ライフサイエンスコーパス: dermal fibroblast

1 d VZV peptides, as well as kill VZV-infected dermal fibroblasts.

2 ct of targeted beta-catenin stabilization in dermal fibroblasts.

3 c function in new matrix deposition by human dermal fibroblasts.

4 K, were down-regulated in LTBP4 mutant human dermal fibroblasts.

5 ernate but inefficient receptor for HSV-1 on dermal fibroblasts.

6 tion and wound-healing properties of patient dermal fibroblasts.

7 lution (particulate matter) on human primary dermal fibroblasts.

8 mparable entry pathways in keratinocytes and dermal fibroblasts.

9 es of human skin epidermal keratinocytes and dermal fibroblasts.

10 ellular retention of collagen XII in patient dermal fibroblasts.

11 ile biocompatibility was verified with human dermal fibroblasts.

12 eticulum (ER) retention of COL4A2 in primary dermal fibroblasts.

13 ession in gingival fibroblasts compared with dermal fibroblasts.

14 quercetin alleviated cellular senescence of dermal fibroblasts.

15 )] mitochondrial DNA (mtDNA) damage in human dermal fibroblasts.

16 y reduced the PDGF-BB-dependent migration in dermal fibroblasts.

17 of the canonical TGFbeta pathway, in primary dermal fibroblasts.

18 ns and other cell types, including patients' dermal fibroblasts.

19 teraction with alphaVbeta3 integrin in human dermal fibroblasts.

20 authentic versican core protein produced by dermal fibroblasts.

21 Decorin was predominantly produced by dermal fibroblasts.

22 ) in keloid fibroblasts compared with normal dermal fibroblasts.

23 human retinal pigmented epithelium cells and dermal fibroblasts.

24 ntial KF apoptosis when compared with normal dermal fibroblasts.

25 ing on keratinocytes and importantly also on dermal fibroblasts.

26 ed the expression of HAS3 and versican V2 in dermal fibroblasts.

27 OA FLS) but not in nondiseased primary human dermal fibroblasts.

28 stinct gene expression signature from non-DP dermal fibroblasts.

29 t activities when transfected in orbital and dermal fibroblasts.

30 votal role in regulation of the ECM genes in dermal fibroblasts.

31 ients were elevated 2-5-fold above wild-type dermal fibroblasts.

32 pecific sites on DNA is greater than that in dermal fibroblasts.

33 a negative regulator of the ERalpha gene in dermal fibroblasts.

34 O1 and p63 in skin-derived keratinocytes and dermal fibroblasts.

35 how wound electric fields guide migration of dermal fibroblasts.

36 e cardiomyocytes compared to co-culture with dermal fibroblasts.

37 t of NF-kappaB to the MMP-1 promoter site in dermal fibroblasts.

38 EDA-dependent fibro-inflammatory response in dermal fibroblasts.

39 in different concentrations on primary human dermal fibroblasts.

40 AP in mediating the profibrotic responses in dermal fibroblasts.

41 that dermal invasion is directly impeded by dermal fibroblasts.

42 drives two waves of gene expression in human dermal fibroblasts.

43 l motility in serum-stimulated primary mouse dermal fibroblasts.

44 and gelatine, was performed by seeding human dermal fibroblasts.

45 nduces collagen production by normal and SSc dermal fibroblasts.

46 growth factor-beta, at least in the case of dermal fibroblasts.

47 Here, we show that in primary normal human dermal fibroblasts, A2AR stimulation with CGS21680 elici

48 In addition, P311 induced dermal fibroblast activation and proliferation.

49 ata suggest that in the Abcc6(-/-) genotype, dermal fibroblasts actively contribute to changes that p

50 investigated the hypothesis that autologous dermal fibroblast (ADF) injection into the AV nodal area

51 nd PDGF-BB (KD=200 nM), enhanced adult human dermal fibroblast (AHDF) survival under serum starvation

52 factor BB (PDGF-BB) and promote adult human dermal fibroblast (AHDF) survival under stress.

53 s ROS induced carbonylation profile in human dermal fibroblasts along with A498 primary site and ACHN

54 Interestingly, in dermal fibroblasts, although complete inhibition of IRAK

55 n (aged 19-81 years) and primary cultures of dermal fibroblast and dermal sheath cells.

56 elialization, and angiogenesis compared with dermal fibroblast and PBS treated wounds.

57 and proliferation of diabetic mouse primary dermal fibroblasts and 3T3 fibroblasts, which express ve

58 ists of a dermal equivalent containing human dermal fibroblasts and a fully stratified, biologically

59 lyplexes also distributed more broadly among dermal fibroblasts and allowed greater interaction with

60 demonstrate that NRG1 is highly expressed by dermal fibroblasts and cancer-associated fibroblasts (CA

61 sing cell death also in normal cells such as dermal fibroblasts and endometrial mesenchymal stem cell

62 Both EZH2 and H3K27me3 were elevated in SSc dermal fibroblasts and endothelial cells compared with h

63 mis, but Ing3A also activated ERK1/2 in skin dermal fibroblasts and endothelial cells.

64 delivery of either autologous or allogeneic dermal fibroblasts and epidermal keratinocytes directly

65 its individual components on matched primary dermal fibroblasts and epidermal keratinocytes from huma

66 al wounds bioprinted with layered autologous dermal fibroblasts and epidermal keratinocytes in a hydr

67 -38 cells and other elastogenic cells, human dermal fibroblasts and fetal bovine chondrocytes.

68 , it does not form elastic fibers with human dermal fibroblasts and forms fewer atypical fibers with

69 ll as in lung cancer-associated fibroblasts, dermal fibroblasts and hepatic stellate cells.

70 Moreover, the patient's dermal fibroblasts and induced pluripotent stem cell (iP

71 that TLY012 selectively blocks activation of dermal fibroblasts and induces DR-mediated apoptosis in

72 pressed in human epidermal keratinocytes and dermal fibroblasts and is regulated via RAR/RXR-mediated

73 rm of IL-36Ra was confirmed in human primary dermal fibroblasts and keratinocytes and in skin equival

74 B1 protein levels decline in senescent human dermal fibroblasts and keratinocytes, mediated by reduce

75 al promoters of TGFbeta signaling in primary dermal fibroblasts and of bleomycin-induced fibrosis in

76 Human neonatal dermal fibroblasts and primary human adult keratinocyte

77 down the expression of 285 lncRNAs in human dermal fibroblasts and quantified cellular growth, morph

78 13 in mediating the induction of collagen in dermal fibroblasts and that blockade with IL-13 antibodi

79 talk between PDGFRbeta and TbetaRI occurs in dermal fibroblasts and that CD44 negatively modulates si

80 B and TGFbeta interact physically in primary dermal fibroblasts and that stimulation with PDGF-BB ind

81 omas, normal melanocytes, keratinocytes, and dermal fibroblasts and utilized The Cancer Genome Atlas

82 rometry in human keratinocytes, melanocytes, dermal fibroblasts, and melanoma cells.

83 sis in systemic sclerosis (SSc; scleroderma) dermal fibroblasts, and such cells in scleroderma skin l

84 r, Fn14, is upregulated in keratinocytes and dermal fibroblasts, and TWEAK induces these cytokines an

85 o, HEp-2, LLC-MK2, primary human and macaque dermal fibroblasts, and U373 human glioblastoma cells.

86 of fibrillin-1 into microfibrils produced by dermal fibroblasts; and (iii) the requirement of the pro

87 Dermal fibroblasts are a simple, relevant, and much unde

88 Instead, dermal fibroblasts are both necessary and sufficient to

89 do not usually develop in adult skin, adult dermal fibroblasts are competent to contribute to DP dur

90 ist for the four neuroectoderm lineages, and dermal fibroblasts are not progenitors for fin ray osteo

91 These results suggest that synthetic dermal fibroblasts are plastic, and that CCN2 is require

92 liferative phase of cutaneous wound healing, dermal fibroblasts are recruited into the clotted wound

93 r is mediated by the lower lineage and upper dermal fibroblasts are recruited only during re-epitheli

94 Dermal fibroblasts are required for structural integrity

95 In skin homeostasis, dermal fibroblasts are responsible for coordinating the

96 trated the value of carefully-phenotyped SSc dermal fibroblasts as a platform for SSc target and drug

97 dary effect of SAg-stimulated PBMCs on human dermal fibroblasts as judged by C/EBP delta expression.

98 ure and ex vivo approaches to identify human dermal fibroblasts as natural host cells that support pr

99 ogether, our findings identify primary human dermal fibroblasts as responder cells to IFNlambda.

100 anced uptake of apoptotic PMN (51%) by human dermal fibroblasts at concentrations as low as 0.1 nM.

101 We found a novel human dermal fibroblast attachment and spreading site on tropo

102 , impaired hydrogel formation, and decreased dermal fibroblast attachment compared to wild-type tropo

103 dipocytes) secrete factors that affect human dermal fibroblast behavior.

104 ntry mechanism suggests that HSV-1 can enter dermal fibroblasts both by direct fusion with the plasma

105 e in the regulation of TGFbeta1 signaling in dermal fibroblasts both in vivo and in vitro.

106 CPyV is able to replicate within a subset of dermal fibroblasts, but MCPyV DNA has also been detected

107 Finally, induction of MMP-1 expression in dermal fibroblasts by CCN1 N-terminal domains resulted i

108 d after the dedifferentiation of human adult dermal fibroblasts by overexpression of pluripotency tra

109 Since dermal fibroblasts can have profound impacts on melanoma

110 .0009) and TGF-beta2 (P=0.0001) secretion in dermal fibroblast cell lines from subjects with FMD comp

111 ir effect on UVA-induced senescence in human dermal fibroblast cells (FB) and the associated mechanis

112 PNIPAAm-conjugated PCL allowed human dermal fibroblast cells (HDF) and mesenchymal stem cells

113 cytotoxic effects on SHSY5Y, MRC5, and human dermal fibroblast cells compared with the dissolved PhIP

114 ed in vitro studies on the response of human dermal fibroblast cells toward pristine titania nanotube

115 This suggests that the properties of dermal fibroblasts change during postnatal development.

116 ECM protein production by normal dermal fibroblasts cocultured with SSc CD8+ T cell super

117 c mice, with selective expression of CCN1 in dermal fibroblasts (COL1A2-CCN1), display accelerated sk

118 t, P4HA1 protein level and C-P4H activity in dermal fibroblasts compared to age-matched control sampl

119 sensitivity to mtDNA damage was observed in dermal fibroblasts compared with keratinocytes at wavele

120 , we examined whether age-related changes in dermal fibroblasts could drive melanoma metastasis and r

121 Primary dermal fibroblast cultures confirmed that matrix metallo

122 is variability, we established primary human dermal fibroblast cultures from several ODDD patients an

123 re assessed ex-vivo in primary human-derived dermal fibroblast cultures.

124 transforming growth factor-beta1-stimulated dermal fibroblasts decreased the formation of contractil

125 Blimp1 ablation in E12.5 mouse dermal fibroblasts delayed HF morphogenesis and growth a

126 We obtained similar findings in dermal fibroblasts, demonstrating that the IFN-gamma/TNF

127 ed in vitro by exposing platelets to TSP2 KO dermal fibroblast (DF)-derived ECM.

128 tained from a patient biopsy by reprograming dermal fibroblasts (DF), hiPSc present the same properti

129 Dermal fibroblasts (dFBs) resist infection by locally di

130 ols cellular properties of keratinocytes and dermal fibroblasts during early stages of skin developme

131 oupled to the surrounding matrix for primary dermal fibroblasts embedded in a 3D fibrin matrix.

132 nd in vitro matrix fiber assembly by primary dermal fibroblasts, EMILIN-1 and -2 are deposited on and

133 rting cell population is combined with human dermal fibroblasts, encapsulated in a fibrin hydrogel an

134 , etc.), in primary cultures of normal human dermal fibroblasts exposed to visible and near infra-red

135 Here we show that senescent dermal fibroblasts express the non-classical MHC molecul

136 sent a long-term cell culture model of human dermal fibroblasts expressing fluorescence-labelled huma

137 We demonstrate that normal dermal fibroblasts expressing high PEDF levels attenuate

138 ombinant human C7 (rhC7) purified from human dermal fibroblasts (FB-rhC7), we showed previously that

139 Experiment 1) and versus nerve-derived SC or dermal fibroblast (Fibro) transplantation (Experiment 2)

140 CD26 expression was increased in dermal fibroblasts following skin wounding but was downr

141 ts5(-/-);Vcan(hdf/+) mice and isolated their dermal fibroblasts for comparison with dermal fibroblast

142 hese results highlight a central function of dermal fibroblasts for skin protection, opening new poss

143 ion and are epigenetically suppressed in SSc dermal fibroblasts, Friend leukaemia integration 1 (Fli1

144 the generation of human ESCs via SCNT using dermal fibroblasts from 35- and 75-year-old males.

145 Dermal fibroblasts from Adamts5(-/-) mice, which lack a

146 their dermal fibroblasts for comparison with dermal fibroblasts from Adamts5(-/-) mice.

147 We show here that cellular defects in dermal fibroblasts from affected individuals are complem

148 Cultured dermal fibroblasts from affected individuals showed acce

149 Cultured dermal fibroblasts from affected individuals showed enha

150 differentiation into specifically of palmar dermal fibroblasts from Dupuytren's patients in to myofi

151 proteomics to characterize the phenotype of dermal fibroblasts from healthy subjects of various ages

152 peripheral myelin protein 22) protein and in dermal fibroblasts from patients with CMT1A, PMP22 aggre

153 teomic analysis of the secretome of cultured dermal fibroblasts from patients with systemic sclerosis

154 eta) signaling biomarkers in plasma and from dermal fibroblasts from patients with VEDS.

155 re, we demonstrate unexpectedly that primary dermal fibroblasts from pre-symptomatic mutation carrier

156 Primary dermal fibroblasts from R258C patients exhibited increas

157 Cultured dermal fibroblasts from the most severely affected infan

158 Dermal fibroblasts from the Syrian hamster (SHD cells) a

159 Dermal fibroblasts from two CMT1A pedigrees with confirm

160 Dermal fibroblasts from two unrelated patients harboring

161 In contrast, dermal fibroblasts from Vcan haploinsufficient (Vcan(hdf

162 We used dermal fibroblasts, from patients with retinal pathology

163 , beta2AR antagonism increased angiogenesis, dermal fibroblast function, and re-epithelialization, bu

164 Estrogen is an important regulator of dermal fibroblast functions, including extracellular mat

165 onstrate that elevated expression of CCN1 by dermal fibroblasts functions as a key mediator of dermal

166 enzyme, indoleamine 2,3-dioxygenase (IDO) in dermal fibroblasts generates a tryptophan-deficient envi

167 Compared to dermal fibroblasts, gingival fibroblasts are less respon

168 Here we show that, compared with dermal fibroblasts, gingival fibroblasts show reduced ex

169 In vitro, OPN(-/-) dermal fibroblasts had decreased migratory capacity but

170 s necessary for reprogramming of human adult dermal fibroblasts (hADFs) into undifferentiated induced

171 tion, angiogenesis, and differentiated human dermal fibroblast (HDF ) function contribute to scarring

172 Human dermal fibroblasts (HDF) were also subjected to PMB stre

173 in steroid-resistant BT-549 cells and human dermal fibroblasts (HDF-a) using AP and WB.

174 ll metabolism for a healthy cell line (Human Dermal Fibroblasts, HDF) and a cervical cancer cell line

175 no-associated virus type 2 (wtAAV2) in human dermal fibroblasts (HDFs) and HeLa cells revealed that j

176 human embryonic stem cells (hESCs) and human dermal fibroblasts (hDFs) derived hiPSCs.

177 ts role in aging, we demonstrated that human dermal fibroblasts (HDFs) from older human subjects were

178 We exposed human dermal fibroblasts (HDFs) to the drug-eluting scaffold a

179 ys both migration and proliferation of human dermal fibroblasts (HDFs) were inhibited by the 5-LO pha

180 Treatment of human dermal fibroblasts (HDFs) with combinations of cell-perm

181 (non-contact) cocultures with primary human dermal fibroblasts (hDFs).

182 enase type 1 (11beta-HSD1) in cultured human dermal fibroblasts (HDFs).

183 iPSCs), as well as in control parental human dermal fibroblasts (HDFs).

184 cal and cell attachment activity using human dermal fibroblasts (HDFs).

185 tumor-promoting epigenetic modifications in dermal fibroblasts, highlighting further the importance

186 vation by various cells (e.g., primary human dermal fibroblasts, HMEC-1 endothelial cells), and unloc

187 chymal stem cells (hMSCs) and human neonatal dermal fibroblasts (hNDFs) within a customized extracell

188 Human dermal fibroblasts, however, are not used for commercial

189 In dermal fibroblasts, IFNlambda induced the expression of

190 ciated matricellular protein, is elevated in dermal fibroblasts in aged human skin.

191 Our findings suggest a crucial role for dermal fibroblasts in regulating the differentiation and

192 alone did not induce CD44v7, but stretching dermal fibroblasts in the presence of OPN increased huma

193 Using healthy dermal fibroblasts in vitro, we analyzed the signaling p

194 00A12 by nearly 70%, which in turn activated dermal fibroblasts in vitro.

195 ated protein, is elevated in aged human skin dermal fibroblasts in vivo and stimulates MMP-1 expressi

196 )-responsive gene expression in SSc skin and dermal fibroblasts, in particular the effect of differen

197 cated that the addition of FnIII-1c to human dermal fibroblasts induced the expression of several inf

198 on repressive function, causes conversion of dermal fibroblasts into CAFs.

199 (MESP) homolog may be used to convert human dermal fibroblasts into cardiac progenitors.

200 d that YAP1 regulates the differentiation of dermal fibroblasts into highly contractile myofibroblast

201 for achieving transdifferentiation of human dermal fibroblasts into induced cardiomyocyte-like cells

202 ession of human ETS2 to convert normal human dermal fibroblasts into replicative cells expressing the

203 KI mice displayed increased infiltration of dermal fibroblasts into the wound environment, increased

204 Unlike strain-matched dermal fibroblasts, intraperitoneally administered ICC-S

205 A balanced turnover of dermal fibroblasts is crucial for structural integrity a

206 that a physiological function of ADAMTS5 in dermal fibroblasts is to maintain optimal versican conte

207 te and sebocyte differentiation, its role in dermal fibroblasts is unclear.

208 MMP-1, principally derived from dermal fibroblasts, is the major protease capable of ini

209 es have successfully demonstrated the use of dermal fibroblasts isolated from DPs as models for PDK4

210 ave now extended our study of HSV-1 entry to dermal fibroblasts isolated from nectin-1- or HVEM-defic

211 to in vitro investigation of patient-derived dermal fibroblasts, laboratory engineered cell lines and

212 C5 in different human cell lines and primary dermal fibroblasts leads to reduced MHC class I expressi

213 noted that versican overexpression in human dermal fibroblasts led to increased SMA expression, enha

214 OL7A1, the defective gene in RDEB, in normal dermal fibroblasts led to increased type XII collagen, t

215 argeted expression of CXCR2 in primary human dermal fibroblasts led to paracrine induction of nuclear

216 d scRNA-seq and exome-seq data from 32 human dermal fibroblast lines, identifying hundreds of differe

217 out the disease process and that analyses of dermal fibroblasts might lead to the discovery of promis

218 Specifically, an increase in 5-HETE enhanced dermal fibroblast migration and collagen deposition.

219 Second, COL7A1 supported dermal fibroblast migration and regulates their cytokine

220 ice with inducible deletion of MMP-14 in the dermal fibroblast (MMP-14(Sf-/-)).

221 g mouse embryonic fibroblast Balb/3T3, human dermal fibroblast NHDF cell lines, and on neoplastic cel

222 mal human epidermal keratinocytes (NHEK) and dermal fibroblasts (NHDF) and to confine all tumorigenic

223 nerated induced pluripotent stem cells using dermal fibroblasts obtained from patients with TSC.

224 performed a non-integrative reprogramming of dermal fibroblasts obtained from two patients with radio

225 sion that also occur, to a lesser extent, in dermal fibroblasts of apparently unaffected skin.

226 Human dermal fibroblasts of both primary and cell-line culture

227 uced pluripotent stem cells (iPSCs) from the dermal fibroblasts of TLR3- and UNC-93B-deficient patien

228 contrast, IL-1alpha-dependent stimulation of dermal fibroblasts optimally stimulates epidermal stem c

229 ivo, we injected CSp-EMV-primed or -unprimed dermal fibroblasts (or CSp-EMVs) in a chronic rat model

230 ic mice were treated either with fibrocytes, dermal fibroblasts, or phosphate buffered saline (PBS) t

231 In vitro, primary dermal fibroblasts (pDFs) from KI mice showed substantia

232 ctivity of ADAMTS5 profoundly influenced the dermal fibroblast phenotype and may regulate a phenotypi

233 Uniform Wnt signaling activity is present in dermal fibroblast precursors preceding hair follicle ini

234 in a transition wherein the abundant CD34(+) dermal fibroblasts present in healthy human skin disappe

235 to provide insights into the utility of SSc dermal fibroblast primary cells for therapeutic target d

236 Furthermore, normal human dermal fibroblasts (primary cells) are also seeded on th

237 Our previous studies show that whereas dermal fibroblasts proliferate in response to TGF-beta1,

238 ing into nonkeratinocytes, particularly into dermal fibroblasts, promotes cellular infiltration and,

239 In vitro, primary dermal fibroblasts readily express podoplanin in respons

240 ha and ERbeta; however, regulation of ERs in dermal fibroblasts remains poorly understood.

241 Dermal fibroblasts represent a heterogeneous population

242 mulation of D1 dopamine receptors present in dermal fibroblasts restores vascular endothelial growth

243 SSc dermal fibroblasts retained most of the molecular diseas

244 Gene-expression profiles of primary dermal fibroblasts revealed overexpression of matrix met

245 Cultured dermal fibroblast samples from patients with major depre

246 VEDS dermal fibroblasts secreted more TGF-beta2, whereas down

247 an increase in dermal sheath proteins in the dermal fibroblast secretome with aging.

248 Affected dermal fibroblasts showed enhanced basal and epidermal g

249 Dermal fibroblasts showed splice-variant mediated skippi

250 In vitro studies using murine and human dermal fibroblasts showed that P311 stimulated TGF-beta1

251 tic endothelial cells (LECs) cocultured with dermal fibroblasts spontaneously organize into a stable

252 IL-1alpha treatment of dermal fibroblasts stimulated CD26 activity, and therefo

253 we show that forced expression of MRTF-A in dermal fibroblasts stimulates contraction of a collagen

254 in all cancer cell lines and normal primary dermal fibroblasts studied.

255 Microbe, Liu et al. (2016) report that human dermal fibroblasts support productive MCPyV infection.

256 extracellular 2-D matrix derived from human dermal fibroblasts supports GAS adherence and biofilm fo

257 f each receptor during HSV-1 entry in murine dermal fibroblasts that were deficient in expression of

258 of MEK1 in the epidermis leads to changes in dermal fibroblasts that, like the skin inflammatory infi

259 inhibitory factor (MIF), more strongly than dermal fibroblasts, thereby creating a MIF gradient in s

260 ol-sensitive PTP1B is affected by ROS in SSc dermal fibroblasts, thereby enhancing the phosphorylatio

261 transforming growth factor-beta signaling in dermal fibroblasts through the down-regulation of thromb

262 al differentiation, is sufficient to convert dermal fibroblasts to a keratinocyte phenotype.

263 es were undertaken by exposing primary human dermal fibroblasts to different concentrations of partic

264 ctin expression and secretion and stimulated dermal fibroblasts to express EDA-fibronectin.

265 It took more than 1 hour for dermal fibroblasts to manifest detectable directional mi

266 est-derived melanocytes and mesoderm-derived dermal fibroblasts, to identify SE differentially methyl

267 e describe the transdifferentiation of human dermal fibroblasts towards the cardiac cell lineage via

268 ineered with iMyoD-hTERT-NHDFs, normal human dermal fibroblasts transduced with genes encoding human

269 f the myofibroblast protein alpha-SMA in SSc dermal fibroblasts treated with a caspase 1 inhibitor.

270 olecular reprogramming trajectories of human dermal fibroblasts using single-cell transcriptomics.

271 re, intraray glia, lateral line, osteoblast, dermal fibroblast, vascular endothelium, and resident bl

272 mal stability of collagen in patient-derived dermal fibroblasts versus age-matched control samples.

273 Using knockout dermal fibroblasts, we confirmed its role for Brucella b

274 By using human dermal fibroblasts, we demonstrate that NO-np increased

275 lasts or selectively targeting Dlk1(+) lower dermal fibroblasts, we found that beta-catenin stabiliza

276 sing primary mouse embryonic fibroblasts and dermal fibroblasts, we show that TGF-beta-mediated, Smad

277 Using patient-matched oral and dermal fibroblasts, we show that TGF-beta1-dependent pro

278 These nuclear abnormalities in patient dermal fibroblast were restored by expression of WT NSMC

279 f freshly isolated and in vitro cultured SSc dermal fibroblasts were characterized using whole transc

280 g cancer (NSCLC) cell lines and normal human dermal fibroblasts were co-cultured.

281 Human dermal fibroblasts were contaminated with gammaFe(2)O(3)

282 mmortalized lines of control and R258C human dermal fibroblasts were established and SM alpha-actin e

283 Isolated dermal fibroblasts were incubated with recombinant IL-13

284 Dermal fibroblasts were isolated from systemic sclerosis

285 beta (TGF-beta) cytokines in patient-derived dermal fibroblasts were measured by ELISA.

286 es from KO mice were more migratory, whereas dermal fibroblasts were more proliferative compared with

287 Dermal fibroblasts were obtained from 2 patients with AR

288 Dermal fibroblasts were obtained from a CPVT patient due

289 Dermal fibroblasts were primed with CSp-EMVs for 24 h fo

290 The functional properties of fibrocytes and dermal fibroblasts were tested by using reverse-transcri

291 Human dermal fibroblasts were used as comparator cells.

292 Our in vitro experiments showed that dermal fibroblasts, which are an important source of gro

293 nd induced a profibrotic phenotype in normal dermal fibroblasts, which was inhibited by an anti-IL-13

294 We aimed to study affected dermal fibroblasts with a view to inform therapeutic stu

295 We measured PIP3 concentrations in dermal fibroblasts with endogenous PIK3CA mutations and

296 r growth, and co-injection of Cav1-deficient dermal fibroblasts with melanoma cells is sufficient to

297 being made to generate neurons directly from dermal fibroblasts with neuron-specific transcription fa

298 ion of cell-derived matrices (CDMs) by human dermal fibroblasts with stable knockdown of COL6A1 revea

299 We show that treatment of human dermal fibroblasts with transforming growth factor-beta

300 r inducing apoptosis of primary normal human dermal fibroblasts without affecting the overall cell vi