戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ss defects and reduced fracture toughness of dental enamel.
2 ew avenues for the transport of materials in dental enamel.
3  separately is not critical for formation of dental enamel.
4 allization of nanofibrous apatite comprising dental enamel.
5 ost abundant secreted proteins in developing dental enamel.
6  in enteroendocrine cells, pineal gland, and dental enamel.
7 nd inhibit caries-like lesion progression in dental enamel.
8 ent satiety signaling, and the maturation of dental enamel.
9                                              Dental enamel, a hierarchical material composed primaril
10 reactive, and their incorporation in forming dental enamel at low concentrations promotes mineralizat
11 netically heterogeneous inherited defects of dental enamel bio-mineralization.
12 growth of carbonated apatite crystals during dental enamel biomineralization.
13 n) and strontium ((87)Sr/(86)Sr) analysis on dental enamel, bulk carbon (delta(13)C) and nitrogen (de
14 s an important role not only in mineralizing dental enamel but also in regulating the expression of E
15      Since type V collagen is not present in dental enamel but is an otherwise widely distributed col
16 s have shown that phytoliths are softer than dental enamel but still act as abrasive agents.
17 arth ratios (AERs) Sr/Ca and Ba/Ca in fossil dental enamel can inform the habitat, residence and life
18            Functional impairment and loss of dental enamel, caused by developmental defects or tooth
19                                              Dental enamel comprises interwoven arrays of extremely l
20 affect the development and mineralization of dental enamel compromising these functions.
21                ORAI1-deficient patients have dental enamel defects and anhidrosis, representing a new
22 describes a heterogeneous group of inherited dental enamel defects reflecting failure of normal amelo
23                                              Dental enamel defects were more common among patients th
24 y asked about oral manifestations, including dental enamel defects, recurrent aphthous ulceration and
25 e low and less variable distributions of its dental enamel delta(13)C values are similar to those fro
26 nase-20 (enamelysin, MMP20) is essential for dental enamel development.
27 ymes are necessary for the mineralization of dental enamel during development, and mutations in the k
28 rders characterized by abnormal formation of dental enamel, either in isolation or as part of a syndr
29 anding mechanical and chemical properties of dental enamel emerge from its complex hierarchical archi
30  conclude that MMP20 plays a nominal role in dental enamel fluorosis.
31 (skeletal fluorosis) and enamel development (dental/enamel fluorosis).
32            Before the role of amelogenins in dental enamel formation can be better understood, one mu
33                                              Dental enamel formation depends upon the transcellular t
34                                              Dental enamel formation is an intricate process tightly
35                                              Dental enamel formation is coordinated by ameloblast dif
36                                              Dental enamel formation requires large quantities of Ca(
37 ecta (AI) is a group of inherited defects of dental enamel formation that shows both clinical and gen
38              Enamelin is critical for proper dental enamel formation, and defects in the human enamel
39               Amelogenesis is the process of dental enamel formation, leading to the deposition of th
40      Because ameloblasts are responsible for dental enamel formation, we used an ameloblast-derived c
41  genetic conditions that result in defective dental enamel formation.
42 ntegrins are known to play critical roles in dental enamel formation.
43  conclude that ameloblastin is essential for dental enamel formation.
44 ith vesicles, and plays an important role in dental enamel formation.
45 namelin is likely to be essential for proper dental enamel formation.
46 rmation and prevention, and the mechanism of dental enamel formation.
47                                              Dental enamel forms by matrix-mediated biomineralization
48                                              Dental enamel forms extracellularly as thin ribbons of a
49 leistocene epoch(7-9), using the proteome of dental enamel from a Stephanorhinus tooth that is approx
50                      The results showed that dental enamel from these patients was essentially normal
51            Here, we analyze the frequency of dental enamel hypoplasia, a growth disruption indicator
52 ns such as dermatitis herpetiformis, anemia, dental enamel hypoplasia, recurrent oral aphthae, short
53 f pRTA exhibit acidemia, corneal edema, weak dental enamel, impacted colons, nutritional defects, and
54  two different methods, the surface of human dental enamel in vitro after being etched.
55                                              Dental enamel is a principal component of teeth(1), and
56                                Fully matured dental enamel is an architecturally and mechanically com
57                                              Dental enamel is comprised primarily of carbonated apati
58                             The formation of dental enamel is dependent upon amelogenins, a family of
59 infiltrate various molecules and resins into dental enamel is highly desirable in dentistry, yet tran
60 n dentistry, yet transporting materials into dental enamel is limited by the nanometric scale of thei
61                                              Dental enamel is one of the most remarkable examples of
62     Unlike other mineralized tissues, mature dental enamel is primarily (> 95% by weight) composed of
63                                      Healthy dental enamel is the hardest and most highly mineralized
64                                              Dental enamel is the hardest tissue in the body and cann
65   As the outermost layer of the tooth crown, dental enamel is the most mineralized tissue in mammals,
66                  Cracks originating from the dental-enamel junction and enamel tufts, crack deflectio
67                                              Dental enamel malformations, or amelogenesis imperfecta
68                      Peptide analysis on the dental enamel of 25 tested individuals confirmed they we
69    We were able to extract proteins from the dental enamel of both individuals (~1600 years old) and
70 here as delta(66)Zn) in bioapatite (bone and dental enamel) of animals from a modern food web in the
71 and trace element ratio analysis measured in dental enamel on a Pleistocene food web in Gabasa, Spain
72 sequencing the proteome of Early Pleistocene dental enamel overcomes the limitations of phylogenetic
73  include gingival enlargement, fibromas, and dental enamel pitting.
74     Amelogenin, the major protein of forming dental enamel, plays a crucial role in the biomineraliza
75 ical role for ACP4 in appositional growth of dental enamel probably by processing and regulating enam
76 to-immunity, muscle hypotonia and defects in dental enamel production and sweat gland function.
77                                              Dental enamel protects against the invasion of bacterial
78         The survival of an Early Pleistocene dental enamel proteome in the subtropics further expands
79                                 We retrieved dental enamel proteome sequences from a 1.9-million-year
80                          Here we present the dental enamel proteomes of H. antecessor from Atapuerca
81 n, the major extracellular matrix protein of dental enamel, regulates the formation of these crystall
82                    The limitations to assess dental enamel remineralization have been overcome by a m
83 ng hydrogen peroxide, is effective to remove dental enamel stains.
84                            The maturation of dental enamel succeeds the degradation of organic matrix
85 the most abundant protein species in forming dental enamel, taken to regulate crystal shape and cryst
86 ) describes a group of inherited diseases of dental enamel that have major clinical impact.
87 ), with and without amine fluoride, on human dental enamel under cariogenic challenge in situ.
88 tide applied to caries-like lesions in human dental enamel under simulated intra-oral conditions of p
89          The ability of CaneCPI-5 to bind to dental enamel was evaluated using atomic force microscop
90 onto a glass-like core substrate (ceramic or dental enamel), was loaded at its top surface with a har
91 veal that proteomic investigation of ancient dental enamel-which is the hardest tissue in vertebrates