ライフサイエンスコーパス: dental pulp

1 ATP-permeable channels, are present in human dental pulp.

2 ohydrolases (NTPDases), are present in human dental pulp.

3 f selective cytokines in the regeneration of dental pulp.

4 r population and not multipotent MSCs in the dental pulp.

5 sitive nociceptors in trigeminal ganglia and dental pulp.

6 ve nociceptors in the trigeminal ganglia and dental pulp.

7 involved in inflammatory hyperalgesia in the dental pulp.

8 erentiation, thus maintaining homeostasis in dental pulp.

9 rom peptidergic afferents innervating bovine dental pulp.

10 e neurons via in vitro superfusion of bovine dental pulp.

11 g trigeminal nerve fibers that innervate the dental pulp.

12 expression by cells derived from the porcine dental pulp.

13 erative population of cells from adult human dental pulp.

14 4 [IL-4], and IL-10) were detected in human dental pulp.

15 rvival function for Msx1 in odontoblasts and dental pulp.

16 ut not by odontoblasts or other cells in the dental pulp.

17 both metabolic and transductive processes in dental pulp.

18 expressed by many cell types throughout the dental pulp.

19 s found in many orofacial tissues, including dental pulp.

20 mity to blood vessels in physiological human dental pulps.

21 2) on the revascularization of severed human dental pulps.

22 ed for the maintenance of stem cell pools in dental pulps.

23 more intense in inflamed compared to healthy dental pulps.

24 the hypothesis that EAA receptors in bovine dental pulp activate a population of peptidergic sensory

25 produced by PDL (periodontal ligament), DPA (dental pulp adult), and GF (gingival fibroblast) cells.

26 Nerve fibers of human dental pulp also contained detectable mu-opioid receptor

27 of versican and link protein coincide in the dental pulp and are enriched in the peripheral area of t

28 as investigated by the immunolabeling of the dental pulp and cultured peripheral neuronal equivalent

29 arying stiffness would regenerate functional dental pulp and dentin when concentrically injected into

30 pulp stem cells (hDPSCs) reside in postnatal dental pulp and exhibit the potential to differentiate i

31 sor population isolated from postnatal human dental pulp and have the ability to regenerate a reparat

32 resorption occurs following infection of the dental pulp and is mediated mainly by IL-1alpha in the m

33 on of the local complement system within the dental pulp and its role in initiating the regeneration

34 nated and unmyelinated nerve fibers in human dental pulp and may play a role in dental pain mechanism

35 letion of Ddr2 in primary cell cultures from dental pulp and PDL inhibited differentiation of cells t

36 ularized for organ-specific functions of the dental pulp and periodontal ligament for a targeted rege

37 ed the survival of undifferentiated cells in dental pulp and promoted the formation of 2.3GFP(+) preo

38 red for ATP release and degradation in human dental pulp and that pannexin channels are involved in e

39 heir innate immunomodulatory activity in the dental pulp and their capability for pulp repair.

40 munohistochemical staining of infected human dental pulp and tissue from experimental dental pulpitis

41 enhanced neovascularization of severed human dental pulps and suggest that topical application of an

42 immune cells in irreversibly inflamed human dental pulp, and extracts of this tissue have significan

43 and distribution of P2X3 receptors in human dental pulp, and their co-localization with other neural

44 y of capsaicin-sensitive neurons innervating dental pulp, and these factors may be significant clinic

45 Inflammatory changes in the dental pulp are accompanied by release of a wide variety

46 Furthermore, Twist-1 functions in dental pulp are dependent on its interaction with Runx2.

47 uilibrium of odontoblast progenitor cells in dental pulp are unknown.

48 nt mesenchymal tissues, the periodontium and dental pulp, are maintained by distinct pools of stem ce

49 that cells in the mesenchymal compartment of dental pulp attenuate osteoclastogenesis.

50 evealed that LYVE-1(+) macrophages colonized dental pulp before birth.

51 materials (CaP) are introduced as potential dental pulp capping materials for deciduous teeth.

52 (TNF-alpha) could be a mediator involved in dental pulp cell differentiation toward an odontoblastic

53 l mesenchymal/papilla cells (FDPCs) or human dental pulp cells (ADPCs) under 2-dimensional or 3-dimen

54 Dental pulp cells (DPC) produce an array of neurotrophic

55 imensional (3D) tissues were engineered from dental pulp cells (DPCs) and assessed as a device for pu

56 e-aspartic acid (RGD) peptides, will bind to dental pulp cells (DPCs) and modulate their differentiat

57 ential for osseointegration engineering with dental pulp cells (DPCs) by testing a hypothesis that DP

58 odontogenic/osteogenic differentiation from dental pulp cells (DPCs) in vitro.

59 Human dental pulp cells (DPCs), adherent cells derived from de

60 ts of light-emitting diode (LED) exposure on dental pulp cells (DPCs).

61 l line-derived neurotrophic factor (GDNF) on dental pulp cells (DPCs).

62 n vitro, Wnt5a was increased 8-fold in human dental pulp cells (HDPCs) after TNF-alpha stimulation co

63 ans ATCC90028, the cytotoxicity toward human dental pulp cells (HDPCs), and the mechanical properties

64 nt cells from donor-matched rat dental pulp (dental pulp cells [DPCs]) and alveolar bone (alveolar bo

65 rscoring the need for therapies that protect dental pulp cells and enhance their regeneration.

66 Dental pulp cells and mouse odontoblast-like cells (MDPC

67 nx2, Gli1, Numb, and Notch expression in the dental pulp cells and odontoblasts of DSPP-null mice whe

68 ficantly ameliorated its cytotoxicity to the dental pulp cells and restored their odontoblast-like ce

69 system to study the interactions between the dental pulp cells and trigeminal neurons.

70 Calcitonin gene-related peptide (CGRP) and dental pulp cells are promising candidates for mediating

71 However, it is not clear whether dental pulp cells can be recruited endogenously for rege

72 Dental pulp cells can differentiate toward an odontoblas

73 ginate hydrogel with pulp ECM components and dental pulp cells followed by differentiation induction

74 Wnt/beta-catenin in dentinogenesis, we used dental pulp cells from a panel of transgenic mice, in wh

75 s OSX, OCN, PTX3 and p65 in odontoblasts and dental pulp cells from DSPP null mouse was lower when co

76 agen hydrogels were seeded either with human dental pulp cells from patients with characterized PHEX

77 Insights into the role of mesenchymal dental pulp cells in attenuating dentin resorption in ho

78 We show that dental pulp cells produce several neurotrophic factors i

79 However, the mechanisms by which dental pulp cells recognize lipopolysaccharides (LPSs) r

80 Long-term G5-RGD treatment of dental pulp cells resulted in enhanced mineralization as

81 Furthermore, adult dental pulp cells supplemented with these two growth fac

82 tudy was that LTA induces VEGF expression in dental pulp cells through TLR2 and PI3k/Akt signaling.

83 amined the effects of continuous exposure of dental pulp cells to FGF2 and showed that the effects of

84 that TNF-alpha stimulates differentiation of dental pulp cells toward an odontoblastic phenotype via

85 Human immortalized dental pulp cells were driven toward an odontoblast phen

86 Primary dental pulp cells were subsequently seeded onto pulp-der

87 Ten percent of the rat dental pulp cells were viable when cultured on the PMMA

88 fferentiation could be achieved by culturing dental pulp cells with their associated ECM.

89 ormed using human skeletal (MG63 and primary dental pulp cells) and salivary gland (HSG) cells.

90 with porcine dental epithelial cells, human dental pulp cells, and human umbilical vein endothelial

91 Thus, endogenous dental pulp cells, including stem/progenitor cells, may

92 Using multiple models, such as dental pulp cells, induced pluripotent stem cell (iPSC)-

93 When compared with dental pulp cells, periodontal neural crest lineage diff

94 vascular endothelial growth factor (VEGF) in dental pulp cells.

95 embryonic cells and populations of postnatal dental pulp cells; however, these cells are unable to co

96 P19L/+) and Dspp(P19L/P19L) mice had smaller dental pulp chambers mimicking DGI type II.

97 human DGI type III characterized by enlarged dental pulp chambers, while the teeth of older Dspp(P19L

98 Moreover, human dental pulp collected from patients with caries lesions

99 tus for ATP release and degradation in human dental pulp, consistent with the involvement of ATP sign

100 strategy for engineering of pre-vascularized dental pulp constructs offering potentially beneficial t

101 Also tested was the hypothesis that dental pulp contains either the B1 or B2 or both BK rece

102 Since the dental pulp contains hyaluronan, versican may bind to hy

103 WNT3a also promoted osteogenesis in dental pulp cultures.

104 ed and adherent cells from donor-matched rat dental pulp (dental pulp cells [DPCs]) and alveolar bone

105 domain peptide (MDP) hydrogel scaffolds with dental pulp-derived cells but were limited in their abil

106 indings suggest that LNGFR(Low+)THY-1(High+) dental pulp-derived cells provide an excellent source of

107 Prospectively isolated, dental pulp-derived LNGFR(Low+)THY-1(High+) cells repres

108 , indicating a functional bioactivity of the dental pulp-derived neurotrophic factors in vivo by resc

109 Based on these findings, we propose that dental pulp-derived neurotrophic factors play an importa

110 arborizations of neurons that innervate the dental pulp (DP) and periodontal tissues (PDL).

111 in mesenchymal stem cells derived from human dental pulp (DP-MSCs) and bone marrow (BM-MSCs).

112 stem cells derived from bone marrow (BMSC), dental pulp (DPSC) and dental apical papilla (SCAP) to e

113 ls (BMSC) with stem cells derived from human dental pulp (DPSC), apical papilla (SCAP) and follicle (

114 e its lack of expression in odontoblasts and dental pulp during tooth development, the BSP-GFPtpz tra

115 During caries, dental pulp expresses a range of pro-inflammatory cytoki

116 e scaffold that mimics the complexity of the dental pulp extracellular matrix (ECM).

117 cretion is evoked from isolated terminals of dental pulp fibers via the bradykinin B2 receptor-depend

118 n in dental pulp stem cells (DPSC) and human dental pulp fibroblasts (HDPF) through mitogen-activated

119 e C5a receptor (C5aR), here we show that all dental pulp fibroblasts, localized beneath the carious i

120 n an elemental neurosensory structure, human dental pulp, following chronic carious insult.

121 astrophic failure, while also protecting the dental pulp from bacterial attack.

122 Thus, this study reports that human dental pulp from healthy extracted teeth can be successf

123 The dental pulp from third molars of a diverse patient group

124 Dental pulps from erupted incisors displayed increased p

125 Dental pulps from sound teeth were CLARITY-cleared, immu

126 ough the regenerative potential of the human dental pulp has been investigated extensively, its antim

127 Dental pulp has intrinsic capacity for self-repair.

128 a multifunctional role in the regulation of dental pulp homeostasis.

129 Finally, Wnt5a participates in dental pulp inflammation in a MAPK-dependent (p38-, JNK-

130 ent and as an inflammatory mediator in human dental pulp inflammation.

131 We found that mice with dental pulp injury have greater Mouse Grimace Scores tha

132 In response to dental pulp injury induced by cavity preparation, LYVE-1

133 We use a dental pulp injury model that exposes the pulp to the ou

134 Visualization of the whole dental pulp innervation and vasculature was achieved.

135 Studies of the developing dental pulp innervation by nerve fibers from the trigemi

136 organogenesis, the timing and patterning of dental pulp innervation require both chemoattractive and

137 play an important role in orchestrating the dental pulp innervation.

138 Preservation of a vital dental pulp is a central goal of restorative dentistry.

139 Because the dental pulp is frequently infected by oral bacteria due

140 Here we tested whether homeostasis in dental pulp is modulated by Twist-1, a nuclear protein t

141 stem cells within the perivascular niche in dental pulps is unclear.

142 Infection of the dental pulp leads to an osteolytic lesion that results f

143 heroids can successfully regenerate vascular dental pulp-like tissue and also highlight the significa

144 en fabricated pre-vascularized, full-length, dental pulp-like tissue constructs by dispensing OD21 ce

145 After 28 d, enhanced dental pulp-like tissue formation was observed with a si

146 PC engineered tissues can regenerate a vital dental pulp-like tissue in a tooth root canal system and

147 In mature rat molar dental pulp, LYVE-1(+) macrophages were the main subset

148 activation of AMPA and kainate receptors in dental pulp may contribute to peripheral release of vaso

149 Up-regulation of VEGF expression in the dental pulp may result in increased intra-pulpal pressur

150 aled that this DSP domain induces endogenous dental pulp mesenchymal cell proliferation, differentiat

151 s is typically accompanied by an increase in dental pulp microvascular density.

152 cytokines and growth factors present in the dental pulp MSC secretome, including hepatocyte growth f

153 re used to demonstrate that the secretome of dental pulp multipotent mesenchymal stromal cells (MSCs)

154 the odontoblast layer, subodontoblast layer, dental pulp nerve bundles, and blood vessels.

155 cific questions regarding development of the dental pulp nerve system to be addressed.

156 nd anatomical simplicity of accessible human dental pulp neurogenic zone to address this conflict.

157 nhanced TRPA1 expression was also evident in dental pulp of carious compared with noncarious teeth.

158 mixed anaerobic infection, we inoculated the dental pulp of mice with six anaerobic pathogens contain

159 The inflammation observed in the dental pulp of teeth with deep caries lesions is charact

160 hronic interstitial polymicrobial infection, dental pulps of MCP-1(-/-) mice and controls were inocul

161 The dental pulps of the first molars were exposed and infect

162 ain insight into the potential population in dental pulps of unerupted and erupted incisors that give

163 ntation with mesenchymal stem cells from the dental pulp on poly-l-lactic acid scaffolds in nude mice

164 the presumptive root furcating region, where dental pulp overgrowth occurred, was increased in K14-Cr

165 te populations, and results highlighted that dental pulp pericytes are already precommitted to an odo

166 This indicated that for dental pulp pericytes, the odontoblast-specific gene Dsp

167 for immediate vascularization of engineered dental pulp poses a major hurdle towards successful impl

168 clearance of bacterial infection within the dental pulp precedes pulpal regeneration.

169 ontoblast-like cell differentiation of human dental pulp progenitor cells (DPSCs).

170 ries left untreated threaten exposure of the dental pulp, providing facile access for bacteria to cau

171 Dental pulp regeneration is a promising approach to rest

172 re major hurdles that need to be overcome in dental pulp regeneration.

173 application of HS to tissue engineering and dental pulp regeneration.

174 Resolvins have been shown to promote dental pulp regeneration.

175 ue constructs in full-length root canals for dental pulp regeneration.

176 carious teeth and plays an important role in dental-pulp regeneration via interaction of the active C

177 ely, topical treatment with RvE1 facilitated dental pulp regenerative properties by promoting Axin2-e

178 emperature and pH of isolated superfused rat dental pulp regulate capsaicin-induced release of calcit

179 argets to manipulate hDPSCs to promote tooth/dental pulp repair and regeneration.

180 Bacterial infections of the dental pulp result in soft tissue and alveolar bone dest

181 Bacterial infections of the dental pulp result in tissue destruction and periapical

182 ange of drug-induced Wnt-activity within the dental pulp, RNA of short-term induced (24-h) molars is

183 In conclusion whole CLARITY-cleared dental pulp samples revealed 3D-morphological neurovascu

184 of developing a biocompatible decellularised dental pulp scaffold, which is able to support dental pu

185 co-cultured with stromal cells (SCs), human dental pulp SCs or human adipose SCs, in natural hydroge

186 -1(+) tissue-resident M2-like macrophages in dental pulp showed dynamism in response to pulp injury,

187 that activation of beta(2)-adrenoceptors in dental pulp significantly reduces exocytosis of neuropep

188 re increased and deposited abnormally in the dental pulp, similar to the hereditary human tooth disor

189 om MEPE (Dentonin or AC-100) could stimulate dental pulp stem cell (DPSC) proliferation and/or differ

190 e adhesion behavior of a population of human dental pulp stem cell (hDPSC) on 64 combinations of nano

191 sults show that macrophages are required for dental pulp stem cell activation and appropriate reparat

192 HAp:Gd/Yb/Tm and HAp:Gd/Eu powders in human dental pulp stem cell cultures indicated their good bioc

193 ntal pulp scaffold, which is able to support dental pulp stem cell repopulation.

194 dothelial growth factor (VEGF) expression in dental pulp stem cells (DPSC) and human dental pulp fibr

195 Dental pulp stem cells (DPSC) are a relatively new alter

196 ne-2-O-beta-d-glucoside (THSG)-treated human dental pulp stem cells (DPSC) on the healing of experime

197 Evidence indicates that dental pulp stem cells (DPSC) secrete neurotrophic facto

198 Dental pulp stem cells (DPSCs) and periodontal ligament

199 Dental pulp stem cells (DPSCs) appear to be a good candi

200 Dental pulp stem cells (DPSCs) are a unique precursor po

201 Dental pulp stem cells (DPSCs) are important in tooth ph

202 In vitro assays with human dental pulp stem cells (DPSCs) assessed adhesion, viabil

203 It is known that dental pulp stem cells (DPSCs) can be induced to differe

204 Human dental pulp stem cells (DPSCs) can be isolated from infl

205 Dental pulp stem cells (DPSCs) can differentiate into va

206 Herein, we hypothesized that dental pulp stem cells (DPSCs) encapsulated in a collage

207 Dental pulp stem cells (DPSCs) have previously demonstra

208 Dental pulp stem cells (DPSCs) have the potential to pol

209 oblastic MC3T3-E1 cells and preodontoblastic dental pulp stem cells (DPSCs) in a dose-dependent manne

210 cumulating evidence has shown the promise of dental pulp stem cells (DPSCs) in bone regeneration, but

211 tial for autologous transplantation of human dental pulp stem cells (DPSCs) in endodontic treatment.

212 differentiation and mineralization of human dental pulp stem cells (DPSCs) in two-dimensional cell c

213 -like cells (ECs) derived from guiding human dental pulp stem cells (DPSCs) into expressing endotheli

214 Dental pulp stem cells (DPSCs) possess immunoregulatory

215 se of scaffold-free microtissue spheroids of dental pulp stem cells (DPSCs) prevascularized by human

216 Post-natal human dental pulp stem cells (DPSCs) represent a unique precur

217 ated into chitosan scaffolds and tested with Dental Pulp Stem Cells (DPSCs) to check their regenerati

218 Here we studied the dental pulp stem cells (DPSCs), a population of adult st

219 owever, there is no study available on human dental pulp stem cells (DPSCs), especially in the inflam

220 Bioactivity was assessed with human dental pulp stem cells (DPSCs), macrophages, an LPS-chal

221 cells have been isolated and characterized: dental pulp stem cells (DPSCs), stem cells from exfoliat

222 terials that can support native functions of dental pulp stem cells (DPSCs), which are capable of reg

223 ogenic differentiation and mineralization of dental pulp stem cells (DPSCs).

224 bone marrow stromal stem cells (BMSSCs) and dental pulp stem cells (DPSCs).

225 capacity, and clonogenic efficiency of human dental pulp stem cells (DPSCs).

226 sive regulatory functions of disease-derived dental pulp stem cells (DPSCs).

227 mesenchymal stem cells, including postnatal dental pulp stem cells (from permanent teeth) and stem c

228 derived from both mineralizing primary human dental pulp stem cells (hDPSCs) and an immortalized muri

229 G1 RSs were injected with human dental pulp stem cells (hDPSCs) and human umbilical vein

230 on the viability and proliferation of human dental pulp stem cells (hDPSCs) by comparing the cellula

231 Human dental pulp stem cells (hDPSCs) reside in postnatal dent

232 ression of C5L2 is highly modulated in human dental pulp stem cells (hDPSCs) undergoing odontoblastic

233 Cytotoxicity was examined on human dental pulp stem cells (hDPSCs).

234 In cultured mouse dental pulp stem cells (mDPSCs), RvE1 facilitated Axin2-

235 The discovery that dental pulp stem cells are capable of differentiating in

236 characteristics of the previously identified dental pulp stem cells can generate mineralized tissue o

237 nterleukin-6 [IL-6]) promote self-renewal of dental pulp stem cells cultured in low-attachment condit

238 Cell viability of human dental pulp stem cells cultured three-dimensionally in g

239 e aim of this study is to investigate if the dental pulp stem cells express C5L2 and if this receptor

240 ctionality and biocompatibility, stimulating dental pulp stem cells proliferation and differentiation

241 Transplantation of dental pulp stem cells stably transduced with small hair

242 sed scaffolds were recellularised with human dental pulp stem cells up to 14 days in vitro.

243 ll-derived IL-6 enhances the self-renewal of dental pulp stem cells via STAT3 signaling and induction

244 ix protein activates Wnt signaling in DPSCs (dental pulp stem cells).

245 derstanding odontoblastic differentiation of dental pulp stem cells, and may be useful in future dent

246 Expressed in bone tissue, dental pulp stem cells, and periodontal ligament cells,

247 also observed in T4-4 preodontoblast cells, dental pulp stem cells, and primary preodontoblasts.

248 5% NaOCl and triple antibiotic paste, ferret dental pulp stem cells, encapsulated in a hydrogel scaff

249 ling decreases the vasculogenic potential of dental pulp stem cells.

250 dhesives eluates were cytotoxic to the human dental pulp stem cells.

251 ulator of the endothelial differentiation of dental pulp stem cells.

252 ster regulator of stem cell self-renewal) in dental pulp stem cells.

253 se activity, and mineralization potential of dental pulp stem cells.

254 H 7 from all the composites were nontoxic to dental pulp stem cells.

255 ing is necessary to maintain self-renewal of dental pulp stem cells.

256 signaling in endothelial differentiation of dental pulp stem cells.

257 iently induce endothelial differentiation of dental pulp stem cells.

258 differentiation and mineralization of human dental pulp stem cells.

259 Human dental pulp stem/progenitor cells (hDPSCs) are attractiv

260 This work is based on a hypothesis that dental pulp stem/progenitor cells can be induced to migr

261 Biocompatibility with human dental pulp stromal cells (HDPSCs) was investigated (MTT

262 her late function of Msx1 in odontoblast and dental pulp survival.

263 useful indication of tissue distress in the dental pulp that could be used to investigate the early

264 been implied to have an irreversibly damaged dental pulp that is beyond repair and warranting root ca

265 populations in the rodent incisor apex, the dental pulp, the alveolar bone, the periodontal ligament

266 adult stem cells have been isolated from the dental pulp, the deciduous tooth, and the periodontium.

267 In the unique microenvironment of the dental pulp, the triad of tissue engineering would requi

268 e by recruiting endogenous stem cells of the dental pulp, through an easy-to-handle delivery vehicle

269 ssess the feasibility of decellularising rat dental pulp tissue and evaluate the ability of such scaf

270 ered centrally and peripherally within whole dental pulp tissue are shown to be biocompatible, preser

271 lization of the spatial relationships of the dental pulp tissue at the whole-organ has remained chall

272 In addition, we show that dental pulp tissue becomes innervated when transplanted

273 Interestingly, grafting the dental pulp tissue into hemisected spinal cord increases

274 veloping dental mesenchyme and whether adult dental pulp tissue maintains its inductive capability re

275 Here, we identified a dental pulp tissue-specific cell population based on the

276 blished the inductive potential of postnatal dental pulp tissue.

277 The presence of stem cells within the dental-pulp tissue as well as their differentiation into

278 lp cells (DPCs), adherent cells derived from dental pulp tissues, are potential tools for cell transp

279 ven achieve de novo regeneration of complete dental pulp tissues.

280 ascularization treatment to regenerate human dental pulp tissues.

281 ated 3 methods of decellularization of human dental pulp to be used as a potential autograft scaffold

282 e present study used isolated superfused rat dental pulp to test the hypothesis that capsaicin recept

283 We therefore investigated human dental pulps to determine if angiogenic changes could be

284 emistry, we detected ecto-AMPase activity in dental pulp, trigeminal ganglia (TG) neurons, and their

285 We further tested if ATP is released from dental pulp upon dentin mechanical or thermal stimulatio

286 rity and extracellular matrix composition of dental pulp varies considerably during tooth development

287 Innervation comprised 40% of the dental pulp volume and the vasculature another 40%.

288 ction, we used an in vivo model in which the dental pulp was inoculated with six anaerobic pathogens,

289 Bovine dental pulp was prepared and stimulated by the superfusi

290 In vitro superfusion of bovine dental pulp was used to evaluate the regulation of iCGRP

291 Rat dental pulps were retrieved and divided into control and

292 trigeminal ganglia and by 26% (p < 0.05) in dental pulp when tissues were pre-treated with [Leu(31),

293 ude that BK evokes iCGRP release from bovine dental pulp which is enhanced by a positive interaction

294 present a unique precursor population in the dental pulp, which has multipotential and can regenerate

295 enamel and dentin provide protection to the dental pulp, which is vital tissue rich with cells, vasc

296 We used CLARITY to study the whole human dental pulp with emphasis on the neurovascular component

297 Subsequent infection of the dental pulp with P. gingivalis caused extensive inflamma

298 s study, the effect of mono-infection of the dental pulp with T. denticola and with polymicrobial "re

299 The preservation of vital dental pulp with vasculature and nerve components remain

300 enchymal stromal cells, extracted from human dental pulp, with no adverse effects on cell viability,