ライフサイエンスコーパス: decay time

1 rized by a fast (tau(1)) and a slow (tau(2)) decay time.

2 and prevents the shortening in NMDAR current decay time.

3 ishes the developmental change in NMDAR-EPSC decay time.

4 diction of brightness, stability, Phi(f), or decay time.

5 ctivity did not significantly alter the EPSC decay time.

6 cholinesterase inhibitor, prolonged the EPSC decay time.

7 cumulation but had no effect on the rise and decay time.

8 velength emission associated with the longer decay time.

9 using a long lifetime standard with a known decay time.

10 the deactivation kinetics by prolonging the decay time.

11 (cis) isomer in DOPC (DPPC) presents a fast decay time.

12 ag-1 and a sharp rise in the autocorrelation decay time.

13 2+ transient amplitude and prolonged the 50% decay time.

14 smission to Bergmann glia and decreased EPSC decay time.

15 ise times, amplitudes, charge transfers, and decay times.

16 to CaN to maintain NMDAR currents with long decay times.

17 of the eIPSC could be distinguished by their decay times.

18 imilar to WT bumps, but with slightly slower decay times.

19 current amplitudes with significantly faster decay times.

20 complexes led to variations in fluorescence decay times.

21 elations with relatively long characteristic decay times.

22 s of more than PhiPL = 90% at short emission decay times.

23 mations that fit the experimentally measured decay times.

24 tes of LOV390 formation but exhibited adduct decay times 1 order of magnitude faster than wild type.

25 netically fast (rise time, 0.32 +/- 0.02 ms; decay time, 1.66 +/- 0.18 ms; mean +/- SD; n = 6 cells),

26 (31%), and prolongation of the Ca(2+) signal decay time (165%) than overexpression (2-fold) of wild t

27 imes greater), and decayed more slowly (half-decay time 189 ms for strontium and 32 ms for calcium).

28 entedly fast conductance cycle (current half-decay time 2-4 ms depending on voltage).

29 tivity exhibit significantly longer AHP half-decay times (24.67 vs. 11.02 ms) and greater AHP amplitu

30 d trimer system, the fluorescence anisotropy decay time (35 fs) is found to be much shorter than that

31 nses were slow (rise time, 1.28 +/- 0.35 ms; decay time, 6.71 +/- 1.46 ms; n = 8 cells).

32 pressure is typically much shorter than the decay time after cessation or decline in the volume of d

33 ecays at intermediate pH values and that the decay time amplitudes are greatly dependent on pH.

34 e different membrane phases via fluorescence decay time analysis, making this new probe versatile for

35 an account for frequency-dependent change of decay time and force potentiation at intermediate stimul

36 d in gas phase by measuring the fluorescence decay time and ion-neutral collision cross sections (CCS

37 Elevating quantal content lengthened EPSC decay time and prolonged both the fast (alpha7-nAChR-med

38 osolic Ca(2+) followed by an increase in the decay time and the spread of the spontaneous Ca(2+) spik

39 wo adjustable parameters instead of the many decay times and amplitudes required in standard analysis

40 that elicited responses show layer-specific decay times and frequency-dependent facilitation.

41 rents in mutant neurons have slower rise and decay times and lower NMDAR/AMPAR current ratios.

42 ic modeling of temperature-dependent singlet decay times and quantum yields of fluorescence, isomeriz

43 cal role in recovering accurate and reliable decay times and resulting diffusion constants.

44 peak parameters including shorter rise time, decay time, and half-width as compared to a bare carbon

45 2+) signaling, the specific effects on peak, decay time, and/or frequency were different.

46 peak amplitudes, prolonged current rise and decay times, and altered responses to benzodiazepine ago

47 that display long emission wavelengths, long decay times, and high quantum yields.

48 etected and analysed for amplitude, rise and decay times, and latency.

49 (A) receptor, the peak amplitudes, 90-to-10% decay times, and total charge transfer of spontaneous mi

50 than 10-fold slower in nucleo-olivary cells (decay time, approximately 25 ms) than in large cells ( a

51 s (reductions of approximately 48% in 10-90% decay time, approximately 40% in tau, and approximately

52 eld and comparable reduction of luminescence decay time are observed.

53 ant glycine receptors, we show that response decay times are accelerated by addition of GABA, a weak

54 efficient scintillation crystals with short decay times are indispensable for improving the performa

55 echnique in which fluorescence excited state decay times are measured as fluorescently labeled cells

56 The decay times are shorter for the purines than for the pyr

57 We also rule out small increases in IPSC decay times (as caused by W170S and R414H) as a possible

58 L-type calcium channel window current, slow decay time at various voltages, and increased late calci

59 es as photodetectors, for both intensity and decay-time based monitoring of the sensing element's PL.

60 rylated by postnatal day 11 as NMDAR current decay times become faster.

61 t on the observation wavelength, with longer decay times being observed at longer wavelengths.

62 The temperature dependence of decay times can be attributed to changes in the viscosit

63 ample, we show that very short synthesis and decay times can perturb the wild-type pattern.

64 amplitude, amplitude, wave speed, rise, and decay times) can be easily extracted.

65 2AP does not exhibit the long fluorescence decay time characteristic of the free nucleoside, sugges

66 taining proteins are well fit using only two decay times compared to three required for Trp.

67 uum has a rise time of less than 80 ns and a decay time component of approximately 300 micros.

68 The absorption spectrum and fluorescence decay time components of the complex at room temperature

69 The second decay time constant (tau 2) was significantly longer in

70 postsynaptic currents (mIPSCs) (predominant decay time constant (tau(decay)), 1.0 ms) in addition to

71 ntly with distance from the soma whereas the decay time constant (taudecay) of Delta[Ca2+] decreases

72 an amplitude of 68 +/- 6 pA, and a weighted decay time constant (tauw) of 15.8 +/- 2.9 ms.

73 +/- 0.59 to -4.15 +/- 0.73 nA with the fast decay time constant accelerating from 0.75 +/- 0.09 ms a

74 However, the miniature IPSC decay time constant and the benzodiazepine potentiation

75 is derived as the inverse of the exponential decay time constant based on a heterogeneous Michaelis-M

76 In contrast, the IPSC decay time constant depended only on the postsynaptic cl

77 However, the decay time constant for STP of the AMPA receptor-mediate

78 tential was approximately 6 min, whereas the decay time constant for STP of the NMDA receptor-mediate

79 e recovery from desensitization is slow (the decay time constant is roughly 500 milliseconds), little

80 ronal firings are asynchronous, the synaptic decay time constant needs to be comparable to that of th

81 been found that the crystal exhibits a fast decay time constant of 1 ns at 7 K.

82 five cells, with a time to peak of 1.0 ms, a decay time constant of 2.3 ms, and a reversal potential

83 al of -83 mV, a time to peak of 2.6 ms and a decay time constant of 6.5 ms.

84 r the catalytic effect of temperature on the decay time constant of a synaptic current.

85 0 and 15 ms after an action potential, had a decay time constant of about 30 ms, and showed no accumu

86 Also, halothane considerably prolonged the decay time constant of evoked IPSCs in pyramidal cells a

87 howed that R(A) resulted in a slowing of the decay time constant of excitatory postsynaptic currents

88 he amplitude but increased its effect on the decay time constant of field EPSPs recorded under condit

89 at 38 h after ischemia; the rising slope and decay time constant of I(A) were accordingly increased a

90 37), but did not alter the amplitude and the decay time constant of mIPSCs.

91 opregnanolone caused an increase in the slow decay time constant of spontaneous GABA-mediated IPSCs i

92 on progressively decreases the amplitude and decay time constant of spontaneous mEPSPs.

93 The average decay time constant of the AR for orientation-tuned cell

94 enzodiazepine agonist zolpidem increased the decay time constant of the IPSCs of immature granule cel

95 velocity, the quick-phase frequency, and the decay time constant of the negative OKAN were dependent

96 ation had no effect on the peak amplitude or decay time constant of the NMDA component, or the I-V re

97 In HD mice, the decay time constant of the perisynaptic Glu concentratio

98 nnection, amplitude, latency, rise time, and decay time constant of the unitary EPSC were not differe

99 ncy, dose-dependent hyperpolarization with a decay time constant on the order of a few seconds.

100 e induced absorption shows quadratic and the decay time constant shows linear dependence on the laser

101 ure/volume measurements (-dP/dtmin, pressure decay time constant tau-Glantz, and passive filling stif

102 a significantly higher frequency and faster decay time constant than those recorded from the medulla

103 9 +/- 0.0299 pA pF(-1) (n = 7 cells) and the decay time constant was tau = 790 +/- 76 ms (n = 5).

104 709 +/- 0.0299 pA pF-1 (n = 7 cells) and the decay time constant was tau = 790 +/- 76 ms (n = 5).

105 and the benzodiazepine potentiation of this decay time constant were both significantly increased in

106 ns (10-100 pA, 10 ms rise time constant, 5 s decay time constant) in the presence of various synaptic

107 cay time, weighted decay time constant, slow decay time constant, and, consequently, the total charge

108 In addition, the decay time constant, but not the amplitude of the mEPSCs

109 ak amplitude, 90-to-10% decay time, weighted decay time constant, slow decay time constant, and, cons

110 f speckle patterns to obtain the exponential decay time constant, tau.

111 of the unitary IPSC: latency, rise time, and decay time constant.

112 i, resulting primarily from a slowing of the decay time constant.

113 12), without affecting the amplitude and the decay time constant.

114 PSCs) without changing the amplitude and the decay time constant.

115 e difference was accounted for by the second decay time constant.

116 r is marked by a significant increase in the decay-time constant for evoked and spontaneous IPSCs and

117 e Pb2+ changed neither the amplitude nor the decay-time constant of the MPSCs, Pb2+-induced changes i

118 but not young, rats exhibit a twofold longer decay time-constant and temporally summate a train of st

119 erculoventral cells had significantly faster decay time constants (0.35-0.40 msec) than did those fro

120 bicuculline methiodide (BMI), and had longer decay time constants (4.5-6.0 ms) that were modulated by

121 , and zolpidem-sensitive mIPSCs had weighted decay time constants (tau(w)) of 4-6 ms.

122 ell recordings at 22 degrees C, the weighted decay time constants (tau(w)) of spontaneous IPSCs (sIPS

123 tants were increased by 195% and evoked IPSC decay time constants by 220% compared with age-matched c

124 The decay time constants change e-fold per 22.1 mV above the

125 e in Ng+/+ compared with Ng-/- mice, but the decay time constants did not differ.

126 Decay time constants of cytosolic Ca2+ transients from c

127 Decay time constants of EPSCs increased (or decreased) i

128 wer IPSCs, with a 2.6-fold difference in the decay time constants of spontaneous IPSCs and a 5.3-fold

129 an inactivating potassium (IA) current with decay time constants of up to 225 ms, and small-amplitud

130 ated quantum states of multiple nuclei, have decay time constants that may exceed T1 by large factors

131 s methods for assessing ventricular pressure decay time constants to test whether sensitivity to slig

132 Spontaneous IPSC decay time constants were increased by 195% and evoked I

133 EPC and MEPC decay time constants were prolonged because of a slowed

134 ication of L-glutamate to nucleated patches, decay time constants were similar at +/-60 mV in the pre

135 current amplitudes, altered desensitization decay time constants, and reduced GlyR clustering and sy

136 ated EPSCs showed an unusually wide range of decay time constants, from <5 to >500 ms.

137 (mIPSCs) without affecting the rise time and decay time constants.

138 ACh concentrations and the synaptic current decay time constants.

139 GABA(A) receptors, isoflurane prolonged the decay-time constants of both eIPSCs and mIPCSs.

140 EPSCs accelerate over development to achieve decay time-constants of 2.5 ms.

141 tal maturation accelerate to sub-millisecond decay time-constants.

142 ak) of fAMPAsEPSCs was 1.5+/-1.05 ms and the decay time could be fitted with a single exponential wit

143 ch as high solubility and short fluorescence decay time, could be obtained from fluorophors composed

144 The decay time course of excitatory postsynaptic currents ge

145 Here, we show that the decay time course of GABAergic inhibitory synaptic curre

146 prolonged (63 +/- 14 %; mean +/- s.e.m.) the decay time course of miniature IPSCs (mIPSCs) without si

147 rong correlation between prolongation of the decay time course of sIPSCs and potentiation of single-c

148 e whether the magnitude of modulation of the decay time course of sIPSCs correlates with the extent o

149 MPARs in stellate cells but did not slow the decay time course of synaptic currents.

150 The rise and decay time course of the sAMPAsEPSCs and NMDAsEPSCs were

151 Furthermore, the decay time course ofEPSCs mediated by GluR2-containing r

152 The averaged macroscopic current exhibited a decay time course which was well described by a single e

153 increasing its width and by slowing down its decay time course.

154 rents (EACs) with no effect on the NMDAR EAC decay time course.

155 receptor-mediated sEPSC with slower rise and decay time courses and larger peak amplitudes (sAMPAsEPS

156 In parallel, NMDA EPSC decay times decreased over a similar developmental time

157 bly phased bends, the relative birefringence decay times depend on the flexibility of each bend, not

158 ay that is generally stepwise linear and the decay time depends strongly on [erg].

159 Although pure GABAergic and glycinergic decay times did not differ depending on HM location, the

160 ive decay paths reduces the overall emission decay time distinctly.

161 e temperature dependence of the luminescence decay time enables intrinsic temperature compensation of

162 ied out, as a function of expected diffusion decay time for a particular solute, and show that use of

163 seconds) was markedly greater than the half decay time for cytosolic Ca2+ sparks (31.2+/-0.56 ms) ob

164 thdrawal is due to a sixfold decrease in the decay time for GABA currents and consequent decreased in

165 The average decay time for PSI-LHCI-LHCII is approximately 65 ps upo

166 The measured rise and decay time for the 1(st) order is 7.62 ms and 6.75 ms, a

167 he formation time for both peaks matches the decay time for the delocalized LMCT excited state.

168 that the decay is exponential with different decay times for other, simpler dipeptides.

169 The decay times for the higher excited states of [Au(13)(dpp

170 A significant correlation of rise and decay times for the overall population of unitary IPSCs

171 d on Floquet modes, which increases the Rabi decay time ([Formula: see text]) in a number of material

172 g by electron transfer predict heterogeneous decay times from 50-500 ps that agree with our experimen

173 ed consistently over a wide range of tension decay times from a few seconds to over 100 s.

174 m2 mass transporting area), 90 s 10-90% rise/decay time glucose electrode, and an on-the-skin electro

175 Long "in-bag" (129)Xe polarization decay times have been measured (T1 approximately 38 min

176 w the carrier-recombination-limited hologram decay time in a photorefractive crystal.

177 er miniature inhibitory postsynaptic current decay time in null mice, with no change in miniature inh

178 ons displayed a larger amplitude and shorter decay time in spontaneously hypertensive rats (SHRs) tha

179 addition, the normal downregulation of NMDAR decay time in sSC neurons at P11 was absent after NMDA t

180 er Ames medium, we also observed >30% longer decay time in the PCP2-null rod bipolar cells.

181 e early and rapid reduction of NMDAR current decay time in visual neurons.

182 However, the invariance of *Trp decay times in ferric and ferrous Mbs raises the questio

183 release, as measured by EPSP amplitudes and decay times in postsynaptic neurons.

184 ontains at least seven transient states with decay times in the range from 10 micros to 200 ms, but t

185 We find that PL decay times increase by a factor of 2 on addition of CsC

186 Under control conditions, GABA(A slow) IPSC decay times increased linearly with membrane depolarizat

187 tic trauma (AT, loud sounds) slow AMPAR-EPSC decay times, increasing GluA1 and decreasing GluA4 mRNA.

188 es, such as (Pro-Pro-Gly)(10), show multiple decay times, indicating multiple scission locations and

189 light response directly, and accelerates the decay time indirectly via the inhibitory network.

190 The observed temperature dependence of the decay time is discussed in terms of two possible mechani

191 Thus, the overall emission decay time is distinctly reduced.

192 When bound to a protein or lipid, the decay time is near 3 microseconds and the quantum yield

193 The spin echo decay time is ~40 mus, both with one and four echo pulse

194 he level of screening achieved at nanosecond decay times is shown to change with the coverage of elec

195 then becomes saturated, whereas the average decay time keeps increasing linearly.

196 mplicated in developmental plasticity, shows decay time kinetics that shorten postnatally as NR2A sub

197 ncreased, while GABA-ACh pairing affects the decay time leading to elevated calcium levels during the

198 l populations, noise can render the measured decay times meaningless for small amplitude Ca2+ sparks.

199 elationship between Ca2+ spark amplitude and decay time might be used to distinguish Ca2+ sparks from

200 curate diffusion constants for both species, decay times must be bounded by adequate minimum and maxi

201 cay schemes and the dramatic drop in channel decay time observed at erg mol % = 50.

202 ltrafast (tens of femtoseconds) hot electron decay times observed experimentally arise from electron-

203 the same order as the S(1)-S(0) nonradiative decay time obtained previously for the (6,4) CNT.

204 somer dissociates through thermolysis with a decay time of 14 min at 296 K to form the [6,6]-closed e

205 rong fluorescence at 1544 nm with a measured decay time of 3 ms and an estimated quantum efficiency o

206 tion time of 1.68 +/- 0.05 ps and an initial decay time of 31.7 ps.

207 ations of OHBI give an estimated first-order decay time of 476 fs for the S(1) state, which is larger

208 itated neural excitability by shortening the decay time of 5-HT(3)R currents, lowering the stimulus t

209 , 5-HT(3B) is responsible for shortening the decay time of 5-HT(3)R-mediated currents.

210 cated by increased rectification and reduced decay time of AMPAR-mediated excitatory postsynaptic cur

211 d triplet pair states, but the rapid singlet decay time of approximately 200 ps in solution-grown sin

212 mately 20-500 fF (mean = 123 fF) with 10-90% decay time of approximately 30-500 ms.

213 , because of the low proton density and fast decay time of bone tissue.

214 a(2+) exchange activity (indexed by the half decay time of caffeine-elicited Ca(2+) transient) by 27%

215 The effect upon EPSC decay time of changing quantal content was 5-10 times mo

216 ampal slices from CIE rats revealed that the decay time of GABAR-mediated miniature inhibitory postsy

217 educed light spread in the detector and fast decay time of GSO.

218 cells in adult female rats and prolongs the decay time of IPSCs in these cells.

219 re, bounding the scrambling time by a finite decay time of local quantum correlations at late times.

220 ion as shown by increasing the frequency and decay time of mEPSCs, and simultaneously inhibiting GABA

221 Amplitude and decay time of mEPSCs, decay time of mIPSCs, and spine si

222 Amplitude and decay time of mEPSCs, decay time of mIPSCs, and spine size were unaltered.

223 In addition, the decay time of NMDA currents was decreased in BDNF(Met/Me

224 a rapid, activity-induced shortening in the decay time of NMDA receptor (NMDAR) currents.

225 i) in cultured neurons SGE-301 prolonged the decay time of NMDAR-dependent spontaneous excitatory pos

226 ith these changes, the current amplitude and decay time of NMDARs in PFC was significantly reduced.

227 s the inhibitory signaling by prolonging the decay time of postsynaptic GABAergic currents upon photo

228 te the relatively slow transfer, the overall decay time of PSI-LHCI-LHCII remains fast enough to assu

229 o MIMG resulted in a decreased intensity and decay time of RuCon A.

230 At the same time, the rise time and decay time of sEPSCs significantly decreased, suggesting

231 without affecting mean current and inhibited decay time of sIPSCs.

232 stimulation or uncaging of IP3 increased the decay time of spontaneous Ca(2+) events without changing

233 The emission maxima and decay time of such tandem luminophores can be readily ad

234 cerebellar synapses selectively prolongs the decay time of synaptic currents, whereas a switch from G

235 d calcium kinetics associated with prolonged decay time of the calcium transient and increased diasto

236 We show how the decay time of the El Nino anomaly in this data set can b

237 simple strategy to control and modulate the decay time of the functionalized Yb(3+)-doped nanopartic

238 ion, both protein kinase A and C shorten the decay time of the glycine current.

239 ns is however limited by the relatively fast decay time of the hyperpolarized spin state together wit

240 n of the gamma oscillation, in which the the decay time of the inhibitory cells is critical to the fr

241 longer times we find an ambient temperature decay time of the Omega Fe-C5' bond of tau ~ 5-6 s, like

242 The decay time of the oscillations is 14.5 +/- 2.7 minutes f

243 delay) is crucial because tdelay defines the decay time of the reactive intermediate.

244 hematite, this recombination exhibits a 50% decay time of ~6 ps, ~10(3) times faster than that of Ti

245 cay displays multiexponential character with decay times of 1.2 and 16 ps, and 0.6, 2.2, and 4.2 ns.

246 able properties overall, which included fast decay times of 2 ns.

247 lipid hydrogen bonds are long lived, showing decay times of 50 ns, and forming strings of lipids, and

248 this issue, we have measured the rotational decay times of a 'gapped-duplex' DNA molecule possessing

249 is described by two kinetic components with decay times of approximately 20 and approximately 200 ns

250 tracellular space, is reflected by increased decay times of neuronal NR2A-mediated NMDA currents.

251 Increasing quantal content also prolonged decay times of pharmacologically isolated alpha7-nAChR-

252 The rise times, areas, half-widths, and decay times of sEPSCs and emEPSCs and interevent interva

253 lay very high luminescence quantum yields at decay times of several tens of mus even in solution unde

254 etectably, in that the amplitudes, areas and decay times of spontaneous miniature EJCs were unchanged

255 size, without affecting the average rise and decay times of synaptic currents.

256 second carbamate is evident from bimodal T2 decay times of the approximately 163 ppm peak, indicatin

257 The characteristic decay times of the autocorrelation functions report the

258 showed that an alteration of amplitudes and decay times of the GABAergic currents to the dentate nuc

259 llows a bi-exponential time dependence, with decay times of the order of picoseconds, indicating that

260 ground state bacteriorhodopsin and the mean decay times of the photocycle M-state intermediates.

261 the similarities between the characteristic decay times of the time correlation function, as obtaine

262 This is reflected in short emission decay times of the triplet states of only 34 mus (1) and

263 The rise- and decay-times of local mIPSCs were also independent of the

264 d at P12-13; (3) the kinetics (rise time and decay time) of both mEPSCs and mIPSCs accelerated with a

265 t pure graphane has a very long nonradiative decay time, on the order of 100 ns, while epoxy- and hyd

266 Nonlinear fitting of the fluorescence decay times provides activation parameters for singlet d

267 Two months after AT the EPSC decay times recovered to control values.

268 s with multiple peaks and increased rise and decay times, reflecting "desynchronized" SV fusion.

269 ependent long-term depression decreased EPSC decay time, revealing a 'late' current that is present w

270 e of the fluctuations controlled by the NMDA decay time scale.

271 ectra (TRES) indicate that this fluorescence decay time should be ascribed to a highly quenched confo

272 ed patches to synaptically released GABA had decay times similar to brief pulse responses.

273 EPSCs in low quantal-content conditions had decay times similar to the time course of receptor deact

274 Fluo-4-measured calcium transient decay time suggested calcium mishandling in CACNA1C-p.R5

275 c membrane input resistance or EPSC rise and decay time suggested that the effects of PPs on EPSCs we

276 large drop in NMDA receptor (NMDAR) current decay time synchronized across all neurons and occurring

277 with a 2-fold faster rise and 7-fold faster decay time (t1/2 of 40 ms) than GCaMP6f, indicating that

278 tor (IP3R), significantly prolonged the half-decay time (t1/2) of IP3-induced Ca2+ transients.

279 th GCaMP6fu displaying fluorescence rise and decay times (t1/2) of 1 and 3 ms (37 degrees C) in vitro

280 ce (TEB) measurement in which the rotational decay times (taugap) of DNA molecules possessing central

281 The multiquantal events exhibit faster decay time than the GluR4 receptor desensitization time

282 Pure GABAergic events had slower decay times than glycinergic events.

283 These events had slower rise times and decay times than sparks and were more heterogeneous.

284 2O8+x characterized by an excited population decay time that maps directly to a discrete component of

285 s that the mutant subunit increases synaptic decay times, thereby prolonging postsynaptic activity.

286 Non-contact luminescence decay time thermometry is often the method of choice for

287 Median decay time to half-life ranged from 13.8 to 15.1 months.

288 s accompanied by a nearly 2-fold increase in decay time, to values that are indistinguishable from th

289 anostructures exhibit extremely long carrier decay times up to 20 micros that are combined with high

290 The two decay times vary only mildly with the water concentratio

291 with membrane depolarization, and this IPSC decay time voltage dependence was not significantly alte

292 IPSC peak amplitude, 90-to-10% decay time, weighted decay time constant, slow decay tim

293 The spike amplitude as well as rise and decay time were comparable with those measured by carbon

294 pproximately 400 msec, whereas rise time and decay time were not altered significantly.

295 Phosphorescence decay times were found to be around ~5 us at 295 K and ~

296 However, faster rising and decay times were obtained for the bright to dark than th

297 In contrast, GlyR IPSC and NMDAR-EPSC decay times were unchanged.

298 by the histograms of the rise times and half-decay times, which revealed modes at 38 and 65 ms, respe

299 opic transmission to glia and decreased EPSC decay time with closely similar time courses.

300 e scaling of the characteristic fluorescence decay time with the vesicle diameter and the buildup of

301 on, significantly increases glycinergic IPSC decay times without causing motor side effects.