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1  The compounds were similar as substrates of deoxycytidine kinase.
2 s a poor phosphorylation substrate for human deoxycytidine kinase, a pro-nucleotide form of the 4-bro
3 s spectrophotometric assay for thymidine and deoxycytidine kinase activities by coupling nucleoside 5
4 tivation was significantly less dependent on deoxycytidine kinase and on nucleoside transporters, and
5 dine (RO-9187) were excellent substrates for deoxycytidine kinase and were phosphorylated with effici
6 EGF and the gemcitabine metabolizing enzyme, deoxycytidine kinase, are specifically bound by HuR in p
7                                 The ratio of deoxycytidine kinase/CDA transcripts significantly corre
8                              We used a human deoxycytidine kinase containing three amino acid substit
9 ation in a heterologous system with a 244-bp deoxycytidine kinase core promoter.
10                                              Deoxycytidine kinase (dCK) activity was approximately 22
11 planation that emerged from enzyme assays of deoxycytidine kinase (dCK) and deoxycytidine monophospha
12 the nucleoside salvage pathway (NSP) enzymes deoxycytidine kinase (dCK) and thymidine kinase (TK1).
13 the cytosol of human cells is carried out by deoxycytidine kinase (dCK) and thymidine kinase 1 (TK1).
14                                              Deoxycytidine kinase (DCK) controls the rate-limiting st
15          We hypothesized that PET probes for deoxycytidine kinase (dCK) could be used to differentiat
16 ch as cladribine, require phosphorylation by deoxycytidine kinase (dCK) for pharmacological activity.
17 n, we report the crystal structures of human deoxycytidine kinase (dCK) in complex with the L-nucleos
18 d inhibition of ribonucleotide reductase and deoxycytidine kinase (dCK) in multiple cancer cell lines
19                                              Deoxycytidine kinase (dCK) is a rate limiting enzyme cri
20                                              Deoxycytidine kinase (dCK) is a rate-limiting enzyme in
21                                              Deoxycytidine kinase (dCK) is an essential nucleoside ki
22                                        Human deoxycytidine kinase (dCK) is responsible for the phosph
23              The physiological role of human deoxycytidine kinase (dCK) is to phosphorylate deoxynucl
24 pancreatic cancer cells, HuR associates with deoxycytidine kinase (dCK) mRNA, which encodes the enzym
25 hough in vitro investigations using purified deoxycytidine kinase (dCK) or deoxyguanosine kinase (dGK
26                                              Deoxycytidine kinase (dCK) phosphorylates a number of nu
27  human-derived reporter genes based on human deoxycytidine kinase (dCK) suitable for clinical PET.
28                                        Human deoxycytidine kinase (dCK) uses nucleoside triphosphates
29 nucleotide purine analog, is a substrate for deoxycytidine kinase (dCK), a key enzyme in the deoxyrib
30        (18)F-FAC retention in cells requires deoxycytidine kinase (dCK), a rate-limiting enzyme in th
31                                              Deoxycytidine kinase (dCK), a rate-limiting enzyme in th
32 r with either deoxyguanosine kinase (dGK) or deoxycytidine kinase (dCK), and is heterotropically acti
33 ilibrative nucleoside transporter 1 (hENT1), deoxycytidine kinase (dCK), and ribonucleotide reductase
34 or activation, 2'-AzCyd is phosphorylated by deoxycytidine kinase (dCK), and we find that expression
35 ancer drug and potent inhibitor of mammalian deoxycytidine kinase (dCK), mitigates Staphylococcus aur
36 In this article, we describe a deficiency in deoxycytidine kinase (DCK), one of the major enzymes of
37  enzymes, ribonucleotide reductase (RNR) and deoxycytidine kinase (dCK), via distinct molecular mecha
38 leukemia (B-CLL) cells have high activity of deoxycytidine kinase (dCK), we hypothesized that these l
39            We wanted to understand why human deoxycytidine kinase (dCK), which is related to HSV1-TK
40 oximately by 2 orders of magnitude in the 2'-deoxycytidine kinase (dCK)-deficient CEM/dCK(-) cell lin
41 step in which they are monophosphorylated by deoxycytidine kinase (dCK).
42 ivation is phosphorylation of the prodrug by deoxycytidine kinase (dCK).
43 o more cytotoxic than gemcitabine HCl in the deoxycytidine kinase deficient (CCRF-CEM/dCK(-/-)) tumor
44 was more active than gemcitabine against two deoxycytidine kinase-deficient cell lines.
45 uman sodium-iodide symporter (hNIS), a human deoxycytidine kinase double mutant (hdCKDM), and herpes
46 y, salvage of dG to its nucleotide forms via deoxycytidine kinase drove potent telomerase activation,
47                                              Deoxycytidine kinase (EC 2.7.1.74, dCK) is central to dr
48                                          Low deoxycytidine kinase expression in tumour biopsies from
49                   The virus expressing human deoxycytidine kinase failed to replicate acutely in neur
50 ncy on the nucleoside salvage pathway enzyme deoxycytidine kinase for the maintenance of a proper bal
51 methionine levels, and/or methylation of the deoxycytidine kinase gene.
52                                Inhibition of deoxycytidine kinase had little effect on radiocarbon in
53 omoter, was superior to thymidine kinase and deoxycytidine kinase in its ability to achieve high leve
54                                              Deoxycytidine kinase is the rate-limiting process in the
55           Inhibition of adenosine kinase and deoxycytidine kinase prevented the accumulation of dATP
56 s on the initial monophosphorylation step by deoxycytidine kinase showed that the catalytic efficienc
57 hymidine kinase, deoxycytidine as does human deoxycytidine kinase, the cytosolic kinase whose amino a
58                                        Human deoxycytidine kinase triple mutant (hdCK3mut) is a nonim
59                                          For deoxycytidine kinase, we coupled the reaction to a thymi
60                               Transcripts of deoxycytidine kinase were 2.6-fold higher in DS compared
61 anscripts of cystathionine-beta-synthase and deoxycytidine kinase were a median 12.5- and 2.6-fold hi
62 cherichia coli cytosine deaminase, and human deoxycytidine kinase were investigated in metastatic hum