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1 of breathing and metabolism in high-altitude deer mice.
2 onse that fails to clear the virus occurs in deer mice.
3 disease were observed in any of the infected deer mice.
4 Nombre virus (SNV) from chronically infected deer mice.
5 99 case patients reported indoor exposure to deer mice.
6 cted in seven different organs of sacrificed deer mice.
7 pas1, including high-altitude populations of deer mice.
8 obic performance in hypoxia in high-altitude deer mice.
11 ve phosphorylation pathways in high-altitude deer mice and by concomitant changes in the expression o
13 n, genome structure and genetic diversity in deer mice and likely facilitate local adaptation across
14 Hopi Hoekstra studied an intercross between deer mice and old-field mice that differ in their mating
17 pproaches: the genetic basis of burrowing in deer mice and transcriptomic analyses of division of lab
20 mice, we sacrificed experimentally infected deer mice at eight time points from day 21 to day 217 po
21 RT-PCR) in the blood of ELISA-positive adult deer mice but not in the blood of ELISA-positive juvenil
22 protein is highly enriched in high-altitude deer mice, but its functional significance is unknown.
25 agent of HPS in North America, propagated in deer mice develop HPS, which is characterized by thrombo
26 results suggest that sperm from promiscuous deer mice discriminate among relatives and thereby coope
27 ecies from four different groups (Peromyscus deer mice, Drosophila flies, mosquitoes, and Nasonia was
28 he lungs or cardiac tissue from SNV-infected deer mice, even at the time of peak viral antigen expres
29 genetic cross between two sister species of deer mice exhibiting large, innate differences in the ac
30 ng gestation and found that lowland-ancestry deer mice expand their placenta and maternal blood space
33 nfection of P. maniculatus, we examined wild deer mice for localization of viral antigens and nucleic
35 eucopus raised in the laboratory and in male deer mice from the subspecies Peromyscus maniculatus bai
36 across neonatal development in eight taxa of deer mice (genus Peromyscus) and compare them with labor
42 Immunohistochemical analysis of SNV-infected deer mice identified viral antigens within lung, liver,
43 cular helper phenotype in some ANDV-infected deer mice, including activation of the interleukin 4 (IL
44 dentified elevated immune gene expression in deer mice infected with ANDV and suggested maturation to
45 enhanced thermogenic performance of highland deer mice is largely attributable to an increased capaci
49 bias in seropositivity was detected in adult deer mice, no significant sex bias in seropositivity was
50 foraging within mature forests; in contrast, deer mice occur in high densities across forest types an
52 wever, in T cells from persistently infected deer mice, only TGF-beta(1) was expressed by all lines,
54 ocial behavior, across two sister species of deer mice (Peromyscus maniculatus and P. polionotus) wit
55 adaptive increases in aerobic performance in deer mice (Peromyscus maniculatus) adapted to the hypoxi
59 ouse-adapted strain of Sin Nombre virus from deer mice (Peromyscus maniculatus) by i.m. inoculation o
61 h American rodent genus Peromyscus, highland deer mice (Peromyscus maniculatus) have greater thermoge
63 -altitude variant of Epas1 in North American deer mice (Peromyscus maniculatus) on the control of bre
64 cated globin genes in natural populations of deer mice (Peromyscus maniculatus) that are adapted to d
65 plasticity in enabling highland and lowland deer mice (Peromyscus maniculatus) to sustain aerobic th
68 n a field experiment, we observed individual deer mice (Peromyscus maniculatus) with known personalit
69 adaptation in the reproductive physiology of deer mice (Peromyscus maniculatus), a rodent species wit
70 ied biosolids, soil, earthworms (Lumbricus), deer mice (Peromyscus maniculatus), and eggs of European
71 evaluated by inoculating them into groups of deer mice (Peromyscus maniculatus), hamsters, and Swiss
72 es not cause disease in chronically infected deer mice (Peromyscus maniculatus), the natural host.
74 the tandemly duplicated beta-globin genes of deer mice (Peromyscus maniculatus), which contribute to
80 83.5% were pilfered by 10 species, including deer mice ((Peromyscus maniculatus) and southern red-bac
84 ere relatively weak in T cells isolated from deer mice, regardless of acute or persistent infection.
88 omparisons of forest and prairie ecotypes of deer mice revealed 13 inversions that contribute to diff
90 conducted on wild-caught, naturally infected deer mice showed a similar pattern of intermittent posit
91 sence of sera from bonded and bond-disrupted deer mice showed that in monogamous Peromyscus polionotu
95 gen levels within the kidney were highest in deer mice that did not have antibodies to SNV but contai
96 field, we show that the light coat color of deer mice that recently colonized the light-colored soil
99 the genetic architecture of parental care in deer mice to discover an important contribution of vasop
100 etween high- and low-altitude populations of deer mice to disrupt linkages between genetic loci so th
101 We acclimated lowland- and highland-ancestry deer mice to normoxia or hypoxia (12.3% O(2)) during ges
102 role in SNV persistence and immune escape in deer mice, we measured the prevalence of virus quasispec
103 address Sin Nombre (SN) virus persistence in deer mice, we sacrificed experimentally infected deer mi
104 nal to the mass of the animal, with juvenile deer mice weighing less than 11 g most likely to be anti
108 lizing antibodies were routinely detected in deer mice which maintained virus RNA in the blood and lu
110 serial blood samples from naturally infected deer mice, which were sequentially analyzed for SNV dive
112 to transmit infection by cohousing infected deer mice with seronegative cage mates, we observed only
114 virus through inoculation of cells or naive deer mice with the secreta or excreta of infected mice w