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1 reated with a selective inhibitor of NQO(1), dicumarol.
2 enten kinetics and was markedly inhibited by dicumarol, a competitive inhibitor of naphthoquinone oxi
3                Intrathecal administration of dicumarol alleviated nerve injury-induced mechanical all
4                                              Dicumarol also inhibited cell growth, plating efficiency
5                                 In addition, dicumarol, an inhibitor of the phase II detoxifying enzy
6      The enzymatic activity was inhibited by dicumarol, anti-coagulant drug, with IC50 of 4 microm.
7  screens and identified an FDA-approved drug dicumarol as a novel and potent VRAC inhibitor.
8                                              Dicumarol decreased clonogenic survival equally in both
9                                              Dicumarol decreased clonogenic survival in the transform
10                                     Although dicumarol has been used as an inhibitor of the two-elect
11 vity is insensitive to rotenone, flavone, or dicumarol; however, it was inhibited by diphenyl iodoniu
12                                              Dicumarol increased intracellular production of O(2)(.-)
13 atalase with adenoviral vectors reversed the dicumarol-induced cytotoxicity and reversed fluorescence
14  increased susceptibility of cancer cells to dicumarol-induced cytotoxicity.
15 susceptibility of pancreatic cancer cells to dicumarol-induced metabolic oxidative stress.
16 ibitable NAD(P)H:menadione oxidoreductase or dicumarol-inhibitable NAD(P)H:dichlorophenolindophenol r
17 ified DT-diaphorase can be assayed as either dicumarol-inhibitable NAD(P)H:menadione oxidoreductase o
18  When DT-diaphorase activity was measured as dicumarol-inhibitable NADPH:dichlorophenolindophenol red
19 bstances of widely different molecular size: dicumarol, insulin and plasmid DNA.
20                                              Dicumarol is a naturally occurring anticoagulant derived
21 ctase NAD(P)H:quinone oxidoreductase (NQO1), dicumarol is also thought to affect quinone-mediated ele
22 H, or NMNH; (ii) is very weakly inhibited by dicumarol or Cibacron blue; (iii) is very potently inhib
23           Moreover, using inhibitors of GST (dicumarol) or MRP1 (sulfinpyrazone), it was shown that i
24 inone oxidoreductase enzymatic activity with dicumarol prior to quinone treatment resulted in increas
25 using mitomycin (trial 1) and mitomycin with dicumarol (trial 2) as an adjunct to radiation therapy i
26  or without mitomycin (trial 1) or mitomycin/dicumarol (trial 2).
27                                              Dicumarol with the mitochondrial electron transport chai
28                                              Dicumarol, with the addition of mitochondrial electron t