ライフサイエンスコーパス: dendrite

1 uted to increased sodium ion mobility in the dendrite.

2 elf-organisation in functionally specialised dendrites.

3 terneurons are a source of these presynaptic dendrites.

4 les dense Li plating and avoids porosity and dendrites.

5 xons innervating their own soma and proximal dendrites.

6 eferentially established on basal vs. apical dendrites.

7 shows the PEC layer successfully intercepts dendrites.

8 red by their formation and growth of lithium dendrites.

9 kt, regulates APP transport in axons but not dendrites.

10 modulates HCN channels in CA1 versus L5 PFC dendrites.

11 through specialized synapses on their distal dendrites.

12 localize microtubule nucleation machinery in dendrites.

13 dendodendritic inputs on their distal apical dendrites.

14 ape of the somata and the disposition of the dendrites.

15 the extension and complexity of their apical dendrites.

16 f spines on apical, but not basal, secondary dendrites.

17 selective transport of proteins to axons and dendrites.

18 ent-producing cells of the skin that possess dendrites.

19 alized Ca2+ transients within the melanocyte dendrites.

20 capacity using a Na-K liquid anode to avoid dendrites.

21 s ends and immobilizes microtubule arrays in dendrites.

22 t fiber activation coupled to MGF collateral dendrites.

23 t has been found to differ between axons and dendrites.

24 y requires a tight control of mRNA levels in dendrites.

25 alization and development of multiple apical dendrites.

26 rotein synthesis in neuronal cell bodies and dendrites.

27 citability is correlated with shorter apical dendrites.

28 ) cause structural remodeling of hippocampal dendrites.

29 Ror was required to position dsh and Axin in dendrites.

30 ei of the BNC and had aspiny or spine-sparse dendrites.

31 oth synchronously and asynchronously onto MC dendrites.

32 protein kinase IIalpha (CaMKIIalpha) mRNA in dendrites.

33 esults in early onset of SEI instability and dendrites.

34 modulation of the cytoskeleton morphology in dendrites, a mechanism associated to neuronal plasticity

35 physical location of synaptic input onto its dendrites, a relationship called the retinotopic map.

36 dendritic branches, formed a second primary dendrite, acquired more mushroom spines, and had enlarge

37 d electrolyte interphase (SEI) preventing Zn dendrite and further suppressing water decomposition.

38 ding glia, but the molecules that coordinate dendrite and glial morphogenesis are mostly unknown.

39 g dual whole-cell recordings from the apical dendrite and soma of layer 5 (L5) pyramidal neurons in t

40 21 (P21), but fail to stabilize, leading to dendrite and synapse loss by P42.

41 indicate that Tiam1 promotes DG granule cell dendrite and synapse stabilization late in development.

42 d electrolyte interphase (SEI) preventing Zn dendrite and water decomposition.

43 ells, preferentially targeting distal apical dendrites and apical dendritic tufts of pyramidal neuron

44 ging to show that APL inhibits Kenyon cells' dendrites and axons, and that both activity in APL and A

45 d by both focal stimulation near GC proximal dendrites and by activating sensory inputs in the glomer

46 GFD) is required for structural integrity of dendrites and cognitive abilities.

47 nucleus, and these calcium responses invade dendrites and dendritic spines by active backpropagation

48 Here, we quantified dendrites and dendritic spines of supragranular pyramida

49 n the formation and long-term maintenance of dendrites and dendritic spines.

50 otes coordination of growth among individual dendrites and engages the autophagy mechanism to sculpt

51 Previous work in this system examined dendrites and glia that develop within epithelia, simila

52 s the structural remodeling of spines on ABN dendrites and impairs memory consolidation.

53 rformance by suppressing the formation of Li dendrites and inactive Li and presenting higher average

54 terrogate the interaction between melanocyte dendrites and keratinocytes, we show that signals from n

55 Our recordings from synapses, dendrites and large neuronal populations in behaving mic

56 ables fast Li-ion diffusion, and inhibits Li dendrites and Li pulverization.

57 ic finding that axons run in parallel to the dendrites and make multiple synaptic contacts support su

58 cellular skin components such as melanocytic dendrites and melanin, FLAME is ready to be translated i

59 tanding of Li electrocrystallization without dendrites and provides guidance for practical applicatio

60 ssing (SST) interneurons selectively inhibit dendrites and regulate synaptic inputs, yet their respon

61 ural network fully integrated with synapses, dendrites and soma, implemented using scalable memristor

62 The stabilization of dendrites and spines during neuronal maturation is essen

63 forms (Kv7.2, Kv7.3, and Kv7.5) on layer III dendrites and spines, similar to M1Rs.

64 n the expression of cytoskeleton proteins in dendrites and spines.

65 e control of pyramidal cells at their apical dendrites and support differential computational propert

66 Notably, DG granule cell dendrites and synapses develop normally in Tiam1 KO mice

67 r by coordinating the dynamics of individual dendrites and that the autophagy mechanism may be levera

68 The growth of sodium dendrites and the associated solid electrolyte interface

69 Both alpha-Al dendrites and the silicon particle sizes were significan

70 ncoding in simultaneously imaged apical tuft dendrites and their respective cell bodies in retrosplen

71 synapses onto cerebellar Purkinje cell (PC) dendrites and trigger distinctive responses known as com

72 g large volume fractions of primary alpha-Al dendrites and ultrafine Al-Si eutectic of lamellar morph

73 and the relative contributions from somata, dendrites, and axons are often unknown.

74 ing charging, they rapidly develop porosity, dendrites, and dead Li that cause poor performance and,

75 echanical tension along the length of axons, dendrites, and glial processes has been proposed as a ma

76 ering the current understanding of potassium dendrites, and highlighting the deep-eutectic K-Na alloy

77 rk shows how glial factors can help to shape dendrites, and identifies a novel molecular mechanism fo

78 th: these neurons had larger nuclei, thicker dendrites, and more dendritic filopodia than all other g

79 macrine cells with long, relatively straight dendrites, and sometimes also axons, that run in a singl

80 h proximal segments on both apical and basal dendrites, and spine density was increased in secondary

81 ing mechanism is key to define and diversify dendrite arbor compartmentalization.

82 Dendrite arbor pattern determines the functional charact

83 aling pathway promotes cortical neuron basal dendrite arborization but also repels axons.

84 ndritic territories, whereas one expands its dendrite arbors to reach new partners.

85 lc7a5 knockdown on mTOR pathway activity and dendrite arbors.

86 Neuronal dendrites are characterized by an anti-parallel microtub

87 oscopy, we found that apical and basolateral dendrites are coordinately sculpted during development.

88 These studies have shown that human dendrites are electrically excitable, exhibiting backpro

89 Synaptic inputs on dendrites are spatially clustered by stimulus feature, b

90 urate neuronal models.SIGNIFICANCE STATEMENT Dendrites are the sites of the synaptic connections amon

91 ropic glutamate receptors of ON bipolar-cell dendrites, are both present in lamprey.

92 the KO were localized more proximally on PC dendrites, as indicated by VGLUT2(+) immunoreactive punc

93 d isolating large segments of their proximal dendrites, as revealed by three-dimensional high-resolut

94 ine populations on basal and proximal apical dendrites, as well as firing rates and ocular dominance,

95 normal bony matrix, but they completely lack dendrites, as well as the characteristic lacuno-canalicu

96 oming action potentials would depolarize the dendrite at multiple sites within a brief time interval.

97 Each cell, however, contributed less dendrites at each depth within the plexus, intermingling

98 months, but cholinergic interneurons showed dendrite attenuation by 6 months.

99 ithin two hours of induction, withdrew their dendrites between twelve hours and one day, appeared ame

100 At dendrite branch points, Axin and dsh colocalized with ea

101 Nucleation sites concentrate at dendrite branch points, but how they localize is not kno

102 In the nervous system, dendrites, branches of neurons that transmit signals bet

103 Whereas the delta:Pdlim5 complex enhances dendrite branching at the expense of elongation, the del

104 PH] domains 1) has been suggested to control dendrite branching by regulating the small GTPase ARF6.

105 arbor, little is known of those that govern dendrite branching versus extension.

106 come from spatially offset ON and OFF layer dendrites, but instead arises from a network of electric

107 l (BC) axons and retinal ganglion cell (RGC) dendrites, but makes the majority of its synapses with t

108 entially innervated either L2 or L3/5 apical dendrites, but not both.

109 nd LiF with high interface energy suppresses dendrites by enhancing the nucleation energy and suppres

110 Here, we report that K dendrites can be healed in situ in a K-metal battery.

111 mplification of organized synaptic inputs in dendrites can endow individual neurons with the ability

112 We propose that in dendrites, canonical Wnt signaling proteins are housed o

113 LC6 cells in Drosophila, a population whose dendrites collectively cover the visual field, but whose

114 indings provide insights into how melanocyte dendrites communicate with neighboring cells and offer a

115 buse tests of Li-metal pouch cells result in dendrites completely penetrating the single-layer separa

116 w rather multipolar morphology lacking thick dendrite contacting central canal lumen.

117 tips of the processes (plus-end-out), while dendrites contain microtubules with a minus-end-out orie

118 tals in the eutectic region and the aluminum dendrites contained a significant number of twins which

119 Microtubule polarity in axons and dendrites defines the direction of intracellular transpo

120 turning towards the axon and exclusion from dendrites depend on Kinesin-2, a plus-end-associated mot

121 We show that these dendrites develop by retrograde extension, in which the

122 Dendrites develop elaborate morphologies in concert with

123 The mixed oriented microtubules promote dendrite development and facilitate polarized cargo traf

124 Our data suggest that these complexes affect dendrite development by differentially regulating the sm

125 Using forward genetic screens, we find that dendrite development requires the adhesion protein SAX-7

126 naptotrophic hypothesis at initial stages of dendrite development, suggest a second mode in which cum

127 not required for axon regeneration or normal dendrite development, suggesting a specific role in dend

128 nizing dendritic microtubules and to control dendrite development.

129 balancing the branching versus extension of dendrites during neural development.

130 apical dendrite without altering basolateral dendrite dynamics.

131 Rac1, and that Rac1 activity is required for dendrite elaboration but not axon growth cone collapse.

132 in neurodevelopment, from axon repulsion to dendrite elaboration.

133 y retrograde extension, in which the nascent dendrite endings anchor to the presumptive nose and then

134 cytoplasmic protein GRDN-1/CCDC88C to anchor dendrite endings at the nose.

135 Microglia dendrites extended between photoreceptors within two hou

136 N-1 acts in glia to non-autonomously promote dendrite extension.

137 that perturbations on the sides of cells (or dendrites) facilitate crystals to change growth directio

138 ndary branches and distal segments on apical dendrites following sleep deprivation.

139 In neurons, dendrites form the major sites of information receipt an

140 lication of a Zn anode is hindered by severe dendrite formation and side reactions.

141 Na dendrite formation mainly originates from the uncontroll

142 s polysulfide the shuttle effect and lithium dendrite formation of lithium-sulfur (Li-S) batteries.

143 ntroduction of Li(x) LM(y) not only prevents dendrite formation, but also eliminates the use of coppe

144 ches reported to date often seek to suppress dendrites formation at the expense of energy density.

145 The Li deposition is morphologically dendrite-free and close to theoretical density when cycl

146 t resolves the above conflict and achieves a dendrite-free and long-term reversible Li metal anode is

147 ach and design principle for next-generation dendrite-free batteries.

148 ) N-LiF composite is used to validate the Li-dendrite-free design criteria, where the highly ionic co

149 Dendrite-free electrodeposition of lithium metal is nece

150 d passivation interphases and contributes to dendrite-free Li deposition and reversible cathode elect

151 s-developed potassium metal anodes exhibit a dendrite-free morphology with high Coulombic efficiency

152 ed to effectively regulate Na nucleation for dendrite-free Na deposition.

153 Dendrite-free plating/stripping is achieved through impr

154 The dendrite-free sodium-potassium (Na-K) liquid alloy compo

155 tion of Li dendrites is investigated, and Li-dendrite-free SSE criteria are reported.

156 ingle glial cell at the nose, reminiscent of dendrite-glia contacts in the mammalian brain.

157 Preferential dendrite growth allows for heightened animal responses t

158 tration sulfolane electrolyte to suppress Li dendrite growth and achieve a high Coulombic efficiency

159 c structure is found to effectively suppress dendrite growth and side reactions of the Zn anode.

160 Our study shows how dendrite growth balances structure-function requirements

161 A generative model of dendrite growth based on competitive synaptogenesis larg

162 lithium-ion diffusion and suppresses lithium dendrite growth but also brings a synergistic effect of

163 d identifies a novel molecular mechanism for dendrite growth by retrograde extension.

164 Lithium dendrite growth during repeated charge and discharge cyc

165 te Li batteries, one approach to suppress Li dendrite growth has been the use of mechanically stiff s

166 Here, we suppress water reduction and Zn dendrite growth in dilute aqueous electrolyte by adding

167 However, Li dendrite growth in solid-state electrolytes (SSEs) still

168 Herein, the Li dendrite growth of metallic lithium anode is suppressed

169 However, uncontrollable dendrite growth on the potassium metal anode has restrai

170 a liquid crystalline electrolyte to suppress dendrite growth with a lithium metal anode.

171 nly limited success due to uncontrollable Li dendrite growth(3-7).

172 fety concern (short-circuiting related to Li dendrite growth).

173 umulative synapse formation inhibits further dendrite growth, and highlight the importance of competi

174 shuttling effects, mismatched interfaces, Li dendrite growth, and the gap between fundamental researc

175 n anodes owing to water decomposition and Zn dendrite growth.

176 is limited by the water decomposition and Zn dendrite growth.

177 anodes suffer from a challenging problem of dendrite growth.

178 is formed on the Li surface, suppressing Li dendrite growth.

179 Neuronal dendrites have specialized actin-rich structures called

180 a potassium cobalt oxide cathode to achieve dendrite healing in a practical full-cell device.

181 oms in K relative to Li metal, which enables dendrite healing to take place at an order-of-magnitude

182 Any plus ends that reach a dendrite, however, do not readily enter, maintaining min

183 l superior olive, with their large bilateral dendrites, however, can detect coincidence of binaural a

184 owever, the uncontrollable growth of lithium dendrites impedes the service of Li anodes in high energ

185 al axonal segment is located distal to where dendrites impinge on axons and is the likely site of AP

186 However, whether dendrites in behaving animals perform independent local

187 tratified, axon-bearing GABAergic cell, with dendrites in both ON and OFF synaptic layers, but with a

188 mammalian cells to amyloid-beta plaques and dendrites in brain tissues and elastic fibers in develop

189 uced microtubule-associated protein 2 (MAP2) dendrites in human neurons.

190 nputs, suggesting a dominant role for apical dendrites in integrating feedback in visual information

191 influences the development of both axons and dendrites in invertebrates.

192 ules are also necessary for the formation of dendrites in melanocytes, and Ilk inactivation reduces m

193 d by overlapping fluorescence from axons and dendrites in surrounding neuropil.

194 ecreased number of LC with fewer and stunted dendrites in the epidermis as well as a decreased number

195 Both antibodies labeled RBC dendrites in the outer plexiform layer and axon terminal

196 s onto GC distal dendrites via their lateral dendrites in the superficial external plexiform layer (E

197 ASICs are localized to cell bodies and dendrites, including the postsynaptic density, and withi

198 t (SAC) proteins in Drosophila neurons after dendrite injury.

199 Furthermore, apical dendrite inputs have larger receptive fields and longer

200 lds and longer response latencies than basal dendrite inputs, suggesting a dominant role for apical d

201 To understand how the dendrite integrates excitatory inputs as a whole, we com

202 rtebrate neurons are typically unipolar with dendrites integrating directly into the axon.

203 -controlled, self-heating at the electrolyte/dendrite interface, which causes migration of surface at

204 When the dendrite is hyperpolarized only, however, mGluR1s boost

205 Consequently, the growth of Li dendrites is eliminated and the direct reduction of the

206 erein, the mechanism for the formation of Li dendrites is investigated, and Li-dendrite-free SSE crit

207 th of planar Li layers, instead of random Li dendrites, is achieved on self-assembled reduced graphen

208 nt and eliminate the growth of wispy lithium dendrites, leading to long cycle life and high Coulombic

209 dulation of dendritic excitability by apical dendrite length and show that the operational repertoire

210 Slc7a5 knockdown effected late phases of GC dendrite maturation and survival.

211 netic epistasis analyses indicate that these dendrite morphogenesis defects result from a deficit in

212 d highlight the importance of competition in dendrite morphogenesis.

213 or few transcription factors often predicts dendrite morphologies and axonal projections to specific

214 a series of electron micrographs of neuronal dendrite morphology explored at three different length s

215 that single afferent fibers follow a single dendrite mostly up to the soma and contact it at multipl

216 neuroinflammation and loss of Purkinje cell dendrites observed within the mutant mice were alleviate

217 +) spikes as prominently found in the apical dendrite of S1 (somatosensory cortex) pyramidal neurons.

218 density of obligatory GluN1-NMDA subunits in dendrites of all sizes and (2) a shift from cytoplasmic

219 led that spine volume was greater in primary dendrites of apical and basal segments, along with proxi

220 atory synaptic input patterns in perisomatic dendrites of CA1 pyramidal cells.

221 various spatial distributions in perisomatic dendrites of CA1 pyramidal neurons in slices from adult

222 regulating glutamatergic synapse assembly on dendrites of central neurons.

223 estigated the synaptic innervation of apical dendrites of cortical pyramidal cells in a region betwee

224 etoes' excitation at the level of individual dendrites of direction-selective ganglion cells.

225 They almost exclusively innervate dendrites of excitatory cells, preferentially targeting

226 otectal synaptic targets are the presynaptic dendrites of GABAergic interneurons, and GAD67-GFP inter

227 The first patch-clamp recordings from the dendrites of human neocortical neurons have recently bee

228 elective and color-selective inputs in basal dendrites of individual V1 neurons.

229 s, and map the excitatory synaptic inputs on dendrites of individual V1 superficial layer neurons wit

230 pecific to L2/3 neurons and absent on apical dendrites of L5 neurons, and is dependent on expression

231 NDNF+ cells also inhibit the apical dendrites of L5 pyramidal tract (PT) cells to suppress a

232 Signal propagation in the dendrites of many neurons, including cortical pyramidal

233 molecules, which surround the cell body and dendrites of many types of neuron and regulate neural pl

234 n cells can form electrical synapses between dendrites of neighboring cells in support of lateral inf

235 pines are tiny membranous protrusions on the dendrites of neurons.

236 that NPY neurons are stellate cells, and the dendrites of NPY neurons in the tonotopically organized

237 sphorylation of FAK (p-FAK) and p-cofilin in dendrites of reinstated animals compared with extinguish

238 on profiles: in contrast to the other types, dendrites of transient Off alpha cells were spatially in

239 mata and long, thick dendrites; their distal dendrites often branched extensively and had long append

240 However, formation of dendrites on such K-metal surfaces is inevitable, which

241 ic RNA interference (RNAi) was performed and dendrites or axons were removed using laser microsurgery

242 neighboring non-neural tissues, resulting in dendrite overgrowth.

243 on along the SEI/Li interface and prevent Li dendrite penetration into the SEI.

244 ined responses, whereas the less susceptible dendrites predominantly respond transiently.

245 arbor growth: Under nutrient stress, sensory dendrites preferentially grow as compared to neighboring

246 The integration of synaptic inputs onto dendrites provides the basis for neuronal computation.

247 y, cognition, and local protein synthesis in dendrites, providing fundamental insight into the neuron

248 Yet little is known about how melanocyte dendrites receive and process information from neighbori

249 egated streams of sensory input: the lateral dendrite receives mechanosensory input while the ventral

250 eives mechanosensory input while the ventral dendrite receives visual input.

251 plasmalemmal distribution of GluN1 in large dendrites receiving mainly inhibitory-type synapses from

252 pendence of the firing rate on the number of dendrites receiving synaptic drive; a significant effect

253 We conclude that Ror promotes dendrite regeneration as part of a Wnt signaling pathway

254 Reduction of gamma-tubulin also rescued dendrite regeneration in backgrounds containing kinetoch

255 In addition, dendrite regeneration was sensitive to partial reduction

256 test whether these proteins function during dendrite regeneration, postmitotic RNA interference (RNA

257 Using a Drosophila model of dendrite regeneration, we performed a candidate screen o

258 sh), Axin, and gilgamesh (gish) also reduced dendrite regeneration.

259 e development, suggesting a specific role in dendrite regeneration.

260 icrotubule nucleation at baseline and during dendrite regeneration.

261 the involvement of Wnt signaling proteins in dendrite regeneration.

262 ng APP transport to the synapse in axons and dendrites remain to be identified.

263 th five crucial ionic currents in the apical dendrites reproduces all features of these neurons.

264 ficient mice shows that they are involved in dendrite self-avoidance, synapse development, dendritic

265 Here, to link dendrite shape with its proprioceptive role, we performe

266 cell viability, and prevented MPP(+)-induced dendrite shrinkage in primary neurons.

267 We use dendrite-soma dual patch-clamp recordings to show that t

268 In our model, a basal-dendrites/somatic compartment included fast-inactivating

269 However, Li dendrites still grow through them(10,11).

270 Moreover, if each axon and dendrite strive to shorten while preserving connectivity

271 rfacial resistance, neglecting the intrinsic dendrite-suppression capability of SSEs.

272 To achieve a high dendrite-suppression capability, SSEs should be thermody

273 al diversity comprising subsets of all known dendrite targeting CCK(+) interneurons in addition to th

274 neurons, approximately one-third of putative dendrite-targeting (somatostatin-expressing) interneuron

275 creased NMDAR currents and reduced firing of dendrite-targeting somatostatin-expressing (SST) GABAerg

276 erized by a more rounded cell body and fewer dendrites than wild-type cells.

277 acetylcholine, and GABA receptors along the dendrite that matched the previously reported EM-reconst

278 rm cells had small somata and numerous short dendrites that formed a dense dendritic arborization; th

279 elopment of the MT cytoskeleton in axons and dendrites: the cross-linking level of MTs by motors and

280 ay from the cell body, whereas in vertebrate dendrites, their orientations are locally mixed.

281 very large irregular somata and long, thick dendrites; their distal dendrites often branched extensi

282 Each cell has two major dendrites thought to receive segregated streams of senso

283 Removal of NUP188 also resulted in aberrant dendrite tiling, suggesting a potential role of NUP188 i

284 ses migration of surface atoms away from the dendrite tips, thereby smoothening the dendritic surface

285 ient surges of microtubule polymerization at dendrite tips; they drive retrograde extension of an act

286 als like NMDA spikes or trains of EPSPs from dendrite to soma.

287 es or trains of EPSPs from the distal apical dendrite to the soma in pyramidal neurons in the ACC, wh

288 Here, we report a new mechanism instructing dendrites to branch versus extend.

289 pecialized branches that climbed along other dendrites to form strong multi-synaptic connections with

290 little is known about mechanisms that allow dendrites to regenerate after injury.

291 erotonergic psychedelics may converge at the dendrites, to both enhance and suppress membrane excitab

292 rectifier K(+) conductances, while an apical-dendrites/trunk compartment included persistent Na(+), h

293 ; (2) axodendritic synapses onto GC proximal dendrites via their axon collaterals or terminals in the

294 (1) dendrodendritic synapses onto GC distal dendrites via their lateral dendrites in the superficial

295 Activity in dendrites was not determined solely by somatic activity

296 The complexity of apical, but not basal dendrites was significantly lower in Foxp2(+/R552H) cort

297 Here, by developing artificial dendrites, we experimentally demonstrate a complete neur

298 structured forms of synaptic potentiation in dendrites, we explored plasticity of glutamate uncaging-

299 tion at the nanoscale of zinc and tubulin in dendrites with a molecular ratio of about one zinc atom

300 owing plus, but not minus, ends increased in dendrites with reduced KT, CPC, and SAC proteins, while

301 ination, leading to retraction of the apical dendrite without altering basolateral dendrite dynamics.