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1 tions (albendazole plus either ivermectin or diethylcarbamazine).
2  10 patients treated for onchocerciasis with diethylcarbamazine.
3 s also a site of action of the anthelmintic, diethylcarbamazine.
4 ted that Brugia malayi TRP-2 is activated by diethylcarbamazine.
5 le oral dose of IDA (ivermectin, 200 mug/kg; diethylcarbamazine, 6 mg/kg; plus albendazole, a fixed d
6              Recent studies, have found that diethylcarbamazine acts on the muscle of adult female Br
7 s with a low (5%) baseline mf prevalence and diethylcarbamazine + albendazole (DA) or ivermectin + di
8 rbamazine + albendazole (DA) or ivermectin + diethylcarbamazine + albendazole (IDA) treatment, elimin
9 ears of mass drug administration (MDA) using diethylcarbamazine and albendazole (DA).
10 dose, triple-drug treatment (ivermectin with diethylcarbamazine and albendazole [IDA]) is superior to
11                     All groups also received diethylcarbamazine and albendazole for lymphatic filaria
12 alence than anticipated, and the addition of diethylcarbamazine and albendazole to scabies treatment.
13 er antifilariasis drugs, namely doxycycline, diethylcarbamazine and albendazole, intradermally.
14 nellosis experienced treatment failure (with diethylcarbamazine and levamisole-mebendazole, respectiv
15 hat the triple-drug therapy with ivermectin, diethylcarbamazine, and albendazole (IDA) is much more e
16 e dose of a triple-drug regimen (ivermectin, diethylcarbamazine, and albendazole) has been shown to c
17 nologic perturbations nor on the efficacy of diethylcarbamazine as a curative agent in loiasis.
18                                              Diethylcarbamazine citrate (DEC) treatment of loiasis is
19 is, topical application of the anthelminthic diethylcarbamazine (DEC) induces clinical and histologic
20                                              Diethylcarbamazine (DEC) is used to treat lymphatic fila
21             Treatment of onchocerciasis with diethylcarbamazine (DEC) or ivermectin is associated wit
22 became undetectable within 1 month following diethylcarbamazine (DEC) treatment and remained negative
23 riple-drug therapy of the antifilarial drugs diethylcarbamazine (DEC), ivermectin (IVM), and albendaz
24 s with Loa loa infection treated with either diethylcarbamazine (DEC), the drug of choice for loiasis
25                                              Diethylcarbamazine (DEC)-medicated salt may overcome the
26 before and after administration of 300 mg of diethylcarbamazine (DEC).
27 ian patients before and after treatment with diethylcarbamazine for Brugia malayi infection, and reco
28  the newly approved ivermectin, albendazole, diethylcarbamazine (IDA) regime for Lymphatic Filariasis
29                                              Diethylcarbamazine is a classic anthelmintic that is use
30                        The mode of action of diethylcarbamazine is still not well understood with the
31            Drugs to treat filariasis include diethylcarbamazine, ivermectin, and albendazole, which a
32  lipoxygenase inhibition was performed using diethylcarbamazine or the 5-lipoxygenase activating-prot
33 IDA]) is superior to a two-drug combination (diethylcarbamazine plus albendazole [DA]) that is widely
34 ngle co-administered dose of ivermectin plus diethylcarbamazine plus albendazole [IDA] is noninferior
35 f a new triple drug regimen (ivermectin plus diethylcarbamazine plus albendazole, IDA) is non-inferio
36                         Our study shows that diethylcarbamazine's action is dependent upon the Brugia
37                             Albendazole plus diethylcarbamazine significantly reduced prevalence of e
38 s and declined (but did not disappear) after diethylcarbamazine therapy in infected patients.
39                               Application of diethylcarbamazine to Bma-trp-2b transfected HEK293 cell
40  was positive for Wolbachia DNA 4-48 h after diethylcarbamazine treatment.
41 ds nor apheresis reduced the efficacy of the diethylcarbamazine used to treat these subjects.