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1 ane CD35 expression in enucleated neutrophil cytoplasts.
2 ntal competence of reconstituted oocytes and cytoplasts.
3 lls that circulate in the blood as anucleate cytoplasts.
4  transfer by electrofusion with simultaneous cytoplast activation; and sequential media.
5 ivation by CPT is dramatically diminished in cytoplasts and in CEM/C2 cells expressing a mutant Topo
6 roteolytic cleavage during apoptosis of both cytoplasts and intact cells.
7 stasis and inflammation, but these anucleate cytoplasts are not thought to synthesize proteins or cyt
8 karyoplasts, whereas the Golgi stacks in the cytoplasts are scattered.
9 sistent with division plane specification at cytoplast boundaries.
10 AMP inhibited FMLP-stimulated aggregation in cytoplasts but not neutrophils.
11 ransiently activated MAPK in neutrophils and cytoplasts, consistent with a role in signaling for neut
12         Enucleation resulted in a mixture of cytoplasts containing or lacking the centrosome.
13                                In fibroblast cytoplasts containing the centrosome, MTs showed dynamic
14  Similar changes were observed in neutrophil cytoplasts depleted of organelles and nucleus.
15 g microtubule dynamics in acentrosomal basal cytoplasts derived from these cells.
16 introduced into an enucleated recipient egg (cytoplast), derived from another female, by karyoplast-c
17 e spindle donors whereas mtDNA came from the cytoplast donors.
18  for DNA, which may disrupt functions of the cytoplast, especially mitochondria.
19 lination (H3Cit), nuclear DNA extrusion, and cytoplast formation.
20              Here we show that by generating cytoplasts from colonocytes, standard fusion techniques
21 mulated significantly more daunorubicin than cytoplasts from U-937 cells.
22 bicin concentrations of 100 ng/mL or higher, cytoplasts from U-A10 cells accumulated significantly mo
23 f the centrosome by examining MT behavior in cytoplasts from which the centrosome was removed.
24 h their capacity to stimulate neutrophil and cytoplast homotypic aggregation.
25 ation of p36 MBP kinase occurs in enucleated cytoplasts, indicating no requirement for a nucleus or f
26 n of the CD95 (Fas/APO-1) molecule on Jurkat cytoplasts induces dramatic PS externalization similar t
27 ortion inadequately reflect the state of the cytoplast, it is also time consuming.
28                              In contrast, in cytoplasts lacking the centrosome, MTs treadmilled-short
29 l preparations, which showed the presence of cytoplast-like structures and microcells containing mito
30 , derived from another female, by karyoplast-cytoplast membrane fusion.
31 ved structures were characterized: migratory cytoplasts, migrasomes, and elongated neutrophil-derived
32 -studied neutrophil EVs: apoptotic vesicles, cytoplasts, migrasomes, and ENDS.
33                                The nontarget cytoplast organellar genomes and metabolites are removed
34 rast to fibroblast cells, in centrosome-free cytoplasts prepared from epithelial cells, MTs displayed
35 ouse (Mus musculus domesticus) were fused to cytoplasts prepared from Mus musculus, Mus spretus, or r
36                                              Cytoplasts prepared from U-A10 and U-937 cells were expo
37      Moreover, the generated nucleus-lacking cytoplast provides an ideal environment to test the feas
38 vated oocytes have also proved successful as cytoplast recipients.
39                      Platelets are anucleate cytoplasts that contain a rich repertoire of RNAs encodi
40 ted the aggregation of egranulate neutrophil cytoplasts that lack the capacity for CD11b/CD18 up-regu
41 mployed neutrophils and enucleate neutrophil cytoplasts to study the activation of the mitogen-activa
42            In addition, motile, granule-poor cytoplasts (U-Cyt) from human blood PMN can exert anti-B
43 ree tubulin subunits (78% in centrosome-free cytoplasts vs. 44% in intact cells) generated by minus-e
44                  Apoptosis of both cells and cytoplasts was associated with proteolytic processing of
45        These cybrids were enucleated and the cytoplasts were electrofused to rhodamine-6G (R-6G)-trea
46 mphoblast cell lines were enucleated and the cytoplasts were fused to a mtDNA-deficient (p 0) lymphob
47 P- and insulin-stimulated MAPK activation in cytoplasts were inhibited by Bt2cAMP, consistent with si
48                                            U-cytoplasts, which have activatable oxidase, killed opson
49 slocation from cytosol to plasma membrane in cytoplasts, with kinetics consistent with events upstrea
50 ed into human mtDNA-less (rhoo) 206 cells by cytoplast x rhoo cell fusion, and 7-31 transformant clon