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1 therapy (triamcinolone, bevacizumab, and/or dexamethasone).
2 ase inhibitor ouabain), and corticosteroids (dexamethasone).
3 olerated dose of melflufen 40 mg plus weekly dexamethasone.
4 enetoclax versus placebo plus bortezomib and dexamethasone.
5 ntly induced by the synthetic corticosteroid dexamethasone.
6 pha, IL-6, and IL-8 at a similar efficacy to dexamethasone.
7 ed in combination with alkylating agents and dexamethasone.
8 global changes in gene expression induced by dexamethasone.
9 of isatuximab with pomalidomide and low-dose dexamethasone.
10 id beta42 oligomers, lipopolysaccharides, or dexamethasone.
11 neurons following explant when treated with dexamethasone.
12 d dexamethasone compared with bortezomib and dexamethasone.
13 and were increased by the aromatase inducer dexamethasone.
14 tly higher levels of virus when treated with dexamethasone.
15 ntiation in the presence of interleukin 4 or dexamethasone.
16 esistant to the anti-inflammatory effects of dexamethasone.
17 omide-dexamethasone, and 153 to pomalidomide-dexamethasone.
18 viral regulatory proteins was stimulated by dexamethasone.
19 e was 40 mg of melflufen in combination with dexamethasone.
20 eekly, plus subcutaneous bortezomib and oral dexamethasone.
21 0 (95% CI, 1.09-4.87) for ciprofloxacin plus dexamethasone.
22 s, including TGF-beta signaling, affected by dexamethasone.
23 e that reflected CFS, rescue medication use (dexamethasone 0.1% 4 times daily), and corneal ulceratio
25 15; lenalidomide 15 mg orally on days 1-21; dexamethasone 12 mg orally on days 1, 18, and 15 every 2
26 entration of EDCs detected in the muscle was dexamethasone (2.37-15.84 ng/g) and (0.77-13.41 ng/g), i
27 t 24 weeks and once per week thereafter) and dexamethasone (20 mg four times per week for the first 2
28 on days 1, 4, 8, and 11 every 21 days), and dexamethasone (20 mg orally on days 1, 2, 4, 5, 8, 9, 11
30 bortezomib (1.3 mg/m(2) once per week), and dexamethasone (20 mg twice per week), or bortezomib (1.3
31 We administered oral selinexor (80 mg) plus dexamethasone (20 mg) twice weekly to patients with myel
32 tuximab 10 mg/kg plus pomalidomide 4 mg plus dexamethasone 40 mg (20 mg for patients aged >=75 years)
33 de 4 mg daily on days 1-21 and oral low-dose dexamethasone 40 mg on days 1, 8, 15, and 22 in 28-day c
34 ral lenalidomide 25 mg on days 1-21 and oral dexamethasone 40 mg on days 1, 8, 15, and 22 of repeated
35 r 30 min on day 1 in 21-day cycles plus oral dexamethasone 40 mg weekly and did not receive melflufen
38 ess frequent with selinexor, bortezomib, and dexamethasone (41 [21%] patients) than with bortezomib a
39 (41 [21%] patients) than with bortezomib and dexamethasone (70 [34%] patients; odds ratio 0.50 [95% C
40 trial, however, revealed that treatment with dexamethasone, a classic synthetic glucocorticoid, enhan
45 domide and dexamethasone or lenalidomide and dexamethasone alone using an interactive voice or integr
47 ciently as a synthetic glucocorticoid (e.g., Dexamethasone) an induced skin atopic dermatitis, an ind
48 s were given a combination of melflufen plus dexamethasone and 13 patients were given single-agent me
49 st one dose of pomalidomide, bortezomib, and dexamethasone and 270 patients received at least one dos
51 t evaluable) with selinexor, bortezomib, and dexamethasone and 9.46 months (8.11-10.78) with bortezom
52 lease like progesterone, ZnSO(4), quercetin, dexamethasone and apomorphine were active in models of A
53 poorer tolerance to chemotherapy, especially dexamethasone and asparaginase, and have increased risk
55 Interestingly, the synthetic corticosteroid dexamethasone and GR or KLF15 alone had little effect on
56 es using lenalidomide and/or bortezomib with dexamethasone and HDIVig led to a significant improvemen
57 asone or cyclophosphamide, lenalidomide, and dexamethasone) and achieved a partial or minimal respons
58 to treatment: 154 to isatuximab-pomalidomide-dexamethasone, and 153 to pomalidomide-dexamethasone.
59 a were randomly assigned 2:1 to carfilzomib, dexamethasone, and daratumumab (KdD) or carfilzomib and
60 pare the efficacy and safety of carfilzomib, dexamethasone, and daratumumab versus carfilzomib and de
62 ginase intensification, the use of induction dexamethasone, and the safe omission of cranial radiothe
63 received at least one dose of bortezomib and dexamethasone, and these patients were included in safet
64 izumab recommended that all patients receive dexamethasone as a component of the prophylactic antieme
65 upports use of pomalidomide, bortezomib, and dexamethasone as a treatment option in patients with rel
66 n multiple myeloma, both in combination with dexamethasone as well as in triplet regimens with additi
67 proval of the XPO1 inhibitor selinexor (plus dexamethasone) as a fifth-line therapy for patients with
68 ) genotype influenced the clinical impact of dexamethasone, as observed in tuberculous meningitis.
69 gned patients to receive oral or intravenous dexamethasone (at a dose of 6 mg once daily) for up to 1
70 randomly assigned to receive bortezomib plus dexamethasone (BD), or BD plus cyclophosphamide (C-BD).
71 the efficacy and safety of bendamustine with dexamethasone (ben-dex) in patients with persistent or p
74 peripheral blood mononuclear cells, MPA and dexamethasone, but not NET-A, upregulated (median [inter
76 tients received bortezomib, doxorubicin, and dexamethasone chemotherapy, and were then followed up fo
78 ced antileukemic activity by quizartinib and dexamethasone combination has been validated using prima
79 n treated with pomalidomide, bortezomib, and dexamethasone compared with bortezomib and dexamethasone
80 benefit of isatuximab plus pomalidomide and dexamethasone compared with pomalidomide and dexamethaso
83 ated with cyclophosphamide, thalidomide, and dexamethasone (CTD) (22.5% [n = 121 of 538] vs 16.1% [n
84 lophosphamide, vincristine, doxorubicin, and dexamethasone (CVAD) induction had higher risk of VTE co
85 tment with cyclophosphamide, bortezomib, and dexamethasone (CVD) versus no intensification treatment
86 yloidosis, cyclophosphamide, bortezomib, and dexamethasone (CyBorD) is considered standard of care.
87 aratumumab plus bortezomib, thalidomide, and dexamethasone (D-VTd) significantly improved rates of st
88 one intravenously daily for 5 days, 10 mg of dexamethasone daily for 5 days or until ICU discharge, p
89 to receive daratumumab plus lenalidomide and dexamethasone (daratumumab group) or lenalidomide and de
90 ith advanced AL receiving either daratumumab/dexamethasone (DD, n = 106) or daratumumab/bortezomib/de
91 We investigated interactions between the GC dexamethasone (DEX) and the bone resorptive agents 1,25(
92 erapy for HLH consists of the glucocorticoid dexamethasone (DEX) and the chemotherapeutic agent etopo
93 eceptor (GR) by the synthetic corticosteroid dexamethasone (DEX) stimulates bovine herpesvirus 1 (BoH
94 ted and divided randomly into control (C) or dexamethasone (Dex) treatment at day 14 out of the 21-da
95 For testing, pregnant mice were administered dexamethasone (DEX), a synthetic GC, in the last trimest
96 ma cell line BWTG3, as evidenced by impaired Dexamethasone (Dex)-stimulated direct GR-dependent gene
99 feasibility of this strategy, five different dexamethasone (Dex, a potent GC)-containing monomers wit
100 study, in human NK cells, the glucocorticoid dexamethasone downregulated LIMK expression, F-actin acc
102 ipants received aprepitant, ondansetron, and dexamethasone during and 2 days after chemotherapy.
103 Among the 45 patients given melflufen plus dexamethasone during phase 2, the most common grade 3-4
107 17beta-estradiol equivalents/L)-, GR (60 ng dexamethasone equivalents/L)-, and PPARgamma-mediated ac
109 reated by treating rats with zoledronate and dexamethasone, extracting teeth, and immediately injecti
110 d, double-blind, placebo-controlled trial of dexamethasone for patients with a symptomatic chronic su
113 no clinical evidence to warrant omission of dexamethasone from guideline-compliant prophylactic anti
114 d in the pembrolizumab plus lenalidomide and dexamethasone group (cardiac arrest, cardiac failure, my
116 ther the pembrolizumab plus pomalidomide and dexamethasone group (n=125) or the pomalidomide and dexa
118 d to the pembrolizumab plus lenalidomide and dexamethasone group (n=151) or the lenalidomide and dexa
119 d in the pembrolizumab plus pomalidomide and dexamethasone group (one each of unknown cause, neutrope
120 up (sepsis) and two (1%) in the pomalidomide-dexamethasone group (pneumonia and urinary tract infecti
121 patient (<1%) in the isatuximab-pomalidomide-dexamethasone group (sepsis) and two (1%) in the pomalid
122 bolism) and two (1%) in the lenalidomide and dexamethasone group (upper gastrointestinal haemorrhage
124 patients (8%) in the isatuximab-pomalidomide-dexamethasone group and 14 (9%) in the pomalidomide-dexa
125 6.2-19.8] for the selinexor, bortezomib, and dexamethasone group and 16.5 months [9.4-19.8] for the b
126 -195 (49%) to the selinexor, bortezomib, and dexamethasone group and 207 (51%) to the bortezomib and
128 ) patients in the selinexor, bortezomib, and dexamethasone group and 62 (30%) in the bortezomib and d
129 performed in 6 of 349 patients (1.7%) in the dexamethasone group and in 25 of 350 patients (7.1%) in
130 ported in 286 of 341 patients (83.9%) in the dexamethasone group and in 306 of 339 patients (90.3%) i
131 curred in 278 of 1417 infants (19.6%) in the dexamethasone group and in 331 of 1406 infants (23.5%) i
132 ) in the pembrolizumab plus lenalidomide and dexamethasone group and pneumonia (eight [6%]) and sepsi
136 1 to the pembrolizumab plus pomalidomide and dexamethasone group or the pomalidomide and dexamethason
139 s in the pembrolizumab plus pomalidomide and dexamethasone group versus 56 (46%) of 121 patients in t
140 s in the pembrolizumab plus lenalidomide and dexamethasone group versus 57 (39%) patients in the lena
141 CI 8.9-13.9) in the isatuximab-pomalidomide-dexamethasone group versus 6.5 months (4.5-8.3) in the p
142 ) in the pembrolizumab plus pomalidomide and dexamethasone group versus 8.4 months (5.9-not reached)
143 dexamethasone group or the pomalidomide and dexamethasone group via an interactive voice response or
144 5 patients in the selinexor, bortezomib, and dexamethasone group vs 35 [17%] of 204 in the bortezomib
146 oup vs 35 [17%] of 204 in the bortezomib and dexamethasone group), fatigue (26 [13%] vs two [1%]), an
148 ne group and 207 (51%) to the bortezomib and dexamethasone group-and the first dose of study medicati
155 sus 6.5 months (4.5-8.3) in the pomalidomide-dexamethasone group; hazard ratio 0.596, 95% CI 0.44-0.8
156 hs (5.9-not reached) in the pomalidomide and dexamethasone group; progression-free survival estimates
157 us 57 (39%) patients in the lenalidomide and dexamethasone group; the most common serious adverse eve
162 9.46 months (8.11-10.78) with bortezomib and dexamethasone (hazard ratio 0.70 [95% CI 0.53-0.93], p=0
165 ctive review of 640 consecutive intravitreal dexamethasone implant injections was conducted from Febr
169 se patients who experienced anterior chamber dexamethasone implant migrations were identified, as wel
170 d to evaluate the effect of the intravitreal dexamethasone implant Ozurdex(R) in patients with differ
171 d DME, primary therapy with the intravitreal dexamethasone implant PRN (for 2 years) resulted in sign
174 different response patterns to intravitreal dexamethasone implants (IDI) in naive and previously tre
175 ear positive relationship with the number of dexamethasone implants before FAc implant (P = .002, R(2
176 mg/m(2) intravenously) with 40 mg of weekly dexamethasone in 28-day cycles until disease progression
177 fety profile of isatuximab plus pomalidomide/dexamethasone in heavily pretreated patients with RRMM.
178 dexamethasone versus standard bortezomib and dexamethasone in patients with previously treated multip
179 We did a phase 1-2 study of melflufen and dexamethasone in patients with relapsed and refractory m
180 dexamethasone compared with pomalidomide and dexamethasone in patients with relapsed and refractory m
181 sone, and daratumumab versus carfilzomib and dexamethasone in patients with relapsed or refractory mu
182 s to evaluate venetoclax plus bortezomib and dexamethasone in patients with relapsed or refractory mu
183 study evaluated isatuximab plus pomalidomide/dexamethasone in patients with relapsed/refractory multi
184 ial evaluated the efficacy of melflufen plus dexamethasone in relapsed and refractory multiple myelom
185 reparation and data analysis, the amounts of dexamethasone in RHS, detected and quantified by STXM an
186 he only inputs are concentration profiles of dexamethasone in skin at three consecutive penetration t
188 nterferon-gamma antibody), administered with dexamethasone, in an open-label, single-group, phase 2-3
189 -2 doses, both alone and in combination with dexamethasone, increased the proportion of single IL-10(
190 with bortezomib (a proteasome inhibitor) and dexamethasone induced high response rates with low rates
192 omoted myotube differentiation and prevented dexamethasone-induced atrophy in myotubes in vitro.
195 in base subunit 1 (SPTLC1)/SPTLC2, decreased dexamethasone-induced plasma and liver triglyceride leve
198 ere significantly associated with changes in dexamethasone-induced transrepression of NF-kappaB.
199 yclopentanedione (ACPD), had lower levels of dexamethasone-induced triglyceride accumulation in plasm
200 R activation by the synthetic corticosteroid dexamethasone influenced virus shedding during reactivat
202 the penetration of the antiinflammatory drug dexamethasone into the skin layers by the one-dimensiona
203 a for retinitis pigmentosa, Dextenza (0.4 mg dexamethasone intracanalicular insert) for ocular inflam
205 Authority of India to examine the safety of dexamethasone intravitreal (DEX) implant over 1 year in
206 ated with different alternatives, among them Dexamethasone intravitreal implant 0.7 mg (DEX 0.7) has
207 r-week regimen of selinexor, bortezomib, and dexamethasone is a novel, effective, and convenient trea
208 sensitivity of erythroid differentiation to dexamethasone is dependent on the developmental origin o
210 ofile of pembrolizumab plus pomalidomide and dexamethasone is unfavourable for patients with relapsed
212 In ENDEAVOR, carfilzomib (56 mg/m(2)) and dexamethasone (Kd56) demonstrated longer progression-fre
213 ation (ASCT) into a carfilzomib-lenalidomide-dexamethasone (KRd) regimen for patients with newly diag
215 ibitor, in combination with lenalidomide and dexamethasone (KRd), has shown promising efficacy in pha
216 .78 ng/g), and in the reproductive organ are dexamethasone (<2.54-37.23 ng/g) and caffeine (0.21-18.9
217 g/g) and (0.77-13.41 ng/g), in the liver was dexamethasone (<2.54-43.56 ng/g) and progesterone (2.23-
220 n-free survival compared with bortezomib and dexamethasone (median 11.20 months [95% CI 9.66-13.73] v
221 Akt1 and Ak2, but not Akt3, impaired GR- and dexamethasone-mediated transactivation of the BoHV-1 imm
223 lenalidomide on days 1-14, and 20 mg of oral dexamethasone on days 1, 2, 4, 5, 8, 9, 11, and 12.
224 to 21 in each cycle, and oral or intravenous dexamethasone on days 1, 8, 15, and 22 of each cycle.
226 Kip1, significantly attenuated the impact of dexamethasone on erythroid differentiation and inhibited
227 a, to evaluate the effect of MPA, NET-A, and dexamethasone on Mtb containment in monocyte-derived mac
229 kedly refractory to the inhibitory effect of dexamethasone on proliferation and IL-2Ralpha expression
230 lophosphamide, vincristine, doxorubicin, and dexamethasone) on courses 1, 3, 5, and 7 alternating wit
231 protocol (cyclophosphamide, thalidomide, and dexamethasone or cyclophosphamide, lenalidomide, and dex
233 e either pembrolizumab plus lenalidomide and dexamethasone or lenalidomide and dexamethasone alone us
234 ipants were randomised to a 2-week course of dexamethasone or placebo in addition to receiving standa
235 ect the effects of illegal administration of dexamethasone or prednisolone or 17beta-estradiol on Cha
236 logical blockade of ER stress in vitro using dexamethasone or the chemical chaperones TUDCA and 4PBA
238 After symptomatic measures and high-dose dexamethasone, patients were randomly assigned to receiv
239 moderate or severe ARDS, use of intravenous dexamethasone plus standard care compared with standard
240 rted in patients who received bortezomib and dexamethasone (pneumonia [n=1], hepatic encephalopathy [
241 s who received pomalidomide, bortezomib, and dexamethasone (pneumonia [n=2], unknown cause [n=2], car
242 CR-like" signaling with multiple TKIs and/or dexamethasone prevented this signaling plasticity and in
243 echanisms of harm and test 2 hypotheses: (1) dexamethasone reduced proinflammatory cytokine concentra
246 lytic PP2A subunit (Ppp2ca) had no effect on dexamethasone responses on plasma and liver triglyceride
247 s during the index admission, treatment with dexamethasone resulted in fewer favorable outcomes and m
248 ients hospitalized with Covid-19, the use of dexamethasone resulted in lower 28-day mortality among t
250 ay is GC-responsive since treating mice with dexamethasone resulted in reduced muscle expression of m
251 at risk for early preterm birth, the use of dexamethasone resulted in significantly lower risks of n
252 C.I. 1.14-1.39), lower doses of concomitant dexamethasone (RR: 1.36; 95% C.I. 1.02-1.81), not receiv
254 combination of lenalidomide, bortezomib, and dexamethasone (RVD) is a highly effective and convenient
255 ur subsequent cycles of len, bortezomib, and dexamethasone (RVD; AHCT + RVD), all followed by len unt
259 The addition of isatuximab to pomalidomide-dexamethasone significantly improves progression-free su
260 this study, we show that the glucocorticoid dexamethasone significantly increased IL-10 production b
261 to receive a 2-week tapering course of oral dexamethasone, starting at 8 mg twice daily, or placebo.
263 emonstrate that the synthetic corticosteroid dexamethasone stimulated explant-induced reactivation fr
264 profile, became phagocytic, and responded to dexamethasone stimulation, characteristic of TM cells.
266 o received daratumumab plus lenalidomide and dexamethasone than among those who received lenalidomide
270 vealed the induction of distinct proteins in dexamethasone-treated PB and CB erythroid progenitors.
271 These findings, therefore, demonstrate that dexamethasone treatment can have direct detrimental off-
272 le isotope-labeling technique, we found that dexamethasone treatment induced the rate of hepatic de n
273 Overall, our results indicate that chronic dexamethasone treatment induces an ANGPTL4-ceramide-PKCz
277 that interleukin-1 signalling inhibition and dexamethasone treatment share therapeutic efficacies and
278 Perturbation of Schlemm's canal cells with dexamethasone treatment, alpha-actinin overexpression, o
283 sion-free survival (PFS) over bortezomib and dexamethasone (Vd) in patients with relapsed/refractory
284 benefit of weekly selinexor, bortezomib, and dexamethasone versus standard bortezomib and dexamethaso
285 ll patients received a 3-drug induction with dexamethasone, vincristine, and pegaspargase (PEG) and w
287 e events (any grade; isatuximab-pomalidomide-dexamethasone vs pomalidomide-dexamethasone) were infusi
288 survival versus bortezomib, thalidomide, and dexamethasone (VTd) in patients with newly diagnosed mul
294 b-pomalidomide-dexamethasone vs pomalidomide-dexamethasone) were infusion reactions (56 [38%] vs 0),
295 utine use; and corticosteroids, specifically dexamethasone, were recommended for temporary symptomati
296 xorubicin, ABT-199 (a Bcl-2 antagonist), and dexamethasone when tested on hematological cancer cells.
298 the efficacy and safety of lenalidomide and dexamethasone with and without pembrolizumab in patients
299 ssed efficacy and safety of pomalidomide and dexamethasone with or without pembrolizumab in patients
300 ly assigned patients (1:1) to bortezomib and dexamethasone with or without pomalidomide using a permu