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1 accuracy compared to urodynamically-observed detrusor overactivity.
2 act symptoms/overactive bladder syndrome/and detrusor overactivity.
3 rusor overactivity and those with idiopathic detrusor overactivity.
4 gaining importance in the pathophysiology of detrusor overactivity.
5  neurogenic and probably also non-neurogenic detrusor overactivity.
6  by targeting alternative pathways affecting detrusor overactivity.
7 guidelines recommend urodynamics to identify detrusor overactivity.
8 ave transformed the management of neurogenic detrusor overactivity.
9                      Approximately half have detrusor overactivity and a smaller number have impaired
10 se results suggest that constipation induced detrusor overactivity and increased excitatory serotonin
11  established in the management of refractory detrusor overactivity and overactive bladder.
12 urrent therapeutic alternatives for managing detrusor overactivity and possible future developments a
13 wn to increase cystometric capacity, inhibit detrusor overactivity and resolve overactive bladder sym
14  treatment, both in patients with neurogenic detrusor overactivity and those with idiopathic detrusor
15 ive in reducing incontinence associated with detrusor overactivity, and repeated treatments appear sa
16 nary incontinence, overflow incontinence and detrusor overactivity are the major categories of urinar
17            Bladders of DKO animals exhibited detrusor overactivity at an early stage: increased frequ
18 mechanism that prostatic inflammation causes detrusor overactivity by using a rat model of chemically
19  injury (SCI)-induced bladder dysfunction is detrusor overactivity (DO).
20 he cystitis occurring in patients, including detrusor overactivity (DO).
21 abetic bladder dysfunction, characterized by detrusor overactivity during the early stage of the dise
22 veloped LUTS including urinary frequency and detrusor overactivity evaluated by awake cystometry.
23 with pressure-flow urodynamics demonstrating detrusor overactivity, in the setting of a clinically re
24                                              Detrusor overactivity is a relatively common yet embarra
25  in our understanding of the pathogenesis of detrusor overactivity is slow but steady.
26                                              Detrusor overactivity is the occurrence of abnormal incr
27                                   Neurogenic detrusor overactivity (NDO) is a well known consequence
28                                   Neurogenic detrusor overactivity (NDO) is among the most challengin
29  multiple sclerosis often develop neurogenic detrusor overactivity (NDO), which currently lacks a uni
30 onventional treatments like drug therapy for detrusor overactivity or sling procedures for female str
31                                              Detrusor overactivity poses a major challenge to physici
32 tract dysfunction, especially for those with detrusor overactivity refractory to anticholinergics, is
33                              We propose that detrusor overactivity results from exaggerated symptomat
34 r hypersensitivity as well as non-neurogenic detrusor overactivity, there is up-regulation of unmyeli
35 ts acting on alternative pathways underlying detrusor overactivity with the intention of improving st