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1 ctivity in melanocyte cultures, resulting in depigmentation.
2 erall survival, in the absence of autoimmune depigmentation.
3  and is frequently accompanied by autoimmune depigmentation.
4 educed pigment deposition in melanocytes and depigmentation.
5 umor immunity and autoimmunity manifested as depigmentation.
6 mmunity were observed, characterized by coat depigmentation.
7  the incidence of retinal pigment epithelial depigmentation.
8  epidermis, melanocytes, leading to areas of depigmentation.
9  treatment significantly limited the rate of depigmentation.
10 zed by a lack of melanin and consequent skin depigmentation.
11 active CD8(+) T cells and prevents epidermal depigmentation.
12  chronic autoimmune disease that causes skin depigmentation.
13 se characterized by melanocyte loss and skin depigmentation.
14  disease of the skin characterized by patchy depigmentation.
15 ived oral antibiotics and were monitored for depigmentation.
16  the lack of an assessment method for active depigmentation.
17 oss of epidermal melanocytes and progressive depigmentation.
18 pigmentation with varying amounts of central depigmentation.
19 ntigen-specific T cells, resulting in patchy depigmentation.
20 cells and spontaneously developed autoimmune depigmentation.
21 ina that progresses to panretinal patches of depigmentation.
22 erse events were diarrhea, fatigue, and hair depigmentation.
23 lative efficacy of these agents in mediating depigmentation.
24 ed protein 2-derived peptide correlated with depigmentation.
25 econd, unknown locus that causes progressive depigmentation.
26 spotting but show no evidence of progressive depigmentation.
27 ype of HPS with minimal cutaneous and ocular depigmentation.
28 ently experience stress to the skin prior to depigmentation.
29 ]), generalized pigment dilution and/or hair depigmentation (18 [44%]), xerosis (8 [20%]), scrotal er
30 rtial or complete retinal pigment epithelial depigmentation; (2) round shape; (3) sharp margins; and
31  3.34, 20.08) for retinal pigment epithelium depigmentation, 3.59 (95% CI: 1.71, 7.57) for increased
32 en (51.7%), retinal pigment epithelium (RPE) depigmentation (34.9%), RPE hyperpigmentation (branching
33                                  Progressive depigmentation accelerates in response to stress.
34 of the cumulative incidence of vitiligo-like depigmentation across studies.
35 of mouse skin, which correlated with reduced depigmentation after vitiligo induction.
36 al deletion of Adar1 in mice leads to global depigmentation and absence of myelin from peripheral ner
37 al melanocytes is shown to cause progressive depigmentation and autoimmune vitiligo.
38 mally thin ONL co-localizing with severe RPE depigmentation and choroidal thinning.
39 s investigation might be of relevance to the depigmentation and degeneration of neuromelanin-pigmente
40 hogenic Cx31, but not wild-type Cx31, causes depigmentation and degeneration of ommatidia that are re
41 ondition is characterized by bilateral acute depigmentation and discoloration of the iris stroma, pig
42 e against TRP-2 peptide, inducing autoimmune depigmentation and further decreasing lung tumor nodules
43 twork predictions on the 4-step scale, while depigmentation and geographic atrophy (GA) areas correla
44 e-transgenic mice developed spontaneous hair depigmentation and had visual defects that progressed wi
45 tion with v-Ha-ras or Ela was accompanied by depigmentation and led to complete loss of msg1 expressi
46 n all transplanted mice, we noted a striking depigmentation and loss of hair follicles.
47 eins (TRP) 1 and TRP-2, we observed striking depigmentation and melanocyte destruction only in the sk
48  chronic autoimmune disorder leading to skin depigmentation and reduced quality of life (QOL).
49 chronic autoimmune disease resulting in skin depigmentation and reduced quality of life.
50 ne the cumulative incidence of vitiligo-like depigmentation and the prognostic value of vitiligo deve
51 ng an injection at an outside hospital, iris depigmentation and thinning, iris recession with retinal
52 arnet (Er:YAG) laser techniques for gingival depigmentation and to evaluate their effect on histologi
53 nocytes leads to autonomous growth in vitro, depigmentation, and in the case of the oncogenes, tumori
54  and globules, tan color, irregularly shaped depigmentation, and irregularly distributed and sized do
55              Photoreceptor degeneration, RPE depigmentation, and retinal remodeling follow.
56  such as rickets, abdominal distention, hair depigmentation, and skin lesions and with a maternal his
57 inning, discrete hyperreflective foci, focal depigmentation, and the presence of suprachoroidal hypor
58 ly, porphyrin biosynthesis and light-induced depigmentation are enhanced by starvation, recapitulatin
59 pigment epitelium, defects/pigment mottling, depigmentation area, subretinal haemorrhage, subretinal
60 5% of MT/ret mice develop a vitiligo, a skin depigmentation attributable to the lysis of normal melan
61 l examination or fundus photography, such as depigmentation, bone-spicule pigmentation, vascular tort
62 ing mouse models of vitiligo consist of hair depigmentation but lack prominent epidermal involvement,
63 strategy for repigmentation in patients with depigmentation conditions such as vitiligo.
64                      Although qualitative SN depigmentation correlated modestly with age (p = 0.02),
65                                           SN depigmentation correlated with speed of 6- and 50-foot w
66  or older who had nonsegmental vitiligo with depigmentation covering 10% or less of total body-surfac
67 capacity to migrate to the skin and suppress depigmentation, despite normal systemic numbers in the s
68                                           SN depigmentation, detected neuropathologically, was correl
69 .1(+) cells and Fc receptor gamma-chain, but depigmentation did not require these components.
70 as been shown in vivo that patients with the depigmentation disorder vitiligo accumulate hydrogen per
71                            Patients with the depigmentation disorder vitiligo have low catalase expre
72        Vitiligo is the most common cutaneous depigmentation disorder worldwide, yet little is known a
73 king the pathogenesis of alopecia to that of depigmentation disorders.
74 immune disorder characterized by progressive depigmentation due to melanocyte destruction by cytotoxi
75 o, and active-specific immunotherapy-induced depigmentation had significant anti-TRP-2 IgG titers.
76 een known for several decades that cutaneous depigmentation, i.e., contact/occupational vitiligo, can
77 ulture, we determined the molecular basis of depigmentation in a mouse melanocyte model system.
78  in reduced melanoblast motility and ventral depigmentation in adult mice.
79 egional magnetic resonance (MR) quantifiable depigmentation in association with PD severity and (b) t
80 herapy also induced tumor rejection and skin depigmentation in B cell-deficient and in CD4(+) T cell-
81 sed the regression of established tumors and depigmentation in lymphopenic hosts.
82 egeneration, attenuated retinal vessels, and depigmentation in mice lacking Sema4A.
83 that simvastatin both prevented and reversed depigmentation in our mouse model of vitiligo, and reduc
84 tical for the progression and maintenance of depigmentation in our mouse model of vitiligo.
85                                Vitiligo-like depigmentation in patients with melanoma may be associat
86                                              Depigmentation in PD follows a distinct spatial pattern,
87 om Bsubtilis interfered with T-cell-mediated depigmentation in the h3TA2 mouse model of vitiligo, sug
88 ed metastatic melanomas and developed patchy depigmentation in their coats.
89 f the 14 known missense mutations that cause depigmentation in these species map to the tyrosine kina
90  that the coexistence of parkinsonism and SN depigmentation in this birth cohort may have resulted fr
91 ocol significantly increased and accelerated depigmentation in this model, accompanied by the inducti
92 gly support a role for HSP70i in progressive depigmentation in vivo.
93 , size, and type; retinal pigment epithelium depigmentation; increased pigment; geographic atrophy; a
94                                The cutaneous depigmentation induced by phenolic derivatives results f
95 aking with three primary objectives: halting depigmentation, initiating repigmentation and sustaining
96 donetwork, multiple blue-gray dots, scarlike depigmentation, irregularly distributed and sized brown
97                                              Depigmentation is accompanied by accumulation of autorea
98       Clinical repigmentation after gingival depigmentation is an outcome of histologic changes in th
99 ution of the microbiome in FH mice, in which depigmentation is mediated by tyrosinase-reactive T cell
100   Identification of new compounds to reverse depigmentation is therefore a pressing need for this dis
101 , occurring in a kindred with more extensive depigmentation, is a novel four-base insertion in exon 2
102  retinal pigment, retinal pigment epithelial depigmentation, neovascular lesions, and geographic atro
103  retinal pigment, retinal pigment epithelial depigmentation, neovascular lesions, and geographic atro
104                                Patients with depigmentation of 0.5% or more of their facial body surf
105       However, loss of Rb resulted in severe depigmentation of hair follicles.
106 iameter with drusen area >/=196350 mum2) and depigmentation of retinal pigment epithelium (slope of -
107 ch the presence of bilateral soft drusen and depigmentation of retinal pigment epithelium was associa
108  by melanocyte loss, which results in patchy depigmentation of skin and hair, and is associated with
109  autoimmune disease in which acquired patchy depigmentation of skin, hair, and mucous membranes resul
110 and induced long term antitumor immunity and depigmentation of skin.
111 ratinocyte-melanocyte contact and results in depigmentation of the dark skinned Yucatan swine, sugges
112 sease of the skin causing disfiguring patchy depigmentation of the epidermis and, less commonly, hair
113                              Bilateral Acute Depigmentation of the Iris (BADI) is a condition which w
114 onic migraine, who later developed bilateral depigmentation of the iris.
115                              Bilateral Acute depigmentation of the is a recently discovered condition
116 ed marked hypopigmentation of both fundi and depigmentation of the regressed tumour in left eye.
117                                              Depigmentation of the substantia nigra and other brainst
118  the progressive fundus hypopigmentation and depigmentation of the tumour remnants first appeared aft
119        The prognostic value of vitiligo-like depigmentation on survival outcome was assessed using ra
120  no reduction in the severity or kinetics of depigmentation or long-lived protection against melanoma
121 5.6); presence of retinal pigment epithelial depigmentation (OR, 9.0; 95% CI, 4.1-19.8); or hyperpigm
122 r scores indicating a greater area of facial depigmentation), or F-VASI75 response, at week 24.
123 stically significant values for Er:YAG laser depigmentation (P = 0.001).
124 al drusen (P<0.001) and mottled FAF with RPE depigmentation (P<0.001).
125 use model (Txnrd1(mel-/-)), which exhibits a depigmentation phenotype consisting of variable amelanot
126 gaging anti-CD25 antibody fully restored the depigmentation phenotype in h3TA2-IFN-gamma(-/-) mice, m
127 ne condition vitiligo, characterized by skin depigmentation, presents challenges for effective treatm
128                             Gingival melanin depigmentation procedures are commonly associated with r
129              Surgical stripping for gingival depigmentation remains the gold standard; however, Er:YA
130              To address this, we developed a depigmentation-repigmentation system using cultured mela
131 go (GV) is a complex disease in which patchy depigmentation results from autoimmune loss of melanocyt
132 combination treatment, 56% (38/68) developed depigmentation, starting at the site of vaccination or c
133 d kindred characterized by unusually limited depigmentation, substitutes a threonine for an alanine a
134 body prevents CD8(+) T-cell accumulation and depigmentation, suggesting a therapeutic potential for t
135 peripheral nerve remyelination and focal fur depigmentation; surviving weak mice had persistent expre
136 ducing melanocytes and results in patches of depigmentation that are visible as white spots.
137 d Drug Administration approved drug used for depigmentation therapy of advanced vitiligo.
138 TRP) 1, are relevant to both autoimmune skin depigmentation (vitiligo) and tumor immunity, because th
139                           Ampicillin-induced depigmentation was accompanied by gut but not skin dysbi
140                                           SN depigmentation was detected in 57 (13.2%) of the cohort.
141  of Treg abundance in preventing progressive depigmentation was evaluated by adoptively transferring
142                                              Depigmentation was evaluated over time, measuring immune
143                                              Depigmentation was found to occur in CD4-depleted mice,
144                               In this model, depigmentation was introduced by gene gun vaccination wi
145                                              Depigmentation was significantly impaired only in IFN-ga
146 ciated with retinal pigment epithelium (RPE) depigmentation, was followed by disorganization and furt
147                  Quantitative measures of SN depigmentation were increased in this birth cohort compa
148 Qualitative indices of substantia nigra (SN) depigmentation were verified in a subset of 40 randomly
149 ulating S100B levels in patients with active depigmentation which were strongly correlated with the e
150 ing offers a fully reversible model for hair depigmentation, which might be used for the studies of h
151                    Treatment produced a skin depigmentation, which was associated with prolonged surv
152 es the human condition by inducing epidermal depigmentation while sparing the hair.
153  in which melanocyte destruction causes skin depigmentation, with 49 loci known from previous GWAS.
154  is characterized by acute bilateral stromal depigmentation, without other pathologic ocular findings
155 nknown origin, is the most frequent cause of depigmentation worldwide, with an estimated prevalence o

 
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