ライフサイエンスコーパス: differentiation marker

1 muscle cells and serves as a skeletal muscle differentiation marker.

2 duced germ cell proliferation, and shifted a differentiation marker.

3 roblast platelet-derived growth factor alpha differentiation marker.

4 to a stronger increase in level of epidermal differentiation markers.

5 ision (cytokinesis), associated with loss of differentiation markers.

6 olecular profile lacking most progenitor and differentiation markers.

7 orally affects calcium-induced expression of differentiation markers.

8 y, and increase the expression of hepatocyte differentiation markers.

9 t-like structures expressing region-specific differentiation markers.

10 f pluripotency and an enhanced expression of differentiation markers.

11 igration, and a decline in the expression of differentiation markers.

12 eroxide levels and reduced the expression of differentiation markers.

13 did not correlate with the expression of the differentiation markers.

14 or progenitor markers and low expression of differentiation markers.

15 a-catenin signaling express higher levels of differentiation markers.

16 correlated with delayed expression of early differentiation markers.

17 s, and increased the expression of epidermal differentiation markers.

18 escued the decreased mRNA levels of terminal differentiation markers.

19 coexpression of PLS3 with a panel of T-cell differentiation markers.

20 s EBV-infected B cells expressed plasma cell differentiation markers.

21 apoptosis, and the expression of Paneth cell differentiation markers.

22 ibroblasts was able to restore expression of differentiation markers.

23 , thus revealing altered expression of these differentiation markers.

24 aspase-3 activity and expression of terminal differentiation markers.

25 ce and upregulated the expression of myeloid differentiation markers.

26 ancers that can present with tissue-specific differentiation markers.

27 Ca(2+)(o)-stimulated expression of terminal differentiation markers.

28 ficantly restored expression of Runx2 and OB differentiation markers.

29 ohistochemistry with specific antibodies for differentiation markers.

30 throkeratoderma, with expansion of epidermal differentiation markers.

31 imbal stem cell (LSC) and corneal epithelial differentiation markers.

32 were different and distinct from changes in differentiation markers.

33 scade controlling the expression of terminal differentiation markers.

34 lthough it could not completely suppress all differentiation markers.

35 iber region, lens fibers express appropriate differentiation markers.

36 and expression of early and late myocardial differentiation markers.

37 s immature neural crest cell markers but not differentiation markers.

38 IP3, and Cai; and induction of keratinocyte differentiation markers.

39 n persist in mouse brain and retain neuronal differentiation markers.

40 d a reciprocal gain in some lineage-specific differentiation markers.

41 tion, and in concomitant induction of neural differentiation markers.

42 eratrol, enhanced expression of keratinocyte differentiation markers.

43 ith changes in hyperplasia, infiltrates, and differentiation markers.

44 ndrocyte maturation and expression of myelin differentiation markers.

45 etraenoic acid attenuated expression of late differentiation markers.

46 ells and ectopic expression of squamous-like differentiation markers.

47 ation of BAF155 leads to the upregulation of differentiation markers.

48 , correlating closely with expression of VSM differentiation markers.

49 rker expression but suppressed certain glial differentiation markers.

50 dermal maturation with reduced expression of differentiation markers.

51 killer cell Ig-like receptors or other late-differentiation markers.

52 and proliferation genes, and an increase in differentiation markers.

53 by premature ectopic expression of neuronal differentiation markers.

54 gulates chromatin and expression of neuronal differentiation markers.

55 cellular markers and through alterations in differentiation markers.

56 e induced to form tubules expressing nephron differentiation markers.

57 C small interfering RNA inhibited osteoblast differentiation marker alkaline phosphatase activity, wh

58 o increased the levels of smooth muscle cell differentiation markers alpha-smooth muscle actin and ca

59 onstrate that butyrate induction of the cell differentiation marker ALPi is mediated through KLF5 and

60 of E-cadherin and the dental epithelial cell differentiation marker amelogenin.

61 d highlights a concomitant increase of beige differentiation marker and a decrease in extracellular m

62 In addition, mRNA levels of the neuroD1 differentiation marker and BDNF, a neurotrophin required

63 o correlate with molecular changes (upstream differentiation marker and downstream effector cell mark

64 hibited the expression of corneal epithelial differentiation marker and promoted holoclone by LEPC.

65 Cytokine, differentiation marker and transcription factor mRNA exp

66 or cell markers with increased expression of differentiation markers and cell cycle exit.

67 id cells decreased the expression of thyroid differentiation markers and cell death and increased cel

68 ively correlate with expression of epidermal differentiation markers and components of the Notch1 pat

69 icient keratinocytes do not express terminal differentiation markers and continue to proliferate even

70 ion, as indicated by decreased expression of differentiation markers and decreased translocation of E

71 )T-FH) can increase the expression of acinar differentiation markers and elevate saliva secretion.

72 docytes increased the expression of podocyte differentiation markers and enhanced cell motility; howe

73 fferentiation by promoting the expression of differentiation markers and enhanced colonic barrier fun

74 Here we show that podocytes rapidly lose differentiation markers and enter the cell cycle in adul

75 the expression of mRNAs encoding chondrocyte differentiation markers and growth factors.

76 xpressed lower levels of putative keratocyte differentiation markers and higher levels of putative li

77 This subpopulation lacks differentiation markers and HLA class I (HLAI) antigens,

78 ding rapid cell-cycle exit, re-expression of differentiation markers and improved filtration barrier

79 This was associated with a rapid loss of differentiation markers and increased expression of CSC

80 s the expression of early and late epidermal differentiation markers and increases the proliferative

81 The expression of terminal differentiation markers and key enzymes mediating epider

82 Pak2 display delayed expression of myogenic differentiation markers and myotube formation.

83 tment with glucocorticoids restores podocyte differentiation markers and normal ultrastructure and im

84 delayed, along with reduced expression of HF differentiation markers and of transcriptional regulator

85 , associated with down-regulation of thyroid differentiation markers and ongoing apoptosis.

86 al transition (MET), increased expression of differentiation markers and presence of partially reprog

87 ritical role in regulating expression of SMC differentiation markers and proliferation of SMCs in viv

88 ted in the reexpression of breast epithelial differentiation markers and repression of EMT transcript

89 f L(1p)M-FH and L(1p)T-FH to increase acinar differentiation markers and restore saliva flow rate in

90 retinogenesis in temporal expression of cell differentiation markers and retinal disease genes, as we

91 ryos, but shortly after birth tenocytes lost differentiation markers and reverted to a more stem/prog

92 H inhibitor brequinar had similar effects on differentiation markers and S-phase arrest, and genetic

93 cantly improved the expression of the acinar differentiation markers and saliva secretion when compar

94 All EFNAs induce epidermal differentiation markers and suppress cell adhesion genes

95 Expression levels of the chondrogenic differentiation markers and transcriptional regulators S

96 phology, hyperplasia, aberrant expression of differentiation markers and transcriptional regulators,

97 general downregulation of luminal/epithelial differentiation markers and upregulation of basal/mesenc

98 Osteoblast differentiation markers and Wnt target gene expression w

99 tro differentiation protocols, expression of differentiation markers, and assessment of the ability o

100 ll densities, cell proliferation, osteoblast differentiation markers, and capillaries in human iliac

101 inflammation and epidermal proliferation and differentiation markers, and it has been unclear whether

102 s RA-induced RARalpha binding, activation of differentiation markers, and the repression of pluripote

103 vealed by the premature expression of muscle differentiation markers, and, especially, by a reduced e

104 transitory state where progenitor and early differentiation markers are co-expressed.

105 ed relative to the wild type, and osteoclast differentiation markers are expressed at earlier time po

106 is dependent on culture conditions, and many differentiation markers are usually absent.

107 th a reduction in the expression of podocyte differentiation markers as compared with the wild-type t

108 sociated with podocyte apoptosis and loss of differentiation markers as well as a faster decline in a

109 nment, and expression of smooth muscle (SMC) differentiation markers, as those have been associated w

110 was able to increase the gene expression of differentiation markers, as well as the activity of MMP-

111 ociation with induction of MYCN, EZH2 and NE differentiation markers (ASCL1, AURKA and SYP) linked to

112 induced differentiation as indicated by cell differentiation markers associated with early (CD38 and

113 e proliferative ventricular zone is lost and differentiation markers become expressed throughout the

114 etween microRNA expression and messenger RNA differentiation markers BMP-4, CK8 and CK14 were analyze

115 P2ry12-/- OCs exhibited intact differentiation markers, but diminished resorptive funct

116 pithelial cells show increased expression of differentiation markers, but loss of progenitor cell mar

117 hermore, ERK inhibition and the induction of differentiation markers by DSG1 required both Erbin and

118 IDO1-TG and analyzed them for stem cell and differentiation markers by real-time polymerase chain re

119 of mutant mice, there was a reduction of the differentiation marker, carbonic anhydrase-1, and failur

120 n) and inhibited the expression of adipocyte differentiation markers (CCAAT/enhancer-binding protein

121 both CD-45/KRT markers (and for the monocyte differentiation marker CD-14).

122 e been shown to overexpress the vasculogenic differentiation markers CD144 (VE-cadherin) and TIE1 and

123 1 and HOXC11 ChIPseq analysis identified the differentiation marker, CD24, and the apoptotic protein,

124 molecules CCR7 and L-selectin as well as the differentiation marker CD27, a phenotype consistent with

125 proportions of CD56dim cells expressing the differentiation marker CD57 and expansion of the NKG2C+

126 8 cells less commonly expressed the terminal differentiation marker CD57, a finding consistent with a

127 CD8(+) T cells did not express the terminal differentiation marker CD57, and fewer HDV-specific than

128 ed by decreased expression of megakaryocytic differentiation marker CD61 and cell cycle behavior.

129 led to stratify and did not express terminal differentiation markers characteristic of basal, interme

130 ulate proinflammatory genes or down-regulate differentiation markers characteristic of RAS-expressing

131 DAC4 reverses miR-1 induction of chondrocyte differentiation markers Col X and Ihh.

132 inhibited the premature expression of muscle differentiation markers, corrected the cytoskeletal abno

133 The expression of Klf5, odontoblast-differentiation markers, Dspp and Dmp1 was co-localized

134 related transcription factor 2, osterix) and differentiation markers (eg, osteopontin, osteocalcin, a

135 In contrast, a homolog of the neural differentiation marker elav, CapI-elav1, is restricted t

136 mation in NC is marked by loss of follicular differentiation markers, expansion of keratin-15-positiv

137 cells, nor does it affect the expression of differentiation markers expressed in lens fibers, althou

138 AT2R knockdown by siRNA suppressed myoblast differentiation marker expression and myoblast different

139 ratinocytes, overexpressed YY1 also inhibits differentiation marker expression induced by calcium, su

140 icantly inhibited AQP3 re-expression-induced differentiation marker expression with calcium elevation

141 Hypoxia-induced changes in stem cell and differentiation marker expression, clone-forming potenti

142 ne deacetylase inhibitor, partially restored differentiation marker expression, suggesting a potentia

143 roughput screen of neutrophil CD (cluster of differentiation) marker expression and a thorough litera

144 T-PEMs have a lower expression of macrophage differentiation markers F4/80, CD68, CD115, and CD11b, w

145 supported by a lower mRNA expression of the differentiation markers; fatty acid binding protein 4, p

146 latum also induced expression of key barrier differentiation markers (filaggrin and loricrin), increa

147 e identification of cell surface proteins as differentiation markers, flow cytometry requires suitabl

148 on of Jak3 resulted in reduced expression of differentiation markers for the cells of both enterocyti

149 tatin (SST), insulin (INS), Glucagon (GCG)], differentiation markers [Forkhead box O1 (FOXO1), Paired

150 3, FoxM1b also represses the mammary luminal differentiation marker FoxA1 by promoter-methylation, an

151 e which encoded the expression of functional differentiation markers from the ATRA-inducible transcri

152 MAPK/ERK and Akt signaling, suppresses VSMC differentiation marker gene expression.

153 human adipocytes, without altering adipocyte differentiation marker gene expression.

154 There was increased expression of osteoblast differentiation marker genes and reduced expression of g

155 Pitx2 induces expression of multiple SMC differentiation marker genes by binding to a TAATC(C/T)

156 , another homeodomain protein, regulates SMC differentiation marker genes in fully differentiated SMC

157 ppression of Pitx2 reduces expression of SMC differentiation marker genes in the early stages of SMC

158 VSMC differentiation marker genes such as SM alpha-actin, cal

159 tion was blocked and down-regulation of VSMC differentiation marker genes was enhanced.

160 The VSMC differentiation marker genes, including alphaSMA, SM22,

161 In DGCR8 cKO embryos and knockout VSMCs, differentiation marker genes, including alphaSMA, SM22,

162 detected by reduced expression of osteoblast differentiation marker genes.

163 ching of VSMCs, including suppression of SMC differentiation marker genes.

164 etylation levels within the promoters of SMC differentiation marker genes.

165 cyte marker gene PPP1R14a and other neuronal differentiation marker genes.

166 However, expression of differentiation markers human chorionic gonadotrophin an

167 xpression for p27(kip1), Atoh1 and hair cell differentiation markers implicating notch signaling in t

168 ntly permits detection of only up to a dozen differentiation markers in a single measurement.

169 nonmalignant pulmonary tissues (n = 285) as differentiation markers in an analysis of DNA methylatio

170 se expression correlates highly with that of differentiation markers in both the bladder and skin, in

171 expression of KLF15 stimulated expression of differentiation markers in both wild-type and HIV-1-infe

172 elayed down-regulation of smooth muscle cell differentiation markers in carotid arteries following in

173 ion negatively correlates with expression of differentiation markers in clinical myeloid leukemia sam

174 alyses confirmed the decreased expression of differentiation markers in D2J osteoblasts.

175 n factor, is required for restoring podocyte differentiation markers in mice and human podocytes unde

176 pression of MAF promoted expression of glial differentiation markers in MPNST cells in vitro, decreas

177 D117, and CD34) and myeloid (CD115 and CD14) differentiation markers in parallel with increased phago

178 ssion, DNA synthesis, expression of neuronal differentiation markers in PC12 cells, and Ras-induced f

179 elates with impaired induction of osteoclast differentiation markers in response to RANKL stimulation

180 ecule RNA FISH to measure mRNA expression of differentiation markers in single cells reveals that sis

181 ong with reduced expression of smooth muscle differentiation markers in the carotids.

182 uces the expression of keratinocyte terminal differentiation markers in the duct luminal cells, which

183 The expression of differentiation markers in these organotypic cultures we

184 temness markers and activation of early cell differentiation markers in treated embryonic stem cells.

185 rphology or expression of smooth muscle cell differentiation markers in vessels of SM22alpha-CreKI(+)

186 els correlate with the expression of various differentiation markers in vitro in response to differen

187 -/-) mice are delayed in their expression of differentiation markers in vitro.

188 )M-FH produce only weak expression of acinar differentiation markers in vivo (e.g., aquaporin-5 and t

189 on lipid accumulation and the expression of differentiation markers, in vitro adipogenesis increased

190 es exhibit increased expression of foam cell differentiation markers including 15-lipoxygenase and le

191 reverses miR-365 stimulation of chondrocyte differentiation markers including Ihh, Col X, and Runx2.

192 erentiation as indicated by reduced terminal differentiation markers, including alkaline phosphatase,

193 gnificantly higher mRNA levels of osteoblast differentiation markers, including COL1A1, ALP, and OC,

194 senger ribonucleic acid levels of osteogenic differentiation markers, including collagen 1alpha1, alk

195 ed proliferation and increased expression of differentiation markers, including ERVW-1.

196 iated with a profound repression of terminal differentiation markers, including filaggrin, an essenti

197 architecture, and downregulation of several differentiation markers, including filaggrin.

198 Using a panel of different terminal differentiation markers, including neurotransmitter synt

199 This study demonstrated important differentiation markers, including pigmentation and West

200 Multiple tissue-specific differentiation markers, including the tightly regulated

201 nflammatory phenotype including a decline in differentiation markers, increased cytokine production,

202 ZBTB16 induced the expression of osteogenic differentiation markers independently of RUNX2.

203 nduced expression and enzyme activity of the differentiation marker intestinal alkaline phosphatase (

204 contact exhibited enhanced expression of the differentiation markers involucrin and keratin 10 compar

205 dify the expression profile of the epidermal differentiation markers involucrin, keratin 10, and fila

206 phic, and has altered expression of terminal differentiation markers involucrin, loricrin, and filagg

207 Expression of neuronal differentiation markers is ablated in both KIF1Bbeta-def

208 and are rescued by DKK1, whereas the loss of differentiation markers is CaMKII dependent.

209 The expression of T cell differentiation markers is known to increase during Myco

210 sphoprotein (DSPP), an important odontoblast differentiation marker, is necessary for tooth developme

211 tory factors NF-kappaB, TSLP, TNF-alpha, and differentiation marker K10 by 94%-98% (P < 0.001) in hum

212 evel of N-cadherin, but a lower level of the differentiation marker K12.

213 USP6 and decreased expression of the corneal differentiation marker K12.

214 of the basal layer marker keratin 14 and the differentiation marker keratin 1 was evident in Aurora-A

215 al cell markers keratin 5 and 14 but not the differentiation marker keratin 4.

216 We examined the requirement of the T cell differentiation marker killer cell lectin-like receptor

217 cardiac progenitor cell markers and reduced differentiation marker levels.

218 hron formation, induces tubule formation and differentiation markers Lim1 and E-cadherin in MM cells,

219 tubule-derived cystic segments that lost the differentiation marker lotus tetragonolobus lectin.

220 also lost the expression of several terminal differentiation markers (Lyve1, Stab1, Stab2, Ehd3, Cd20

221 NPs decreases the expression of the neuronal differentiation markers MAP2 and NeuN and downregulates

222 Expression and localization of the acinar differentiation marker MIST1 were altered in Irf6-null s

223 A/Io increased expression of the goblet cell differentiation marker MUC2; these changes were attenuat

224 increased the expression of the goblet cell differentiation marker mucin 2 (MUC2).

225 ly members MAL, MAL2, and myeloid-associated differentiation marker (MYADM) regulate the function and

226 and decreased expression of SC proliferation/differentiation markers (MyoD, myogenin, and active-Notc

227 ing myofibers and expression of the myoblast differentiation markers myogenin and embryonic myosin he

228 nterference reduced the expression levels of differentiation markers (myogenin, myosin heavy chain, t

229 in maintaining the expression level of a CE differentiation marker, N-cadherin, and the hexagonal ce

230 Hes5, with a reduction of the spermatogonial differentiation marker, Neurog3 expression in the mutant

231 olecular markers including the early cardiac differentiation marker Nkx2.5.

232 Dentin sialophosphoprotein (Dspp) as a differentiation marker of odontoblasts is regulated by B

233 luding the expression of prestin, a terminal differentiation marker of outer hair cells, although man

234 ent does not affect expression of macrophage differentiation markers or macrophage biological functio

235 Progressive loss of the myoepithelial cell differentiation markers p63, calponin, and alpha-smooth

236 found that expression of the late adipocyte differentiation marker peroxisome proliferator-activated

237 sociated with HCs, including the terminal HC differentiation marker prestin.

238 The expression of well-known differentiation markers profilaggrin, loricrin, and kera

239 Our study included analysis of germ cell differentiation markers, proliferation markers, and cell

240 populations marked by varying levels of the differentiation marker prostate-specific antigen (PSA).

241 egregated with the template DNA, whereas the differentiation markers prosurfactant protein-C and pan-

242 tress fibers and expression of myofibroblast differentiation marker proteins.

243 It was shown that one of the most powerful differentiation markers proved to be Mn content.

244 h (PSA(+)) and low (PSA(-/lo)) levels of the differentiation marker PSA.

245 ted at 7 T in six C57BL/6 mice and in immune differentiation marker (recombination activation gene [R

246 However, while the apoptosis and differentiation markers remained unchanged at this age,

247 Expression of endocrine differentiation markers required sustained NEUROG3 expre

248 and immunohistochemistry of sebaceous gland differentiation markers revealed reduced peroxisome prol

249 he Gata4-expressing cells eventually express differentiation markers showing commitment to liver deve

250 3 (Slfn3) correlated with the levels of the differentiation markers SI, Dpp4, Glut2, and villin.

251 M population did not upregulate the terminal differentiation marker sialic acid-binding immunoglobuli

252 this specifically prevents expression of the differentiation marker smooth muscle alpha-actin.

253 essed genes in KCOT were squamous epithelial differentiation markers SPRR1A, KRTDAP, and KRT4, as wel

254 However, terminal differentiation markers such as beta- or gamma-crystalli

255 s stem cell markers such as Prom1 as well as differentiation markers such as Ck19 and Hnf4a.

256 DMEM, induced dark pigmentation and promoted differentiation markers such as CRALBP and MerTK.

257 (N3) transcription, inducing HES5 and early differentiation markers such as involucrin (IVL) and cyt

258 ced mRNA and protein expression of epidermal differentiation markers such as keratin 1, keratin 10, a

259 ndent manner that is similar to induction of differentiation markers such as keratin 10 and involucri

260 CD271 expression concomitantly with loss of differentiation markers such as melan-A and tyrosinase,

261 The gene expression for odontogenic differentiation markers such as osteocalcin, bone sialop

262 Foxi3 cKO there was an early upregulation of differentiation markers, such as p21, Fgf15 and Sfrp5.

263 ermal architecture with proper expression of differentiation markers, suggesting that although kerati

264 nin correlated with crypt progenitor but not differentiation markers, suggesting that the Wnt/beta-ca

265 s, with an increase in apoptosis and thyroid differentiation markers, suggesting that these cell line

266 During EMT, the expression of differentiation markers switches from cell-cell junction

267 podocytes, ET-1 caused loss of the podocyte differentiation marker synaptopodin and acquisition of t

268 Delta/-) ESCs re-initiates the expression of differentiation markers T and Gata-6.

269 tive Klf9 target genes include proliferation/differentiation markers that also show circadian express

270 on of panels of N-glycoproteins as potential differentiation markers that are currently not accessibl

271 d by the absence of major corneal epithelial differentiation markers, that is, K3 and K12 keratins, i

272 directly modulate expression of Paneth cell differentiation markers through its effects on TCF4/beta

273 spatial distribution of GJIC and osteogenic differentiation markers throughout 3D constructs.

274 d thyrocytes increased expression of thyroid differentiation markers, thyroglobulin, thyroid-stimulat

275 Analysis of stem cell and differentiation marker transcripts, as well as Oct 3/4 p

276 A induced the expression of lineage-specific differentiation markers Tuj1 and GFAP and reduced the ex

277 messenger RNA expression of PDL osteoblastic differentiation markers: type I collagen, alkaline phosp

278 f cell cycle control molecules and beta-cell differentiation markers upon diabetogenic challenges, an

279 ng the injury downregulate the expression of differentiation markers, upregulate markers of the pre-o

280 ositive cells had lost expression of the end-differentiation marker, urocortin-3, or appeared to co-e

281 n to assay the expression of the enterocytic differentiation markers villin, sucrase-isomaltase, gluc

282 The expression of epidermal keratinocyte differentiation markers was affected in the Klf4CN corne

283 Transcription of dental differentiation markers was as follows: Dentin matrix pr

284 The expression of differentiation markers was evaluated by quantitative PC

285 ll differentiation by calcium, expression of differentiation markers was impaired in both DeltaNp63al

286 The expression of differentiation markers was quantified by enzyme-linked

287 A panel of putative stem cell and differentiation markers was used to analyze the epitheli

288 idase IV (DPP-IV), two well known intestinal differentiation markers, was examined.

289 Among those differentiation markers, we show that Sprr2a transcripti

290 stasis, chondrocyte maturation, and terminal differentiation markers were all up-regulated versus iso

291 Gene expression of leptin and selected differentiation markers were analyzed during preadipocyt

292 erentiation and the expression of osteogenic differentiation markers were assessed by real-time rever

293 nd Paneth cell lysozyme, whereas enterocytic differentiation markers were reduced.

294 KLF4 transcriptional targets, including late differentiation markers, were reduced in HPV E6 and E7 r

295 on concurrent with decreased expression of a differentiation marker when compared with untreated cell

296 cient precursors normally express osteoclast differentiation markers when exposed to M-CSF and recept

297 inamide inhibited expression of keratinocyte differentiation markers, whereas a SIRT1 activator, resv

298 ir expression of mRNA for several osteoclast differentiation markers, whereas ES significantly reduce

299 roxide levels and promoted the expression of differentiation markers, whereas overexpression decrease

300 6 rescued the reduced expression of terminal differentiation markers, whereas transfection of AnxA6-/