ライフサイエンスコーパス: dead

1 utpatients, 42 hospitalized and 21 intubated/dead).

2 sionally thinks that she would be better off dead.

3 free survival (PFS), progressive disease, or dead.

4 t that MGCs are specialized for clearance of dead adipocytes in obesity.

5 lammation, defined by macrophages encircling dead adipocytes, as well as extracellular matrix (ECM) r

6 binding of signaling effectors, and a kinase dead allele of Fgfr2 that broadly phenocopies the null m

7 e complex interactions of the living and the dead among these successful Late Pleistocene foragers.

8 near the crack to die, as indicated by live-dead and caspase staining.

9 time saving approach for detection of live, dead and drug-resistant bacteria.

10 ecent discovery that the metabolite cargo of dead and dying cells ingested through efferocytosis by m

11 e present an overview of the phagocytosis of dead and dying cells, a process known as efferocytosis.

12 The discovery in dead and dying farmed salmon of previously unrecognised

13 ms and facilitate the discrimination between dead and living microbes.

14 ranscriptomic sequencing and surveillance of dead and moribund cultured Chinook salmon revealed a nov

15 of melatonin was carried out using both live/dead and MTT assays.

16 carbon that remains resident on the site as dead and partially 'pyrolysed' (i.e. burnt) material tha

17 remotely operated vehicles commonly observe dead and preserved remains in brine pools.

18 s associated with inflammation in both brain-dead and sham subjects relative to naive controls.

19 ividual historical isolates (both extant and dead) and collections of historical isolates.

20 GRK2-K220R (kinase dead) and GRK2-S670A (unphosphorylated, constitutively a

21 sively weaker responses across the live ant, dead ant and alarm odours treatments respectively.

22 llowing four odour treatments: alarm odours, dead ants, live ants and nest material.

23 t elevated viable LGCs numbers and decreased dead/apoptotic cell counts.

24 onse is recorded until the cells are totally dead as predicted with viability tests.

25 s targeted to differentiate between live and dead as well as antibiotic resistant bacteria.

26 day 88, viable ECD was measured using a live/dead assay, and endothelial expression of Na/K ATPase, C

27 ased scaffolds was evaluated by MTT and live/dead assay.

28 = 0.010) in patients with COVID-19 who were dead at Day 28.Conclusions: Profound global lymphopenia

29 19.0, 95% CI 4.8-74.8) and when the dog was dead at the time of the assessment (OR = 20.7, 95% CI 6.

30 conformation), but not FAK/Arg(454) (kinase-dead), augmented Rab5-GTP levels in FAK-null fibroblasts

31 s (bTB) in Wales, an All Wales Badgers Found Dead (AWBFD) survey was carried out from 2014-2016.

32 hours, the membranes were dyed with the Live/Dead BacLight Bacterial Viability fluorescence stain and

33 icroscopy and the bacteria staining kit Live/Dead BacLight(TM) to quantify viable LAB populations in

34 The percentage of dead bacteria also decreased as a function of hydrophili

35 We also detected the ratio of live/dead bacteria and bacterial composition in the biofilms

36 sor was also able to differentiate live from dead bacteria eliminating the false positive results.

37 dity, vegetation density, and the density of dead bamboo culms.

38 l reason lungs from otherwise eligible brain dead (BD) organ donors are not transplanted.

39 ralysis virus (CBPV), extracted from sick or dead bees, was studied by mobility measurements via elec

40 esent a novel method for estimating standing dead biomass at a fine spatial resolution that combines

41 ximately 95.1 million bone-dry tons (BDT) of dead biomass resulted from 2012-2017 mortality, with a l

42 Of the standing dead biomass, 29% meets minimum constraints for potentia

43 s: contact with the infectious, contact with dead bodies, and transmission by sexual behaviour with c

44 New candidate genes include Dead box helicase 1, a Tim Barrel domain protein and the

45 Plasmid DNA was not detectably bound by DEAD box helicase 60 (DDX60) protein, despite a similar

46 ubunits and nucleolar proteins including the DEAD box helicases, DHX9, DDX5 and DDX17.

47 ilies such as the ribosomal proteins and the DEAD box helicases, we identified novel high-confidence

48 the gene encoding the sole Asp-Glu-Ala-Asp (DEAD)-box RNA helicase in Synechocystis sp. PCC 6803, cr

49 ing early spliceosome formation requires the DEAD-box ATPase PRP5(2-7).

50 and highly abundant family of RNA-dependent DEAD-box ATPases (DDXs)(3) are regulators of RNA-contain

51 e-mRNA splicing in eukaryotes requires three DEAD-box ATPases (Prp5, Prp28 and Sub2), the molecular m

52 The DEAD-box family of proteins are ATP-dependent, RNA-bindi

53 TH/DDX25) is a testis specific member of the DEAD-box family of RNA helicases expressed in meiotic an

54 sp) perturbed self-association and inhibited DEAD-box helicase 3 (X-linked) (DDX3) ATPase activity.

55 DEAD-Box Helicase 3 X-Linked (DDX3X) is frequently mutat

56 RNA-binding protein (RBP)/helicase DDX3 (DEAD-box helicase 3 X-linked) is a key component of stre

57 DEAD-box helicase 5 (DDX5) is a founding member of the D

58 ur of these sfRNA-interacting host proteins, DEAD-box helicase 6 (DDX6) and enhancer of mRNA decappin

59 nal intrinsically disordered region (IDR) of DEAD-box helicase Ddx4, as a test case, to assess roles

60 Recently, we found that the DEAD-box helicase DDX43 contains a KH domain in its N-te

61 Yeast DEAD-box helicase Ded1 stimulates translation initiation

62 ting with G4s and demonstrate that the DDX42 DEAD-box helicase is a newly discovered G4-binding prote

63 DEAD-box helicase proteins perform ATP-dependent rearran

64 SrmB is a DEAD-box helicase that acts early in the ribosome assemb

65 ix between the two RecA domains of the Prp28 DEAD-box helicase.

66 DEAD-box helicases (DDXs) regulate RNA processing and me

67 We demonstrate that the DEAD-box helicases eIF4A2 and DDX6 interact directly wit

68 gomerization domain (NOD)-like receptors and DEAD-box or DEAH-box RNA helicases in an orchestrated RN

69 denosinetriphosphatase/RNA helicase X-linked DEAD-box polypeptide 3 (DDX3X) emerged as a novel therap

70 ly, we identified the ATPase activity of the DEAD-box protein Dhh1 as a key regulator of PB dynamics

71 that, through ATP-dependent RNA binding, the DEAD-box protein eIF4A reduces RNA condensation in vitro

72 tron ribozyme by CYT-19, a Neurospora crassa DEAD-box protein that functions as a general chaperone f

73 Dbp5 is an essential DEAD-box protein that mediates nuclear mRNP export.

74 DEAD-box proteins (DBPs) comprise a large family of prot

75 ortant role for eIF4A, and potentially other DEAD-box proteins, as ATP-dependent RNA chaperones that

76 DEAD-box RNA helicase DDX21 (also named nucleolar RNA he

77 The human DEAD-box RNA helicase DDX3X is an essential cofactor for

78 Yeast DEAD-box RNA helicase Ded1 appears to promote translatio

79 elicase 5 (DDX5) is a founding member of the DEAD-box RNA helicase family, a group of enzymes that re

80 hat DDX5, one of the founding members of the DEAD-box RNA helicase family, is extremely proficient at

81 Ded1 is a DEAD-box RNA helicase with essential roles in translatio

82 itiation factor 4A (eIF4A), an ATP-dependent DEAD-box RNA helicase, is a critical component of the eI

83 lasmic roles as a modulator of ATP-dependent DEAD-box RNA helicases involved in messenger (m)RNA expo

84 Gle1 is a conserved, essential regulator of DEAD-box RNA helicases, with critical roles defined in m

85 F), that result in kinase-impaired or kinase-dead BRAF have the highest mutation frequency in BRAF ge

86 Several studies have reported that kinase-dead BRAF variants amplify mitogen-activated protein kin

87 t only they are told that their loved one is dead but are also asked to consider organ donation.

88 The expression of wild-type and catalytic-dead but not oligomerization-attenuated NME3 restored mi

89 equires knowledge not only of when trees are dead, but when they begin dying - having passed the poin

90 CRISPR-Cas12a, the invention of the nuclease-dead Cas molecules (dCas9 and dCas12a) offers a platform

91 e map of the energetic landscape of nuclease-dead Cas12a (dCas12a) from Francisella novicida as it in

92 t a CRISPR interference system using a DNase-dead Cas12a to significantly repress endogenous gene exp

93 PRi/a depends on continued expression of the dead Cas9 (dCas9) effector and guide RNA (gRNA), which c

94 eates DNA double strand breaks, targeting of dead Cas9 (dCas9) is presumed not to be mutagenic, as dC

95 ciated virus (AAV) vectors encoding nuclease-dead Cas9 and a single-guide RNA targeting CUG repeats r

96 ssing a single guide RNA and a catalytically dead Cas9 fused to the KRAB repressor domain to suppress

97 n be impeded by targeted binding of nuclease-dead Cas9, by transcription through repetitive Igh switc

98 s the replisome to bypass an inactive (i.e., dead) Cas9 (dCas9) R-loop barrier.

99 f mammalian genomes, we made use of a set of dead-Cas9 base editor (dBE) variants that allow editing

100 general principle whereby signals within the dead cell as well as the environment are integrated by s

101 nes and growth factors, and they phagocytose dead cell debris, a process that is critical for resolut

102 ingle cell PD resulted in parallel events of dead cell exfoliation and migration of intact neighbouri

103 ctate dehydrogenase cytotoxicity assay, live-dead cell staining and transmission electron microscopy

104 r, Clec9A interactions are altered following dead cell uptake to favor antigen presentation.

105 ator of Clec9A and the cross-presentation of dead cell-derived antigens by mouse dendritic cells.

106 ll receptor Clec9A facilitates processing of dead cell-derived antigens for cross-presentation and th

107 also known as CLEC9A), a receptor that binds dead-cell debris and facilitates XP of corpse-associated

108 nge, T6SS activity becomes self-limiting, as dead cells accumulate in its way, forming "corpse barrie

109 ows clear differentiation between living and dead cells after 3 h of inducing apoptosis.

110 nesis and daily in adult organisms, clearing dead cells and associated cellular debris is important i

111 l for cell growth dependent on the uptake of dead cells and cell debris.

112 osis occurs within epithelia, the harmful or dead cells are apically extruded from tissues to maintai

113 r-ligand interactions to clear pathogens and dead cells from a host.

114 s TAZ, Notch, and hedgehog; how clearance of dead cells in NASH via efferocytosis may affect inflamma

115 ts exhibit growth defects and an increase in dead cells in root meristems after CPT treatment demonst

116 her microbes adhering to particles were live/dead cells or inert particles.

117 oral administration, since a minor number of dead cells were found in red color under a fluorescent m

118 to heat or radiation: heated spheroids shed dead cells within four days of heating and displayed fas

119 dead cells) across a wide linear range (10-1000 cells/mu

120 create volume differences between living and dead cells, and then, the differences were characterized

121 ntenance of tissue homeostasis by scavenging dead cells, cell debris and lipoprotein aggregates via p

122 o their role as danger signals released from dead cells, IL-1 family cytokines can be secreted in the

123 This prevented phagocytic removal of dead cells, prevented maturation of monocyte-derived mac

124 olecular techniques to distinguish live from dead cells.

125 minutes of injury, where they rapidly engulf dead cells.

126 cell viability and the distribution of live/dead cells.

127 amyloid occurred in clustered capsules with dead cells.

128 ing width measurement series from living and dead conifers that grew in undisturbed, high-elevation s

129 mical measurements of hundreds of living and dead conifers to reconstruct the impact of heavy industr

130 ar fly, Drosophila melanogaster, exposure to dead conspecifics in the environment induced cues that w

131 that Drosophila have an ability to perceive dead conspecifics in their environment and suggest conse

132 The effects of exposure to dead conspecifics on aversiveness and lifespan required

133 ost cases to a similar extent as in a kinase-dead control.

134 et growth, and accelerated disintegration of dead corals.

135 human macrophages that had ingested live or dead Cryptococcus neoformans cells, or inert beads.

136 s, or adult LV myocytes isolated from "redox dead" (Cys17Ser) PKARIalpha knock-in mice and their wild

137 ctivation systems based on nuclease inactive dead (d)Cas9 fused to transcriptional transactivation do

138 Functional analyses using CRISPR/dead(d)Cas9 targeting of the hHS-8-NFAT site in the huma

139 tabulated as percentage of insects that were dead/deformed, infertile, or fertile and subjected to ch

140 ters and increases in both the proportion of dead diatoms and the diatom assemblage sedimentation rat

141 he AASP and the sinking of the senescent and dead diatoms helps drive carbon sequestration.

142 The expression of kinase-dead DNA-PKcs abrogates cNHEJ(2).

143 owever, most mice that both expressed kinase-dead DNA-PKcs and lacked the tumour suppressor TP53 deve

144 d-ligation, whereas expression of the kinase-dead DNA-PKcs protein (DNA-PKcs(KD/KD) ) abrogates end-l

145 ble the phagocytic pruning of these 'locally dead' domains by macrophages, most notably by microglia

146 anence standard for death and respecting the dead donor rule.

147 nger hepatectomy times than those from brain-dead donors [50 minutes (35 to 68) vs 40 minutes (32 to

148 Nonfailing hearts from brain-dead donors show marked differences in circadian clock g

149 amples recovered from nonheart failure brain-dead donors showed acrophase shifts, or weak or complete

150 ired from the nonfailing ventricles of brain-dead donors.

151 mparable to outcomes from contemporary brain-dead donors.

152 iability compromises retrieval in most brain-dead donors.

153 T1L knockout cells, but not in catalytically dead DOT1L cells, revealing a role of DOT1L in promoting

154 In this study, we isolated the tambaqui's dead end gene (dnd) homolog (tdnd) and used it as a mole

155 on on solvent accessibility of lysine sites, dead end iqPIR cross-linked products can provide protein

156 sex is commonly regarded as an evolutionary dead end.

157 s for logic operations is not engaged into a dead end.

158 It thereby induces the assembly of a dead-end 3' processing complex, blocking mRNA 3' cleavag

159 of pre-existing monomeric IgG1 nuclei into a dead-end aggregate, rather than through macromolecular c

160 X-ray crystal of the AldC C291A mutant in a dead-end complex with octanal and NAD(+) reveals an apol

161 FASPR lies in the optimal combination of the dead-end elimination and tree decomposition with a well

162 er, how nonselected T cells proceed in their dead-end fate is not clear.

163 pread zoonosis which in nature exhibits both dead-end infections and varying levels of sustained tran

164 pandemics such as COVID-19; in other cases, dead-end infections or smaller epidemics result.

165 sphate carboxylase/oxygenase (Rubisco) forms dead-end inhibited complexes while binding multiple suga

166 ly capture ~500 single cells inside isolated dead-end nanoliter compartments using simple pipette inj

167 is achieved by removing trapped regions and dead-end pathways using a simple algorithm.

168 ipolar cycloaddition, while the formation of dead-end prenylated flavin mononucleotide cycloadducts o

169 y that creates connected pathways as well as dead-end regions.

170 not undergo reductive elimination meaning a "dead-end route".

171 Standard dead-end sample filtration is used to improve sample pur

172 se systemic infections are presumed to be a "dead-end" for P. aeruginosa and to have no impact on tra

173 A high-resolution reconstruction of a "dead-end" intermediate at 3.6 angstrom allowed us to vis

174 melanogaster to retreat when it encounters a dead-end, we identified a pair of ascending neurons, the

175 We identify Dead-end1 (Dnd1), a master regulator of vertebrate germl

176 tes away from states that are photosynthetic dead ends.

177 e accurate choices between live channels and dead ends.

178 ound obstacles and extricate themselves from dead-ends (for reviews, see [1-3]).

179 Some of these channels lead to tiny, dead-ends in the state space: the persistence of complex

180 omplementation of KO cells with WT or enzyme-dead FAM173A indicated that the enzymatic activity of FA

181 ix was associated primarily with emptying of dead fibers.

182 ctivity using a commercially available "live/dead" fluorescent dye.

183 ailure - best predicting when trees had been dead for some time, rather than when they were dying.

184 idual activity retained in a reported kinase-dead form, LKB1K78I, was sufficient to hamper tumor aggr

185 Integration rates were found higher in dead fry than in live fingerlings, indicating either off

186 Studies of the protease-dead full-length Mt2 (Mt2S762A) and the truncated Mt2 th

187 y-down/rewetting cycle, bacteria on and near dead fungal hyphae were more metabolically active than t

188 The DEAD/H-box helicase ME31B showed the highest affinity fo

189 discuss pathways through which stressed and dead hepatocytes instigate the profibrogenic crosstalk w

190 he incidence of chromosomal abnormalities of dead hybrid embryos were described.

191 candidiasis-ectodermal dystrophy-like kinase-dead Ikkalpha knockin mice and patients with autoimmune

192 inflammation, and skin phenotypes in kinase-dead Ikkalpha knockin mice.

193 Dead/intubated(Day28) subjects exhibited an opposite pat

194 Expression of kinase-dead (KD) ATM protein solely accelerates lymphomagenesis

195 the latter harboring an allele with a kinase-dead (KD) mutation.

196 ced PKARIalpha disulfide formation in "redox dead" knock-in mouse hearts resulted in larger infarcts

197 pharmacological inhibitors and RIPK1 kinase-dead knockin mice.Measurements and Main Results: HMGB1 l

198 high reactivity induces dendrite growth and dead Li formation during repeated charge/discharge proce

199 g leads to continuous Li dendrite growth and dead Li formation, which causes safety risks and short l

200 ssolution, the formation of dendritic and/or dead Li lies as a fundamental barrier to the practical i

201 hey rapidly develop porosity, dendrites, and dead Li that cause poor performance and, all too often,

202 Little-to-no dead Li was observed when FEC is used as an additive.

203 ll-cell lithium metal battery to be tracked: dead lithium and SEI formation can be quantified by NMR

204 The methodology allows inactive or "dead lithium" formation during plating and stripping of

205 kinase assays and by expressing WT or kinase-dead MAPK kinase kinase 19 (MAP3K19) in the HEK293T cell

206 scued with exogenous WT MELK, but not kinase-dead MELK complementation.

207 Rapid SYBR green I/propidium iodide live/dead microbial cellular discrimination assay for Schizos

208 ce method could be applied for the fast live/dead microbial discrimination in antimicrobial assays.

209 These data from dead minds open up an untapped and highly diverse subjec

210 ow that BRCA1-/-, CtIPS332A/-/- and nuclease-dead MRE11D20A/- mutants display increased sensitivity t

211 reover, BRCA1-/-, CtIPS332A/-/- and nuclease-dead MRE11D20A/- mutants failed to resume DNA replicatio

212 onstitutively active mTOR (mTORca) or kinase-dead mTOR (mTORkd) in the heart.

213 Dead museum specimens are finding new life, providing cr

214 -type TPL2 rescues the phenotype, its kinase-dead mutant does not.

215 decrease in Ig production, PI3Kdelta kinase-dead mutant donor bone marrow-derived GC B cells still s

216 , expression of NO66, but not of demethylase-dead mutant NO66, decreased H3K4me3 and H3K36me3 and sup

217 ssion of active PPM1F, but not a phosphatase-dead mutant.

218 Brain-dead (n = 12) and sham (n = 5) rhesus macaques were main

219 as been primarily hindered by dendritic and "dead" Na formation that leads to low Coulombic efficienc

220 erexpression of active Nek2A, but not kinase-dead Nek2A, prematurely displaced DAs from the interphas

221 it large quantities of energy in the form of dead nestlings.

222 In neurodegenerative diseases, debris of dead neurons are thought to trigger glia-mediated neuroi

223 rsed by expression of wild-type or catalytic-dead NME3.

224 ibonucleic acid (cfDNA) released from either dead or damaged cells serves as a key autoantigen in rhe

225 atients with modified Rankin Scale data were dead or dependent at 1 year.

226 segregate from incompatible partners (e.g., dead or different sized cells), and (iii) pair with othe

227 and Europe was more virulent; the number of dead or diseased wild birds found and the severity of pa

228 ted by immune cells or passively released by dead or dying cells that can bind to pathogen recognitio

229 nt trial, targeted mild hypothermia in brain-dead organ donors significantly reduced the incidence of

230 Pitting, the removal of dead parasites from their host erythrocyte, has been stu

231 llum level were significantly lower in brain-dead patients (p = 0.019 for HC-WM, p < 0.001 for other

232 Pneumonia occurs in one-third of brain-dead patients and appears as the main reason for lung no

233 Family members of brain dead patients experience an unprecedented situation in w

234 R/Cas9-based system to express catalytically dead PfClpP, which showed that PfClpP oligomerizes as a

235 ion of the disorder, phagocytic clearance of dead photoreceptor cell bodies has a protective role by

236 Muller glia phagocytose dead photoreceptor cells in a mouse model of retinal deg

237 Catalytically dead PLD2 robustly blocks anesthetic TREK-1 currents in

238 tivity and is more accurate than exonuclease-dead Polepsilon.

239 oarsening during plating/stripping, possibly dead potassium metal on stripped surfaces, and rapid fai

240 of wild-type PPIP5K1; transfection of kinase-dead PPIP5K1 was ineffective.

241 TP could feed upon dead prey floating at the surface while WTS preyed mainl

242 T mice, as well as enzymatically inactivated Dead-PRSS1R122H mice, we unraveled that increased trypsi

243 ed 3 different mechanisms that create kinase-dead pseudokinases, including D2017A, which further emph

244 ytokine release from living (hyperactive) or dead (pyroptotic) cells.

245 0.05), primarily through a reduction in days dead (rate ratio, 0.847 [95% CI, 0.728-0.986]; P=0.03).

246 rats subjected to rapid-onset BD, and brain-dead rats treated with E2 (280 ug/kg, intravenous) (BD-E

247 Using U(S)3 deletion and U(S)3 kinase-dead recombinant MDV, we identified U(S)3-responsive MDV

248 tablished that variant BRAF(D594G), a kinase-dead representative of class 3 mutation-derived BRAF var

249 NA) complex through the co-administration of dead-RNAs (dRNAs), truncated guide RNAs that direct Cas9

250 ller glial cells phagocytosed cell bodies of dead rod photoreceptors albeit at a lower frequency.

251 Isolated from the Dead Sea in 1975, Haloferax volcanii thrives in high sal

252 The discovery of the 2,000-year-old Dead Sea Scrolls had an incomparable impact on the histo

253 The bitumen can be sourced to the Dead Sea using biomarkers, evidencing a trade in this ma

254 fusion abnormality and increased physiologic dead space (dynamic compliance, 33.7 +/- 14.7 ml/cm H(2)

255 antly lower PaO2/FIO2 and higher physiologic dead space compared with extrapulmonary acute respirator

256 Pulmonary dead space fraction (Vd/Vt) is an independent predictor

257 t presents large variability, while elevated dead space fraction may be a specific pathophysiological

258 erference (CRISPRi) by binding catalytically dead SpCas9 (dSpCas9) to the region upstream of a gene o

259 ibition of Src or overexpression of a kinase-dead Src mutant prevented the effect of leptin, whereas

260 distinct viability assays: PrestoBlue, Live/Dead stain and a novel luciferase-based assay.

261 Cellular viability was assessed using LIVE/DEAD stain kit and the recellularised scaffolds were fur

262 rmed by formation of formazan crystals, live-dead staining and caspase-3 activity assay.

263 acterium E. coli was semi-quantified by Live/Dead staining and fluorescence microscopy, and visualise

264 nt and detachment were also observed by Live-Dead staining and phase contrast imaging.

265 Antibacterial testing performed with live/dead staining indicated that the acetic acid-solubilized

266 Live/dead staining showed that after 1 and 3 days in culture,

267 Live/Dead stains showed the scaffold successfully supported c

268 bycatch in fisheries and 44 live or freshly dead stranded turtles from the west coast of North and C

269 n prevents the formation of inactive sulfur (dead sulfur).

270 aria, that invade necrotic wound and feed on dead tissue.

271 terodimer with one arm that is catalytically dead, to extend the lifetime of the ATP/ADP state by pre

272 quality-adjusted life-years (QALY; range: 0 [dead] to 0.5 [full health at 6 months]).

273 on (EQ-5D-5L) for quality of life (range: 0 [dead] to 1 [full health]); and quality-adjusted life-yea

274 Drought mortality increased dead tree and surface fuel carbon in all treatments, whi

275 ng a tree-ring database of surviving and now-dead trees from 118 sites (22 species, >3,500 trees).

276 ass energy production that utilizes standing dead trees removed for safety concerns.

277 Living and dead trees transport and emit CH(4) produced in soils; l

278 ansport, and emission of CH(4) by living and dead trees, and the spatial and temporal dynamics of the

279 n live trees, but a fewer number of standing-dead trees, associated with the annual operation of larg

280 knockdown; however, reintroduction of kinase-dead TTK did not.

281 inducing cell death; however, the resulting dead tumor cells, or debris, may paradoxically stimulate

282 how that wild-type MDV U(S)3, but not kinase-dead U(S)3 (U(S)3-K220A), increases CREB phosphorylation

283 Using an enzymatic dead version of PRMT5 and a PRMT5-specific inhibitor, we

284 to alter NMII RLC localization, while a GAP-dead version of RN-tre partially restored phosphomyosin

285 We also generated a catalytically dead version of the tyrosinating enzyme TTL (TTL-E331Q)

286 Further confinement of kinase-dead versions of Lck suggests that Lck confinement was n

287 .5 (P < 0.005) for the two predicted groups (dead versus alive 1 year after ECG) over a 25-year follo

288 statement: 'When my IBS is bad, I wish I was dead' versus 4 and 7% of patients with mild and moderate

289 tection features on a fused silica chip in a dead volume-free manner, all extra-column peak dispersio

290 nt was sampled into microcapillaries using a dead volume-reduced world-to-chip interface.

291 Grouped under the same "dead-water" vocabulary, two wave-making resistance pheno

292 This outbreak resulted in numerous dead wild birds with severe pathology.

293 fect soil carbon stocks, despite an elevated dead wood density relative to undisturbed forests, but i

294 it CH(4) produced in soils; living trees and dead wood emit CH(4) produced inside trees by microorgan

295 events generate large quantities of standing dead wood, raising concern over catastrophic wildfire an

296 tribute between 58 and 64% of mass loss from dead wood.

297 Proportions of dead worms also did not differ between the 4 groups (P =

298 Proportions of dead worms also did not differ between the 4 groups (p=0

299 field, the continuous separation of live and dead yeast cells as well as the yeast cells with targete

300 NETs formed a barrier "dead zone," confining bacteria to the external corneal e