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1  as well as for the lateral segregation of a diglyceride.
2 lly active lipids phosphatidic acid (PA) and diglyceride.
3 wn products inositol 1,4,5-trisphosphate and diglyceride.
4 effect on phosphatidic acid, but not that on diglyceride.
5 his lethal process is potently suppressed by diglyceride.
6 des and in 2-3-fold accumulation of label in diglycerides.
7 al correlate for IL-1-generated ether-linked diglycerides.
8     Here, we report that the 1-O-alkyl ether diglyceride, 1-O-hexadecyl-2-acetyl-sn-glycerol (HAG), l
9        The main hydrolysis products were 1,2-diglycerides, 2-monoglycerides, glycerol and fatty acids
10                      Nephrocyte knockdown of diglyceride acyltransferase 1 (DGAT1), overexpression of
11 Decreasing TG synthesis by deleting neuronal diglyceride acyltransferases (DGATs) and enhancing PL sy
12 lase, and three out of five putative type II diglyceride acyltransferases (DGATs), the enzymes that c
13                           Two ether glucosyl diglyceride analogs were synthesized, and their antiprol
14 ine (ara-C) stimulates the formation of both diglyceride and ceramide in the acute myelogenous leukem
15                Therefore, the utilization of diglyceride and modified PPI can serve as structural enh
16 g change being observed in the modulation of diglyceride and monoglyceride levels in BAT.
17 , with lesser amounts of cholesteryl esters, diglyceride and other phospholipids.
18 h complete DB position characterization of 9 diglycerides and 24 triglycerides.
19 of natural ceramides with those of saturated diglycerides and an unsaturated ceramide demonstrates th
20 e fatty acid (FA) profile of monoglycerides, diglycerides and cholesteryl esters from retroperitoneal
21 R and attributed to partial incorporation of diglycerides and free fatty acids into gum bilayers afte
22 ers, dimers, oxidized triglyceride monomers, diglycerides and free fatty acids, and induction period
23 tty acids, although small proportions of 1,3-diglycerides and of 1-monoglycerides were also found.
24                Here we show higher levels of diglycerides and triglycerides are adversely associated
25  Harmonic REIMS spectra, high intensities of diglycerides and triglycerides were observed.
26 centrations of two phosphatidylcholines, two diglycerides and two acyl-carnitines were significantly
27                                          1,2-Diglycerides and vegetable stanols and/or sterols have b
28       Octanol, 1,2-dioctanoyl-sn-glycerol (a diglyceride), and N-hexanoyl-D-erythrosphingosine (a cer
29 vine cardiolipin, chloroplast monogalactosyl-diglyceride, and dioleoyl phosphatidylethanolamine as a
30 fatty acids and their esters, monoglyceride, diglyceride, and triglyceride.
31  such as phosphatidylcholine, sphingomyelin, diglycerides, and triglycerides were detected and identi
32 revealed increases in oleic acid (OA, 18:1), diglycerides, and triglycerides.
33 cates cytokine receptor-induced ether-linked diglycerides as second messengers that inhibit the bioac
34 pases are differentially activated such that diglyceride breakdown is approximately four times faster
35 gment ions corresponding to loss of the full diglyceride chain as well as the remaining headgroup bou
36 80 degrees C, which might be due to its high diglyceride content.
37      Interestingly, hepatic triglyceride and diglyceride contents were normalized to chow-fed levels
38 ificant changes in inflammation, ceramide or diglyceride contents, endoplasmic reticulum stress, or a
39 , diacetyl tartaric acid esters of mono- and diglycerides (DATEM), and/or modified DATEMs with differ
40                                              Diglyceride DG(18:1/20:0)-sodium adduct and protonated o
41 ding to TG synthesis and steatosis by way of diglyceride (DG) generation.
42  (PEt) instead of phosphatidic acid (PA) and diglyceride (DG).
43 estigation of de novo triglycerides (TG) and diglycerides (DG) during the ovarian previtellogenic (PV
44 t age-related changes in triglycerides (TG), diglycerides (DG), phosphatidylcholine, phosphatidyletha
45                    In addition, digalactosyl diglyceride (DGDG) 36:4 was shown to be an effective mar
46 t of sea urchin membrane vesicle PtdIns into diglycerides enriched in long chain, polyunsaturated spe
47  in addition to increasing triglycerides and diglycerides, especially enriched with unsaturated fatty
48 n in 1-monoglycerides and an increase in 1,2-diglycerides, especially in the margarines with higher w
49 aracterized by high levels of triglycerides, diglycerides, fatty acids, ceramides, and oxidized fatty
50 atty acids, together with triacylglycerides, diglycerides, free fatty acids and ergosterol in salmon
51 rolysis, mobilization of intracellular Ca2+, diglyceride generation, and redistribution of protein ki
52                                          CDP-diglyceride hydrolase (Cdh) catalyzes the same reaction
53                                          The diglyceride-hydrolyzing lipase was purified from the ext
54 te that production of membrane-destabilizing diglycerides in membranes enriched in PtdIns may facilit
55 ation and higher levels of triglycerides and diglycerides in the myocardium, aggravating oxidative st
56 and the rate of palmitate incorporation into diglycerides in white muscle was 93% higher in obese rat
57 ut affecting the induced increase in nuclear diglyceride, indicating that the increase in nuclear PLD
58 nterleukin-1 receptor-generated ether-linked diglycerides inhibit immunoprecipitated protein kinase C
59 ramide because, in contrast to the bacterial diglyceride kinase, ceramide is not phosphorylated under
60 with nonprogressors, with simultaneous lower diglycerides, lysophosphatidylcholines, triglycerides, a
61                  Cell-permeable ether-linked diglycerides mimic the effects of interleukin-1 to induc
62 nounsaturated FA (MUFA) in the monoglyceride diglyceride (Mono-Di) fraction.
63 characterized by increased triglycerides and diglycerides, n3 polyunsaturated fatty acid depletion, a
64 ent score [NES], 2.68), PEs (NES, 2.48), and diglycerides (NES, 1.65) were positively associated, whe
65 in pre-emulsions containing triglyceride- or diglyceride-oil on the emulsifying and gelling propertie
66 ol, treating human fibroblasts with octanol, diglyceride, or ceramide stimulated the rapid inactivati
67  complex of poly(ethylene argininylaspartate diglyceride) (PEAD) polycation, heparin, and cargo insul
68 olism, including decreased triglycerides and diglycerides, phosphatidylcholine/phosphatidylserine rat
69 phosphorylated under conditions specific for diglyceride phosphorylation.
70                                        Polar diglyceride promoted protein adsorption and fostered int
71  < 0.0001) without increased accumulation of diglycerides, resulting in significant improvement of th
72 dothelin, selectively generates ether-linked diglyceride species (alkyl, acyl- and alkenyl, acylglyce
73 hat saroglitazar also reduced triglycerides, diglycerides, sphingomyelins and ceramides.
74 itory profile of these ether-linked glucosyl diglycerides strengthens the hypothesis that such glycol
75 of sterols, fatty acids, monoglycerides, and diglycerides that are not detected in IR-MALDI MSI exper
76 ctive peptides stimulate distinct species of diglycerides that differentially regulate protein kinase
77 s an HFA with a FA from triglyceride (TG) or diglyceride to produce FAHFAs.
78                                 The block in diglyceride to triglyceride conversion could not be expl
79 ed defects in FA uptake and in conversion of diglycerides to triglycerides that are similar to those
80 sity lipoprotein cholesterol, triglycerides, diglycerides, total and monounsaturated fatty acids, fat
81  phosphatidic acids but reduced abundance of diglycerides, triglycerides, and phosphatidylglycerol li
82                                    Combining diglyceride-type PPI(U) and PPI(pH) emulsions with TG in
83 ated fatty acids beta-sitosterol, sn-1 and 3 diglyceride values.
84 r ones, such as 1- and 2-monoglycerides, 1,2-diglycerides, vegetable stanols and sterols, and sorbic
85 ol (DPG) vesicles (but not with digalactosyl diglyceride vesicles) as demonstrated by flotation in su
86  PLD2 and PAP2b act sequentially to generate diglyceride within this specialized membrane compartment