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1  of the bud form both the dental papilla and dental follicle.
2 oth eruption in rats and is localized in the dental follicle, a loose connective tissue sac that is n
3 c protein-1 (MCP-1) gene is expressed in the dental follicle, a loose connective tissue sac that must
4 the mouse, Rspo3 is largely expressed in the dental follicle and alveolar mesenchyme while Rspo2 expr
5                   It has been shown that the dental follicle and bone remodeling are essential for to
6 d hormone-related peptide (PTHrP) are in the dental follicle and on the root surface, and deletion of
7 icant accumulation of aberrant fibers in the dental follicle and periodontium.
8 m the primary enamel knot, contribute to the dental follicle, and also the apical part of the papilla
9 tage, indicating that the dental papilla and dental follicle are still not defined populations at thi
10 ion, an influx of mononuclear cells into the dental follicle at an early post-natal age, may be initi
11 e expression of osteoprotegerin (OPG) in the dental follicle at day 3 enables the osteoclastogenesis
12 ctor-1 (CSF-1) is maximally expressed in the dental follicle at day 3, it was hypothesized that CSF-1
13                                              Dental follicle cells (DFCs) are neural crest cell-deriv
14 lopment, progenitor cell populations such as dental follicle cells are characterized by an open H3K4m
15 s study to determine if MCP-1 is secreted by dental follicle cells in culture and if its secretion is
16 onents onto the surface of the dentin before dental follicle cells penetrate the HERS network to cont
17 interfering RNA specific for CSF-1 mRNA into dental follicle cells resulted in an up-regulation of OP
18 f the CSF-1 receptor mRNA levels in cultured dental follicle cells reveals that high concentrations o
19 livered via polymer sponges, while implanted dental follicle cells seem to inhibit periodontal healin
20 to evaluate the ability of cementoblasts and dental follicle cells to promote periodontal regeneratio
21 gnal transduction level, we treated cultured dental follicle cells with either phorbolmyristate aceta
22 that during development of the periodontium, dental follicle cells, when appropriately triggered, hav
23 e initiated by the secretion of MCP-1 by the dental follicle cells.
24 ial stellate reticulum cells and mesenchymal dental follicle (DF) cells surrounding the teeth.
25                                          The dental follicle (DF) differentiates into the periodontal
26          The alveolar bone develops from the dental follicle (DF) during tooth root formation due to
27                                          The dental follicle (DF), most often associated with unerupt
28 ptor protein shows that it is present in the dental follicle early post-natally and is either absent
29 ion in cultured pluripotent cells from human dental follicles (hDFC).
30 -1 down-regulates OPG gene expression in the dental follicle in vivo.
31   Dental mesenchymal progenitor cells in the dental follicle lie at the heart of the coupling of thes
32 the gene expression of OPG is reduced in the dental follicle of the first mandibular molar of the rat
33 osteoprotegerin (OPG) gene expression in the dental follicle of the rat first mandibular molar.
34 ene expression was found to be higher in the dental follicle of the tl/tl mutants than in normals.
35 , stem cells from apical papilla (SCAP), and dental follicle progenitor cells (DFPCs).
36                                          The dental follicle regulates the alveolar bone resorption n
37          Ddr2 expression was detected in the dental follicle/sac and dental papilla mesenchyme of dev
38 nd c-fos, both of which are expressed in the dental follicle, the tissue that is required for eruptio
39 recruit mononuclear cells (monocytes) to the dental follicle, where these cells, in turn, fuse to for