis of endothelial cells, and are likely triggered

203 MUC-2, preventing parasite contact-dependent cytolysis of epithelial cells.

204 ti-Gal antibodies caused complement-mediated cytolysis of GalT(+) target cells, with extensive cytoly

205 demonstrated Ag-specific, non-MHC-restricted cytolysis of h5T4-positive B16 and CT26 tumor cells in v

206 Finally, in vitro IFN-alpha2a-activated NK cytolysis of HCV-infected target cells was in part depen

207 p blood agar and quantitative measurement of cytolysis of human lung epithelial cells.

208 xia-dependent E1A expression and conditional cytolysis of hypoxic but not normoxic cells.

209 pression, viral replication, and conditional cytolysis of hypoxic, but not normoxic cells.

210 - and alphaMB2 integrin-dependent eosinophil cytolysis of IL3-primed blood eosinophils seeded on heat

211 Cytolysis of IL3-primed eosinophils was dependent on the

212 regulation of surface CD107a, proliferation, cytolysis of infected cells, and suppression of viral re

213 tor and memory cells, a critical step in the cytolysis of infected cells.

214 Viral clearance involves immune cell cytolysis of infected cells.

215 ag and pol after vaccination, which suggests cytolysis of infected cells.

216 ertain infections and malignancies by direct cytolysis of infected or transformed cells and by secret

217 responses to anthrax infection contribute to cytolysis of LeTx- resistant macrophages.

218 factors other than LeTx are involved in the cytolysis of LeTx-resistant macrophages in vivo.

219 SV-1) is equally effective in promoting PBMC cytolysis of leukemic cells and is 1000- to 10 000-fold

220 Mechanistically, UV-HSV-1 stimulates PBMC cytolysis of leukemic cells, partly via Toll-like recept

221 blocked LT-mediated caspase-1 activation and cytolysis of LT-sensitive (Fischer and Brown-Norway) rat

222 Although LeTx-induced cytolysis of macrophages plays an important role in the

223 NKp80-AICL interaction promotes cytolysis of malignant myeloid cells, but also stimulate

224 ent in that they stimulate proliferation and cytolysis of mature T cells (classifying the variant pep

225 ainst MHC-matched tumor targets and enhanced cytolysis of MHC mismatched tumor targets.

226 nd expressed NKG2d, a marker associated with cytolysis of microbially infected and neoplastic cells.

227 K deficiency leads to a global defect in the cytolysis of multiple targets.

228 linked to anthrax lethal toxin (LT)-mediated cytolysis of murine macrophages.

229 Mechanistically, cytolysis of neuroblastoma was mediated through natural

230 onal cells of the immune system that promote cytolysis of pathogen-infected cells and nascent tumors.

231 Lkt-induced cytolysis of PMNs results in the release of cytotoxic co

232 tingly, the DeltaRD1 mutants failed to cause cytolysis of pneumocytes, a phenotype that had been prev

233 equently, SLAMF7-CAR T cells conferred rapid cytolysis of previously untreated and R/R primary myelom

234 ontrol in the absence of BCMA, we maintained cytolysis of primary tumor expressing both BCMA and TACI

235 In addition, the hemolysis and cytolysis of recombinant beta-hemolysin were markedly en

236 Inhibition of Lkt-induced cytolysis of ruminant leukocytes by CD18 peptide analogs

237 inds to the intact signal peptide and causes cytolysis of ruminant leukocytes, resulting in acute inf

238 convertase activity and complement-dependent cytolysis of sheep blood erythrocytes.

239 r intermediate filament assembly, leading to cytolysis of suprabasal keratinocytes and secondary hype

240 essary and sufficient to mediate Lkt-induced cytolysis of target cells.

241 peptide and abrogation of leukotoxin-induced cytolysis of target cells.

242 f serine kinases ERK1/2 and AKT and enhanced cytolysis of target cells.

243 In contrast, HVEM activation of BTLA reduced cytolysis of target cells.

244 ls function via cytokine secretion or direct cytolysis of target cells; dendritic cells (DCs) are tho

245 ), which subsequently induces activation and cytolysis of these cells.

246 vage of the signal peptide and abrogation of cytolysis of transfectants expressing bovine CD18 carryi

247 sduction, we show that NKG2D is required for cytolysis of tumor cells, including autologous tumor cel

248 ent of novel miR-targeted therapy to promote cytolysis of tumor cells.

249 e to the adenovirus vector or virus-mediated cytolysis of tumor cells.

250 activation targets MAPK and AKT during early cytolysis of tumor target cells.

251 Our studies demonstrate that ESAT6 causes cytolysis of type 1 and type 2 pneumocytes.

252 n with bacterial vaginosis and revealed that cytolysis of vaginal epithelial cells is associated to L

253 ion, NKG2D was required for NK cell-mediated cytolysis on adenovirus-infected targets.

254 surface proteins induce complement-dependent cytolysis or Ab-dependent cell-mediated cytotoxicity of

255 hat might mediate rejection by either direct cytolysis or by inducing apoptosis of the donor corneal

256 did not have either measurable CMV-specific cytolysis or secretion of IFN-gamma without in vitro sti

257 Though CVB is well known to disseminate via cytolysis, recent reports have revealed a second pathway

258 and IFN-gamma production, but not increased cytolysis, required recognition of influenza-infected DC

259 roliferation and cytokine secretion, but not cytolysis, specifically associated with synaptic accumul

260 F-H1 knockdown augmented complement-mediated cytolysis, suggesting a role for GEF-H1 and RhoA in prot

261 tumor effector function including tumor cell cytolysis, T(C)1 cytokine production, and zetakine-regul

262 We report that adhesion-induced eosinophil cytolysis takes place through RIPK3-MLKL-dependent necro

263 ion with LF results in a macrophage-specific cytolysis that is not well understood.

264 selves diminishes target cell sensitivity to cytolysis, thereby reducing the lytic potency of IFN-gam

265 ular endothelial cells via perforin-mediated cytolysis, thereby severely compromising vascular integr

266 even greater variability, from no detectable cytolysis to 80% or 90% cytolysis of Ect1 and BPH-1, res

267 iparum and Toxoplasma gondii cause host cell cytolysis to facilitate parasite release and disease pro

268 for cytoplasmic vacuolization and subsequent cytolysis to occur.

269 fic CD8(+) T cells require perforin-mediated cytolysis to protect animals from infection.

270 ed the contribution of perforin-mediated CTL cytolysis to protective immunity against recombinant Lm

271 provoked interferon-gamma production and/or cytolysis upon stimulation with HLA-A*02:01pos malignant

272 ridium perfringens enterotoxin (CPE) induces cytolysis very rapidly through binding to its receptors,

273 kine interferon-gamma (IFN-gamma) and induce cytolysis via releasing factors such as perforin, which

274 Cytolysis was abrogated by concanamycin A and EGTA but n

275 Furthermore, cytolysis was completely blocked when we prevented attac

276 HUVEC cytolysis was dependent on granule exocytosis, as demons

277 and propidium iodide labeled target-specific cytolysis was determined by flow cytometry.

278 e identified events and pathways involved in cytolysis was measured.

279 The enhanced cytolysis was mediated through the perforin/granzyme pat

280 c CD4 T cell clones were cytolytic, but that cytolysis was not likely critical for controlling C. mur

281 However, Lkt-induced cytolysis was observed only with transfectants expressin

282 Direct evidence for pDC cytolysis was obtained by rapid and selective pDC deplet

283 This cytolysis was repressed by the cytoprotectant glycine, p

284 Cytolysis was triggered by NKG2D recognition of stress-i

285 feration as readout but not when target cell cytolysis was used.

286 MTX-dependent release of mIL-1beta (but not cytolysis) was inhibited by the elimination of the trans

287 signalling pathways potentially involved in cytolysis were assessed using inhibitors.

288 MTX-induced cytokine release and cytolysis were both abrogated in the absence of extracel

289 sphorylation, cytoplasmic vacuolization, and cytolysis were observed in eosinophils under in vivo inf

290 and 1.4 nU when the ultrasonic and chemical cytolysis were used, respectively.

291 macrophages became sensitive to LeTx-induced cytolysis when these cells were activated by bacterial c

292 hesion to the integrin ligand ICAM-1 and for cytolysis, whereas phosphorylation of Tyr378 was require

293 s susceptible than human PMNs to PVL-induced cytolysis, whereas rabbit PMNs, like those of humans, ar

294 everal different mechanisms, one of which is cytolysis, which is associated with release of intact gr

295 Reduced ROCK signalling preceded cytolysis, which was associated with eosinophil adhesion

296 tibility of U87 glioma cells to CTL-mediated cytolysis while ICAM-1 mRNA levels remained stable.

297 re resistant to GzmA-mediated DNA damage and cytolysis, while cells overexpressing NM23-H1 are more s

298 iving within neutrophils promotes neutrophil cytolysis, with release of host-derived molecules that p

299 carcinoma cells underwent rapid and complete cytolysis within 1 hour.

300 A sharp increase in cytolysis within 24 h was observed at MOI > or = 25.