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1 tants such as ozone, particulate matter, and diesel exhaust.
2 e exposure (P = 0.02), but not sandstorms or diesel exhaust.
3 myocytes as a result of in utero exposure to diesel exhaust.
4 ) formed from the photochemical oxidation of diesel exhaust.
5 for secondary organic aerosol formation from diesel exhaust.
6  susceptibility through prenatal exposure to diesel exhaust.
7 y childhood exposures, including exposure to diesel exhaust.
8 ic material (ACM), that were challenged with diesel exhaust.
9 d at rest in a randomized, balanced order to diesel exhaust (200 mug/m(3) particulate matter with an
10 se in the ischemic burden during exposure to diesel exhaust (-22+/-4 vs. -8+/-6 millivolt seconds, P<
11 tudy, 30 healthy men were exposed to diluted diesel exhaust (300 microg/m3 particulate concentration)
12 exposed, in two separate sessions, to dilute diesel exhaust (300 mug per cubic meter) or filtered air
13 nvestigated electrode-assisted deposition of diesel exhaust aerosol (DEA) on human lung epithelial ce
14                                    Moreover, diesel exhaust affected A. mellifera in a way that reduc
15                         In utero exposure to diesel exhaust air pollution has been associated with in
16                  Exposure to allergen alone, diesel exhaust alone, or allergen and diesel exhaust tog
17 and suspected human carcinogen identified in diesel exhaust and air pollution.
18 y to this environmental contaminant found in diesel exhaust and ambient air pollution.
19 late carbon is obtained by increasing mobile diesel exhaust and area-source particulate carbon emissi
20  the increase was similar during exposure to diesel exhaust and exposure to filtered air (P=0.67).
21  cardiac myocytes after in utero exposure to diesel exhaust and found that the promoter for Mir133a-2
22 a, we estimated SOA formation potential from diesel exhaust and predict the contribution from UCM vap
23 en the same lung was exposed to allergen and diesel exhaust but separated by approximately 4 weeks, s
24  +/- 2% of gasoline exhaust and 26 +/- 1% of diesel exhaust by mass.
25 sks associated with occupational exposure to diesel exhaust characterized by elemental carbon (EC) co
26 utagenicity and the genotoxicity of complete diesel exhaust compared to an organic exhaust particle e
27 eratures as low as ~160 (o)C under simulated diesel exhaust conditions while using 5 times less Pt-gr
28                   Under laboratory-simulated diesel exhaust conditions, this mixed-phase oxide materi
29 tigated how acute exposure to a high-dose of diesel exhaust (containing 19.8 and 17.5 ppm of NO and N
30 sly demonstrated that short-term exposure to diesel exhaust (DE) for 1 h induced a marked leukocytic
31 nt mice were exposed to filtered air (FA) or diesel exhaust (DE) on embryonic days (E) 9-17.
32 s (SHRs) were exposed to 150 or 500 mug/m(3) diesel exhaust (DE) or filtered air (FA).
33    We hypothesized that a single exposure to diesel exhaust (DE) would increase the risk of adverse c
34                                   Rationale: Diesel exhaust (DE), an established model of traffic-rel
35                                              Diesel exhaust (DE), in addition to generating other pol
36 ne whether exposure to allergen, exposure to diesel exhaust (DE), or coexposures modulate miRNA, gene
37                                  Exposure to diesel exhaust did not aggravate preexisting vasomotor d
38       Air pollution, primarily consisting of diesel exhaust emissions, has increased at a similar rat
39 hose observed in laboratory studies of fresh diesel exhaust emissions.
40                                              Diesel exhaust enhances allergic inflammation, and pollu
41 stantially reduce SOA formation potential of diesel exhaust, except at low speed operations.
42 nalyses, which demonstrated that only in the diesel exhaust exposed honey bees was there a significan
43                                              Diesel exhaust exposure decreased honey bees' ability to
44 ht to investigate the effect of allergen and diesel exhaust exposure on bronchial epithelial DNA meth
45                  With emerging evidence that diesel exhaust exposure poses distinct risks to human he
46 -term air pollution (AP) exposure, including diesel exhaust exposure, is increasingly being recognize
47 experimental evidence on cigarette smoke and diesel exhaust exposure.
48 est potential reductions result from reduced diesel exhaust fluid (DEF) usage due to lower NO(x) emis
49 le organic compounds (VOCs) were measured in diesel exhaust from three heavy-duty trucks equipped wit
50                                              Diesel exhaust gaseous sulphuric acid (GSA) concentratio
51 e and compare ozone production potentials of diesel exhaust, gasoline exhaust, and nontailpipe gasoli
52                                              Diesel exhaust has been considered to be a probable lung
53 n an urban environment, inhalation of dilute diesel exhaust impairs 2 important and complementary asp
54 ganic exhaust particle extract from the same diesel exhaust in a bacterial and a eukaryotic system, t
55     We confirmed that short-term exposure to diesel exhaust in healthy subjects is associated with ac
56 er-controlled exposure study to allergen and diesel exhaust in humans, and measured single-site (CpG)
57 We conducted a controlled exposure to dilute diesel exhaust in patients with stable coronary heart di
58 +/-3 years) were exposed to filtered air and diesel exhaust in the presence or absence of a particle
59  adult hearts from mice that were exposed to diesel exhaust in utero and that have subsequently under
60             No association was found between diesel exhaust inhalation and flow-mediated dilation.
61  study aim was to investigate the effects of diesel exhaust inhalation on vascular and endothelial fu
62                  Compared with filtered air, diesel exhaust inhalation was associated with reduced va
63                                              Diesel exhaust inhalation, which is the model traffic-re
64  from gasoline and diesel vehicles, and find diesel exhaust is seven times more efficient at forming
65 ominant nitropolycyclic hydrocarbon found in diesel exhaust, is a mutagen and tumorigen.
66                       Short-term exposure to diesel exhaust leads to increased eosinophil activation
67 s of 1-nitropyrene (1-NP), a highly specific diesel exhaust marker, at the neighborhood scale.
68                                  Exposure to diesel exhaust may play a role in the development and pr
69 ergents, tobacco, ozone, particulate matter, diesel exhaust, nanoparticles, and microplastic on the i
70                                Sulfur driven diesel exhaust nucleation particle formation processes w
71 ow or inflammatory markers after exposure to diesel exhaust or air.
72 ression between honey bees exposed to either diesel exhaust or clean air across the entire duration o
73 studies have reported an association between diesel exhaust particle (DEP) exposure, allergic sensiti
74 ent for established asthma in the context of diesel exhaust particle (DEP) exposure.
75                   5-Aza-2'-deoxycytidine and diesel exhaust particle exposure in human bronchial epit
76 ell-established allergic adjuvant effects of diesel exhaust particle exposure.
77 in the pathogenesis of lung injury caused by diesel exhaust particles (DEP) and bacterial lipopolysac
78                                              Diesel exhaust particles (DEP) and their organic constit
79                        The redox activity of diesel exhaust particles (DEP) collected from a light-du
80                                              Diesel exhaust particles (DEP) contain organic chemicals
81                               Ambient PM and diesel exhaust particles (DEP) contain redox cycling org
82 of the mice were co-exposed to mycotoxins or diesel exhaust particles (DEP) during pregnancy.
83                                      Inhaled diesel exhaust particles (DEP) exert proinflammatory eff
84 Increased exposure to air pollutants such as diesel exhaust particles (DEP) has been proposed as one
85                                              Diesel exhaust particles (DEP) have strong, selective Th
86 cies are involved in the adjuvant effects of diesel exhaust particles (DEP) in a murine OVA sensitiza
87 demonstrate that methanol extracts made from diesel exhaust particles (DEP) induce apoptosis and reac
88 usly shown that in vivo nasal challenge with diesel exhaust particles (DEP) induces both quantitative
89 el, here we showed that maternal exposure to diesel exhaust particles (DEP) predisposed offspring to
90 and either urban particulate matter (UPM) or diesel exhaust particles (DEP) via the airways and asses
91 dies have correlated inflammatory effects of diesel exhaust particles (DEP) with its organic constitu
92             We hypothesized that exposure to diesel exhaust particles (DEP), a major component of urb
93 effects of particulate pollutants, including diesel exhaust particles (DEP), are related to their con
94       Exposure to traffic pollution, notably diesel exhaust particles (DEP), increases risk for asthm
95 nce that particulate air pollutants, such as diesel exhaust particles (DEP), potentiate chronic infla
96        Ambient particulate matter, including diesel exhaust particles (DEP), promotes the development
97 ncluding ambient particulate matter (PM) and diesel exhaust particles (DEP).
98 encephalic neuron-glia cultures treated with diesel exhaust particles (DEP; 0.22 microM) (5-50 microg
99 ntly exposed via oropharyngeal aspiration to diesel exhaust particles (DEP; 50 mug x 6 doses) or vehi
100                                              Diesel exhaust particles (DEPs) and SHS can interact wit
101                                              Diesel exhaust particles (DEPs) are a major component of
102                                              Diesel exhaust particles (DEPs) are a major component of
103                                              Diesel exhaust particles (DEPs) are major air pollutants
104                                              Diesel exhaust particles (DEPs) can deposit onto the res
105                   Oxidant pollutants such as diesel exhaust particles (DEPs) can initiate and exacerb
106                            The inhalation of diesel exhaust particles (DEPs) is associated with incre
107 re given repeated intranasal applications of diesel exhaust particles (DEPs) or PBS.
108                         Here, we report that diesel exhaust particles (DEPs), a major constituent of
109  pollution particulate matter, predominantly diesel exhaust particles (DEPs), increases the risk of a
110                                  Exposure to diesel exhaust particles (DEPs), which are major constit
111 xposure to environmental pollutants, such as diesel exhaust particles (DEPs).
112 igh levels of airborne particulates, such as diesel exhaust particles (DEPs).
113 of traffic-related particulate matter (e.g., diesel exhaust particles [DEPs]) is associated with acut
114 /L [-0.2-19.6], p=0.01) after challenge with diesel exhaust particles and allergens.
115      Further work investigated how pollutant diesel exhaust particles could modify these transcriptio
116                                          The diesel exhaust particles enhancement was largest in pati
117 mon lipophilic pollutants benzo[a]pyrene and diesel exhaust particles impact on the activation of lip
118 y with allergen alone and with allergen plus diesel exhaust particles in a randomised order at separa
119                    Insights into the role of diesel exhaust particles in patients with severe asthma
120 f temperature on the number concentration of diesel exhaust particles in the nucleation mode in a pre
121 STM1 and GSTP1 modify the adjuvant effect of diesel exhaust particles on allergic inflammation.
122 re key regulators of the adjuvant effects of diesel exhaust particles on allergic responses.
123 4.7] vs 7.4 nmol/L [1.2-12.3], p=0.02) after diesel exhaust particles plus allergen challenge.
124                                   Similarly, diesel exhaust particles showed a marginal inhibitory ef
125                         The model pollutant, diesel exhaust particles, can participate with allergens
126 combined prenatal exposure to air pollution (diesel exhaust particles, DEP) and maternal stress (MS)
127 t has been described that exposure to ozone, diesel exhaust particles, or tobacco smoke exacerbates a
128                         Examples of UFPs are diesel exhaust particles, products of cooking, heating,
129 anin in conjunction with adjuvant intranasal diesel exhaust particles.
130  species and detoxify xenobiotics present in diesel exhaust particles.
131  nasal allergic responses in the presence of diesel exhaust particles.
132  higher levels of carbon dioxide, ozone, and diesel exhaust particles.
133                We investigated the impact of diesel exhaust particulate extract (DEPe) exposure on th
134 on in the pathophysiology due to exposure to diesel exhaust particulate matter (DEP).
135           The particle trap markedly reduced diesel exhaust particulate number (from 150 000 to 300 0
136 d the inflammatory response to inhalation of diesel exhaust particulates (DEP) in normal volunteers.
137                          We investigated how diesel exhaust particulates (DEPs) aggravate asthma-like
138                         In utero exposure to diesel exhaust particulates is associated with an altere
139 uated, with rank-ordered responses of CFA1 > diesel exhaust PM > crystalline silica; TRP melastatin-8
140 an health, the need for fine-scale models of diesel exhaust pollutants is growing.
141 ent aftertreatment system for the removal of diesel exhaust pollutants.
142 hybrid modeling was successful in predicting diesel exhaust pollution at a very fine scale and identi
143 oraging for flowers; we investigated whether diesel exhaust pollution could interrupt these floral od
144  (NPAHs) to identify fine-scale gradients in diesel exhaust pollution in two Seattle, WA neighborhood
145 identified from oilseed rape, was exposed to diesel exhaust pollution.
146                     Brief exposure to dilute diesel exhaust promotes myocardial ischemia and inhibits
147                                              Diesel exhaust-related vasoconstriction was primarily ob
148     Pretreatment with antioxidants augmented diesel exhaust-related vasoconstriction with a mean chan
149      Compared with filtered air, exposure to diesel exhaust resulted in a significant reduction in BA
150 age in activities that result in exposure to diesel exhaust, solvents, welding fumes, and other respi
151 was applied to PM samples from woodsmoke and diesel exhaust, the model accurately predicts HMW PAH co
152 alone, diesel exhaust alone, or allergen and diesel exhaust together (coexposure) led to significant
153 s show promise for a considerably lower-cost diesel exhaust treatment system.
154                   The effective SOA yield of diesel exhaust was similar to that of unburned diesel fu
155 P<0.001) infusions 2 hours after exposure to diesel exhaust, which persisted at 6 hours.
156 ents were created in a chamber that combined diesel exhaust with an urban-like mixture.
157 rrhythmia during or after exposure to dilute diesel exhaust, wood smoke, ozone, concentrated ambient
158             To determine whether exposure to diesel exhaust would alter their tolerance to a subseque
159            Ignoring secondary chemistry from diesel exhaust would lead to underestimates of both orga

 
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