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1 lial protein, was increased in parallel with delta power.
2      TNFR KO mice showed higher baseline SWS delta power.
3 cortex changes electroencephalographic (EEG) delta power.
4 nd that those reductions reduce cortical EEG delta power.
5 sals with corresponding changes in sigma and delta power.
6 ases nonrapid eye movement sleep amounts and delta power.
7 tes the neuronal "down state" and raises the delta power.
8 tified possible biomarkers such as Theta and Delta powers.
9 epileptic spikes in LFP and enhancing delta (delta) power.
10  leads showed increasing alpha (8-12 Hz) and delta power (0-4 Hz) and in the occipital leads delta po
11 gonersen treatment were electroencephalogram delta-power (2-4 Hz) and domains of the Bayley Scales of
12 an even greater postdeprivation reduction in delta power (60-75%) and a concomitant increase in wakef
13 leep depth (N3 > N2) and correlates with EEG delta power, a measure of slow wave activity.
14 response latencies and decreased mid-frontal delta power across all tasks.
15 cluding fragmented sleep and disturbances in delta power after sleep deprivation, all without observa
16 oss bilaterally) caused a 60-70% decrease in delta power and a 50-60% decrease in nonrapid-eye-moveme
17              5-MeO-DMT led to an increase of delta power and a decrease of theta power in the HP LFPs
18  circadian network, are associated with NREM delta power and AD tauopathy progression.
19 n C19 patients compared to control (enhanced delta power and attenuated alpha-beta power).
20         Esports athletes demonstrated higher delta power and decreased alpha power at Fpz and Cz comp
21 tween conditions, was predicted by increased delta power and decreased sigma power in RS compared wit
22     We test the hypothesis that reduced NREM delta power and increased tauopathy are associated with
23  increasing sleep electroencephalogram (EEG) delta power and is decreased with increasing sleep EEG b
24 ep (non-rapid eye-movement sleep with higher delta power and lower body temperature).
25              Optogenetic treatment increased delta power and reduced sleep fragmentation in APP mice.
26 nd theta synchrony were reduced in patients; delta power and synchrony better distinguished between g
27  manual restraint can increase sleep and EEG delta power and that increases in sleep may persist acro
28 onist, reduces cortical electroencephalogram delta power and transiently inhibits spontaneous seizure
29  Lesions resulted in increases in slow-wave (delta) power and decreases in high-frequency (beta 2) po
30 ally significant improvements on deep sleep (delta power) and sleep consolidation at doses as low as
31 ortical ACh release, behavioral arousal, EEG delta power, and sleep.
32 eprivation there is a diminished increase in delta power, and the mice catch up little on lost sleep.
33 ntensity [i.e., nonrapid eye movement (NREM) delta power] and increased rapid eye movement sleep time
34 of nNOS, they are unable to up-regulate NREM delta power appropriately.
35                        The increase of NREMS delta power as a function of previous wake duration vari
36 ring REO, soccer players displayed increased delta power at Fpz and midline frontal (Fz) and reduced
37          After the intervention, the average delta power at the frontal region increased in the activ
38                            The difference of delta power between asynchronous and synchronous conditi
39 sk, individuals with aphantasia showed lower delta power-brain activity linked to regulating sensory
40 at selective REM sleep deprivation increased delta power but decreased theta power during the residua
41 ly compensated for the SD-induced deficit in delta power, but the Per3(4/4) and wild-type mice did no
42 increased baseline NREM sleep by 4% and NREM delta power by 15%, and decreased REM sleep by 10%.
43 el cortex during REM concomitant with strong delta power, challenging the view of a wakefulness-like
44 sitions between responsive states, while the delta power/connectivity changes were consistent with th
45                                          GPe delta power correlated with OCD symptom severity, establ
46                                              Delta power density (PD) was unchanged between age 9 and
47 delta and theta power, and an increase in LC delta power during correct responses in the rCPT.
48 els of non-REM sleep (stage N3), and reduced delta power during daytime.
49 nd the hypothalamus positively regulates EEG delta power during non-rapid eye movement sleep (NREMS)
50                                              Delta power during NREM sleep was increased in both grou
51 REM) sleep time, NREM bout duration, and EEG delta power during NREM sleep, an index of preexisting h
52 Mice receiving conditioning stimuli had more delta power during NREM sleep, whereas mice receiving fe
53 h voltage NREM sleep, sleep bout length, and delta power during NREM sleep.
54  M1, mNE and mFS significantly increased EEG delta power during NREM, but M2-3, NE and FS alone did n
55                      The initial increase in delta power during NREMS after sleep deprivation was gre
56 ereas the initial LPS-induced suppression of delta power during NREMS was greater in PKR(-/-) mice.
57                                              Delta power during NREMS, a measure of sleep depth, was
58 e thus propose that slow waves, reflected in delta power during RS, act to restore brain function, th
59 phy (EEG) theta power during wakefulness and delta power during sleep, were greater in the Per3(5/5)
60 ollowed by increased sleep and increased EEG delta power during sleep.
61 s in increased electroencephalographic (EEG) delta power during subsequent non-rapid eye movement sle
62 frequency power in the electroencephalogram (delta power) during non-rapid eye movement sleep reflect
63 vely in TDW rather than all waking, predicts delta power dynamics both in Hcrt(ko/ko) and WT mouse ba
64  causes AS-like increases in neocortical EEG delta power, enhances seizure susceptibility, and leads
65 idation, (ii) persistent enhancement in NREM delta power especially in the frontal and parietal regio
66 m (EEG) correlate of sleep pressure, non-REM delta power, failed to increase.
67 ta power (0-4 Hz) and in the occipital leads delta power greater than alpha power.
68     Compensatory elevation in NREM sleep EEG delta power has been typically observed following prolon
69 al and auditory prosodies leads to increased delta power in left motor cortex and correlates with per
70               Further analysis revealed that delta power in long NREM bouts prior to NREM-REM transit
71  rapid eye movement sleep and a reduction of delta power in non-rapid eye movement sleep.
72 ed for some lower-frequency metrics, such as delta power in parietal regions.
73  volunteers; (v) a decrease in the sleep EEG delta power in patients.
74 Effective forms of ECT resulted in increased delta power in prefrontal regions, and this change was a
75 , shortened sleep latency, and increased EEG delta power in rats.
76 TDW maintenance in baseline wake and blunted delta power in SWS, reproducing, respectively, narcoleps
77 ly 43% during the dark period, and increased delta power in the EEG during NREM sleep by approximatel
78 ed audiovisual asynchrony, and (2) increased delta power in the left motor cortex in response to audi
79 similar extent (>99%), and, as expected, the delta power increase during recovery sleep was quantitat
80          Power spectral analysis showed that delta power increased from responsiveness to unresponsiv
81 ep depth (lower nonrapid eye movement [NREM] delta power), increased NREM-to-REM transitions, hindere
82 dullary lamina, induces significant cortical delta power increases greater than stimulation within hi
83 synaptic strength and electroencephalography delta power indicating macro-level sleep pressure by dev
84 restriction (CSR) studies suggests that NREM delta power is not progressively increased despite of ac
85 peated sessions whereas the magnitude of EEG delta power may vary across sessions.
86 tive behavioral assays as well as normalized delta power measured by electroencephalogram was observe
87                                      Greater delta power, more negative 1/f slope, and lower power ra
88 w-frequency activity as reflected by greater delta power, more negative 1/f slope, and lower theta/de
89 7; p < 0.001) and total electroencephalogram delta power (r = 0.79; p < 0.001) but not to rapid-eye-m
90 rmates, spontaneous waking fails to induce a delta power reflecting prior waking duration.
91 nimals failed to exhibit a compensatory NREM delta power response during the 4-h sleep opportunities
92                               This divergent delta power response is consistent with the known cortic
93 heep did not show the increase in NREM sleep delta power seen in unaffected sheep.
94 mals showed a massive increase in NREM sleep Delta power, similarly to that occurring in natural torp
95                      By analyzing changes in delta power, sleep homeostasis (also referred to as "sle
96 ity of pink noise and other sounds to elicit delta power, slow oscillatory power, and N3 sleep.
97  hM4Di receptors significantly increased EEG delta power spectrum and slightly decreased wakefulness.
98 ignificantly and lastingly decreased the EEG delta power spectrum, produced low-delta non-rapid eye m
99 nt as WT mice, with similar increases in EEG delta power, suggesting that their homeostatic control o
100 nt to SD was proportional to the increase in delta power that occurs in inbred strains: the strain th
101                 Although both drugs affected delta power, these changes did not correlate with increa
102 tatic sleep drive into up-regulation of NREM delta power through an NO-dependent mechanism.
103 t paired mice exhibited significantly higher delta power throughout the dark period.
104                       REM sleep frontal high delta power was a negative correlate of intelligence.
105                                     Notably, delta power was also associated with imagery vividness (
106 dose of mirtazapine (p = 0.42), but NREM EEG delta power was increased by more than 30% at all doses
107 9S1/SvImJ) F2 mouse population in which NREM delta power was measured (N = 98).
108                           However, Theta and Delta power was slightly higher with the dry electrodes
109 tion to wild-type mice there is a rebound in delta power when they enter normal NREM sleep, reminisce
110 show that about 73% and 66% of the Theta and Delta powers which are selected as biomarkers are increa
111 le in modulating arousal states and NREM EEG delta power, which is widely recognized as a marker for
112                                 Intracranial delta power widely increased across five lobes during SW

 
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