ライフサイエンスコーパス: diet-induced

1 ing normal neurogenesis with that altered by diet-induced accelerated aging in adult zebrafish.

2 myeloid-Klf6 deficiency significantly curbs diet-induced adipose tissue inflammation, obesity, gluco

3 deficient mouse models are protected against diet-induced adiposity, hepatic steatosis, and hyperglyc

4 s for p32 are resistant to age- and high-fat diet-induced ailments, including obesity, hyperglycemia,

5 e value of CC mice in combination with HF/HS diet-induced alterations as an approach to study the sus

6 independent models of renal failure, adenine diet induced and 5/6 nephrectomy.

7 Muscle-CB1R ablation prevented diet-induced and age-induced insulin resistance by incre

8 ly in the liver, protects mice from high-fat diet-induced and aging-induced obesity and hepatic fat a

9 ndpoints in Lep(ob/ob) mice with established diet-induced and biopsy-confirmed NASH (ob/ob-NASH).

10 disease reversal study was performed in the diet-induced animal model of NAFLD (DIAMOND).

11 lpha/gamma agonist would improve NASH in the diet-induced animal model of NAFLD.

12 and increased VSM PTH1R signaling mitigates diet-induced arteriosclerosis in LDLR(-/-) mice.

13 elet function, vasculature inflammation, and diet-induced atherosclerosis and myocardial infarction.

14 ch Cav1/caveolae control the pathogenesis of diet-induced atherosclerosis are still not clear.

15 in both models, disturbed flow- and high fat diet-induced atherosclerosis, whereas Nck2 deletion did

16 h in vitro and in vivo with a mouse model of diet-induced atherosclerosis.

17 LR) and Arhgef1 were protected from high-fat diet-induced atherosclerosis.

18 s and protected Ldlr(-/-) mice from high-fat diet-induced atherosclerosis.

19 of rapamycin complex 1 seems involved in the diet-induced beneficial effects, as also strengthened by

20 ogenesis was sufficient to protect mice from diet-induced body-weight gain.

21 ect interspecies electron transfer (MIET and DIET), induced by the conductive GAC promote the overall

22 However, the exact pathways linking diet-induced changes (e.g., hyperlipidemia) and the ensu

23 LLTS reversed HS diet-induced changes at both these sites.

24 Few studies have analyzed how diet-induced changes in the microbiota influence lupus.

25 y composition are unlikely to be mediated by diet-induced changes in the taxonomic composition of the

26 ce of cholesterol metabolism by the host for diet-induced changes of the gut microbiota and energy me

27 CHADN reduced the diet-induced defect in net hepatic glucose balance by 37

28 ypothesize this IgA decrease is secondary to diet-induced dysbiosis.

29 means of managing cholesterol metabolism and diet induced dyslipidaemia, as well as insulin sensitivi

30 erm-free zebrafish are resistant to high fat diet induced EEC silencing.

31 Herein, we investigate the effect of diet-induced elevated levels of brain Hcy on the phenoty

32 ty liver disease; 3) DKO mice demonstrate HF diet-induced elevations of plasma leptin, resistin, fed-

33 ZIP14 during pharmacologically and high-fat diet-induced ER stress using Zip14(-/-) (KO) mice, which

34 Unexpectedly, high fat diet induced extensive atherosclerosis in miR-144 knocko

35 is in WAT, and also promotes age-related and diet-induced fat mass gain and insulin resistance.

36 yte-specific IMP2 deficiency promotes modest diet-induced fatty liver by impairing fatty acid oxidati

37 salutary role in the protection against the diet-induced fatty liver disease.

38 yslipidemia, it protected mice from high-fat diet-induced glucose intolerance and insulin resistance

39 n-induced beta-cell destruction and high-fat diet-induced glucose intolerance.

40 postprandial glucose clearance in states of diet-induced glucose intolerance.

41 s to insulin either in culture or in vivo in diet-induced, glucose-intolerant mice rendered them resi

42 stress induced in mice by feeding a high-fat diet induced greater DNA damage in osteoblast of Fto (Oc

43 The ketogenic diet induced hepatic lipid oxidation and ketogenesis, an

44 of integrin signaling in the pathogenesis of diet-induced hepatic insulin resistance.

45 l oxidation of fat in the liver and reversed diet-induced hepatic steatosis and insulin resistance.

46 cific disruption of Hif2a, in which high-fat-diet-induced hepatic steatosis and obesity were substant

47 SLC13A5 deletion protects mice from high-fat diet-induced hepatic steatosis and that mutation of the

48 Furthermore, established high fat diet-induced hepatic steatosis was effectively reduced w

49 This study showed that diet-induced hepatosteatosis, hyperlipidemia, and insuli

50 adipose tissue and have shown that high-fat diet-induced (HFD-induced) insulin resistance is mitigat

51 However, diet-induced high Hcy resulted in a significant increase

52 al diet affects HMO biosynthesis and how any diet-induced HMO alterations influence the infant gut mi

53 epatic DGAT2 deficiency successfully reduces diet-induced HS and supports development of DGAT2 inhibi

54 lin sensitivity both in in vitro and in vivo diet-induced hyperinsulinemic mouse model.

55 not in Nkx2.1-lineage neural cells, promoted diet-induced hyperphagia and obesity in both male and fe

56 To explore the potential role of diet-induced inflammation, we examined the association b

57 Diet-induced insulin resistance (IR) adversely affects h

58 r dysfunction relative to dietary obesity or diet-induced insulin resistance in male mice.

59 sis in both beta cells and adipocytes during diet-induced insulin resistance in mice.

60 uorophore sodium fluorescein (NaFl), whereas diet-induced insulin resistance increased permeability t

61 vealed protection against BBB breakdown with diet-induced insulin resistance, despite comparable meta

62 ized BBB permeability in wild-type mice with diet-induced insulin resistance.

63 glucose production in rodents with high fat diet-induced insulin resistance.

64 and increased as expected in the setting of diet-induced insulin resistance.

65 eased the susceptibility to high cholesterol diet-induced liver injury and abolished the protective e

66 , we show that Zbtb20 ablation protects from diet-induced liver steatosis and improves hepatic insuli

67 ediators of whole-body glucose regulation in diet-induced metabolic disease.

68 K2 kinase inactivation also reduced high-fat diet-induced metabolic diseases.

69 ge proinflammatory activation and preventing diet-induced metabolic dysfunction.

70 tatus at baseline, E3FAD mice showed greater diet-induced metabolic impairments.

71 enteric lymphatic vessels from high-fructose diet-induced metabolic syndrome (MetSyn) rats exhibited

72 Maternal diet-induced mild iron deficiency decreased offspring pe

73 rbates or alleviates, respectively, high-fat diet-induced mitochondrial dysfunction, hepatosteatosis,

74 C57BL6/J mice were subjected to a diet-induced model of CKD by delivery of adenine for six

75 In a diet-induced mouse model of non-alcoholic fatty liver di

76 show that CSE knockout exacerbated high-fat diet-induced mouse obesity as well as its related insuli

77 Here we used a diet-induced murine model of T2D to investigate the unde

78 Diet-induced NAFLD was associated with a nearly doubled

79 induced NASH or methionine-choline deficient diet-induced NASH in mice.

80 sis, inflammation, and fibrosis in a Western diet-induced NASH mouse model.

81 As expected, betaine prevented MCD diet-induced NASH.

82 lic steatohepatitis (NASH), and in mice with diet-induced NASH.

83 line-deficient l-amino acid-defined high-fat diet-induced NASH.

84 in vivo lipopolysaccharide- and atherogenic diet-induced NF-kappaB activation.

85 c trajectories of hepatic macrophages during diet-induced non-alcoholic steatohepatitis (NASH).

86 estigated high-fat/high-carbohydrate (HF/HC) diet-induced nonalcoholic fatty liver disease (NAFLD) in

87 lar and interstitial macrophages in high-fat diet induced obese mice were lower than regular chow die

88 eated regular chow diet-fed mice or high-fat diet induced obese mice with lipopolysaccharide (LPS) or

89 glucose, lipid and cholesterol metabolism in diet induced obese rodents.

90 ious studies have reported that treatment of diet-induced obese (DIO) male mice with adropin(34-76) (

91 ine GIPR antibody (muGIPR-Ab) that protected diet-induced obese (DIO) mice against body weight gain a

92 metabolism in perfused livers from lean and diet-induced obese (DIO) mice and validated the HP obser

93 halamic expression of Ctbp2 was increased in diet-induced obese (DIO) mice as compared with age-match

94 Gubra-Amylin NASH (GAN) diet-induced obese (DIO) mice represent a model of fibro

95 st reduction in body weight in both lean and diet-induced obese (DIO) mice, when compared with both v

96 Grb14-shRNA) improves glucose homeostasis in diet-induced obese (DIO) mice.

97 agent with profound anti-obesity effects in diet-induced obese (DIO) mice.

98 adigm reduced high-fat intake and obesity in diet-induced obese (DIO) mice.

99 nsulin secretion and decrease body weight in diet-induced obese (DIO) mice.

100 Here we report that in the NTS of high-fat diet-induced obese (DIO) rats, the apoA-IV mRNA level is

101 s compared to monotherapies in both lean and diet-induced obese (DIO) rats.

102 not on improving insulin sensitivity in both diet-induced obese and lean mice.

103 Diet-induced obese animals received either Lactobacillus

104 ng body weight gain relative to control in a diet-induced obese dog model, suggesting the importance

105 ial fibrosis were significantly increased in diet-induced obese mice as compared with controls.

106 al duration, and reversed atrial fibrosis in diet-induced obese mice as compared with controls.

107 n acetylation-defective SIRT3-K57R mutant in diet-induced obese mice decreased acetylation of mitocho

108 Diet-induced obese mice lacking lncOb show increased fat

109 mpromised intestinal BCRP functions and that diet-induced obese mice recapitulate these outcomes.

110 am signals TNFSF11A or NDUFAB1 in the MBH of diet-induced obese mice reverses mitochondrial elongatio

111 GM-CSF neutralization in diet-induced obese mice significantly reduced immunosupp

112 Pacing-induced AF in 100% of diet-induced obese mice versus 25% in controls (P<0.01)

113 Diet-induced obese mice were randomized to VSG or sham s

114 s food intake, body weight, and adiposity in diet-induced obese mice when administered once daily for

115 We further found that diet-induced obese mice with mild hyperbilirubinemia hav

116 In diet-induced obese mice, expression of EAT gene induces

117 Diet-induced obese mice, including wild-type or whole bo

118 Here we show that in hypercaloric diet-induced obese mice, persistently activated microgli

119 that ob/ob mice, as well as leptin-resistant diet-induced obese mice, show significant reductions of

120 In diet-induced obese mice, the coadministration of a perip

121 and puncture model of sepsis in lean and in diet-induced obese mice, we demonstrate that obese diabe

122 verexpression of NRG4 reduced weight gain in diet-induced obese mice, while overexpression of ANGPTL8

123 ucyl-phenylalanine, are elevated in high-fat diet-induced obese mice.

124 nd whole-body insulin resistance in high-fat-diet-induced obese mice.

125 increased methylation in adipose tissue from diet-induced obese mice.

126 miR-7 expression is altered in the brains of diet-induced obese mice.

127 ood glucose and improve glucose tolerance in diet-induced obese mice.

128 osure relieved hepatic steatosis in high-fat diet-induced obese mice.

129 worsens metabolic dysfunction in genetic and diet-induced obese mice.

130 underlying electrophysiological mechanisms a diet-induced obese mouse model was used.

131 ebrafish and in live tissues from a high-fat diet-induced obese mouse model.

132 titis (NASH) improve liver histopathology in diet-induced obese mouse models of biopsy-confirmed NASH

133 For this purpose, we used diet-induced obese rats and rats administered thapsigarg

134 Diet-induced obese rats were randomized to isocaloric di

135 ust alteration in the myocardial proteome of diet-induced obese rats, even before functional impairme

136 Diet-induced obese rats, which remained relatively hyper

137 es, adipose tissue macrophages isolated from diet-induced obese Ucp2(DeltaLysM) mice showed decreased

138 ockdown causes hypophagia and weight loss in diet-induced obese wild-type mice; however, these effect

139 Oral administration of Pep19 into diet-induced obese Wistar rats significantly reduces adi

140 ctions to leptin receptor-deficient (db/db), diet-induced obese, and control mice; pancreatic islets

141 paralleled profiles from long-term high-fat diet induced obesity in males.

142 to the gut microbiota retards development of diet induced obesity in wild-type mice.

143 oside A and sucralose on NASH using high fat diet induced obesity mouse model by substituting fructos

144 We used a mouse model of maternal-diet induced obesity to define predictive correlations b

145 of Dennd5b results in resistance to western diet induced obesity, changes in plasma lipids, and redu

146 ion of Hdac3 (Hdac3(IKO)) protects mice from diet induced obesity.

147 n weight gain and energy balance in high fat diet induced obesity.

148 improves diabetic dyslipidemia in mice with diet-induced obesity (DIO mice).

149 B1) antagonists have been shown to attenuate diet-induced obesity (DIO) and associated inflammation,

150 lin sensitivity and fasting blood glucose in diet-induced obesity (DIO) and db/db mouse models.

151 t protein-coding mRNAs, are repressed during diet-induced obesity (DIO) and refeeding, whilst nutrien

152 However, obese humans and mice with diet-induced obesity (DIO) are resistant to leptin becau

153 Its reduced levels in diet-induced obesity (DIO) contribute to hyperleptinemia

154 stemically quenched the blood sugar level in diet-induced obesity (DIO) diabetic mice, it reduced ost

155 We report that diet-induced obesity (DIO) in mice increased plasma and

156 ought to establish whether the propensity to diet-induced obesity (DIO) is associated with addictive-

157 In the diet-induced obesity (DIO) mouse model, activation of li

158 sing three different mouse models of obesity-diet-induced obesity (DIO), leptin receptor (LepR)-null,

159 In mice subjected to diet-induced obesity (DIO), we observed similar increase

160 , pADORA(1) signaling facilitates a high-fat diet-induced obesity (DIO).

161 ular mechanisms underlying susceptibility to diet-induced obesity (DIO).

162 Maternal diet-induced obesity alters muscle mitochondrial functio

163 mice were completely protected from high-fat diet-induced obesity and accompanying metabolic impairme

164 enes in BAT, and are protected from high-fat diet-induced obesity and development of insulin resistan

165 y weight but specifically prevents excessive diet-induced obesity and ensuing metabolic impairments.

166 male zebrafish increased the propensity for diet-induced obesity and fasting hyperglycemia in adulth

167 Diet-induced obesity and food allergies increase in tand

168 fuel oxidation and thermogenesis, leading to diet-induced obesity and glucose intolerance.

169 issues were resistant to developing high-fat diet-induced obesity and had significantly reduced white

170 Mice globally lacking Them2 are resistant to diet-induced obesity and hepatic steatosis, and exhibit

171 lism and thermogenesis, and protects against diet-induced obesity and hepatic steatosis.

172 1)R deletion (B(1) (-/-)) protects mice from diet-induced obesity and improves insulin and leptin sen

173 tivation of Hh signaling suppresses high-fat-diet-induced obesity and improves whole-body glucose tol

174 t Opn3-knockout (Opn3-KO) mice were prone to diet-induced obesity and insulin resistance.

175 ed energy expenditure and are protected from diet-induced obesity and insulin resistance.

176 ases energy expenditure and protects against diet-induced obesity and insulin resistance.

177 Aifm2, thus, can ameliorate diet-induced obesity and insulin resistance.

178 overexpression of the IR protects mice from diet-induced obesity and its effects on glucose metaboli

179 pothesis that Nnmt deletion protects against diet-induced obesity and its metabolic consequences in m

180 e that LCN2 is dispensable for both high fat diet-induced obesity and its therapeutic reduction by ce

181 epresents a promising approach to ameliorate diet-induced obesity and leptin resistance.

182 rgy expenditure and amelioration of high-fat-diet-induced obesity and markedly improved glucose toler

183 geted activation of Hh signaling ameliorates diet-induced obesity and may be explored for pharmaceuti

184 dy suggests that blocking of CB1 ameliorates Diet-Induced Obesity and metabolic disorder by modulatin

185 ies additional novel therapeutic targets for diet-induced obesity and metabolic disorder.

186 egulated microbiota protect BALB/c mice from diet-induced obesity and metabolic dysfunction.

187 Finally, moderate alcohol prevented high-fat diet-induced obesity and metabolic dysfunction.

188 xpression of SH2B1 protects against high fat diet-induced obesity and metabolic syndromes.

189 pe mice, NaHS treatment ameliorates high fat diet-induced obesity and metabolism disorders, indicatin

190 ntrolling the proliferation of beta cells in diet-induced obesity and suggest that selective targetin

191 ic insulin resistance and hepatosteatosis in diet-induced obesity are associated with various metabol

192 mproves insulin sensitivity substantially in diet-induced obesity by both peripheral and central mech

193 R in adipose tissue controls the response to diet-induced obesity by promoting adipose tissue expansi

194 Diet-induced obesity causes hyperinsulinemia and diminis

195 Here we show that mice with diet-induced obesity display mislocalization of Par3, a

196 exercise on type II diabetes risk under a HF diet-induced obesity environment.

197 aches of leptin therapy for the treatment of diet-induced obesity have been ineffective.

198 Diet-induced obesity impaired AT1-ILC killing ability.

199 MH-specific inhibition of TBK-1 in mice with diet-induced obesity impaired glucose metabolism and AKT

200 logy and satellite cell dynamics compared to diet-induced obesity in irradiated muscle, and have impl

201 oting AgRP neurons during the development of diet-induced obesity in mice.

202 on of Id1 causes age-associated and high-fat diet-induced obesity in mice.

203 ative stress in the metabolic alterations in diet-induced obesity in rats.

204 Estrogens protect against diet-induced obesity in women and female rodents.

205 Maternal diet-induced obesity increased miR-126 expression howeve

206 High-fat diet-induced obesity is a major risk factor for osteoart

207 ate and testing its effectiveness to prevent diet-induced obesity later in life.

208 sulin sensitivity and glucose control in the diet-induced obesity mouse model after both acute and ch

209 In a diet-induced obesity mouse model, daily subcutaneous adm

210 ong-acting GLP-1 analog is demonstrated in a diet-induced obesity mouse model.

211 , are present in naive CD4(+) T cells from a diet-induced obesity murine model and that elevated O-Gl

212 Relative to the overweight and diet-induced obesity regimens, CR decreased body weight,

213 Diet-induced obesity resulted in increased muscle fibros

214 ocytic genes from islets of rodent models of diet-induced obesity that significantly overlap with clo

215 h was slightly inhibited and protection from diet-induced obesity was less complete.

216 Our findings suggest that the combination of diet-induced obesity with other risk factors may increas

217 ting) and excessive energy storage (high-fat diet-induced obesity) blunt the effect of leptin.

218 5(-/-) (double knock-out (DKO)) mice show HF diet-induced obesity, adipocyte hypertrophy, and present

219 to test the role of high dietary fat intake, diet-induced obesity, and associated changes in gut micr

220 proves insulin sensitivity, protects against diet-induced obesity, and elicits the browning of white

221 aling and insulin action that manifests with diet-induced obesity, as insulin action is preserved to

222 role in different models of NCDs, including diet-induced obesity, atherosclerosis, and inflammation-

223 r steady state and under metabolic stress by diet-induced obesity, but we observed increases in both

224 r steady state and under metabolic stress by diet-induced obesity, but we observed increases in proli

225 Monoacylglycerol lipase deficiency affects diet-induced obesity, fat absorption, and feeding behavi

226 -) mice would exhibit altered progression of diet-induced obesity, fatty liver, and insulin resistanc

227 cific deletion of P2Y(6)R protects mice from diet-induced obesity, improving glucose tolerance and in

228 nvestigated the effects of Lcn2 depletion on diet-induced obesity, inflammation, and PDAC development

229 er of the EAT gene to mice prevents high-fat diet-induced obesity, insulin resistance and fatty liver

230 Importantly, CD81 loss causes diet-induced obesity, insulin resistance, and adipose ti

231 rexpression (Adipo-TFEB) were protected from diet-induced obesity, insulin resistance, and metabolic

232 rovide mechanistic insights of MGL's role in diet-induced obesity, lipid metabolic disorder, and regu

233 e receptor (Drd1)-null mice are resistant to diet-induced obesity, metabolic disease, and circadian d

234 der to determine whether miR-146a influences diet-induced obesity, mice that were either wild type (W

235 y of LCN2 altered neither the development of diet-induced obesity, nor the ability of celastrol to pr

236 sis was tested by comparing the wild-derived diet-induced obesity- (DIO-) resistant mouse strain WSB/

237 o evidence for its involvement in developing diet-induced obesity.

238 ds and undergoes extensive remodeling during diet-induced obesity.

239 me, and glucose metabolism in the context of diet-induced obesity.

240 ic lymphangiogenesis during 16-week high-fat diet-induced obesity.

241 ssue inflammation has on metabolic health in diet-induced obesity.

242 impairs adaptive thermogenesis and promotes diet-induced obesity.

243 d adipocyte mitochondrial quality control in diet-induced obesity.

244 arged beta cell area and hyperinsulinemia in diet-induced obesity.

245 gene Cd36 and promoting lipid absorption and diet-induced obesity.

246 impaired glucose homeostasis associated with diet-induced obesity.

247 ythmicity in metabolism and is implicated in diet-induced obesity.

248 at mass loss on a normal diet after high fat diet-induced obesity.

249 hR-deficient females (VEH) were resistant to diet-induced obesity.

250 Thy1-knockout mice are prone to diet-induced obesity.

251 s in reduced weight gain and protection from diet-induced obesity.

252 nd increases food intake leading to high-fat diet-induced obesity.

253 the MARC2 KO mice were resistant to high-fat diet-induced obesity.

254 se fails to correct metabolic dysfunction in diet-induced obesity.

255 mma activity enhanced insulin sensitivity in diet-induced obesity.

256 eta cell function and islet compensation for diet-induced obesity.

257 t-reducing effects of leptin in rodents with diet-induced obesity.

258 sion of miR-26a protected mice from high-fat diet-induced obesity.

259 decreased energy expenditure, and increased diet-induced obesity.

260 deficiency-induced colitis, and ameliorates diet-induced obesity.

261 at diet (HFD), IRMOE mice are protected from diet-induced obesity.

262 ation of hepatic Dpp4 in young mice prone to diet-induced obesity.

263 electrophysiological properties observed in diet-induced obesity.

264 inflammation in the NAcc in animal models of diet-induced obesity.

265 ts may define a prothrombotic risk factor in diet-induced obesity.

266 sm in the TME, detailing how it changes with diet-induced obesity.

267 IF-1alpha and its target genes in ATM during diet-induced obesity.

268 erexpression inhibits lipolysis and promotes diet-induced obesity.

269 e of Drd1 expression within the SCN restores diet-induced overconsumption, weight gain, and obesogeni

270 and carbohydrates consumption) combined with diet-induced overweight/obesity on the risk of periodont

271 n better glucose tolerance during a high-fat diet-induced regain phase (all, P < .05).

272 l-like receptors TLR2 and TLR4 inhibited the diet-induced replication of beta cells in mice and human

273 In a high-fat, high-cholesterol diet-induced rodent model of NAFLD, we observed a progre

274 Both diets induced severe hepatic steatosis in the LTKO mice

275 ammadeltaT cell recruitment protects against diet-induced SH and accelerates disease resolution.

276 The NASH diet induced significant changes in Kupffer cell enhance

277 fter energy restriction, both the MHP and LF diets induced similar significant decreases in adiposity

278 Mice with chemically induced fibrosis or diet-induced steatohepatitis given nintedanib or aspirin

279 inflammation and hyperglycemia in mice with diet-induced steatohepatitis.

280 We used a mouse model of simple, diet-induced steatosis and assessed the impact of exerci

281 ockout in mice alleviates the development of diet-induced steatosis and fibrosis and causes activatio

282 r; LCR) displayed susceptibility to high fat diet-induced steatosis in association with reduced hepat

283 manner consistent with findings in patients, diet-induced steatosis increases circulating PCSK9 level

284 es lipid catabolism and ameliorates high-fat-diet-induced steatosis.

285 hod in mice and dogs and allows us to detect diet-induced subtle changes in ApoAI turnover in mice.

286 eceptor (Insr) deletion model, we found that diet-induced T reg dysfunction is driven by T reg-intrin

287 ration, and heat production during cold- and diet-induced thermogenesis.

288 The NGR represents an animal model of diet-induced Type 2 Diabetes Mellitus (T2DM), exhibiting

289 tozocin-induced type 1 diabetes and high fat diet-induced type 2 diabetes mouse models and liver-spec

290 stress in mice completely abolishes high fat diet-induced upregulation of TRIP-Br2 in BAT.

291 exogenous rGDF11, but not rGDF8, can reduce diet-induced weight gain and improve metabolic homeostas

292 Asxl2DeltaLysM) were completely resistant to diet-induced weight gain and metabolically normal despit

293 c MyD88 or IRAK2 deficiency reduced high-fat-diet-induced weight gain, increased energy expenditure a

294 total cholesterol levels and suppression of diet-induced weight gain.

295 Diet-induced weight loss (WL) is usually accompanied by

296 group were also evaluated before and after a diet-induced weight loss of 10%.RESULTSThe contribution

297 We also evaluated the effect of diet-induced weight loss on insulin secretion in people

298 Diet-induced weight loss restores IEL number and CD103/C

299 approach to aiding weight maintenance after diet-induced weight loss.

300 r into the developmental origins of obesity, dieting-induced weight gain, and anorexia nervosa.