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1 ere increased post-repair, especially during diastolic filling.
2  in RV systolic function and RV and LV early-diastolic filling.
3 tion, delayed untwisting, and impaired early diastolic filling.
4 , critically linking systolic contraction to diastolic filling.
5 uscle stiffness; an important determinant of diastolic filling.
6 f myocardial oxygen consumption and impaired diastolic filling.
7 consequence of left heart anatomy and aid LV diastolic filling.
8 ad in patients with CTEPH causes abnormal LV diastolic filling.
9 systolic ejection time, which may compromise diastolic filling.
10 ay be an increase in stretch due to enhanced diastolic filling.
11 r valve disease, due to impaired ventricular diastolic filling.
12  implantable ventricular assist device on LV diastolic filling.
13 ents stored energy that may facilitate early diastolic filling.
14 oppler echocardiography in the assessment of diastolic filling.
15  after transplantation with respect to RV/LV diastolic filling.
16  < 0.0001), increased atrial contribution to diastolic filling (0.47 +/- 0.09 vs. 0.30 +/- 0.08 m/s,
17   LV systolic function (S' z score) and late-diastolic filling (A' z score) improved to normal in 11
18 rast, captopril almost completely normalized diastolic filling abnormalities (E velocity 82 +/- 5 cm/
19                                              Diastolic filling abnormalities after pericardiectomy co
20 e of subclinical contractile dysfunction and diastolic filling abnormalities are both predictive of s
21 l systolic function, elevated BNP levels and diastolic filling abnormalities might help to reinforce
22 nction but markedly improves the restrictive diastolic filling abnormalities that are seen in untreat
23 ed in left ventricular systolic function and diastolic filling after exercise in the control groups.
24 pairs left ventricular systolic function and diastolic filling after exercise.
25 duced ischemia impairs systolic function and diastolic filling after exercise.
26 nce, but the viscoelastic forces that resist diastolic filling and become elevated in human HF are po
27 w, which should improve the understanding of diastolic filling and function of the heart.
28 arked by the hemodynamic pattern of impaired diastolic filling and restrictive cardiomyopathy.
29 on-invasive MRI of early, chronic changes in diastolic filling and systolic ejection in both the left
30 the passive elastance of the RV walls during diastolic filling and the active elastance produced by t
31 ties during early (E wave) and late (A wave) diastolic filling, and myocardial performance index.
32 ties during early (E-wave) and late (A-wave) diastolic filling, and the myocardial performance index.
33 s with MI showed progressively restricted LV diastolic filling as assessed by transmitral Doppler rec
34 s in RT-CMR left ventricular total and early diastolic filling at rest and during exercise stress (P
35 sional transmural strains during systole and diastolic filling, at 1 and 12 weeks postoperatively.
36 rly diastolic filling rate [E wave] and late diastolic filling [atrial contraction] rate [A wave]) ti
37                                  arametrized diastolic filling-based E-wave analysis (k, c or DTs and
38  likely to impact functional transitions and diastolic filling behavior during development of the hea
39                    Noninvasive assessment of diastolic filling by Doppler echocardiography provides i
40  dilated cardiomyopathy significantly impact diastolic filling by lowering myocardial stiffness.
41 during beta-adrenergic stimulation to defend diastolic filling by means of an increased cardiomyocyte
42 se of abnormal left ventricular (LV) Doppler diastolic filling characteristics in chronic thromboembo
43  pressure overload and left ventricular (LV) diastolic filling characteristics in patients with chron
44                                              Diastolic filling characteristics remain abnormal in a s
45 s, preserved ejection fraction, and impaired diastolic filling, characterized by reduced deceleration
46 dance of dTyr-MT in the myocardium, improved diastolic filling, compliance, cardiac output, and strok
47                                        Early diastolic filling decreased after four days of pacing fr
48 r hypertension, E/A, a diagnostic measure of diastolic filling, decreases, and isovolumic relaxation
49 3 mm Hg; P=0.0007), suggesting inadequate LV diastolic filling, despite high pulmonary capillary wedg
50 ults provide a standard reference concerning diastolic filling dynamics by trimester.
51 d to identify abnormal left ventricular (LV) diastolic filling dynamics, inherent limitations suggest
52 c function (S' z score -2.7+/-0.8), RV early-diastolic filling (E' z score -2.3+/-1.1), and LV early-
53 illing (E' z score -2.3+/-1.1), and LV early-diastolic filling (E' z score -2.3+/-1.1).
54 mproved to normal in 11 to 30 days, LV early-diastolic filling (E' z score) in 4 to 6 months, and RV
55 The peak rate of left ventricular (LV) early diastolic filling (E) and velocity of the mitral annulus
56 ales, but abnormal fractional shortening and diastolic filling (E/A ratio) patterns were more common
57  IHH (P > 0.236), whereas the passive/active diastolic filling (E/A) ratio was reduced (P = 0.022), p
58     Isovolumic relaxation time (IVRT), early diastolic filling (E/A), myocardial performance index (M
59 an energy-saving/efficient mode: it improves diastolic filling (enhanced lusitropy - lowering HRT), m
60               It is associated with impaired diastolic filling, even in seemingly asymptomatic obese
61 function was quantified via the parametrized diastolic filling formalism that yields relaxation/visco
62 r) by analyzing E-waves via the parametrized diastolic filling formalism.
63 ection fraction, cardiac output, heart rate, diastolic filling function, and ventricular mass.
64 tic resonance spectroscopy, left ventricular diastolic filling (heart rate normalized time to peak fi
65  (E' z score) in 4 to 6 months, and RV early-diastolic filling in 6 to 9 months (P<0.001 for all on l
66 lated work was associated with greater early diastolic filling in men only.
67 me (ACE) inhibition on left ventricular (LV) diastolic filling in postinfarction heart failure.
68 bnormalities after the race included altered diastolic filling, increased pulmonary pressures and rig
69                These results suggest that RV diastolic filling is altered in young adults who were bo
70                           This impairment in diastolic filling is related to both abnormalities of th
71                     Peak velocities of early diastolic filling, late diastolic filling, late to early
72  velocities of early diastolic filling, late diastolic filling, late to early filling ratio, decelera
73 cs of the cardiac left ventricle (LV) during diastolic filling may play a critical role in dictating
74 ansmural gradients of oxygen consumption and diastolic filling, may play a role in the cycle of the f
75 ion, we hypothesized that improvements in LV diastolic filling might contribute to the overall benefi
76 le of echocardiography in the "Evaluation of Diastolic Filling of Left Ventricle in Health and Diseas
77                                              Diastolic filling of the left ventricle (E/A ratio) was
78 ied approach to understanding the process of diastolic filling of the left ventricle and interpreting
79 multiple interrelated factors that determine diastolic filling of the left ventricle, these flow velo
80 d augmentation in lusitropy, indicating that diastolic filling of the right heart is not passive.
81 s a comprehensive, noninvasive evaluation of diastolic filling of the ventricle, myocardial relaxatio
82            Patients with persistent abnormal diastolic filling on Doppler echocardiography had had sy
83                                              Diastolic filling pattern at late follow-up was normal i
84                                              Diastolic filling pattern itself influences chamber pres
85 There was a significant relationship between diastolic filling patterns and symptomatic status (chi2
86                                              Diastolic filling patterns at rest were assessed by Dopp
87 raphy has been used to examine the different diastolic filling patterns of the left ventricle in heal
88                                              Diastolic filling patterns were also similar.
89 s with symptoms had higher BNP levels in all diastolic filling patterns.
90 ex >33 ml/m(2), ratio of mitral inflow early diastolic filling peak velocity to early diastolic mitra
91 g ratio, deceleration time of early filling, diastolic filling period and atrial filling fraction wer
92                   At similar declines in the diastolic filling period, end-diastolic volume index (ED
93 delity catheters to compare left ventricular diastolic filling pressures (pre-A wave left ventricular
94               Elevated left ventricular (LV) diastolic filling pressures represent a key therapeutic
95  blood pressure (-1 vs. +7 mm Hg), and early diastolic filling rate (+1.7 vs. +2.4 end-diastolic volu
96 V ejection fraction (p = 0.006) and LV early diastolic filling rate (p = 0.001), which decreased over
97 articipants at systolic and diastolic (early diastolic filling rate [E wave] and late diastolic filli
98            The only gender difference was in diastolic filling rate, which was similar in the young m
99 a, LV longitudinal strain rate, and LV early diastolic filling rate.
100 ongitudinal strain, diastolic-strain rate or diastolic-filling rate.
101 During 20-W exercise, lower left ventricular diastolic filling rates (r=0.58; P<0.001), lower left ve
102  cardiac parameters, including reduced early diastolic filling rates as well as a higher prevalence o
103 al relaxation, pseudonormal, and restrictive diastolic filling, respectively.
104       Heart rate, BP, systolic function, and diastolic filling responses to parasympathetic withdrawa
105 ved for measures of LV systolic function and diastolic filling (RHO from 0.71 to 0.57).
106 ventional echocardiographic and parametrized diastolic filling stiffness (k) and relaxation (c) param
107                  The changes in longitudinal diastolic filling strains between 1 and 12 weeks, howeve
108 ns beat by beat in each patient, but overall diastolic filling tended to normalize with an increase o
109 gnose and treat the various abnormalities of diastolic filling that occur in patients with heart dise
110 1), together with a significant reduction in diastolic filling time (377 +/- 138 ms to 300 +/- 118 ms
111 locities, E/A ratio, deceleration time [DT], diastolic filling time [DFT], and isovolumic relaxation
112 n in relation to right ventricular ejection, diastolic filling time as a ratio of cycle length, and s
113 EF are typically exertional, optimization of diastolic filling time by controlling heart rate may del
114 7), isovolumic relaxation time increased and diastolic filling time was shortened.
115                                              Diastolic filling variables obtained with the SPECT soft
116 ucing the mitral ratio of peak early to late diastolic filling velocity (E/A).
117 dices reflective of atrial pressures and the diastolic filling volume.
118                                              Diastolic filling was determined by a simulated atrial p
119 ntly, the atrial contribution to ventricular diastolic filling was substantially higher in the YG whe
120            Prolongation of transmitral early diastolic filling wave deceleration time was associated
121 eudonormal, and restrictive left ventricular diastolic filling were associated with hazard ratios of

 
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