ライフサイエンスコーパス: dermatitis

1 tions were proposed (eg food allergy, atopic dermatitis).

2 for future prevention or treatment of atopic dermatitis.

3 tion such as psoriasis and contact or atopic dermatitis.

4 m development and subsequent risk for atopic dermatitis.

5 howed no evidence of association with atopic dermatitis.

6 ts and adults with moderate-to-severe atopic dermatitis.

7 is associated with reduced asthma and atopic dermatitis.

8 published datasets from subjects with atopic dermatitis.

9 females was associated with more seborrheic dermatitis.

10 evoked scratching in a mouse model of atopic dermatitis.

11 mouse model of IL-22-induced psoriasis-like dermatitis.

12 included, of whom 636 (14.5%) had seborrheic dermatitis.

13 factors were not associated with seborrheic dermatitis.

14 lesion formation and intense itch in atopic dermatitis.

15 ema area and severity index (EASI) in atopic dermatitis.

16 ics in adults with moderate to severe atopic dermatitis.

17 and long-term effect in children with atopic dermatitis.

18 a mouse model of IL-23 mediated psoriasiform dermatitis.

19 ough MRGPRB2 drives itch in allergic contact dermatitis.

20 of these NPs in a model of allergic contact dermatitis.

21 that enter the skin and cause itch in atopic dermatitis.

22 dant capacity are associated with seborrheic dermatitis.

23 nagement of asthma, food allergy, and atopic dermatitis.

24 ls are downregulated in patients with atopic dermatitis.

25 nfection to occur in association with atopic dermatitis.

26 eterious effects of PAR2 in models of atopic dermatitis.

27 b in patients with moderate to severe atopic dermatitis.

28 l profile changes that result in response to dermatitis.

29 sent similar characteristics, such as atopic dermatitis.

30 ts and adults with moderate-to-severe atopic dermatitis.

31 identify new strategies for targeting atopic dermatitis.

32 approach for antipruritic therapy in atopic dermatitis.

33 philic esophagitis, food allergy, and atopic dermatitis.

34 he development of WD-associated psoriasiform dermatitis.

35 on is a feature of skin aging and eczematous dermatitis.

36 d by donor monocytes to generate a GVHD-like dermatitis.

37 nd related phenols, which can induce contact dermatitis.

38 the innate immune system in allergic contact dermatitis (ACD) has traditionally been confined to the

39 tiple preclinical models of allergic contact dermatitis (ACD), a pruritic inflammatory skin disorder,

40 Using a mouse model of allergic contact dermatitis (ACD), we tested the effects of treatment wit

41 mmatory diseases, including allergic contact dermatitis (ACD).

42 dye that is known to cause allergic contact dermatitis (ACD).

43 T(RM)) cells play a role in allergic contact dermatitis (ACD).

44 and in the placebo plus erlotinib group were dermatitis acneiform (20 [9%]) and increased alanine ami

45 re hypertension (52 [24%]; grade 3 only) and dermatitis acneiform (33 [15%]), and in the placebo plus

46 e-limiting toxicities (grade 2 hypertension, dermatitis acneiform, and memory impairment in patient 1

47 EKTI exonic mutations associated with atopic dermatitis (AD) affect the protease inhibitory activity

48 Atopic dermatitis (AD) affects up to 20% of children and adults

49 Atopic dermatitis (AD) affects up to 20% of children worldwide

50 lk allergy, and/or moderate to severe atopic dermatitis (AD) and a positive egg/milk skin prick test

51 Atopic dermatitis (AD) and food allergy (FA) are associated wit

52 ed in the pathophysiology of not only atopic dermatitis (AD) and psoriasis (PSO) but also lupus eryth

53 Our current understanding of atopic dermatitis (AD) and psoriasis pathophysiology is largely

54 Molecular studies in atopic dermatitis (AD) are largely restricted to patients with

55 rmal skin and skin form patients with atopic dermatitis (AD) are unknown.

56 ip and participants were examined for atopic dermatitis (AD) at enrolment.

57 Moderate-to-severe atopic dermatitis (AD) has been associated with significant dis

58 r the treatment of moderate to severe atopic dermatitis (AD) in adults.

59 racial/ethnic differences in incident atopic dermatitis (AD) in childhood, few studies have examined

60 Farm exposures may reduce the risk of atopic dermatitis (AD) in children, but this is controversial a

61 Spontaneous development of atopic dermatitis (AD) in NC/Nga (NC) mice has been attributed

62 1beta-HSD1 affects the development of atopic dermatitis (AD) in vitro and in vivo.

63 Atopic dermatitis (AD) is a chronic inflammatory skin disease w

64 Atopic dermatitis (AD) is a chronic inflammatory skin disorder

65 Atopic dermatitis (AD) is a common chronic inflammatory skin di

66 Atopic dermatitis (AD) is a common inflammatory skin condition

67 Atopic dermatitis (AD) is a common skin disease affecting up to

68 Atopic dermatitis (AD) is a common, chronic, inflammatory skin

69 Atopic dermatitis (AD) is a common, complex, and highly heritab

70 t research advancements indicate that atopic dermatitis (AD) is a complex disease characterized by di

71 Atopic dermatitis (AD) is a complex inflammatory disorder with

72 Atopic dermatitis (AD) is a highly heterogeneous disease, both

73 Atopic dermatitis (AD) is a highly prevalent chronic inflammato

74 Atopic dermatitis (AD) is a highly prevalent, itchy inflammator

75 Atopic dermatitis (AD) is a highly pruritic chronic inflammator

76 Atopic dermatitis (AD) is a prevalent disease worldwide and is

77 Atopic dermatitis (AD) is a prevalent inflammatory skin disease

78 Atopic dermatitis (AD) is a severe inflammatory skin disease.

79 Atopic Dermatitis (AD) is a T cell-mediated chronic skin diseas

80 Atopic dermatitis (AD) is a T helper (Th)2-biased disease with

81 Atopic dermatitis (AD) is among the most common chronic inflamm

82 Population studies suggest that atopic dermatitis (AD) is associated with an increased risk of

83 Atopic dermatitis (AD) is associated with epidermal barrier def

84 Atopic dermatitis (AD) is characterized by a skin barrier defec

85 Atopic dermatitis (AD) is more common among African American ch

86 Atopic dermatitis (AD) is the most common chronic inflammatory

87 Atopic dermatitis (AD) is the most common inflammatory skin dis

88 actor attachment protein receptors in atopic dermatitis (AD) is unknown.

89 assessed molecular changes in chronic atopic dermatitis (AD) lesions, little is known about the trans

90 skin sites in diseased skin, such as atopic dermatitis (AD) lesions.

91 hma, allergic rhinitis, or both after atopic dermatitis (AD) onset.

92 31 receptor alpha subunit involved in atopic dermatitis (AD) pathogenesis.

93 IL-13 has an important role in atopic dermatitis (AD) pathogenesis.

94 Atopic dermatitis (AD) patients are often colonized with Staphy

95 The fundamental defect(s) that drives atopic dermatitis (AD) remains controversial.

96 Skin transcriptome studies in atopic dermatitis (AD) showed broad dysregulation as well as "i

97 Atopic dermatitis (AD) shows differential clinical presentation

98 , allergic conjunctivitis (AC), and allergic dermatitis (AD) were defined from self-completed questio

99 tion in skin lesions of patients with atopic dermatitis (AD) were recently reported.

100 The genetic background of Atopic Dermatitis (AD) with chronic pruritus is complex.

101 The nonlesional skin of children with atopic dermatitis (AD) with peanut allergy (PA) is associated w

102 3A, KIF3A, have been associated with atopic dermatitis (AD), a chronic inflammatory skin disorder.

103 years) for the assessment of current atopic dermatitis (AD), allergic rhinitis (AR), asthma and sens

104 ng, who is best known for his work in atopic dermatitis (AD), along with many other contributions in

105 adults and young children with early atopic dermatitis (AD), but chronologic changes in the blood of

106 health care utilization for pediatric atopic dermatitis (AD), but do not account for disease severity

107 ed risk factor for the development of atopic dermatitis (AD), but several aspects of this association

108 pathogenic cytokine in patients with atopic dermatitis (AD), but the molecular effects of IL-22 anta

109 Atopic dermatitis (AD), characterized by pruritis and cutaneous

110 ronic inflammatory conditions such as atopic dermatitis (AD), chronic obstructive pulmonary disease (

111 e the effect of cheese consumption on atopic dermatitis (AD), food allergy (FA), allergic rhinitis, a

112 tice guideline "systemic treatment of atopic dermatitis (AD)," we critically appraised evidence on sy

113 play a role in the pathophysiology of atopic dermatitis (AD).

114 e for uncontrolled moderate-to-severe atopic dermatitis (AD).

115 atched case-control study on incident atopic dermatitis (AD).

116 ratinocytes (KCs) in association with atopic dermatitis (AD).

117 n T cell-mediated diseases, including atopic dermatitis (AD).

118 ing epithelial barrier dysfunction in atopic dermatitis (AD).

119 ia found on the skin of patients with atopic dermatitis (AD).

120 ns and symptoms of moderate to severe atopic dermatitis (AD).

121 e assessments are not standardized in atopic dermatitis (AD).

122 e subsequent onset and progression of atopic dermatitis (AD).

123 for the treatment of mild-to-moderate atopic dermatitis (AD).

124 recommended as adjunctive therapy for atopic dermatitis (AD).

125 inflammatory skin diseases, including atopic dermatitis (AD).

126 is presents as one of the symptoms of atopic Dermatitis (AD).

127 evealed multiple loci associated with atopic dermatitis (AD).

128 psoriasis (n = 30), and patients with atopic dermatitis (AD; n = 16).

129 ents with inflammatory skin diseases (atopic dermatitis [AD] and alopecia areata [AA]).

130 01; 25.2% vs 15.1%, P < .001; asthma, atopic dermatitis [AD] and rhinitis, respectively).

131 bsence of common allergic conditions (atopic dermatitis [AD], IgE-mediated food allergy [IgE-FA], ast

132 Atopic eczema (atopic dermatitis, AD) is characterized by disrupted skin barri

133 biopsies promote our understanding of atopic dermatitis/AD pathomechanisms in infants/toddlers with e

134 pattern-derived PCA factors, and seborrheic dermatitis adjusted for confounders.

135 iRNAs in allergic diseases, including atopic dermatitis, allergic rhinitis, and asthma.

136 ergen and aeroallergen sensitization, atopic dermatitis, allergic rhinitis, asthma, and challenge-pro

137 to be associated with development of atopic dermatitis, allergic sensitization, and asthma.

138 (e.g., Dexamethasone) an induced skin atopic dermatitis, an induced psoriasis-like inflammation, a ho

139 as little as 4 weeks with a WD promoted mild dermatitis and accumulation of IL-17A-producing gammadel

140 CRSwNP and other type 2 diseases (eg, atopic dermatitis and asthma).

141 ion, and has been approved for use in atopic dermatitis and asthma.

142 Atopic dermatitis and bronchial asthma are common diseases in c

143 l findings are noted in management of atopic dermatitis and bronchial asthma.

144 se genes constitute a risk factor for atopic dermatitis and eczema-related asthma.

145 to vitiligo to lupus erythematosus to atopic dermatitis and food allergy.

146 as a disease-modifying treatment for atopic dermatitis and itch.

147 ession of VCAM1 is upregulated in spongiotic dermatitis and lupus and is associated with a dense peri

148 We find that atopic dermatitis and psoriasis can be classified by distinct m

149 te inflammatory skin diseases such as atopic dermatitis and psoriasis.

150 minated skin inflammation that mimics atopic dermatitis and sensitizes the airways for antigen challe

151 Dermatitis and SPT reactivity were more prevalent among

152 stratum corneum (SC) of patients with atopic dermatitis and their impaired skin barrier and water-hol

153 Patients with atopic dermatitis and those with more severe disease had higher

154 ze, urticaria, rhinitis and visible flexural dermatitis), and effect modification by Sm exposure.

155 skin diseases, including ichthyosis, atopic dermatitis, and a multitude of clinical eczema variants.

156 aneous diseases (including psoriasis, atopic dermatitis, and alopecia areata) and eight other immune-

157 lls drive diseases such as psoriasis, atopic dermatitis, and alopecia.

158 th food allergies, allergic rhinitis, atopic dermatitis, and asthma.

159 is approved for treatment of asthma, atopic dermatitis, and chronic sinusitis with nasal polyposis.

160 n following transplants, treatment of atopic dermatitis, and dry eye disease.

161 rom diseases as diverse as psoriasis, atopic dermatitis, and erythrokeratodermia variabilis, suggesti

162 atology clinical trials in psoriasis, atopic dermatitis, and hidradenitis have been suspended, termin

163 aracteristics, incidence of allergic contact dermatitis, and incidence of wound complications.

164 g that is used to treat psoriasis and atopic dermatitis, and is thought to function through regulatio

165 with a particular focus on asthma and atopic dermatitis, and provide insights into the roles of these

166 anding about sleep, itch, scratching, atopic dermatitis, and psoriasis.

167 Given concerns such as contact dermatitis, antibiotic resistance, and healthcare costs

168 Itch, inflammation, and atopic dermatitis are associated with activation of PAR2.

169 Several types of psoriasiform dermatitis are associated with increased IL-36 cytokine

170 Allergic skin diseases, such as atopic dermatitis, are clinically characterized by severe itchi

171 s, including asthma, food allergy and atopic dermatitis, are increasing in prevalence, particularly i

172 deep insight into the pathogenesis of atopic dermatitis as a ceramide-deficient disease.

173 de effective treatments for allergic contact dermatitis-associated chronic pruritus.

174 tors for adult allergic rhinitis were atopic dermatitis, asthma and asymptomatic sensitization to pol

175 factor for allergic diseases such as atopic dermatitis, asthma, allergic rhinitis, food allergy, con

176 c and inflammatory disorders, such as atopic dermatitis, asthma, rheumatoid arthritis, colitis, and c

177 Bovine digital dermatitis (BDD), an infectious disease of the bovine fo

178 found in the incidence and severity of neck dermatitis between the two arms.

179 Both mouse models showed dermatitis, blepharitis, and splenomegaly.

180 e history: He has been suffering from atopic dermatitis, bronchial asthma, and food allergies since c

181 These polymorphisms associate with atopic dermatitis but how they affect Ca2+ signalling and cell

182 the association between C-section and atopic dermatitis by age four and examine potential sources of

183 rean delivery was not associated with atopic dermatitis by age four in this large US cohort.

184 Canine atopic dermatitis (cAD) is a common hereditary clinical syndrom

185 ften suffering from an IgE-mediated allergic dermatitis caused by bites of midges (Culicoides spp).

186 tivity (IBH) in horses is a chronic allergic dermatitis caused by insect bites.

187 salinity persisting weeks to months, and (2) dermatitis characterized grossly by patchy skin pallor t

188 r cofactor in atopic diseases such as atopic dermatitis, chronic rhinosinusitis with nasal polyps, an

189 logicals for the treatment of asthma, atopic dermatitis, chronic rhinosinusitis with nasal polyps, or

190 2%), grade 2; and 1 (0.5%) grade 3 radiation dermatitis (Common Terminology Criteria for Adverse Even

191 ation is ~50% higher in children with atopic dermatitis compared to children from a high-risk allergy

192 eurons under both naive and allergic contact dermatitis conditions.

193 mice characterized by chronic proliferative dermatitis (cpdm), which is propagated by TNFR1-induced

194 ugh 3 years of age for development of atopic dermatitis; data on allergic sensitization and asthma we

195 iction tasks from ulcerative colitis, atopic dermatitis, diabetes, to many cancer subtypes for a tota

196 n a young girl being treated for both atopic dermatitis, diagnosed at 1 year of age, and bronchial as

197 diagnosed the patient with allergic contact dermatitis due to the EITC and BITC present in nitrile r

198 increase the risk of food allergy and atopic dermatitis/eczema but may prevent peanut and egg allergy

199 and the development of food allergy, atopic dermatitis/eczema, asthma, and allergic rhinitis?

200 the risk of developing food allergy, atopic dermatitis/eczema, or childhood asthma.

201 or of transcription 3 (STAT3) mutations have dermatitis, enhanced IgE production despite a relative l

202 are predisposed to mount an allergic contact dermatitis, especially at hapten threshold levels elicit

203 Guidelines on the use of dupilumab in atopic dermatitis follow the GRADE approach in formulating reco

204 link between skin barrier impairment, atopic dermatitis, food allergy, allergic asthma, and allergic

205 t- and food-allergic sensitization or atopic dermatitis had higher circulating memory Treg cells, but

206 r some chronic itch disorders such as atopic dermatitis has given widespread recognition to the impor

207 c rhinitis, asthma, food allergy, and atopic dermatitis has increased dramatically during the last de

208 reliable biomarkers in psoriasis and atopic dermatitis has lagged behind therapeutic progress, we cr

209 Research into the pathogenesis of atopic dermatitis has led to the testing and development of num

210 matory diseases, including asthma and atopic dermatitis, has established the need for effective IL-33

211 itrile rubber glove-induced allergic contact dermatitis have not been fully elucidated.

212 cosal inflammatory disorders, such as atopic dermatitis, have been associated with an impaired epithe

213 Celiac disease (CD), dermatitis herpetiformis (DH), gluten ataxia (GA), wheat

214 II deficiency, TG2 in celiac disease, TG3 in dermatitis herpetiformis, TG4 in autoimmume polyglandula

215 In atopic dermatitis, IL-19 was significantly elevated, correlated

216 Atopic dermatitis imposes a significant burden on patients, fam

217 ted in adults with moderate-to-severe atopic dermatitis in a phase 2b trial.

218 ered that MrgprA3(+) neurons respond to skin dermatitis in a way that is unique from other sensory ne

219 -based estimates on the prevalence of atopic dermatitis in adults vary widely.

220 required for the development of psoriasiform dermatitis in an IL-23 intradermal injection model.

221 versus ever being fed human milk and atopic dermatitis in childhood or 2) the duration of any human

222 ilk feeding and allergic rhinitis and atopic dermatitis in childhood.

223 = .001), a 17% greater risk of ever allergic dermatitis in children (OR, 1.17; 95% CI, 1.04-1.32; P =

224 to distinguish allergic and irritant contact dermatitis in human skin.

225 Atopic dermatitis in infancy comprises three immunological endo

226 34% greater risk of ever or current allergic dermatitis in infants up to 2 years of age (OR, 1.34; 95

227 (cpdm) , where cpdm = chronic proliferative dermatitis in mice) with and without melanoma tumor allo

228 y were to determine the prevalence of atopic dermatitis in the population of the United States, the d

229 high prevalence and disease burden of atopic dermatitis in this population.

230 a stronger inflammation in allergic contact dermatitis, indicating a regulatory role of CD163.

231 ed itch in a mouse model of allergic contact dermatitis induced by squaric acid dibutylester.

232 cted in the skin in steady-state, psoriasis, dermatitis, infection, and malignant skin diseases.

233 Information on atopic dermatitis, inhalant- and food-allergic sensitization, a

234 he individual diseases, children with atopic dermatitis, inhalant-, and food-allergic sensitization h

235 d >=12 years) with moderate-to-severe atopic dermatitis (Investigator Global Assessment score >=3, Ec

236 Atopic dermatitis is a chronic inflammatory skin disease charac

237 Atopic dermatitis is a common inflammatory skin disorder charac

238 Atopic dermatitis is a complex, chronic inflammatory skin disor

239 Chronic dermatitis is a hallmark of Dedicator of cytokinesis 8 (

240 Atopic Dermatitis is an inflammatory skin disease associated wi

241 Atopic dermatitis is associated with increased risk of multiple

242 Atopic dermatitis is dominated by a single microbe (Staphylococ

243 ood allergies, allergic rhinitis, and atopic dermatitis is limited.

244 matitis, the Patient-Oriented Scoring Atopic Dermatitis-itch, the Patient-Oriented Scoring Atopic Der

245 A distinct ulcerative dermatitis known as "freshwater skin disease" is an emer

246 ore food allergen sensitization on an atopic dermatitis-like skin lesion, followed by intragastric al

247 are more likely to have occupational contact dermatitis, mainly due to wet work.

248 The use of dupilumab on atopic dermatitis may lead to less risk of infection of skin an

249 g and inflammatory responses in mouse atopic dermatitis models.

250 t of patients with moderate-to-severe atopic dermatitis (msAD).

251 Some patients with severe dermatitis, multiple allergies, and metabolic wasting (S

252 Nickel-induced allergic contact dermatitis (nACD) remains a major occupational skin diso

253 ients with CP of inflammatory origin (atopic dermatitis), neuropathic origin (brachioradial pruritus)

254 s the most common seasonal pruritic allergic dermatitis of horses occurring upon insect bites.

255 lated ACD and 7 hairdressers without contact dermatitis on day 4 after patch testing with 1% PPD in p

256 ureus infection, including those with atopic dermatitis or cancer.

257 in 137 patients with chronic itch and atopic dermatitis or psoriasis.

258 eous gene expression in patients with atopic dermatitis or psoriasis.

259 o allergic skin inflammation, such as atopic dermatitis or urticaria, are poorly defined.

260 the association between C-section and atopic dermatitis overall and when stratified by demographic an

261 l disease (IBD) (P = 1.17 x 10-4) and eczema/dermatitis (P = 2.81 x 10-3), as well as associations be

262 ly shown that caspase-1 and -11 promoted the dermatitis pathology of cpdm mice and mediated cell deat

263 measured IL-19 in baricitinib-treated atopic dermatitis patients.

264 ity assessment tool for psoriasis and atopic dermatitis patients.

265 ated biomarkers in moderate to severe atopic dermatitis patients.

266 igh vitiligo, high psoriasis, and low atopic dermatitis polygenic risk scores (PRSs) were associated

267 Current treatments for seborrheic dermatitis provide only temporary relief.

268 ered in various dermatoses, including atopic dermatitis, psoriasis, and rosacea.

269 atment models of oxazolone- and DNFB-induced dermatitis, PZ-235 significantly attenuated skin thicken

270 showed modest efficacy and decreased atopic dermatitis-related biomarkers in moderate to severe atop

271 A biopsy substudy evaluated atopic dermatitis-related biomarkers.

272 l phenotype of irritant and allergic contact dermatitis remains challenging.

273 re radiation-induced oral mucositis and neck dermatitis, respectively.

274 was evidence that C-section increased atopic dermatitis risk among certain subgroups (eg firstborns,

275 ive association between C-section and atopic dermatitis [RR(95%CI): 1.06(1.03, 1.10)], this effect wa

276 Stasis dermatitis (SD) is a common disease in the elderly popul

277 is-itch, the Patient-Oriented Scoring Atopic Dermatitis-sleep, and the Numerical Rating Scale of pain

278 TMEM79 is a predisposition gene for Atopic dermatitis, suggesting deregulation of Wnt/FZD signaling

279 atified approach to the management of atopic dermatitis, supporting the use of targeted treatments wi

280 ntibody (dupilumab) on two cases with atopic dermatitis that was refractory to conventional managemen

281 rophils are key initiators of itch in atopic dermatitis, the most prevalent chronic itch disorder.

282 Measure, the Patient-Oriented Scoring Atopic Dermatitis, the Patient-Oriented Scoring Atopic Dermatit

283 tinctly representing a switch from an atopic dermatitis to a healthy skin phenotype.

284 to shift the ceramide profile from an atopic dermatitis to a healthy skin phenotype.

285 n primary and secondary prevention of atopic dermatitis to achieve the desired outcome.

286 AD, the Mechanisms of Progression of Atopic Dermatitis to Asthma in Children (MPAACH).

287 Contact dermatitis tremendously impacts the quality of life of s

288 evalence (95% confidence interval) of atopic dermatitis was 7.3% (5.9-8.8).

289 sociated with anxiety and depression, atopic dermatitis was associated with suicidal ideation, and th

290 cal diagnosis of asthma, rhinitis and atopic dermatitis was retrieved for each participant.

291 The rate of contact dermatitis was similar (0.5% vs. 0.5%; P = 1.00), as was

292 om patients with DOCK8 deficiency and atopic dermatitis were profiled on a cytokine/chemokine panel f

293 t intake was associated with less seborrheic dermatitis, whereas high adherence to a "Western" dietar

294 biopsies from patients and mice with atopic dermatitis, whereas their inhibition attenuated scratchi

295 levels are increased in patients with atopic dermatitis, which commonly precedes asthma in the atopic

296 KLK6 causes generalized, severe psoriasiform dermatitis with spontaneous development of debilitating

297 lion adults would have a diagnosis of atopic dermatitis, with 6.6 million meeting criteria for modera

298 nt cause of T cell-mediated allergic contact dermatitis worldwide.

299 in barrier and prevention or treating atopic dermatitis would have beneficial effects on prevention o

300 olybacterial bovine wound infection 'digital dermatitis', Zn/Cu-shellac adhered rapidly and robustly