1 ntial genes that cannot be evaluated by gene
deletion analysis.
2 gPHO4, and CgMSN5 in the PHO pathway through
deletion analysis.
3 ins within the I-II loop were then mapped by
deletion analysis.
4 which we confirmed experimentally by serial
deletion analysis.
5 oligotyping, biochemical testing, or genomic
deletion analysis.
6 hese elements were authenticated by mutation/
deletion analysis.
7 and p79, using a yeast two-hybrid screen and
deletion analysis.
8 g critical cis-regulatory elements, requires
deletion analysis.
9 tributions to nucleosome positioning through
deletion analysis.
10 fine the promoter region upstream of idsA by
deletion analysis.
11 ion of this sequence is validated by in vivo
deletion analysis.
12 By
deletion analysis,
a region between nucleotides -145 and
13 Sequence and
deletion analysis allowed us to map LamR binding to two
14 (iii) The
deletion analysis also revealed that residues 596-638, w
15 Deletion analysis also reveals a functional requirement
16 tart site, which we identified by sequential
deletion analysis and by comparison of human and mouse g
17 Deletion analysis and characterization of the purified B
18 Deletion analysis and FR-alpha/SV40 promoter chimeras sh
19 Deletion analysis and generation of transgenic worms com
20 y, by combining the results from the somatic
deletion analysis and genetic linkage analysis, we were
21 Deletion analysis and mRNA reporter assays show that a c
22 Interestingly,
deletion analysis and mutation of the hypoxia responsive
23 Deletion analysis and mutational studies were done to ma
24 Deletion analysis and NMR spectroscopy revealed that an
25 Deletion analysis and point mutagenesis demonstrate that
26 Deletion analysis and point mutations indicate that pres
27 ntragenic PTEN mutations be offered clinical
deletion analysis and promoter-mutation analysis, respec
28 Deletion analysis and site-directed mutagenesis identifi
29 Here we employ 5' and 3'
deletion analysis and site-directed mutagenesis of the a
30 Sequential
deletion analysis and site-directed mutagenesis of the I
31 regulatory regions function in the same way,
deletion analysis and site-directed mutagenesis of the O
32 A combination of
deletion analysis and site-directed mutagenesis revealed
33 Deletion analysis and site-directed mutagenesis revealed
34 We identified the NLS of LEDGF/p75 through
deletion analysis and site-directed mutagenesis.
35 This site was confirmed by
deletion analysis and specific detection with a custom-g
36 Deletion analysis and the activity of hybrid promoter co
37 Through a combination of pull-down assays,
deletion analysis,
and isothermal titration calorimetry,
38 Random-linker insertion mutagenesis,
deletion analysis,
and site-directed mutagenesis of hydr
39 of hypoxia-responsive genes, COX-2 promoter
deletion analysis,
and site-directed mutagenesis, we ide
40 gene were cloned by PCR and characterized by
deletion analysis by using a reporter assay.
41 Promoter
deletion analysis,
by particle-bombardment transient tra
42 Promoter
deletion analysis characterizes a putative RA-responsive
43 nteraction with its binding partners through
deletion analysis,
co-immunoprecipitation, two-hybrid as
44 Site-specific mutagenesis and
deletion analysis confirm that the hydrophobic residues
45 ' genetic elements of the dbpBA operon using
deletion analysis,
coupled with luciferase reporter assa
46 Deletion analysis defined a 25 kb fragment in the RAD50
47 Deletion analysis defined the 5' upstream HS cluster reg
48 Deletion analysis defined the minimal binding motif of t
49 amping and confocal microscopy, coupled with
deletion analysis,
demonstrate that MMP23-PD suppresses
50 Deletion analysis demonstrated that binding to the minim
51 Deletion analysis demonstrated that removal of the regio
52 Previously,
deletion analysis demonstrated that several effectors ha
53 Further,
deletion analysis demonstrated that TAZ binds to the NH(
54 osidase reporter in a proportional manner, a
deletion analysis demonstrated that the AOX1 5'UTR conta
55 with MET1 in vitro and in vivo, and further
deletion analysis demonstrated that the carboxyl-termina
56 A
deletion analysis demonstrated that the low affinity was
57 Extensive
deletion analysis demonstrated that the majority of sile
58 This
deletion analysis demonstrated that the Pen-2 cytosolic
59 r constructs, reverse transcriptase PCR, and
deletion analysis demonstrated that the UpxYs do not aff
60 Furthermore, systematic
deletion analysis demonstrates that N-terminal amino aci
61 of VR occurs within a (G/C)(14) element, and
deletion analysis demonstrates that the reaction is inde
62 Moreover, this pX
deletion analysis demonstrates that WT pX function is mo
63 Deletion analysis determined that the essential elements
64 Promoter
deletion analysis,
electrophoresis mobility shift assays
65 he assay proved as accurate as the reference
deletion analysis for all 192 isolates and detected and
66 Moreover, nucleotide (nt)
deletion analysis found twelve deletion sites throughout
67 ing site-directed mutagenesis and systematic
deletion analysis from the 5' and the 3' ends of the PAP
68 Deletion analysis further established that the cell line
69 Promoter
deletion analysis further indicates that enhancer elemen
70 Deletion analysis further refined FSN-1 binding to a con
71 Structure-based
deletion analysis further shows the presence of a Wnt si
72 Deletion analysis,
fusion proteins, and point mutations
73 ere isolated, identified and genotyped using
deletion analysis,
Hain(R) Genotype MTBC, spoligotyping
74 ce of each region in gene expression through
deletion analysis has been hampered by the cellular requ
75 Deletion analysis has further shown that domains C-termi
76 5'- and 3'-
deletion analysis has identified the minimal region requ
77 teins is a key element of these circuits and
deletion analysis has implicated the conserved C-termina
78 Large-scale gene
deletion analysis has shown that over 80% of the ~6200 p
79 Deletion analysis,
heterologous promoter assays, and sit
80 Deletion analysis identified a 14-amino acid GRTH sequen
81 Promoter
deletion analysis identified a 280 bp segment of the BON
82 Deletion analysis identified a cyclic AMP receptor prote
83 Deletion analysis identified a dominant-inhibitory segme
84 Deletion analysis identified a Rhox5-responsive element
85 Deletion analysis identified a short segment of 10 amino
86 Deletion analysis identified a short stretch between nuc
87 Deletion analysis identified amino acids 730-758 of hTRP
88 Deletion analysis identified an octamer binding site tha
89 s were identified in the RNase-L 3'-UTR, and
deletion analysis identified positive and negative regul
90 The Nmp4/CIZ promoters are autoregulated and
deletion analysis identified regions that drive P(1) and
91 Sequential
deletion analysis identified that a 250 bp DNA fragment
92 Deletion analysis identified the differentiation-respons
93 Deletion analysis identified the region between residues
94 Deletion analysis identified the second domain (D2) with
95 Deletion analysis identified the Tie2 binding motif to b
96 Deletion analysis identified three elements correspondin
97 A previous
deletion analysis identified two ~100 bp fragments that
98 5'
deletion analysis implied a second upstream positive reg
99 Promoter
deletion analysis in 293T, BaF3, BaF3-p210, and K562 cel
100 Deletion analysis in HoxB BAC reporters reveals that the
101 Deletion analysis in Mt demonstrated that papA5 is requi
102 Systematic
deletion analysis in S. aureus RN6390 is consistent with
103 Deletion analysis in vivo, chemical cross-linking, and m
104 Finally, genetic
deletion analysis in yeast supported the role of a MEK-l
105 Functional
deletion analysis,
in situ mutagenesis and electromobili
106 spoligotyping and MIRU-VNTR, the addition of
deletion analysis increased the number of distinct patte
107 Biochemical mutagenesis and
deletion analysis indicate that this region mediates int
108 Domain
deletion analysis indicated that Bif-1 interacted with p
109 Deletion analysis indicated that independent domains of
110 Gene
deletion analysis indicated that only the cel3B gene pro
111 Deletion analysis indicated that promoter constructs lac
112 Deletion analysis indicated that the C-terminal coiled-c
113 Domain
deletion analysis indicated that the N-terminal domain o
114 Deletion analysis indicated that the noncoding exon 1 re
115 Deletion analysis indicated that the Rpt1 and Rpt2 motif
116 Furthermore, promoter
deletion analysis indicated that the sequence up to -543
117 Deletion analysis indicated that TPRs 2-6 of OGT interac
118 Deletion analysis indicates that both the N-terminal DED
119 prescreening followed by gene sequencing and
deletion analysis is feasible and may be desirable.
120 Deletion analysis is used to demonstrate important roles
121 Instead, by
deletion analysis it was discovered that a 46-bp region,
122 Promoter
deletion analysis lead to the identification of a potent
123 Deletion analysis localized the complementing activity (
124 Limited proteolysis, mass spectrometry and
deletion analysis localized the dRP lyase active site to
125 Progressive
deletion analysis located the SM22 responsive region of
126 Deletion analysis mapped p53-mediated repression to the
127 Deletion analysis mapped the centrosomal localization si
128 Functional
deletion analysis mapped the oxidative stress response e
129 Deletion analysis mapped the PIAS-interacting domain of
130 Deletion analysis mapped the SIRT1-binding domain of cor
131 Deletion analysis mapped this bend to amino acids 611 to
132 rected mutagenesis, DNase I footprinting and
deletion analysis of 5276 bp of 5' proximal -1b flanking
133 Through
deletion analysis of a -907/+70-bp 5' upstream region of
134 In frame
deletion analysis of Asc10 was used to identify a second
135 ith the corresponding MRE11 orthologues, and
deletion analysis of AtNBS1 defines a region towards the
136 By
deletion analysis of beta(2Deltag), we have now identifi
137 A
deletion analysis of Bir1 identified two regions importa
138 Deletion analysis of both proteins reveals discrete and
139 Prediction based
deletion analysis of both the promoter elements confirme
140 Deletion analysis of C/EBPalpha indicated that the C ter
141 Deletion analysis of CBS indicates that the C-terminal r
142 By
deletion analysis of cluster 19-1, the largest genomic d
143 Deletion analysis of CXCR4 promoter identified a seven-b
144 cids 1182-1186 and 69-73, respectively), and
deletion analysis of DRIP150 showed that regions contain
145 Extensive
deletion analysis of DRIP205 shows that multiple domains
146 Deletion analysis of EBNA3C identified a motif within am
147 A
deletion analysis of elements within the transcript reve
148 Deletion analysis of Gli1 indicated that multiple domain
149 Deletion analysis of green fluorescent protein (GFP)-ERK
150 Our 5'
deletion analysis of HOXA9 promoter shows that NF-kappaB
151 (b)
Deletion analysis of human osteocalcin and bone sialopro
152 Deletion analysis of Ins2 promoter identifies a sequence
153 Deletion analysis of IRAK4 indicates the essential struc
154 Clonal
deletion analysis of OPN promoter-luciferase constructs
155 Deletion analysis of p39 showed that muskelin binds to t
156 Deletion analysis of Pmga using single-copy Pmga-gusA re
157 Deletion analysis of POP2 revealed that the first alpha-
158 Deletion analysis of prolamine RNA sequences indicates t
159 Deletion analysis of Redbeta revealed that removal of ju
160 Based on a
deletion analysis of RID to determine the minimal functi
161 Nevertheless,
deletion analysis of Rns demonstrated that the first 60
162 By
deletion analysis of Spo7, we identified a hydrophobic L
163 Deletion analysis of SPT5 supports our biochemical data,
164 Deletion analysis of the 3' end of dctD identified the m
165 Progressive
deletion analysis of the 3,221-bp sequence revealed that
166 Deletion analysis of the 5' flanking, approximately 3.0
167 Deletion analysis of the 5' UTR(COX4) revealed the prese
168 Using DNA
deletion analysis of the 5'-flanking region of promoter
169 Deletion analysis of the alpha2-subunit reveals that the
170 We performed a detailed
deletion analysis of the C-terminal region of Qin (amino
171 Deletion analysis of the CDK4 promoter revealed a 231-bp
172 Scanning
deletion analysis of the cytoplasmic domain (FlhAc) demo
173 Nase I footprinting analysis, coupled with a
deletion analysis of the farAB promoter region, indicate
174 Deletion analysis of the GALGT2 promoter identified a 45
175 Deletion analysis of the human gene identified an intron
176 Deletion analysis of the key DNA binding elements in the
177 Deletion analysis of the long and conserved 3'-UTR has r
178 Deletion analysis of the m-Tnk1 promoter reveals the pre
179 utation testing by full sequencing and large
deletion analysis of the MLH1, MSH2, and MSH6 genes was
180 Deletion analysis of the Npr1 promoter and luciferase as
181 Detailed
deletion analysis of the NtPMT1a gene promoter showed th
182 Deletion analysis of the ORF1 IRES indicates that RNA st
183 Deletion analysis of the OsACBP2 5'-flanking region reve
184 Deletion analysis of the PKCdelta promoter mapped the Na
185 Using
deletion analysis of the previously defined INO promoter
186 Deletion analysis of the PrfA regulon and complementatio
187 Deletion analysis of the promoter region revealed that b
188 We performed promoter
deletion analysis of the rat CYP3A9 promoter and identif
189 Deletion analysis of the region indicates the presence o
190 Deletion analysis of the RPL27 promoter in transgenic pl
191 Deletion analysis of the sigmaNS RNA binding domain and
192 Deletion analysis of the sku genes indicated that all Ku
193 Deletion analysis of the tethering arms provides strong
194 Deletion analysis of the upstream sequence reveals that
195 Deletion analysis of the various functional domains of A
196 Deletion analysis of the vrg6 promoter identified the up
197 Deletion analysis of this intervening DNA segment has no
198 Deletion analysis of TLS-ERG in both mouse L-G myeloid p
199 Deletion analysis of transgenic embryos reduced this fra
200 Here we use a comparative
deletion analysis of two family members (ETR-3 and CELF4
201 In this work, we show through
deletion analysis of unmodified E. coli tRNA(Phe) that t
202 We performed a systematic
deletion analysis of YopM in Yersinia pseudotuberculosis
203 In spite of multiple episodes of set
deletion, analysis of the ratio of silent substitutions
204 suppressor mutation sites, binding data, and
deletion analysis onto the FliM(M) surface defines regio
205 23 in K-12, similar to results of the nested-
deletion analysis performed with EHEC.
206 Mutation and
deletion analysis permitted isolation and identification
207 t E47 is phosphorylated in vitro by p38, and
deletion analysis predicts that the critical amino acid(
208 Deletion analysis reveal a requirement for the N-termina
209 Subsequent mutation/
deletion analysis revealed a progesterone receptor half-
210 Deletion analysis revealed that a CsgB molecule missing
211 Promoter-
deletion analysis revealed that NF-kappaB was a necessar
212 Deletion analysis revealed that stimulation of endocytos
213 Genomic
deletion analysis revealed that strains of Mycobacterium
214 Deletion analysis revealed that the ability to activate
215 Deletion analysis revealed that the binding site is loca
216 Deletion analysis revealed that the core promoter activi
217 Deletion analysis revealed that the leader sequence or t
218 Deletion analysis revealed that the lethal effect of dep
219 Deletion analysis revealed that the luminal region of AT
220 Deletion analysis revealed that the N- and C-terminal mo
221 Deletion analysis revealed that the responsive region fo
222 Deletion analysis revealed that there are two potential
223 Deletion analysis revealed that three different ORF34 do
224 Deletion analysis revealed that transactivation involves
225 Electrophoretic mobility shift assay and
deletion analysis revealed three C/EBP binding sites tha
226 Deletion analysis revealed two classes of rhythmic V2a i
227 Deletion analysis revealed two DNA fragments from -2,725
228 Deletion analysis revealed two sites in the CTR of soybe
229 Deletion analysis reveals that relatively short regulato
230 Systematic
deletion analysis reveals that targeting activity is spr
231 Deletion analysis reveals that the EGF repeats and the t
232 Deletion analysis reveals that the Myo19 tail is necessa
233 A domain
deletion analysis reveals the molecular anatomy of Spt4/
234 nal studies in combination with domain-based
deletion analysis show that the cytosolic KinA forms a h
235 Upstream promoter
deletion analysis showed that a 200- and 412-bp region o
236 Deletion analysis showed that binding of Smad3 to ICD4 w
237 Deletion analysis showed that sequences located up to 75
238 Deletion analysis showed that the CTP transferase domain
239 Deletion analysis showed that the Dlx3 interacting domai
240 Deletion analysis showed that the last C-terminal 10 ami
241 Deletion analysis showed that the leucine zipper domain
242 Deletion analysis showed that the N-terminal 198 of 403
243 Deletion analysis showed that the only genes essential f
244 several Smad binding sites in the promoter;
deletion analysis showed that the region between -274 an
245 Domain
deletion analysis showed that the sterile alpha-motif of
246 Deletion analysis showed that the tRNA(Ser) TPsiC stem-l
247 Deletion analysis shows that that the first 125 amino ac
248 Deletion analysis shows that the caspase-like domain of
249 Domain
deletion analysis shows that the N-terminal DNA-binding
250 Deletion analysis shows that the N-terminal domain of Cl
251 highly homologous to those in K. pneumoniae
Deletion analysis shows that these cit genes are essenti
252 5'-
deletion analysis,
site-directed mutagenesis, and transa
253 5'-
deletion analysis,
site-directed mutagenesis, and transa
254 ll isolates were further characterized using
deletion analysis,
spoligotyping and MIRU-VNTR analysis.
255 Deletion analysis suggested that the N-terminal 67 and C
256 by E2F1 in transient transfections; further,
deletion analysis suggested that the region spanning the
257 Promoter
deletion analysis suggests that ligand-specific receptor
258 Deletion analysis suggests that the 2C central and C-ter
259 Although
deletion analysis supported the requirement of binding s
260 compare these hypotheses in the context of a
deletion analysis that further explores the biphasic dyn
261 This study describes a
deletion analysis that investigates which parts of YidC
262 We demonstrate by
deletion analysis that the extension contributes to DNA
263 It was previously shown by
deletion analysis that the N terminus of Phd was require
264 Through
deletion analysis,
the dimerization interface was mapped
265 By 5'
deletion analysis,
the element(s) responsible for this i
266 By
deletion analysis,
the region between nucleotides -296 t
267 ent lesions, we carried out a candidate gene
deletion analysis to identify genes whose mutation confe
268 sed a reverse yeast two-hybrid selection and
deletion analysis to identify NSI mutants that failed to
269 sed a reverse yeast two-hybrid selection and
deletion analysis to identify NSP mutants that were impa
270 In this study, we performed a
deletion analysis to identify the critical sequences in
271 the present study, we performed a systematic
deletion analysis to identify the signal required for tr
272 Finally, we used domain
deletion analysis to investigate the function of the C-t
273 Deletion analysis to map the IN-binding domain on RT rev
274 ed in SK-N-SH cells were shown by 5'- and 3'-
deletion analysis to play a crucial role in both cell li
275 We used a
deletion analysis to search for functional RNA domains w
276 Here, we use
deletion analysis to show that the LN domain region of n
277 was accomplished in Bacillus subtilis, where
deletion analysis uncovered two cis-elements within the
278 B12-responsive element has been localized by
deletion analysis using a reporter gene assay to a 70-bp
279 Therefore, we used VP40
deletion analysis,
virus-like particle-release assays, a
280 Deletion analysis was facilitated by an improved method
281 ectional promoter activity in a model plant,
deletion analysis was performed for seven rice promoters
282 ificial chromosome (BAC)-based reporter gene
deletion analysis was performed in transgenic mice.
283 Germline PTEN mutation/
deletion analysis was performed.
284 ation scanning, including promoter and large
deletion analysis,
was performed for all subjects.
285 Using
deletion analysis,
we also identify a novel N-terminal d
286 Through
deletion analysis,
we demonstrated that the alpha-(1-->2
287 Through mutation and
deletion analysis,
we have analyzed which specific domai
288 Through a
deletion analysis,
we have identified a cis-acting eleme
289 Using
deletion analysis,
we have identified the PELP1 COOH-ter
290 Through promoter
deletion analysis,
we have mapped a distal E-box element
291 Using promoter
deletion analysis,
we identified a region 1.1 kb upstrea
292 Combined with
deletion analysis,
we identified DNA elements as short a
293 By
deletion analysis,
we identified splicing silencers loca
294 By dissecting its promoters with progressive
deletion analysis,
we identified the sequence between -1
295 Through
deletion analysis,
we identify an active region responsi
296 Using
deletion analysis,
we show that part of this purine-rich
297 By
deletion analysis,
we show that the binding of soluble f
298 rapid amplification of cDNA ends (RACE) and
deletion analysis were used to identify the disA promote
299 all-angle x-ray scattering (SAXS) and domain
deletion analysis were used to obtain solution structura
300 f alanine-scanning, limited proteolysis, and
deletion analysis,
which show that the two reactions dep