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1 licle-stimulating hormone, testosterone, and dehydroepiandrosterone sulfate.
2 , but not by phenobarbital or the steroid PP dehydroepiandrosterone sulfate.
3 allo-tetrahydrocortisol, and the presence of dehydroepiandrosterone sulfate.
4 itor oxythiamine (OT) and the G6PD inhibitor dehydroepiandrosterone-sulfate (0.5 microM each) exerted
6 hydroepiandrosterone (+14.4%, P = 0.02), and dehydroepiandrosterone sulfate (+7.2%, P = 0.26) compare
8 inhibitory steroids pregnenolone sulfate and dehydroepiandrosterone sulfate act through a common inte
9 PREGS potentiation of mEPSCs was mimicked by dehydroepiandrosterone sulfate and (+)-pentazocine but n
10 ibition of OATP1B3-1B7-mediated transport of dehydroepiandrosterone sulfate and identified several in
11 ports taurocholic acid, the adrenal androgen dehydroepiandrosterone sulfate, and thyroid hormone, as
12 physical activity, social class, education, dehydroepiandrosterone sulfate, androstanediol glucuroni
13 ational study found lower, but still normal, dehydroepiandrosterone sulfate concentrations in statin-
15 terol, high-density lipoprotein cholesterol, dehydroepiandrosterone sulfate, cortisol, systolic and d
16 in saliva and found significantly increased dehydroepiandrosterone sulfate (DHEA-S) and reduced estr
18 body of research has provided evidence that dehydroepiandrosterone sulfate (DHEA-S) is involved in a
19 monstrate the utility of the nanosensor with dehydroepiandrosterone sulfate (DHEA-S), a small-molecul
20 easing hormone, adrenocorticotropic hormone, dehydroepiandrosterone sulfate (DHEA-S), and cortisol.
21 ing retinoic acid and the endogenous steroid dehydroepiandrosterone sulfate (DHEA-S), it also has a c
22 port that chronic exposure to high levels of dehydroepiandrosterone sulfate (DHEA-S; converted in viv
23 iveness and lower levels of androstenedione, dehydroepiandrosterone-sulfate (DHEA-S) and free PSA%, a
24 tisol, dehydroepiandrosterone (DHEA), and/or dehydroepiandrosterone-sulfate (DHEA-S) concentrations.
25 inding protein B [S100B]), stress (cortisol, dehydroepiandrosterone sulfate [DHEA-S] epinephrine, nor
26 that endogenous sex hormones (testosterone, dehydroepiandrosterone sulfate [DHEA-S], and estradiol)
27 udy to determine whether hormone levels (E2, dehydroepiandrosterone-sulfate [DHEA-S], and testosteron
29 tmenopausal serum levels of testosterone and dehydroepiandrosterone sulfate (DHEAS) and subsequent ri
30 ements of serum cortisol, corticotropin, and dehydroepiandrosterone sulfate (DHEAS) are used to diagn
31 adult male mice with the neuroactive steroid dehydroepiandrosterone sulfate (DHEAS) at 1 and 40 mg/kg
32 associations between obesity indicators and dehydroepiandrosterone sulfate (DHEAS) at 7 y of age and
33 (DU-14) to rats for 15 days increased plasma dehydroepiandrosterone sulfate (DHEAS) concentrations by
34 could identify hits that sense steroids like dehydroepiandrosterone sulfate (DHEAS) down to 1.3 uM wi
36 th PCOS have insulin resistance and elevated dehydroepiandrosterone sulfate (DHEAS) levels, which sup
37 (r > -0.6), bioavailable testosterone (BT), dehydroepiandrosterone sulfate (DHEAS), and the ratio of
38 ed inversely with plasma androstenedione and dehydroepiandrosterone sulfate (DHEAS), averaged across
39 terone, sex hormone-binding globulin (SHBG), dehydroepiandrosterone sulfate (DHEAS), luteinizing horm
40 neurosteroids such as pregnenolone sulfate, dehydroepiandrosterone sulfate (DHEAS), pregnanolone, an
41 characterized by the usual overproduction of dehydroepiandrosterone sulfate (DHEAS), whose detection
45 sterone, free testosterone, androstenedione, dehydroepiandrosterone sulfate (DHEAS)], sex hormone bin
46 rone sulfate, androstenedione, testosterone, dehydroepiandrosterone sulfate, follicle-stimulating hor
47 s of estrone, estradiol, testosterone, SHBG, dehydroepiandrosterone sulfate, free estradiol, and free
48 +CD38+HLA-DR+, and CD8+CD38+HLA-DR+ T cells, dehydroepiandrosterone sulfate, free testosterone, homeo
50 We examined testosterone, estradiol, and dehydroepiandrosterone sulfate in relation to incident A
52 served enantioselectivity for the actions of dehydroepiandrosterone sulfate indicates that its inhibi
53 re found for endogenous estrogens, cortisol, dehydroepiandrosterone sulfate, insulin, glucagon, or fo
54 , via administration of the enzyme substrate dehydroepiandrosterone sulfate or the enzyme inhibitor C
55 1.16-3.57; P = 0.01), lower plasma levels of dehydroepiandrosterone-sulfate (OR per 1-ln decrease = 2
56 1.16-3.57; P = 0.01), lower plasma levels of dehydroepiandrosterone-sulfate (OR per 1-ln decrease = 2
60 iol, estrone, estrone sulfate, testosterone, dehydroepiandrosterone sulfate, prolactin, and sex hormo
61 tic levels of testosterone, androstenedione, dehydroepiandrosterone sulfate, sex hormone-binding glob
62 impact of SLCO2B1 expression level on DHEAS (dehydroepiandrosterone sulfate) uptake was evaluated in
63 baseline SA (testosterone, androstenedione, dehydroepiandrosterone sulfate), was measured by ultrase
64 androstenedione, dehydroepiandrosterone, and dehydroepiandrosterone sulfate were associated with risk
65 androstenedione, dehydroepiandrosterone, and dehydroepiandrosterone sulfate were determined by liquid
66 a suggestion that dehydroepiandrosterone and dehydroepiandrosterone sulfate were inversely associated
67 androgens testosterone, androstenedione, and dehydroepiandrosterone sulfate were negatively associate
68 ten-3beta,17beta-diol disulfate 1 and 2, and dehydroepiandrosterone sulfate, were the next most stron
69 on of ERK1 and ERK2; however, the steroid PP dehydroepiandrosterone sulfate, which does not induce im