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1 lial protein, was increased in parallel with delta power.
2 TNFR KO mice showed higher baseline SWS delta power.
3 cortex changes electroencephalographic (EEG) delta power.
4 nd that those reductions reduce cortical EEG delta power.
5 sals with corresponding changes in sigma and delta power.
6 ases nonrapid eye movement sleep amounts and delta power.
7 tes the neuronal "down state" and raises the delta power.
8 tified possible biomarkers such as Theta and Delta powers.
9 epileptic spikes in LFP and enhancing delta (delta) power.
10 leads showed increasing alpha (8-12 Hz) and delta power (0-4 Hz) and in the occipital leads delta po
11 gonersen treatment were electroencephalogram delta-power (2-4 Hz) and domains of the Bayley Scales of
12 an even greater postdeprivation reduction in delta power (60-75%) and a concomitant increase in wakef
15 cluding fragmented sleep and disturbances in delta power after sleep deprivation, all without observa
16 oss bilaterally) caused a 60-70% decrease in delta power and a 50-60% decrease in nonrapid-eye-moveme
21 tween conditions, was predicted by increased delta power and decreased sigma power in RS compared wit
23 increasing sleep electroencephalogram (EEG) delta power and is decreased with increasing sleep EEG b
26 nd theta synchrony were reduced in patients; delta power and synchrony better distinguished between g
27 manual restraint can increase sleep and EEG delta power and that increases in sleep may persist acro
28 onist, reduces cortical electroencephalogram delta power and transiently inhibits spontaneous seizure
29 Lesions resulted in increases in slow-wave (delta) power and decreases in high-frequency (beta 2) po
30 ally significant improvements on deep sleep (delta power) and sleep consolidation at doses as low as
32 eprivation there is a diminished increase in delta power, and the mice catch up little on lost sleep.
33 ntensity [i.e., nonrapid eye movement (NREM) delta power] and increased rapid eye movement sleep time
36 ring REO, soccer players displayed increased delta power at Fpz and midline frontal (Fz) and reduced
39 sk, individuals with aphantasia showed lower delta power-brain activity linked to regulating sensory
40 at selective REM sleep deprivation increased delta power but decreased theta power during the residua
41 ly compensated for the SD-induced deficit in delta power, but the Per3(4/4) and wild-type mice did no
43 el cortex during REM concomitant with strong delta power, challenging the view of a wakefulness-like
44 sitions between responsive states, while the delta power/connectivity changes were consistent with th
49 nd the hypothalamus positively regulates EEG delta power during non-rapid eye movement sleep (NREMS)
51 REM) sleep time, NREM bout duration, and EEG delta power during NREM sleep, an index of preexisting h
52 Mice receiving conditioning stimuli had more delta power during NREM sleep, whereas mice receiving fe
54 M1, mNE and mFS significantly increased EEG delta power during NREM, but M2-3, NE and FS alone did n
56 ereas the initial LPS-induced suppression of delta power during NREMS was greater in PKR(-/-) mice.
58 e thus propose that slow waves, reflected in delta power during RS, act to restore brain function, th
59 phy (EEG) theta power during wakefulness and delta power during sleep, were greater in the Per3(5/5)
61 s in increased electroencephalographic (EEG) delta power during subsequent non-rapid eye movement sle
62 frequency power in the electroencephalogram (delta power) during non-rapid eye movement sleep reflect
63 vely in TDW rather than all waking, predicts delta power dynamics both in Hcrt(ko/ko) and WT mouse ba
64 causes AS-like increases in neocortical EEG delta power, enhances seizure susceptibility, and leads
65 idation, (ii) persistent enhancement in NREM delta power especially in the frontal and parietal regio
68 Compensatory elevation in NREM sleep EEG delta power has been typically observed following prolon
69 al and auditory prosodies leads to increased delta power in left motor cortex and correlates with per
74 Effective forms of ECT resulted in increased delta power in prefrontal regions, and this change was a
76 TDW maintenance in baseline wake and blunted delta power in SWS, reproducing, respectively, narcoleps
77 ly 43% during the dark period, and increased delta power in the EEG during NREM sleep by approximatel
78 ed audiovisual asynchrony, and (2) increased delta power in the left motor cortex in response to audi
79 similar extent (>99%), and, as expected, the delta power increase during recovery sleep was quantitat
81 ep depth (lower nonrapid eye movement [NREM] delta power), increased NREM-to-REM transitions, hindere
82 dullary lamina, induces significant cortical delta power increases greater than stimulation within hi
83 synaptic strength and electroencephalography delta power indicating macro-level sleep pressure by dev
84 restriction (CSR) studies suggests that NREM delta power is not progressively increased despite of ac
86 tive behavioral assays as well as normalized delta power measured by electroencephalogram was observe
88 w-frequency activity as reflected by greater delta power, more negative 1/f slope, and lower theta/de
89 7; p < 0.001) and total electroencephalogram delta power (r = 0.79; p < 0.001) but not to rapid-eye-m
91 nimals failed to exhibit a compensatory NREM delta power response during the 4-h sleep opportunities
94 mals showed a massive increase in NREM sleep Delta power, similarly to that occurring in natural torp
97 hM4Di receptors significantly increased EEG delta power spectrum and slightly decreased wakefulness.
98 ignificantly and lastingly decreased the EEG delta power spectrum, produced low-delta non-rapid eye m
99 nt as WT mice, with similar increases in EEG delta power, suggesting that their homeostatic control o
100 nt to SD was proportional to the increase in delta power that occurs in inbred strains: the strain th
106 dose of mirtazapine (p = 0.42), but NREM EEG delta power was increased by more than 30% at all doses
109 tion to wild-type mice there is a rebound in delta power when they enter normal NREM sleep, reminisce
110 show that about 73% and 66% of the Theta and Delta powers which are selected as biomarkers are increa
111 le in modulating arousal states and NREM EEG delta power, which is widely recognized as a marker for