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1                   Treatment usually involves dietary modification.
2 ithelial TLR4 that could be reversed through dietary modification.
3 e aorta and use it to assess the response to dietary modification.
4 easily achieved in the general population by dietary modification.
5  to influence cardiovascular disease risk by dietary modification.
6  including active therapeutic approaches and dietary modification.
7 increased significantly from baseline during dietary modification.
8 pies in glioma treatment with a non-invasive dietary modification.
9  including the role of physical activity and dietary modification.
10 e to the financial implications of drugs and dietary modifications.
11 icrobiome composition changes in response to dietary modifications.
12 cal conditions, myopathy may be mitigated by dietary modifications.
13 ound the world for patients nonresponsive to dietary modifications.
14 ng early development in response to maternal dietary modifications.
15 ists (e.g., tirzepatide) as well as specific dietary modifications.
16 inical trials are needed to encourage use of dietary modification across the cancer continuum.
17  injections, regular glucose monitoring, and dietary modifications; all of which are severely impacte
18 ve significant dyslipoproteinemia after both dietary modification and fish oil supplementation.
19                                              Dietary modification and increased physical activity are
20                                           If dietary modification and phlebotonics are ineffective, g
21 in atherosclerotic plaques, and confirm that dietary modification and statin therapy reduce MMP activ
22 hibitor (MPI) and to determine the effect of dietary modification and statin treatment on MMP activit
23 jor categories of interventions are covered: dietary modification and supplementation, herbal product
24 estyle changes in Western societies, such as dietary modifications and increased antibiotic use.
25  symptom recognition and response, fluid and dietary modification, and lifestyle management.
26 are model in which behavioral interventions, dietary modification, and medications are considered as
27 ation of residual kidney function, dialysis, dietary modification, and medications.
28       Pediatric patients, patients receiving dietary modification, and patients with impaired glucose
29 iation of new subspecialist care, medication/dietary modifications, and furthering life-saving proced
30 ty-optimizing dietary changes, more feasible dietary modifications, and optimized vegan dietary chang
31 tric emptying in the absence of obstruction; dietary modifications are first-line treatment.
32                 For most patients, lifestyle/dietary modifications are helpful in reducing GERD sympt
33 ials of the effects of specific nutrients or dietary modifications are not always feasible, they prov
34                                              Dietary modifications are often sufficient in the manage
35 vention studies are needed to assess whether dietary modification around the time of conception can r
36  suggested that environmental modifications, dietary modifications, artificial tears and lubricants,
37 ditions, behavioral factors, and hormone and dietary modification assignments.
38                                              Dietary modification can affect sleep quality.
39 ion trials are testing whether postdiagnosis dietary modification can influence breast cancer recurre
40 changes such as weight loss in obese men and dietary modification can lessen urgency, nocturia, and i
41                We conclude the following: 1) Dietary modification can reduce spontaneous IDDM express
42           However, behavioral techniques and dietary modifications can be effective in treatment of I
43 ses could open a new era of treatment, where dietary modifications can be used as an adjunct to boost
44                                              Dietary modifications can limit tumour-specific nutritio
45                             In addition, the dietary modification component of the Women's Health Ini
46 th better survival, suggesting postdiagnosis dietary modification consistent with type II diabetes pr
47                            We tested whether dietary modification (DM) altered the risk for incident
48 lyses of the Women's Health Initiative (WHI) Dietary Modification (DM) clinical trial that evaluated
49                                          The dietary modification (DM) clinical trial, within the Wom
50                The Women's Health Initiative Dietary Modification (DM) Trial was a randomized control
51 in women (n ~ 9000) not participating in the dietary modification (DM) trial, which focused on the re
52 nd 100-day-old HC rats in response to the HC dietary modification during the suckling period.
53 nd iNOS expression, and that weight loss via dietary modification effectively reverses these deleteri
54               Multiple interventions such as dietary modification, exercise, antioxidants, and antihy
55                                              Dietary modifications, exercise, and medications have be
56 tical review of the most recent evidence for dietary modifications, food supplements, and herbs in pr
57 n MP density were obtained within 4 weeks of dietary modification for most, but not all, subjects.
58                               We report that dietary modification from a soy-based diet to a casein-b
59                 Early detection coupled with dietary modification greatly reduces pathology, but the
60 loss in obese men with diabetes and LUTS and dietary modification has also been shown to be effective
61                         However, evidence on dietary modification in preventing cognitive decline fro
62      We have also analyzed the importance of dietary modification in primary prevention.
63                         The effectiveness of dietary modification in reducing low-density lipoprotein
64                    These results show that a dietary modification in the early postnatal life of the
65 opathy and warrant further investigations of dietary modification in the management of these conditio
66 gned to lower plasma cholesterol by means of dietary modifications in accordance with recommendations
67 iabetes and decreased excretion in CKD or by dietary modifications in these disease groups.
68 alities for obtaining weight loss in obesity-dietary modification, increasing physical activity, phar
69 novel index [Women's Health Initiative (WHI) Dietary Modification Index (DMI)] of diet quality was as
70   Participants were randomly assigned to the dietary modification intervention (n = 19,541; 40%) or t
71          Women were randomly assigned to the dietary modification intervention group (40% [n = 19,541
72 ry fat intake was significantly lower in the dietary modification intervention group compared with th
73                                              Dietary modification is central to obesity treatment.
74              These findings suggest that, if dietary modification is effective in reducing GERD, subs
75               When MP density increased with dietary modification, it remained elevated for at least
76                                              Dietary modification lasted up to 15 weeks.
77  the effect of the Women's Health Initiative Dietary Modification low-fat and increased fruit, vegeta
78 tabolism, suggesting that multi-generational dietary modifications may be driving reduced prevalence
79 tries, but epidemiological data suggest that dietary modification might reduce these by as much as 90
80                       Finally, we review how dietary modifications modulate hepatic gene expression p
81 ding women involves lifestyle modifications, dietary modifications, non-pharmaceutical remedies and p
82     Beyond well-known interventions (such as dietary modifications), novel therapeutics (such as smal
83       This review examines the potential for dietary modification of raft structure and function in t
84 ther evidence may support physical activity, dietary modification, omega-3 fatty acid supplementation
85 couraging clinical research on the impact of dietary modifications on COPD-related clinical outcomes.
86 decline, or AD itself, could be prevented by dietary modification or food fortification.
87 athways are amenable to manipulation through dietary modifications or drug therapies.
88                          Therefore, a single dietary modification overwhelms vital host antimicrobial
89                        Most importantly this dietary modification partially rescued overall neurologi
90 For several AADs, including phenylketonuria, dietary modification prevents physiological deterioratio
91 tance given the comparative ease of maternal dietary modifications relative to the low likelihood of
92 alculations of body-weight change and of the dietary modifications required for weight-loss maintenan
93 examines the current indications for various dietary modification strategies in patients with CKD (eg
94                   Mounting evidence supports dietary modifications, such as the low-fermentable oligo
95                                      Certain dietary modifications that appear to be effective in mod
96 ced bronchoconstriction (EIB) may respond to dietary modification, thereby reducing the need for phar
97                        Asthma may respond to dietary modification, thereby reducing the need for phar
98                                              Dietary modification therefore has the promise of reduci
99                                              Dietary modifications through decreasing fructose intake
100 ed lactose intolerance may result in adverse dietary modifications; thus, more studies are needed to
101 ic obesity have been focused on exercise and dietary modifications to reduce fat while maintaining mu
102                    Our findings suggest that dietary modifications to reduce insulinemic potential ma
103                           Treatment involves dietary modification, to reduce lymphatic dilation in re
104 en enrolled in the Women's Health Initiative dietary modification trial comparison group and 59,105 w
105 articipants in the Women's Health Initiative Dietary Modification Trial completed a doubly labeled wa
106    Purpose Earlier Women's Health Initiative Dietary Modification trial findings suggested that a low
107 led onto the Women's Health Initiative (WHI) Dietary Modification trial from 1993 to 1998 were random
108                The Women's Health Initiative Dietary Modification Trial tested the effects on chronic
109  PARTICIPANTS: The Women's Health Initiative Dietary Modification Trial, a randomized controlled tria
110 5 203 women in the Women's Health Initiative Dietary Modification Trial, of which 23 776 participants
111 mone therapy trial, and randomization to the dietary modification trial, the predictive model include
112 articipated in the Women's Health Initiative Dietary Modification Trial.
113 tion phases of the Women's Health Initiative Dietary Modification Trial.Participants comprised 48,835
114 articipated in the Women's Health Initiative Dietary Modification Trial; 40% (19,541) were randomized
115                                 Although the dietary modification trials did not show any significant
116 pinach and corn, three types of responses to dietary modification were identified: Eight "retinal res
117 ain management (typically acetaminophen) and dietary modification with a clear liquid diet.
118 results highlight the potential need to pair dietary modification with microbial therapies to control
119         Management approaches should balance dietary modification with recognition of psychological f
120 p new knowledge about specific nutrients and dietary modifications within a framework of interaction

 
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