ライフサイエンスコーパス: dietary sodium

1 gainst hypertension in response to increased dietary sodium.

2 re different from the chronic effects of low dietary sodium.

3 onsume nearly twice the recommended level of dietary sodium.

4 gets for packaged foods and other sources of dietary sodium.

5 vide information and strategies for reducing dietary sodium.

6 Dietary sodium affects function of the beta-2 adrenocept

7 itors in patients with CKD; however, whether dietary sodium also associates with progression to ESRD

8 Furthermore, the BP effect of dietary sodium among individuals taking antihypertensive

9 were used to identify top-ranking sources of dietary sodium among the population aged 2 y and older b

10 lifestyle counseling to achieve weight loss, dietary sodium and alcohol reduction, and increased phys

11 Evidence relating dietary sodium and blood pressure comes from a variety o

12 nstrated a dose-response association between dietary sodium and blood pressure in humans.

13 ences in magnesium metabolism in response to dietary sodium and calcium during rapid bone growth.

14 has identified a direct relationship between dietary sodium and cardiovascular disease (CVD), special

15 s was used to assess the association between dietary sodium and CFR.

16 We examined the relation between habitual dietary sodium and coronary flow reserve (CFR), which is

17 Both dietary sodium and dietary potassium depletion substanti

18 Dietary sodium and energy intake were estimated at basel

19 emia have traditionally been associated with dietary sodium and fat intakes, respectively; however, t

20 pressure over the life span and response to dietary sodium and highlights experimental models invest

21 ere is abundant evidence that a reduction in dietary sodium and increase in potassium intake decrease

22 Due to the importance of dietary sodium and its paucity within many inland enviro

23 verse sample of Hispanic/Latino adults, high dietary sodium and low dietary potassium were independen

24 larify the health consequences of changes in dietary sodium and potassium and that characterize adher

25 Current intake of dietary sodium and potassium fails to meet guideline rec

26 Whether dietary sodium and potassium intake affect CKD progressi

27 effective in achieving population goals for dietary sodium and potassium intake in the USA.

28 Mean dietary sodium and potassium intakes and the mean potass

29 We examined the relation between dietary sodium and potassium intakes and the ratio of so

30 h has the potential to improve estimation of dietary sodium and potassium intakes in observational st

31 Dietary sodium and potassium intakes were averaged from

32 authors have improved the estimation of mean dietary sodium and potassium intakes.

33 identified 8 novel loci for BP responses to dietary sodium and potassium intervention and cold press

34 Blood pressure (BP) responses to dietary sodium and potassium intervention and cold press

35 Associations between dietary sodium and potassium with cardiovascular disease

36 study aimed to evaluate the relationship of dietary sodium and potassium with incident cardiovascula

37 d in analyses that controlled for body mass, dietary sodium and ratio of sodium to potassium, and alc

38 High intakes of dietary sodium are associated with elevated blood pressu

39 ent animal and human studies have implicated dietary sodium as a risk factor in MS, whereby high sodi

40 We suggest that the presence of dietary sodium at distal exchange sites did not enhance

41 reduced sodium intake reported in RCTs with dietary sodium at the 2000 to 3000 mg/d range as opposed

42 sted within pairs: a 1000-mg/d difference in dietary sodium between brothers was associated with a 10

43 in the absence of an increase in BP, excess dietary sodium can adversely affect target organs, inclu

44 Dietary sodium can alter peripheral and central neurotra

45 itivity refers specifically to the effect of dietary sodium chloride (salt) intake on BP.

46 Most dietary sodium comes from meats (including poultry and f

47 The majority (71%) of US dietary sodium comes from restaurant and packaged foods.

48 um/water excretion in response to changes in dietary sodium concentration, but likely becomes critica

49 agnitude of these associations suggests that dietary sodium consumption is unlikely to be an importan

50 ading to a state of high aldosterone despite dietary sodium consumption.

51 epinephrine levels were inversely related to dietary sodium content.

52 Because dietary sodium depletion markedly increases ouabain-inse

53 same extent as WT mice, even during profound dietary sodium depletion, as a result of the upregulatio

54 (+) exchange mRNA abundance was increased by dietary sodium depletion.

55 After 8-day dietary sodium deprivation, immunoreactivity for c-Fos (

56 on in the HSD2 neurons was not influenced by dietary sodium deprivation.

57 ns and percentiles of usual intakes of daily dietary sodium (dNa) and potassium (dK) and 24-h urine e

58 Increasing dietary sodium drives the thirst response.

59 Here, we show that high dietary sodium exacerbates EAE in a strain- and sex-spec

60 Most Americans consume dietary sodium exceeding age-specific government-recomme

61 itures, chronic supraphysiological intake of dietary sodium, excessive alcohol consumption, and psych

62 Patients with high dietary sodium exhibited a blunted antiproteinuric effec

63 and diastolic BP across the entire range of dietary sodium exposure.

64 Dietary sodium, fluid, and SSB intakes were assessed wit

65 Excess dietary sodium has been linked to elevations in blood pr

66 Population-wide restriction of dietary sodium has been recommended.

67 Access to excess dietary sodium has heightened the risk of cardiovascular

68 n with prepared meals, investigating optimal dietary sodium in heart failure comes with challenges, i

69 Soy sauce is a major source of dietary sodium in Malaysia.

70 e, but debate over recommendations to reduce dietary sodium in the general population continues.

71 function studies and responses to changes in dietary sodium in the PKGIalpha mutant mice are normal.

72 would result from a substantial lowering of dietary sodium in the US population could reduce cardiov

73 We identified top sources of dietary sodium in US children from birth to age 24 mo.

74 We studied the effect of different levels of dietary sodium, in conjunction with the Dietary Approach

75 With the data now available concerning dietary sodium, including the minimal and specific blood

76 ack girls than in white girls, regardless of dietary sodium intake (P < 0.001).

77 CI: 0.46, 1.62 servings/d), P = 0.0004], and dietary sodium intake [SMD: -0.39 (-0.58, -0.20), P = 0.

78 y aims to describe the levels and sources of dietary sodium intake among Nigerian adults to inform th

79 The relationship between dietary sodium intake and blood pressure (BP) has been t

80 of SSBs, identifying a possible link between dietary sodium intake and excess energy intake.

81 the combined adverse influences of excessive dietary sodium intake and increased serum uric acid duri

82 Excess dietary sodium intake and retention lead to hypertension

83 therefore typically based on restriction of dietary sodium intake and the use of diuretic agents to

84 recommendations for universal restriction of dietary sodium intake are based on associations of sodiu

85 and in high-income countries argues against dietary sodium intake being a culprit of curtailing life

86 Most dietary sodium intake came from home cooked foods, nearl

87 In POTS patients, high dietary sodium intake compared with low dietary sodium i

88 In contrast, dietary sodium intake did not affect NCC activity in kno

89 Finally, dietary sodium intake did not alter urinary potassium ex

90 ion Examination Survey, a 1 g per day higher dietary sodium intake estimated using dietary recall que

91 rambled ODN-treated rats, chronic changes in dietary sodium intake evoked an endogenous, hypothalamic

92 esent the best available methods to estimate dietary sodium intake from casual urine samples.

93 Excess dietary sodium intake has been associated with death and

94 Reduction of dietary sodium intake has been identified as a priority

95 Since dietary sodium intake has been identified as a risk fact

96 Excessive dietary sodium intake has known adverse effects on intra

97 ongoing controversy about the importance of dietary sodium intake in blood pressure control, conside

98 the use of spot urine specimens to estimate dietary sodium intake in patients with CKD and research

99 The policy of lowering dietary sodium intake in the general population may need

100 For decades, dietary sodium intake in the United States has remained

101 high dietary sodium intake compared with low dietary sodium intake increases plasma volume, lowers st

102 animal studies both have suggested that high dietary sodium intake increases stroke mortality.

103 Dietary sodium intake is most precisely ascertained by u

104 Estimation of dietary sodium intake is problematic.

105 These findings suggest that restriction of dietary sodium intake may be a cost-effective and low-ri

106 The extent to which dietary sodium intake may confer alterations in the infl

107 Evidence suggests that dietary sodium intake needs reduction.

108 The high dietary sodium intake of US children and adolescents may

109 emiologic evidence regarding the outcomes of dietary sodium intake on mortality remains limited for l

110 DCT is essential for mediating the effect of dietary sodium intake on NCC activity is unknown.

111 Dietary sodium intake regulates the thiazide-sensitive N

112 tion, including its recommendation to reduce dietary sodium intake to <1500 mg/d.

113 Mean (SD) dietary sodium intake was 4512 (2632) mg/d in Black indi

114 Dietary sodium intake was assessed by self-report questi

115 Dietary sodium intake was assessed using a validated foo

116 Overall, mean dietary sodium intake was low in 0-5.9-mo-old children,

117 Dietary sodium intake was not significantly associated w

118 Dietary sodium intake was positively associated with flu

119 alysis of the cross-sectional association of dietary sodium intake with hypertension-related outcomes

120 contradictory results on the association of dietary sodium intake with risk of CVD, and this relatio

121 ied, the results will facilitate tracking of dietary sodium intake within populations over time and i

122 fferences in urinary sodium, an indicator of dietary sodium intake, are associated with blood pressur

123 of ethanol ingestion, pack-years of smoking, dietary sodium intake, dietary calcium intake, blood lea

124 duct (CD) cells varies widely in response to dietary sodium intake, GFR, circulating hormones, neural

125 participants had a significant reduction in dietary sodium intake, increased physical activity, and

126 te changes in blood pressure with changes in dietary sodium intake, is an independent risk factor for

127 at investigated the BP effects of changes in dietary sodium intake, we conducted a dose-response meta

128 regulates sodium levels during increases in dietary sodium intake.

129 e sodium excretions are reliable markers for dietary sodium intake.

130 uals should routinely decrease (or increase) dietary sodium intake.

131 the home accounted for approximately 70% of dietary sodium intake.

132 egulated, preventing its suppression by high dietary sodium intake.

133 measures, perceived thirst or xerostomia, or dietary sodium intake.

134 iet quality, fruit and vegetable intake, and dietary sodium intake.

135 o be the second largest contributor to total dietary sodium intake.

136 ic signaling system responding to changes in dietary sodium intake.

137 High (dietary) sodium intake has convincingly been proven to i

138 rces of sodium to achieve a 30% reduction in dietary sodium intakes were estimated.

139 However, whether dietary sodium interacts with sex or genetics remains un

140 affected individuals might be sensitive to a dietary sodium intervention.

141 alt) Study, blood pressure (BP) responses to dietary sodium interventions are explored.

142 Excess dietary sodium is a leading cause of death and disabilit

143 timulation of Kir4.1/Kir5.1 by low intake of dietary sodium is essential for NCC upregulation, and in

144 Habitual dietary sodium is inversely associated with CFR independ

145 Dietary sodium is positively associated with blood press

146 Dietary sodium is positively associated with fluid consu

147 alterations have been reported in rats whose dietary sodium is restricted during pre- and postnatal d

148 of this study was to determine if short-term dietary sodium loading impairs cutaneous microvascular f

149 These data demonstrate that dietary sodium loading impairs cutaneous microvascular f

150 During long-term dietary sodium loading, metoprolol CHF rats retained 28+

151 Similarly, reduction in dietary sodium may be beneficial for children who are sa

152 We hypothesized that dietary sodium may modulate the effect of systolic blood

153 Dietary sodium (mg/d) and potassium (mg/d) intake were a

154 t of NaCl with KCl in top-ranking sources of dietary sodium modeled in this study can result in decre

155 e 3', 5'-monophosphate (cGMP) in response to dietary sodium (Na) depletion alone, or Na depletion or

156 Raising metHb levels with dietary sodium nitrite can further decrease daytime core

157 An increase in dietary sodium of 1000 mg/d was associated with a 10.0%

158 Our results demonstrate that the effects of dietary sodium on autoimmune neuroinflammation are sex s

159 ese observations indicate that the effect of dietary sodium on blood pressure is modulated by other c

160 The effect of dietary sodium on calcium retention and the influence of

161 The effect of changes in dietary sodium on calcium retention was tested in a rand

162 l novel mechanism for the adverse effects of dietary sodium on the cardiovascular system.

163 which has important policy implications for dietary sodium policy implementation.

164 nce models were used to assess the effect of dietary sodium, potassium, and the potassium to sodium r

165 f numerous studies, the associations between dietary sodium, potassium, and the ratio of dietary sodi

166 We assessed the relationship between dietary sodium, potassium, and the sodium-to-potassium r

167 sed on Dietary Guidelines, the corresponding dietary sodium-potassium ratio was either 0.49 (2300/470

168 Dietary sodium recommendations are debated partly due to

169 n the data support a single public policy on dietary sodium recommendations?

170 yle modification, consisting of weight loss, dietary sodium reduction and potassium supplementation,

171 This research underscores the importance of dietary sodium reduction in maintaining lower blood pres

172 Dietary sodium reduction significantly lowered BP in the

173 /hydrochlorothiazide (5 mg/50 mg daily) with dietary sodium restriction (60 mmol per day).

174 is study sought to determine the efficacy of dietary sodium restriction (DSR) for improving vascular

175 erobic exercise (daily walking) and moderate dietary sodium restriction (sodium intake <100 mmol/day)

176 18; 62 +/- 9 years, mean +/- SD) or moderate dietary sodium restriction (SR) (n = 17; 65 +/- 10 years

177 Dietary sodium restriction also increased jejunal inters

178 PGE(2) and 6-keto-PGF(1alpha) in response to dietary sodium restriction and Ang II infusion.

179 x, or volume overload should be managed with dietary sodium restriction and diuretics at the lowest e

180 These findings suggest that dietary sodium restriction and/or targeting MD signaling

181 Dietary sodium restriction combined with unilateral chor

182 Dietary sodium restriction during pre- and postnatal dev

183 Early dietary sodium restriction has profound influences on th

184 Dietary sodium restriction has several clinical benefits

185 In HFPEF animal models, dietary sodium restriction improves ventricular and vasc

186 Dietary sodium restriction in animals enhances the chron

187 Distal diuretics are noninferior to dietary sodium restriction in reducing BP and extracellu

188 whether distal diuretics are noninferior to dietary sodium restriction in reducing BP in patients wi

189 By comparison, dietary sodium restriction instituted during pre- and po

190 and identification of communities for which dietary sodium restriction is most likely to be benefici

191 Dietary sodium restriction may be associated with improv

192 Previous work demonstrated that dietary sodium restriction may induce these early functi

193 D receptor activator paricalcitol (PARI) and dietary sodium restriction on residual albuminuria in CK

194 Dietary sodium restriction or acute aldosterone infusion

195 In WT mice, dietary sodium restriction or ANG II infusion increased

196 e during basal conditions and in response to dietary sodium restriction or infusion of Ang II.

197 ni nerve section or sham section followed by dietary sodium restriction or maintenance on control die

198 However, severe dietary sodium restriction promotes insulin resistance (

199 Sodium intake was assessed using the Dietary Sodium Restriction Questionnaire (DSRQ) and spot

200 Dietary sodium restriction reduced sodium excretion from

201 In conclusion, moderate dietary sodium restriction substantially reduced residua

202 Thus, dietary sodium restriction throughout pre- and postnatal

203 xpansion (VE)] or chronic stressful stimuli (dietary sodium restriction vs. supplementation) in consc

204 L/min/1.73 m2 and are younger than 50 years, dietary sodium restriction, weight management, and adequ

205 Dietary sodium restriction, which activates RAAS, unique

206 he solitary tract (NTS) as a result of early dietary sodium restriction.

207 hat these factors may become evident through dietary sodium restriction.

208 significant downregulation was observed with dietary sodium restriction.

209 Cl with KCl in the 18 top-ranking sources of dietary sodium results in a predicted sodium intake of 2

210 The 2010 Dietary Guidelines emphasized that dietary sodium should be limited to 2300 mg/d, with a lo

211 use of potassium-based NaCl replacers in top dietary sodium sources.

212 appetite and the blood pressure response to dietary sodium through a mineralocorticoid receptor-depe

213 n and related diseases by reducing excessive dietary sodium through national dietary policymaking.

214 The relationship of dietary sodium to blood pressure was modified by age, ra

215 es and the epidemiological research relating dietary sodium to BP and cardiovascular health outcomes,

216 Evidence links dietary sodium to hypertension and cardiovascular diseas

217 Dietary potassium and the molar ratio of dietary sodium to potassium were not associated with cha

218 dietary sodium, potassium, and the ratio of dietary sodium to potassium with blood pressure are not

219 within food groups that contribute the most dietary sodium to the US diet.

220 We examined the association between dietary sodium, total fluid, and SSB consumption in a na

221 The highest quintile of dietary sodium was associated with an increased OR for a

222 to study changes of renal AT(1) receptors by dietary sodium was developed that uses positron emission

223 Across quintiles of sodium consumption, dietary sodium was inversely associated with CFR (P-tren

224 creates a deficit in the ability to excrete dietary sodium, which promotes an increase in plasma vol

225 was used to determine the effect of race and dietary sodium with calcium intake as a covariate.

226 nces ADRB2 protein expression independent of dietary sodium, yet the haemodynamic consequences appear