ライフサイエンスコーパス: cytomegalovirus infection

1 thelial cells, an important cell type during cytomegalovirus infection.

2 ure death of endothelial cells during murine cytomegalovirus infection.

3 mphasis on the study of postnatally acquired cytomegalovirus infection.

4 ovirus in breast milk will acquire a primary cytomegalovirus infection.

5 r agents such as valganciclovir for treating cytomegalovirus infection.

6 val, graft function, and incidence of AR and cytomegalovirus infection.

7 sign of new drugs for the treatment of human cytomegalovirus infection.

8 stomy, long-term mechanical ventilation, and cytomegalovirus infection.

9 a hallmark quality of the T cell response to cytomegalovirus infection.

10 on beyond the immediate-early phase of human cytomegalovirus infection.

11 venous ganciclovir treatment for symptomatic cytomegalovirus infection.

12 coenzyme A reductase inhibitor use and prior cytomegalovirus infection.

13 em also developed superimposed rejection and cytomegalovirus infection.

14 h coincided with host survival during murine cytomegalovirus infection.

15 or prevent other systemic manifestations of cytomegalovirus infection.

16 stinal dysmotility as a consequence of acute cytomegalovirus infection.

17 daches, posttransplant diabetes, and chronic cytomegalovirus infection.

18 significantly higher incidence of antecedent cytomegalovirus infection.

19 ischemic events and incidence of antecedent cytomegalovirus infection.

20 t driven by systemic inflammation or chronic cytomegalovirus infection.

21 n and liver at homeostasis and during murine cytomegalovirus infection.

22 h pathway known as ferroptosis, during human cytomegalovirus infection.

23 haematogenous liver and lung metastasis, and cytomegalovirus infection.

24 coordinate the NK cell response during mouse cytomegalovirus infection.

25 nd calcium signaling in the context of mouse cytomegalovirus infection.

26 K) cells are key mediators in the control of cytomegalovirus infection.

27 d monocytes show enhanced resilience against cytomegalovirus infection.

28 osttransplant hemodialysis, and a history of cytomegalovirus infection.

29 losis disease among individuals with a prior cytomegalovirus infection.

30 ore, we hypothesized it may also function in cytomegalovirus infection.

31 phoid differentiation associated with latent cytomegalovirus infection.

32 K cell (NK(eff)) populations following mouse cytomegalovirus infection.

33 y long-term, memory-like responses to murine cytomegalovirus infection.

34 not essential for the IFN response to human cytomegalovirus infection.

35 , respectively, is in development to prevent cytomegalovirus infection.

36 s early as fetal life, such as in congenital cytomegalovirus infection.

37 immunologic memory to secondary exposures to cytomegalovirus infection.

38 imilar incidence of neutropenia and reported cytomegalovirus infection.

39 K cell function in vivo in response to mouse cytomegalovirus infection.

40 pregnancies complicated by congenital human cytomegalovirus infection.

41 or up to six 4-week cycles at doses used for cytomegalovirus infection.

42 defective in protecting the host from mouse cytomegalovirus infection.

43 served after Listeria monocytogenes or mouse cytomegalovirus infection.

44 expansion of Ly49H(+) NK cells during mouse cytomegalovirus infection.

45 of viral-induced proliferation during murine cytomegalovirus infection.

46 9 to 2.88; p = .001) as high as that without cytomegalovirus infection.

47 re 15 infections in 12 patients, including 5 cytomegalovirus infections.

48 , 0.92 [95% confidence interval, .76-1.11]), cytomegalovirus infection (0.94 [.71-1.24]), herpes simp

49 ir were also less likely to have extraocular cytomegalovirus infections (0, vs. 10.3 percent in the t

50 aft loss (0 vs. 6), patient death (0 vs. 3), cytomegalovirus infection (15% vs. 12%), and 1-year seru

51 us leukaemia relapse (20%vs 7%, p=0.009) and cytomegalovirus infection (28%vs 17%, p=0.023) than was

52 nary tract infection (3/7), hematuria (3/7), cytomegalovirus infection (3/7), and immunosuppression w

53 ome were systemic infection (71% trials) and cytomegalovirus infection (62% trials), respectively.

54 ce of infection (pneumonia, 19.0% vs. 26.1%; cytomegalovirus infection, 9.5% vs. 26.1%; and sepsis, 2

55 Most evidence suggests that early cytomegalovirus infection accelerates HIV disease progre

56 confidence interval [CI], 1.08-34.86), prior cytomegalovirus infection (adjusted OR, 5.65; 95% CI, 0.

57 In one patient with recurrent cytomegalovirus infections, administration of IL-2 parti

58 as discussed at the 2018 workshop entitled "Cytomegalovirus Infection: Advancing Strategies for Prev

59 Control of cytomegalovirus infection after allo-BMT was also impair

60 aribavir or valganciclovir for first-episode cytomegalovirus infection after hematopoietic cell trans

61 articipants developed clinically significant cytomegalovirus infection after letermovir was discontin

62 rotects mice from retinitis caused by murine cytomegalovirus infection after supraciliary inoculation

63 s for VL in renal transplant recipients were cytomegalovirus infection after transplantation (odds ra

64 Cytomegalovirus infection after transplantation and livi

65 Seven (11.7%) patients experienced cytomegalovirus infection, all of whom responded to trea

66 ymphocytic choriomeningitis virus and murine cytomegalovirus infections also induced this trafficking

67 study assesses the prevalence of congenital cytomegalovirus infection among newborns screened in Min

68 ffer significantly was a higher incidence of cytomegalovirus infection among patients with chronic he

69 days after HSCT) with clinically significant cytomegalovirus infection, analysed using the full analy

70 d in 47% of patients, with 4 presenting with cytomegalovirus infection and 4 (age, 42-59 years) diagn

71 gs offer a new physiopathologic link between cytomegalovirus infection and allograft dysfunction in r

72 Cytomegalovirus infection and bacterial infection occurr

73 sitivity, posttransplantation HCV treatment, cytomegalovirus infection and center, female sex was an

74 igorously initiates and amplifies the active cytomegalovirus infection and cooperates with activated

75 Underlying liver disease, cytomegalovirus infection and disease, portal vein throm

76 ial role of adaptive immunity in controlling cytomegalovirus infection and disease, we systematically

77 s simplex virus, varicella-zoster virus, and cytomegalovirus infection and disease.

78 DC), and natural killer (NK) cells to murine cytomegalovirus infection and found distinct functions a

79 nologic complications of transfusion such as cytomegalovirus infection and graft-versus-host disease.

80 nses and plasma cell expansion during murine cytomegalovirus infection and modestly restrains immune

81 arent increased risk of infection, including cytomegalovirus infection and reactivation.

82 CpG1) allele are highly susceptible to mouse cytomegalovirus infection and show impaired infection-in

83 he expansion of non-Vy9Vd2 yd T cells during cytomegalovirus infection and their contribution to vira

84 is at the highest risk for developing active cytomegalovirus infection and to determine its effects o

85 d in two nongenetic models of iron overload (cytomegalovirus infection and treatment with ferric ammo

86 to investigate the association between prior cytomegalovirus infection and tuberculosis disease.

87 elial cells only in patients who experienced cytomegalovirus infection and were more frequent within

88 Cytomegalovirus infection and, specifically, late presen

89 d in natural killer (NK) cells responding to cytomegalovirus infection, and consider the requirements

90 (OIs) and cancers, including Kaposi sarcoma, cytomegalovirus infection, and herpes simplex virus infe

91 s, toxoplasmosis, other infections, rubella, cytomegalovirus infection, and herpes simplex virus infe

92 hepatosplenomegaly, uncontrolled EBV and/or cytomegalovirus infection, and increased incidence of B-

93 more than one factor (liver transplantation, cytomegalovirus infection, and rifampin use) when compar

94 One route by which cytomegalovirus infections are acquired in newborns is v

95 Although primary maternal cytomegalovirus infections are associated with higher ri

96 of regulation was found in vivo with murine cytomegalovirus infection as a physiologic model of NK c

97 psis trials should consider including active cytomegalovirus infection as a prospective covariate.

98 ase was also higher among children acquiring cytomegalovirus infection before age 3 months (adjusted

99 uly ineffective; they showed that imbalanced cytomegalovirus infection between arms would cause false

100 ith an imbalance on the proportion of active cytomegalovirus infection between study arms could lead

101 The G1 arrest introduced by cytomegalovirus infection blocks S-phase entry after ser

102 Four patients (13%) developed cytomegalovirus infection, but none had tissue-invasive

103 en after age 1 year was higher in those with cytomegalovirus infection by age 6 weeks (adjusted HR 4.

104 products also diminishes the transmission of cytomegalovirus infections by that route.

105 These results suggest that a cytomegalovirus infection can be severe only when the vi

106 Congenital toxoplasmosis (CT) and cytomegalovirus infection (cCMV) may cause significant m

107 The exact contribution of congenital cytomegalovirus infection (cCMVI) to permanent hearing l

108 enous thrombosis) and overimmunosuppression (cytomegalovirus infection) complications.

109 Risk factors, such as acute rejection and cytomegalovirus infection, contribute to the development

110 The results raise the possibility that cytomegalovirus infection contributes to structural brai

111 l, letermovir reduced clinically significant cytomegalovirus infections (CS-CMVi) and all-cause morta

112 y was performed using the Spanish Congenital Cytomegalovirus Infection Database.

113 In mice, systemic murine cytomegalovirus infection decreased serum erythropoietin

114 o previous history of clinically significant cytomegalovirus infection, defined as initiation of pre-

115 with cell-mediated immunity and freedom from cytomegalovirus infection demonstrates the importance of

116 dy reveals how protection and disease during cytomegalovirus infection depend on viral strain and dos

117 These results suggest that the outcome of cytomegalovirus infection depends on the presence of oth

118 organ-transplant recipients with persistent cytomegalovirus infection developed morphologic abnormal

119 rapidity and extent to which drug resistant cytomegalovirus infection develops has been elucidated.

120 By doing so, we find that latent cytomegalovirus infection drives DNA methylation variati

121 agents, older donor age, posttransplantation cytomegalovirus infection, elevated very low density lip

122 movir prophylaxis for clinically significant cytomegalovirus infection from 100 days to 200 days foll

123 Pooled findings showed that individuals with cytomegalovirus infection had a higher risk of tuberculo

124 Previous studies of the immune control of cytomegalovirus infection have primarily focused on anal

125 LA matching, immunosuppression regiments and cytomegalovirus infection, heterotopic heart transplanta

126 Cytomegalovirus infection, HIV/hepatitis B virus coinfec

127 liver disease (HR, 2.18; 95% CI, 1.29-3.67), cytomegalovirus infection (HR, 1.89; 95% CI, 1.08-3.3),

128 patients early rejection occurred in 13 and cytomegalovirus infection in 5.

129 re features were not observed during chronic cytomegalovirus infection in an independent cohort.

130 Cytomegalovirus infection in both species can expand a p

131 We use human cytomegalovirus infection in glucose-free conditions as

132 cidofovir to placebo for prophylaxis against cytomegalovirus infection in hematopoietic cell transpla

133 Human cytomegalovirus infection in individuals lacking a fully

134 We aimed to evaluate the acquisition of cytomegalovirus infection in infancy and the development

135 ted and increase in frequency in response to cytomegalovirus infection in mice.

136 e, we investigate the consequences of murine cytomegalovirus infection in newborn mice on NK cells.

137 ver, recent studies have demonstrated active cytomegalovirus infection in nonimmunosuppressed intensi

138 the incidence of late clinically significant cytomegalovirus infection in patients at risk.

139 each viral and host factor involved in human cytomegalovirus infection in primary human fibroblasts t

140 pathway offers full protection against mouse cytomegalovirus infection in the absence of the other.

141 Human cytomegalovirus infection in the presence of the cyclin-

142 ere was no excess incidence of malignancy or cytomegalovirus infection in this prolonged follow-up pe

143 e documented in 11 patients (15%), including cytomegalovirus infection in three patients (4%).

144 Human cytomegalovirus infection in transplant recipients has b

145 n this study, we investigated the effects of cytomegalovirus infection in Trp53 heterozygous mice.

146 diators of NK cell-mediated control of mouse cytomegalovirus infection in vivo.

147 ication, were dependent on AIM2 during mouse cytomegalovirus infection in vivo.

148 flammatory drugs might help to control human cytomegalovirus infections in conjunction with other ant

149 ldom of consequence in healthy term infants, cytomegalovirus infections in low-birth-weight premature

150 ograft vasculopathy progression and reducing cytomegalovirus infections in maintenance heart transpla

151 ler (NK) cells are crucial in the control of cytomegalovirus infections in mice and humans.

152 arize recent studies of breast-milk-acquired cytomegalovirus infections in newborns, particularly in

153 in 46 [30%] and 42 [28%], respectively), and cytomegalovirus infection (in 39 [26%] and 31 [21%]).

154 It has been suggested that cytomegalovirus infection increases the risk of ischemic

155 nt hepatitis), and cryptococcal (symptomatic cytomegalovirus infection) infections.

156 Cytomegalovirus infection is a frequent and life-threate

157 velopment of a vaccine to prevent congenital cytomegalovirus infection is a major public health prior

158 Because cytomegalovirus infection is a risk factor for invasive

159 One common sign of human cytomegalovirus infection is altered liver function.

160 of intrinsically more aggressive disease; 3) cytomegalovirus infection is associated with earlier ons

161 opulations of CD34(+) cells.IMPORTANCE Human cytomegalovirus infection is associated with severe dise

162 Cytomegalovirus infection is present in the majority of

163 d high disease severity, in whom the rate of cytomegalovirus infection is up to 36%.

164 Cytomegalovirus infection is very common and leads to th

165 During murine cytomegalovirus infections known to target spleen and li

166 Cytomegalovirus infections located in posterior eye segm

167 ; lessons learnt in the areas of imaging and cytomegalovirus infection; long-term psychological outco

168 Prenatal cytomegalovirus infection may cause pregnancy complicati

169 We hypothesize that active cytomegalovirus infection may confound survival outcomes

170 Our data support the concept that cytomegalovirus infection may drive NK-cell development

171 -line use of brincidofovir for prevention of cytomegalovirus infection may preserve downstream option

172 mbalanced proportion of patients with active cytomegalovirus infection may severely compromise the re

173 Lastly, in MZ twins discordant for cytomegalovirus infection, more than half of all paramet

174 in cells with viral inclusions diagnostic of cytomegalovirus infection, not tumor as the thallium SPE

175 after cessation of IS and no tissue-invasive cytomegalovirus infections occurred.

176 Active cytomegalovirus infection occurs frequently in nonimmuno

177 Human cytomegalovirus infection of colon-cancer cells (Caco-2)

178 Human cytomegalovirus infection of human fibroblasts induces t

179 Rhesus cytomegalovirus infection of rhesus macaques has emerged

180 Human cytomegalovirus infection of the developing central nerv

181 liver pathology was evaluated during murine cytomegalovirus infections of mice.

182 The effect of human cytomegalovirus infection on cellular DNA synthesis in h

183 The effects of cytomegalovirus infection on HIV-exposed infants are poo

184 munosuppression, and infection, specifically cytomegalovirus infection, on the development of chronic

185 he transcriptional level during acute murine cytomegalovirus infection or after repetitive polyinosin

186 ent of patients with resistant or refractory cytomegalovirus infection or cytomegalovirus disease is

187 infection (OR = 7.75; 95% CI = 1.60-37.57), cytomegalovirus infection or disease (OR = 2.67; 95% CI

188 roportion of lung transplants that developed cytomegalovirus infection or disease during the 180-day

189 cant differences were found in occurrence of cytomegalovirus infection or disease, Pneumocystis carin

190 sociated with basiliximab and no evidence of cytomegalovirus infection or posttransplant lymphoprolif

191 ctivating stimuli, including TNFalpha, human cytomegalovirus infection, or double-stranded DNA, eEF2

192 The incidence of cytomegalovirus infection (P<.05) and posttransplant dia

193 rosing cholangitis (P=0.009) and symptomatic cytomegalovirus infection (P=0.008).

194 ificant increase in the frequency of primary cytomegalovirus infection (P=0.045), and a decrease in c

195 NAemia was not significantly associated with cytomegalovirus infection (P=0.937).

196 (P=0.0053), cold storage time (P=0.019), and cytomegalovirus infections (P=0.002).

197 leading to opportunistic infections such as cytomegalovirus infection, periocular nerve involvement

198 Human cytomegalovirus infection perturbs multiple cellular pro

199 Gaps remain in understanding the role that cytomegalovirus infection plays in HIV-exposed infants.

200 In other specimens, preservation injury, cytomegalovirus infection, post-transplant lymphoprolife

201 nt (PAK or PTA [vs. SPK], RR=3.02, P=0.002), cytomegalovirus infection posttransplant (RR=2.41, P=0.0

202 e, Ly49H(+) NK cells that responded to mouse cytomegalovirus infection primarily developed from ENKPs

203 variety of clinical manifestations of human cytomegalovirus infection probably results from both vir

204 uinone oxido-reductase), we found that human cytomegalovirus infection protected cells from rotenone-

205 Both cytomegalovirus infection rates (BD 21% vs. ED 8%, P=0.0

206 than groups A (37%) or B (23%) (P < 0.001); cytomegalovirus infection rates were 35%, 20% and 23%, r

207 One patient who presented with cytomegalovirus infection received a second infusion of

208 Although the incidence of cytomegalovirus infection remained unchanged, a signific

209 ta in the literature suggest that congenital cytomegalovirus infection remains a research priority, b

210 hy fetuses and in 47 fetuses with congenital cytomegalovirus infection respectively).

211 lymphocytic choriomeningitis virus or murine cytomegalovirus infections resulted in profound splenic

212 Viral disease, particularly cytomegalovirus infection, results in significant morbid

213 times related to acute Epstein-Barr virus or cytomegalovirus infection, sometimes months before typic

214 fect of sex, age, body mass index (BMI), and cytomegalovirus infection status.

215 on interferon gamma production during murine cytomegalovirus infection, stimulating it in conventiona

216 the localization of IE1 and IE2 during human cytomegalovirus infection, suggesting a principle common

217 ctive advantages during in vivo responses to cytomegalovirus infection, suggesting that receptor dens

218 n the setting of a short-term (4-day) murine cytomegalovirus infection, terminally differentiated NKs

219 t day 90) were more likely to have had prior cytomegalovirus infection than those with early-onset as

220 cific humoral and T-cell responses following cytomegalovirus infection that control virus replication

221 viously been demonstrated that, during human cytomegalovirus infection, the viral IE2 86 and IE2 40 p

222 rmal clearance of Listeria monocytogenes and cytomegalovirus infections, the mice displayed a profoun

223 the active recruitment of neutrophils during cytomegalovirus infection, thereby providing for efficie

224 , deceased donor, early rejection, and early cytomegalovirus infection to estimate hazard ratios for

225 We use a mouse model of cytomegalovirus infection to show that, like T cells, NK

226 acebo group developed clinically significant cytomegalovirus infection (treatment difference -16.1% [

227 Mouse cytomegalovirus infection triggers NK receptor-induced,

228 In the AMR cohort, two cases of cytomegalovirus infection, two cases of BK virus infecti

229 c immunomonitoring and routine screening for cytomegalovirus infection until discharge from the inten

230 er age 6 months in children with and without cytomegalovirus infection using Cox regression and hazar

231 verall mortality rate associated with active cytomegalovirus infection was 1.93 times (95% CI, 1.29 t

232 The overall rate of active cytomegalovirus infection was 17% (95% confidence interv

233 Incidence of cytomegalovirus infection was 4.2% at 12 months, and 99.

234 Congenital cytomegalovirus infection was associated with birthweigh

235 In allogeneic patients, active human cytomegalovirus infection was associated with HHV-6 isol

236 Cell cycle progression during cytomegalovirus infection was investigated by fluorescen

237 ion by activated NK cells in an acute murine cytomegalovirus infection was significantly reduced desp

238 In an application to cytomegalovirus infection, we obtain insights into the r

239 In response to murine cytomegalovirus infection, we show that circulating NK c

240 The rates of cytomegalovirus infection were significantly lower in th

241 ces of histologic hepatitis C recurrence and cytomegalovirus infection were similar in each group.

242 at hepatitis B, and cidofovir, used to treat cytomegalovirus infections) were alleviated by the early

243 imics the outcome seen in humans with latent cytomegalovirus infection, where reactivation of virus o

244 ted NK cell activation across tissues during cytomegalovirus infection, which generates antigen-speci

245 the mother increases the rate of congenital cytomegalovirus infection, while maternal antiretroviral

246 owed a marked and significant correlation of cytomegalovirus infection with active tuberculosis (adju

247 at high risk of late clinically significant cytomegalovirus infection (with no previous history of c

248 Therapies for refractory cytomegalovirus infections (with or without resistance [