ライフサイエンスコーパス: diastolic volume

1 nulus, E/E' septal annulus, left ventricular diastolic volume).

2 adjustment for baseline left ventricular end-diastolic volume.

3 e of 15% or more in the left ventricular end-diastolic volume.

4 ed with the postoperative decrease in LV end-diastolic volume.

5 iated with greater RV mass and larger RV end-diastolic volume.

6 ump failure revealed a rapid decrease in end diastolic volume.

7 left ventricular end-systolic volume and end-diastolic volume.

8 fraction, and indexed right ventricular end-diastolic volume.

9 ncing the saline-mediated increase in LV end-diastolic volume.

10 Allogeneic MSCs reduced LV end-diastolic volumes.

11 s associated with lower left ventricular end-diastolic volume (0.01 mm/mL; P<0.01), a lower left vent

12 tio of elevated left ventricular mass to end-diastolic volume (0.02 g/mL per unit).

13 s of end-systolic volume (0 +/- 3.3 ml), end-diastolic volume (- 0.4 +/- 2.0 ml) and ejection fractio

14 as percent reduction in left ventricular end-diastolic volume 1 year after CRT-D implantation) and to

15 with stable LV chamber volumes (DeltaLV end-diastolic volume, 1+/-1 mL/m(2); P=0.48).

16 women exhibited RV cavity dilatation (RV end-diastolic volume, +1.0 mL per BMI point increase; P<0.00

17 ated right ventricles (right ventricular end-diastolic volume=101+/-26 mL/m(2)) with good systolic fu

18 and 65+/-4%; P<0.05), greater indexed LV end-diastolic volume (102+/-34 versus 84+/-14 and 85+/-16 mL

19 t associated with changes in LV volumes (end-diastolic volume 106 +/- 15 versus 110 +/- 22 mL; end-sy

20 ared with patients with GLS</=-15.0% (LV end-diastolic volume 123+/-44 versus 106+/-36 mL and 121+/-4

21 and increased echocardiographic volumes (end-diastolic volume, 126+/-39 versus 112+/-33 mL/BSA(1.3),

22 e was mild with significant decreases in end-diastolic volume (139 to 107 mL; P=0.03) and left atrial

23 A significant reduction of LV end-diastolic volume (149.7 +/- 41.4-140.1 +/- 43.9 mL; P=0.

24 of exercise training (LA volumes 55%; LV end diastolic volumes 15% at 24 months versus baseline; P<0.

25 s a larger increase in right ventricular end-diastolic volume (15 mL; 95% confidence interval, 3-28 m

26 rized by dilation of the left ventricle (end-diastolic volume, 156+/-26 versus 172+/-28 mL, P<0.001),

27 ore favorable remodeling over 1 year (LV end-diastolic volume =157+/-34 to 150+/-38 mL) compared with

28 se of 15 mL at >500 ng/mL, p=0.0026) and end-diastolic volumes (16 mL, p=0.0096) that might have been

29 ncing the saline-mediated increase in LV end-diastolic volume (+17+/-1 versus +10+/-2 mL; P=0.016).

30 significant decrease in left ventricular end-diastolic volume (-18 mL; P=0.009) and end-systolic volu

31 Reduced pump speed caused increased LV end-diastolic volume (190+/-80 versus 165+/-71 mL, P<0.0001)

32 .80; P<0.00001), and tended to reduce LV end-diastolic volume (-2.26 mL; 95% confidence interval, -4.

33 sed E-wave velocity and left ventricular end-diastolic volume, 2) exhibit a higher plasma volume, and

34 CMR at 1 year demonstrated a decrease in end diastolic volume (208.7+/-20.4 versus 167.4+/-7.32 mL; P

35 odeling (>20% change in left ventricular end-diastolic volume; 21.91 [2.75-174.29]; P=0.004).

36 +/- 72 to 145 +/- 23 ml, p < 0.001), LV end-diastolic volume (242 +/- 85 ml to 212 +/- 63 ml, p < 0.

37 iated with a significant reduction of LV end-diastolic volume (-25.1 +/- 26.0 ml vs. -1.5 +/- 25.4 ml

38 22 years were inversely associated with end-diastolic volume (-3.0 mL per unit mean hemoglobin A(1c)

39 luid loading increased right ventricular end-diastolic volume (+31 +/- 13 mL; p = 0.004), right ventr

40 val, -11.57 to -6.25; P<0.00001), and LV end-diastolic volume (-5.23 mL; 95% confidence interval, -7.

41 r age was associated with lower volumes (end-diastolic volume, -5 mLdecade; end-systolic volume, -3 m

42 Changes in end-diastolic volume (-6+/-14 versus -4+/-16 mL; P=0.67) at

43 ficant attenuation in the increase in LV end-diastolic volume (64.8 mL [60.7-71.3] to 83.5 mL [74.7-9

44 6+/-4.3 cm(2); P=0.02), and left atrium (end-diastolic volume, 65+/-19 versus 72+/-19; P=0.02).

45 s in the highest quintile had smaller LV end-diastolic volumes (68+/-13 versus 58+/-12 mL/m(2); P<0.0

46 in; p = 0.03), but no difference in peak end-diastolic volume (77 +/- 18 ml vs. 77 +/- 17 ml; p = 0.5

47 associated with a small right ventricle (end diastolic volume, 79.8+/-13.2 versus 88.5+/-11.8 mL/m(2)

48 Patisiran also led to increased end-diastolic volume (8.3+/-3.9 mL, P=0.036), decreased glob

49 .0%) and improvement in left ventricular end-diastolic volume (-8.0 mL versus -12.7 mL), whereas New

50 more than mild FMR despite increased LV end-diastolic volume (82 +/- 22, 86 +/- 23, and 51 +/- 12 cm

51 ic treatment decreased right ventricular end-diastolic volume (-84 +/- 11 mL; p < 0.001), right ventr

52 rophy was driven by LV dilation (DeltaLV end-diastolic volume, 9+/-3 mL/m(2); P=0.004) with stable LV

53 : -4.3 [11.3] versus 7.4 [11.8], P=0.02; end-diastolic volume: -9.1 [14.9] versus 7.4 [15.8], P=0.02)

54 ere calculated from echocardiographic LV end-diastolic volume accounting for the integral of pump flo

55 V ejection fraction and left ventricular end-diastolic volume, although correlation coefficients were

56 LV end-diastolic volume, an index of LV remodeling, increased b

57 c function, measured by left ventricular end-diastolic volume and cardiac output.

58 the right ventricle were used to measure end-diastolic volume and ejection fraction.

59 results showed a decrease of indexed RV end-diastolic volume and end-systolic volume (98 ml/m(2) to

60 RV volumes (end-diastolic volume and end-systolic volume), stroke volume

61 women were classified as having abnormal end-diastolic volume and internal diameter by ASE 2015 cutof

62 d with higher (more adverse) LV mass, LV end diastolic volume and left atrial volume, but not with ot

63 placebo did not change left ventricular end-diastolic volume and left ventricular mass nor did it im

64 ated the progressive deterioration in LV end-diastolic volume and LV end-systolic volume.

65 s the change in indexed left ventricular end diastolic volume and LVEF.

66 Indexed left ventricular end-diastolic volume and mass correlated with O2max (r=0.45,

67 ); P<0.001) and indexed left ventricular end-diastolic volume and right ventricular end-diastolic vol

68 During PEI, LV end-diastolic volume and stroke volume were increased in bot

69 here is a linear relationship between LV end-diastolic volume and the effective regurgitant orifice a

70 and G6PDX mice but significantly greater end diastolic volume and wall thinning in G6PDX mice.

71 n in CHF rats increased cardiac systolic and diastolic volumes and further increased the elevated LVE

72 ate and midterm reductions in indexed RV end-diastolic volumes and RV end-systolic volumes (RVESVi) (

73 We quantified associations with RV mass, end-diastolic volume, and ejection fraction after control fo

74 +/- 13 years of age), noncontrast 3D EF, end-diastolic volume, and end-systolic volume had significan

75 ographic data, risk factors for scar, LV end-diastolic volume, and LV mass.

76 vascular risk factors, calcium score, LV end-diastolic volume, and mass in addition to resting heart

77 ociated with preserved stroke volume, LV end-diastolic volume, and mass/volume ratio as measured by c

78 ffective regurgitant orifice, indexed LV end-diastolic volume, and right ventricular systolic pressur

79 left ventricular ejection fraction, systolic/diastolic volumes, and wall motion indexes.

80 t relationship between GLS groups and LV end-diastolic volume at 3 and 6 months (adjusted for clinica

81 ependently of load/extrinsic forces, the end-diastolic volume at a common end-diastolic pressure on t

82 ed LV compliance, measured as reduced LV end-diastolic volume at an idealized LV end-diastolic pressu

83 l was causally associated with higher LV end-diastolic volume (beta = 1.85 ml; 95% confidence interva

84 ric remodeling characterized by lower LV end-diastolic volume (beta=-0.21), higher concentricity (bet

85 beta=-0.02/%; P=0.015), left ventricular end-diastolic volume (beta=0.01/mL; P<0.0001), and left vent

86 aneous fat was associated with higher LV end-diastolic volume (beta=0.48), reduced concentricity (bet

87 beta=1.218; adjusted P=0.007), higher LV end-diastolic volume (beta=0.811; adjusted P=0.007), higher

88 End-systolic volume and end-diastolic volume/body surface area (BSA) were 62 (45-101

89 r end-diastolic volume (left ventricular end-diastolic volume/body surface area, 104+/-13 and 69+/-18

90 /-18 mL/m(2); P<0.001; right ventricular end-diastolic volume/body surface area, 110+/-22 and 66+/-16

91 point variables for primary outcomes: LV end-diastolic volume/body surface area, LV ejection fraction

92 on fraction (P<0.01), reduced stroke and end-diastolic volumes (both P<0.001), decreased peak E' velo

93 [7.3-28.5], then decreased by 14 months (end-diastolic volume/BSA(1.3), end-systolic volume/BSA(1.3),

94 d increased with increasing RV mass/BSA, end-diastolic volume/BSA, and T1 mapping and with decreasing

95 Ees/Ea decreased with increasing RV end-diastolic volume/BSA, mass/BSA, and pulmonary arterial s

96 higher ratio of left ventricular mass to end-diastolic volume (by 8%).

97 to -0.23; P=0.028) and left ventricular end-diastolic volume (coefficient, 7.85; 95% confidence inte

98 ndices ([RA+aRV]/[fRV+LA+LV]) and fRV/LV end-diastolic volume corresponded only to some parameters.

99 In both men and women, LV end-diastolic volume decreased (-9.8 and -13.3 mL per decade

100 Left ventricular end-diastolic volume decreased (from 125.1 +/- 40.1 ml to 10

101 odeling was defined as an increase in LV end-diastolic volume (DeltaEDV>0) between discharge and foll

102 e mean left ventricular end systolic and end diastolic volumes did not differ between the groups.

103 mL of CHAM (n=5) or saline (n=13) and LV end-diastolic volume (EDV) and MMP/TIMP profiles in the MI r

104 LV end-diastolic volume (EDV) and stroke volume (SV) were decre

105 13.0 mL; P < 0.0001), difference between end-diastolic volume (EDV) at rest and stress (DeltaEDV[stre

106 tensive versus conventional treatment in end diastolic volume (EDV), end systolic volume, stroke volu

107 al function was assessed by measuring LV end-diastolic volume (EDV), end-systolic volume (ESV), and e

108 erall bias and limits of agreement of LV end-diastolic volume (EDV), end-systolic volume (ESV), and E

109 yzed to provide the reference limits for end-diastolic volume (EDV), end-systolic volume (ESV), eject

110 End-diastolic volume (EDV), end-systolic volume (ESV), strok

111 including the end-systolic volume (ESV), end-diastolic volume (EDV), stroke volume (SV), and ejection

112 ventricular ejection fraction (LVEF) and end-diastolic volume (EDV).

113 c (ePAD) pressures and an increase in LV end diastolic volume (EDV).

114 olume (ESV: Pearson r = 0.99, P < .001), end-diastolic volume (EDV: r = 0.97, P < .001), and ejection

115 Filling was assessed by changes in LV end-diastolic volume (EDV; conductance catheter technique).

116 LV end-diastolic volumes (EDV) were measured by echocardiograph

117 fication based on LV dilatation (high LV end-diastolic volume [EDV] index) and concentricity (mass/en

118 ar events and a dilated LV (increased LV end-diastolic volume [EDV] indexed to body surface area) at

119 diographic measures of EF: cardiac size (end-diastolic volume [EDV]); contractile function (the end-s

120 efficacy end points included changes in end-diastolic volume, end-systolic volume, and ejection frac

121 However, changes in end-diastolic volume, end-systolic volume, and LVEF did not

122 Biventricular end-diastolic volume, end-systolic volume, stroke volume, an

123 demonstrated increasing left ventricular end-diastolic volumes, end-systolic volumes, stroke volumes,

124 Offline analysis generated 3-dimensional end-diastolic volume, ESV, SV, and free-wall RV longitudinal

125 justment for city attenuated the RV mass/end-diastolic volume findings.

126 ter than 20% relative increase in the LV end-diastolic volume from 1 to 12 wk (percentage injected do

127 [95% CI, 28.2-45.8]) and reduction in LV end-diastolic volume from 171.0 to 143.2 mL (difference, 31.

128 8 mm Hg (p = 0.016) and a decrease in LV end-diastolic volume from 172 +/- 37 ml to 158 +/- 38 ml (p

129 ow-up documented a stepwise reduction in end-diastolic volume (from 147 mL [IQR, 95-191 mL] to 127 mL

130 >/=10 U, combined with a decrease in LV end-diastolic volume >/=10% at follow-up.

131 tion for heart failure, left ventricular end-diastolic volume >/=125 mL/m(2), and left atrial volume

132 fined as an increase in left ventricular end-diastolic volume >/=20% at 6 months.

133 -losing enteropathy, ventricular indexed end-diastolic volume >125 mL/body surface area raised to the

134 CMR-derived left ventricular end-diastolic volume >246 mL had good, although lower, discr

135 rovement in the indexed left ventricular end diastolic volume >=10% at 12 months.

136 achycardia, low blood pressure, enlarged end-diastolic volume, high ejection fraction, and high cardi

137 encing MACE showed higher left ventricle end-diastolic volume, higher left ventricle end-systolic vol

138 strain (HR, 1.63; P=0.005), exercise LV end-diastolic volume (HR, 1.38; P=0.048), and resting RV str

139 A reduced left ventricular end diastolic volume, impaired early filling, and decreased

140 ase and dilatation, but left ventricular end-diastolic volume improved because of reduced blood retur

141 line to 12 months, left ventricular (LV) end-diastolic volume improved from 161 +/- 56 ml to 143 +/-

142 Left ventricular end-diastolic volume improved from 172 ml to 140 ml and end-

143 ion initially induced an expansion of LV end-diastolic volume in IPAH (+7%; P < 0.05), whereas end-di

144 P<0.001), BMI was not correlated with RV end-diastolic volume in men (R=0.04; P=0.51).

145 167), and after replacing LV mass by LV end-diastolic volume in the regression models.

146 ntal value of LVGLS for prediction of LV end-diastolic volume increase (0.14 [95% confidence interval

147 After intervention, LV mass and end-diastolic volume increased and exercise capacity improve

148 Right ventricular end diastolic volume increased by 49.4% after transannular p

149 trol animals, LV end-systolic volume and end-diastolic volume increased from 6 to 30 weeks (median an

150 of trastuzumab, indexed left ventricular end diastolic volume increased in patients treated with peri

151 Subsequently, RV end-systolic and end-diastolic volume increased more in both groups, resultin

152 In the exercise group, both LA and LV end diastolic volumes increased proportionately (19% and 17%

153 the Frank-Starling relationship, greater end-diastolic volume increases ventricular output.

154 ery at 5 years, 90% for left ventricular end-diastolic volume index <100 mL/m(2) versus 48% for >/=10

155 scular lung water (<10 mL/kg) and global end-diastolic volume index (<850 mL/m) in the transpulmonary

156 Patients with transplants had lower end-diastolic volume index (59.3+/-15.2 ml/m(2) vs. 71.4+/-1

157 shortening (B=-0.1; P=0.0001), lower LV end-diastolic volume index (B=0.6; P=0.0001), and lower LV e

158 01), obesity (beta=1.3 mL/m(2), P<0.01), end-diastolic volume index (beta=0.4 mL/m(2), P<0.0001), Chi

159 vs 3.8 L/min/m(2); P = .001), and global end-diastolic volume index (GEDVI) (726 vs 775 mL/m(2); P =

160 The left atrial end-diastolic volume index (LAEDVI), representing the minimu

161 come was mean change from baseline in LV end-diastolic volume index (LVEDVI) at 6 months.

162 rations and left ventricular (LV) EF, LV end-diastolic volume index (LVEDVI), LV end-systolic volume

163 ll, stable reduction in left ventricular end-diastolic volume index (P<0.001), with a concomitant sma

164 stable increase in the right ventricular end-diastolic volume index (P<0.001).

165 Stroke volume index (P=0.015) and end-diastolic volume index (P=0.001) decreased during exerci

166 e index (P<0.0001), and left ventricular end-diastolic volume index (P=0.0015).

167 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak exerci

168 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak exerci

169 n change from baseline right ventricular end-diastolic volume index and a 429 ml (P < 0.001) reductio

170 Left ventricle end-diastolic volume index and end-systolic volume index wer

171 reater ECE is associated with reduced LV end-diastolic volume index and LV end-diastolic mass index i

172 between groups in baseline values for LV end-diastolic volume index and LV end-systolic volume index

173 ed in a greater reduction of LV systolic and diastolic volume index and LV mass index as compared wit

174 Empagliflozin reduced LV end-diastolic volume index by 8.2 (95% CI, -13.7 to -2.6) mL

175 The left ventricular end-diastolic volume index decreased (90.1 +/- 28.2 ml/m(2)

176 Resting left ventricular end-diastolic volume index increased (P=0.001) within the si

177 Right ventricular end-diastolic volume index measured by means of cardiovascul

178 threshold of 227% or a left ventricular end-diastolic volume index of 58 ml/m(2) identified patients

179 CMR-derived end-diastolic volume index showed a weaker association with

180 a greater extent of LGE and a higher LV end-diastolic volume index than other groups, but levels of

181 hange from baseline in right ventricular end-diastolic volume index versus placebo.

182 All patients were hypovolemic (global end-diastolic volume index<680 mL/m) on enrollment.

183 improvements in LV ejection fraction, LV end-diastolic volume index, and LV end-systolic volume index

184 ympathetic denervation, left ventricular end-diastolic volume index, creatinine, and no angiotensin i

185 re no significant differences in mean LV end-diastolic volume index, end-systolic volume index and LV

186 tors (age, body mass index, diabetes, LV end-diastolic volume index, LGE, EF) (hazard ratio = 2.051 p

187 vascular magnetic resonance measures (LV end-diastolic volume index, LV ejection fraction), diuretic

188 87.3+/-18.7 mL/m(2) at 3-year follow-up (end-diastolic volume index, P=0.0056; end-systolic volume in

189 ft atrial volume index, left ventricular end-diastolic volume index, peak E wave, and the presence of

190 volume variation monitoring, and global end-diastolic volume index.

191 tic peptide, and larger left ventricular end-diastolic volume index.

192 olume index, and larger left ventricular end-diastolic volume index.

193 (LV mass index: r = 0.35, p < 0.0001; LV end-diastolic volume index: r = 0.43, p < 0.0001) and LVEF (

194 imal NC/C ratio and preceding changes in end-diastolic volume indexed (EDVi) to body surface area and

195 ejection fraction (LVEF) and left atrial end-diastolic volume indexed to body surface area, were asse

196 ratio, left ventricular end-systolic and end-diastolic volume indexes (LVESVI and LVEDVI), left atria

197 nt improvements in both LV end-systolic and -diastolic volume indexes.

198 ) ejection fraction, LV end-systolic and end-diastolic volumes, infarct size, and major adverse cardi

199 CMR-derived ventricular indexed end-diastolic volume is an independent predictor of transpla

200 s defined as (LA end-systolic volume--LA end-diastolic volume) / LA end-systolic volume.

201 d-diastolic volume and right ventricular end-diastolic volume (left ventricular end-diastolic volume/

202 nderwent TTE and CMR, and left ventricle end-diastolic volume, left ventricle end-systolic volume, an

203 provements in LV end-systolic volume, LV end-diastolic volume, left ventricular ejection fraction, le

204 rcentage of myocardium, left ventricular end-diastolic volume, left ventricular end-systolic volume,

205 ctors of death were: larger left ventricular diastolic volume, left ventricular systolic volume and E

206 t in TAVR patients, and low left ventricular diastolic volume, low stroke volume, and greater severit

207 s; and smaller left ventricular systolic and diastolic volumes, low stroke volume, smaller EOA, and p

208 e predicted by a preoperative indexed RV end-diastolic volume </=158 mL/m(2) and RVESVi </=82 mL/m(2)

209 reduction in LV wall stress, reduced LV end-diastolic volume, LV end-systolic volume and increase in

210 ide significant loci (3 loci each for LV end-diastolic volume, LV end-systolic volume, and LV mass to

211 percent change in left ventricular (LV) end-diastolic volume, LV end-systolic volume, LV ejection fr

212 de association studies of 6 LV traits-LV end-diastolic volume, LV end-systolic volume, LV stroke volu

213 Automatic quantification of LV end-diastolic volume, LV myocardium mass, LV trabeculation,

214 LV function, who showed reduction in LV end-diastolic volume, LV wall stress, no change in LV end-sy

215 thickening fraction, LV end-systolic and end-diastolic volumes, LV ejection fraction) and hemodynamic

216 Significant decreases in LV end-diastolic volumes, LV end-systolic volumes, cardiac outp

217 were obtained with MRI: Left ventricular end-diastolic volume (LVDV) was 40 mL (LVDV per body surface

218 ff values for change in left ventricular end-diastolic volume (LVEDV) and LV end-systolic volume (LVE

219 o 50% or greater, whose left ventricular end-diastolic volume (LVEDV) had normalised, and who had an

220 mong patients with available baseline LV end-diastolic volume (LVEDV) measures, 188 received biventri

221 ion fraction (LVEF) and left ventricular end diastolic volume (LVEDV) on cardiac magnetic resonance i

222 ed as percent change in left ventricular end-diastolic volume (LVEDV), was analyzed in 3 prespecified

223 rformance according to the median PET LV end-diastolic volume (LVEDV), with smaller LVs defined as ha

224 ume [EDV] index) and concentricity (mass/end-diastolic volume [M/EDV](2/3)) in hypertensive patients.

225 es were significantly smaller in DM, (LV end-diastolic volume/m(2): -3.46 mL/m(2) [-5.8 to -1.2], P=0

226 5.8 to -1.2], P=0.003, right ventricular end-diastolic volume/m(2): -4.2 mL/m(2) [-6.8 to -1.7], P=0.

227 normalization of LV stroke volume and LV end-diastolic volume/mass ratio, there was a persistent sign

228 CMR showed higher left ventricle end-diastolic volume (mean difference: 43+/-22.5 mL), higher

229 decreased the temporal variability of LV end-diastolic volume measurements by the 2D biplane method.

230 right/left-volume index was defined from end-diastolic volume measurements in CMR: total right/left-v

231 reased from pre-Norwood to pre-stage II (end-diastolic volume [milliliters]/body surface area [BSA](1

232 nance (1.5 T) to measure RV mass (g), RV end-diastolic volume (mL), RV mass/volume ratio, and LV dias

233 Left ventricular systolic and diastolic volumes, New York Heart Association functional

234 .002), exercise indexed left ventricular end-diastolic volume (odds ratio, 1.04; P=0.026), exercise e

235 t ventricular (LV) ejection fraction, LV end-diastolic volume, or New York Heart Association (NYHA) f

236 ; 95% confidence interval, -0.2 to 2.1), end-diastolic volume, or systolic volume were observed compa

237 t not CT-significantly overestimated the end-diastolic volume (p < 0.001), whereas 2D Echo and 3D Ech

238 redictor of increase in left ventricular end-diastolic volume (p < 0.001).

239 y associations with baseline and DeltaLV end diastolic volume (P<0.0001 for each) and not wall thickn

240 ere observed for the ratio of LV mass to end-diastolic volume (P=0.02) and with hyperinflation measur

241 on left ventricular (LV) mass (P=0.016), end-diastolic volume (P=0.029), and end-systolic volumes (P=

242 02), and increased ventricular dilation (end-diastolic volume, P=0.01).

243 (Delta ejection fraction: P<0.04, Delta end-diastolic volume: P<0.02, Delta end-systolic volume: P<0

244 and IMH correlated with the change in LV end-diastolic volume (Pearson's rho of 0.64, 0.59, and 0.66,

245 Indexed RV end-diastolic volume plotted against neoaortic stroke volume

246 nd-systolic volumes (RVESVi) (indexed RV end-diastolic volume pPVR versus immediately after PVR versu

247 ic pressure (LVEDP) and left ventricular end-diastolic volume (preload) in CHF rats, which was not ob

248 ial lidocaine paradoxically decreased LV end-diastolic volume (preload) in CHF rats, which was not ob

249 ted QRS area for heart size using the LV end-diastolic volume (QRSarea/LVEDV).

250 c geometry, defined by higher LV mass to end-diastolic volume quartiles, were associated with higher

251 ) nor PR volume (PRV) correlated with RV end-diastolic volume (r = 0.36; p = 0.15 and r = 0.37; p = 0

252 s a significant predictor of increase in end-diastolic volume (r = 0.51, P < 0.05).

253 ction correlated closely with indexed RV end-diastolic volume (R = 0.79, p < 0.001) and modestly with

254 95), end-systolic volume (r = 0.93), and end-diastolic volume (r = 0.90), and slightly lower correlat

255 ne strongly correlated with reduction of end diastolic volume (r(2)=0.69, P=0.04) and end systolic vo

256 chanical dyssynchrony correlated with RV end-diastolic volume (r=0.39; P=0.03).

257 overall explained variance for volumes (end-diastolic volume, R(2)=0.43; end-systolic volume, R(2)=0

258 ional decline in the RV/left ventricular end-diastolic volume ratio (P=0.05) and trended toward impro

259 % CI: 0.04, 0.36)] and a greater RV mass/end-diastolic volume ratio conditional on LV parameters.

260 ociated with greater RV mass and RV mass/end-diastolic volume ratio conditional on the LV; however, a

261 ociated with greater RV mass and RV mass/end-diastolic volume ratio relative to the LV.

262 ures of PM2.5, whereas those for RV mass/end-diastolic volume ratio were attenuated.

263 e to assess LV remodeling (LV mass-to-LV end diastolic volume ratio), function, tissue characterizati

264 e directly associated with LV mass to LV end-diastolic volume ratio, a marker of cardiac remodelling

265 of LVH and abnormal/normal LV mass to LV end-diastolic volume ratio.

266 ection fraction, LV mass, and LV mass to end-diastolic volume ratio.

267 , LV end-systolic volume, and LV mass to end-diastolic volume ratio; 4 loci for LV ejection fraction,

268 The LA:LV end diastolic volumes ratio did not change from baseline to

269 who developed asymmetry, the wall thickness/diastolic volume ration remained normal (0.09+/-0.02 mmm

270 n LV mass index and the ratio of LV mass:end-diastolic volume (relative wall mass).

271 ercentiles of LV end systolic volume, LV end diastolic volume, relative wall and septal thickness, LV

272 dels showed that the addition of indexed end-diastolic volume resulted in a significantly improved en

273 fraction, and indexed right ventricular end-diastolic volume resulted in significant improvements in

274 57% increase in LV mass (no change in LV end diastolic volume, resulting in an increase in the LV mas

275 Right ventricular end-diastolic volume (RVDV) was 262 mL (RVDV/BSA, 164 mL/m(2

276 Right ventricular end-diastolic volume (RVEDV) was reduced in COPD compared wi

277 f 15 to 30 mL in 3DTTE right ventricular end-diastolic volume; sample sizes were 2x to 2.5x those req

278 Systolic function (EF, end-systolic and end-diastolic volumes, stroke volumes) was not different in

279 tion was 34+/-17%, and right ventricular end-diastolic volume was 114+/-39 cc/m(2).

280 ejection fraction was 31.3 +/- 9.3%, LV end-diastolic volume was 192.7 +/- 71 ml, and effective regu

281 LV end-diastolic volume was 2-fold (P=0.005) higher in MR patie

282 LA end-diastolic volume was increased in patients with mitral r

283 In CAP-treated dogs, end-diastolic volume was unchanged; ejection fraction increa

284 rct size, LV end-systolic volume, and LV end-diastolic volume were analyzed with random-effects meta-

285 roke volume (SV), ejection fraction, and end-diastolic volume were assessed by echocardiography.

286 rct size, LV end-systolic volume, and LV end-diastolic volume were estimated with random-effects meta

287 ulmonary capillary wedge pressure and LV end-diastolic volume were measured at baseline, during decre

288 By CMR, right ventricular end-diastolic volumes were 118 +/- 31 cc/m(2), 108 +/- 22 cc

289 End-systolic and end-diastolic volumes were calculated using blood pool image

290 sed significantly during exercise, while end-diastolic volumes were essentially unchanged.

291 Both the LV and RV end-systolic and LV end-diastolic volumes were increased compared with reference

292 low-up, left atrial and left ventricular end-diastolic volumes were significantly more reduced in pat

293 iated with greater RV mass and larger RV end-diastolic volume with or without further adjustment for

294 ions of RV mass and, to a lesser extent, end diastolic volume with PM10-2.5 mass among susceptible po

295 12 weeks of age and decreased left ventricle diastolic volume with subsequent reduced SV compared to

296 decreased left ventricular (LV) systolic and diastolic volumes with little effect on LV end-diastolic

297 eased LV weight/body weight ratio and LV end diastolic volume (WT, 50.8 mul; CatA-TG, 61.9 mul).

298 ocardiogram, the median left ventricular end-diastolic volume z score was +1.7 (range, -1.3 to +8.2),

299 ormal values, left and right ventricular end-diastolic volume z scores were mildly enlarged (0.48+/-1

300 LV end-diastolic volume z-score increased (P=0.02), LV mass was