ライフサイエンスコーパス: deoxynucleotide triphosphate

1 ernary complex of enzyme*gapped DNA*dNTP (2'-deoxynucleotide triphosphate).

2 ed because the reaction lacked the requisite deoxynucleotide triphosphate.

3 orrelated with a rapid decrease in available deoxynucleotide triphosphates.

4 novel checkpoint responsive to low levels of deoxynucleotide triphosphates.

5 lon nor theta influenced the Km of alpha for deoxynucleotide triphosphate and only slightly decreased

6 Biotinylated deoxynucleotide triphosphates are used in the primer ext

7 a nicking enzyme together with input DNA and deoxynucleotide triphosphates at 55 degrees C.

8 ginine 67 are functionally equivalent to the deoxynucleotide triphosphate binding residues arginine 5

9 to detect the template-dependent binding of deoxynucleotide triphosphates by DNA polymerases.

10 o acid that lies above the nucleobase of the deoxynucleotide triphosphate (dNTP) and is expected to p

11 RNA template-DNA primer duplex and incoming deoxynucleotide triphosphate (dNTP) at 3.0-A resolution.

12 primer, and DNA template in the presence of deoxynucleotide triphosphate (dNTP) complementary to the

13 Deoxynucleotide triphosphate (dNTP) concentrations have

14 In primary cell types, intracellular deoxynucleotide triphosphate (dNTP) levels are tightly r

15 s suggest a possible mechanism for importing deoxynucleotide triphosphate (dNTP) molecules into SRLV

16 The corresponding 2'-deoxynucleotide triphosphate (dNTP) of isopropyl oxime e

17 se controlling the size of the intracellular deoxynucleotide triphosphate (dNTP) pool, a limiting fac

18 -121.6-fold lower binding affinity (K(d)) to deoxynucleotide triphosphate (dNTP) substrates than HIV-

19 RRM2 of ribonucleotide reductase, perturbing deoxynucleotide triphosphate (dNTP) supply.

20 n Arg668 and the ring oxygen of the incoming deoxynucleotide triphosphate (dNTP) using a combination

21 he 3'-OH on the sugar moiety of the incoming deoxynucleotide triphosphate (dNTP), we examined how thi

22 mispaired template:primer in the absence of deoxynucleotide triphosphate (dNTP).

23 compounds are competitive inhibitors of the deoxynucleotide triphosphate (dNTP).

24 ested to play an important role in supplying deoxynucleotide triphosphates (dNTP) for DNA repair duri

25 th protonation of the gamma-phosphate of the deoxynucleotide triphosphate(dNTP) via a solvent water m

26 Deoxynucleotide triphosphates (dNTPs) are essential for

27 ductase (RNR) supplies the balanced pools of deoxynucleotide triphosphates (dNTPs) necessary for DNA

28 ctively incorporate Watson-Crick base-paired deoxynucleotide triphosphates (dNTPs) over incorrectly p

29 tial event in the HIV-1 life cycle, requires deoxynucleotide triphosphates (dNTPs) to fuel DNA synthe

30 cluded the design and synthesis of unnatural deoxynucleotide triphosphates (dNTPs), engineering a sui

31 -limiting enzyme in the de novo synthesis of deoxynucleotide triphosphates (dNTPs), is a potential ta

32 activity of the protein-dephosphorylation of deoxynucleotide triphosphates (dNTPs)-implicates enzymat

33 etroviral RNA into DNA by depleting cellular deoxynucleotide triphosphates (dNTPs).

34 cation blocker alpha-amanitin, NTPs (but not deoxynucleotide triphosphate [dNTPs]) templated at downs

35 ype oligonucleotides were mixed with various deoxynucleotide triphosphates in the presence of Sr(2)(+

36 ent with the high ratio of ribonucleotide to deoxynucleotide triphosphates in tissues, and that riboa

37 These agents cause imbalances in deoxynucleotide triphosphate levels and the accumulation

38 es processing matched or mismatched template-deoxynucleotide triphosphate pairs from 22 degrees to 85

39 whose products supply the mitochondria with deoxynucleotide triphosphate pools needed for DNA replic

40 de reductase and the consequent depletion of deoxynucleotide triphosphate pools result in a cellular

41 r the removal of HAP from purine pools, from deoxynucleotide triphosphate pools, and from DNA, and we

42 lls into S-phase under conditions of altered deoxynucleotide triphosphate pools, particularly an incr

43 bstrate was observed in reactions containing deoxynucleotide triphosphates required to make full-leng

44 Mus81 enables survival of deoxynucleotide triphosphate starvation, UV radiation, a

45 hus far been considered to be a reduction of deoxynucleotide triphosphate synthesis by inhibition of

46 nus to the alpha-beta bridging oxygen of the deoxynucleotide triphosphate; this neutralizes the evolv

47 in myeloid cells by hydrolyzing the cellular deoxynucleotide triphosphates to a level below that whic

48 nterococcus faecalis is a distant homolog of deoxynucleotide triphosphate triphosphohydrolases (dNTPa