ライフサイエンスコーパス: dilatation

1 It is associated with ascending aorta dilatation.

2 r eye-11.6%, both eyes-1.2%) following pupil dilatation.

3 s, pericardial effusion, and coronary artery dilatation.

4 aracatinib no longer increased acetylcholine dilatation.

5 AJs, (iii) AJ width, and (iv) acetylcholine dilatation.

6 eatures, including prevalence of aortic root dilatation.

7 l functions, including endothelium-dependent dilatation.

8 ression was used to examine risk factors for dilatation.

9 ade resulted in an increase in flow-mediated dilatation.

10 a media thickness or endothelium-independent dilatation.

11 ones demonstrating the greatest degree of RV dilatation.

12 markedly impaired NO-dependent flow-mediated dilatation.

13 vant pressure gradient compared with balloon dilatation.

14 end-diastolic pressure, and left ventricular dilatation.

15 ifty percent of patients had ascending aorta dilatation.

16 ignificant elevation of VEGF and ventricular dilatation.

17 ion was associated with less ascending aorta dilatation.

18 scle cells in a process termed flow-mediated dilatation.

19 dilatation and 20% having right ventricular dilatation.

20 ely associated with the degree of esophageal dilatation.

21 dal changes different from a mere sinusoidal dilatation.

22 of PCV may include choroidal congestion and dilatation.

23 The primary outcome was the rate of dilatation.

24 nversion in V6, and evidence of right atrial dilatation.

25 and stricture, and the need for anastomotic dilatation.

26 ndependently associated with ascending aorta dilatation.

27 d sympathetic beta(1) -adrenoceptor-mediated dilatation.

28 primary endpoint was the rate of aortic root dilatation.

29 the subvalvular apparatus, with late annular dilatation.

30 inguishing between acute and chronic urinary dilatations.

31 improved endothelial function (flow-mediated dilatation: +1.45%; 95% CI: 0.83%, 2.1%; P = 0.003), sys

32 Both FNIII1H,8-10 and FNIII1H induced dilatations (12.2 +/- 1.7 mum, n = 12 and 17.2 +/- 2.4 m

33 binding region significantly diminished the dilatation (3.2 +/- 1.8 mum, n = 10).

34 h neither AS nor AI, 37% had ascending aorta dilatation (4% severe).

35 tio (11 vs. 7; P < 0.001), and a left atrial dilatation (40 vs. 29 mL/m(2) ; P = 0.011).

36 basal LVOTO (70-120 mm Hg), and left atrial dilatation (44-57 mm).

37 increased scar thickness, and attenuated LV dilatation 7 days after myocardial infarction.

38 Most patients with progressive RV dilatation (8/14, 57%) had moderate (2 patients) or seve

39 more likely than females to have aortic root dilatation (92% versus 84%), aortic regurgitation (55% v

40 on initial imaging (eg, main pancreatic duct dilatation, a solid component, or mural nodule) require

41 entricular free wall (DDD) pacing lead to LV dilatation, a thinned septum, and thickened lateral wall

42 ophy (adjusted odds ratio 2.1; P<0.0001), LV dilatation (adjusted odds ratio 2.2; P<0.0001), and righ

43 ratio 2.2; P<0.0001), and right ventricular dilatation (adjusted odds ratio 2.2; P<0.0001).

44 Echocardiography revealed increased LV dilatation, altered hypertrophic remodeling and exacerba

45 with 26% of patients having left ventricular dilatation and 20% having right ventricular dilatation.

46 aneous vasomotor tone, endothelium-dependent dilatation and adrenergic vasoconstriction increased at

47 accompanied by progressive left ventricular dilatation and adverse cardiac remodeling.

48 sembles Kawasaki disease (KD), with coronary dilatation and aneurysm occurring in some.

49 stigated using brachial artery flow-mediated dilatation and carotid artery intima-medial hyperplasia.

50 s to spontaneous-onset AF preceded by atrial dilatation and conduction abnormalities.

51 iffuse alveolar damage (70/70) and capillary dilatation and congestion (70/70), often accompanied by

52 cesses of diffuse alveolar damage, capillary dilatation and congestion, and microthrombosis.

53 pathologic observations, including capillary dilatation and congestion, interstitial edema, diffuse a

54 oaded heart, leading to increased mortality, dilatation and contractile dysfunction in mice.

55 Our novel findings, including T-tubule dilatation and disorganization, associated with defects

56 ve SMCs render the aortic wall vulnerable to dilatation and dissection rather than prevent disease pr

57 The most common cardiac pathology was RV dilatation and dysfunction (observed in 39% of patients)

58 d collagen quality, thereby blunting cardiac dilatation and dysfunction after MI.

59 ration of nonmyocyte cells, left ventricular dilatation and dysfunction, and slightly improved surviv

60 cross-linking are associated with cardiac LV dilatation and HF.

61 regulatory factor of brachial flow-mediated dilatation and highlight the importance of the simultane

62 cal activity is also associated with cardiac dilatation and hypertrophy in a healthy adult population

63 s and prevented progressive left ventricular dilatation and hypertrophy, whereas adoptive transfer of

64 o inhibit sensory neurotransmission) blocked dilatation and increased constriction during PNS.

65 With perivascular sensory nerve stimulation, dilatation and inhibition of sympathetic vasoconstrictio

66 en secondary in nature and caused by annular dilatation and leaflet tethering from adverse right vent

67 ew and simple composite parameter of both LV dilatation and LV forward flow able to accurately predic

68 Following treatment with sequential dilatation and maintenance H2-blocker therapy, she achie

69 Extensive changes of dilatation and obstruction in nearly all airway generati

70 Aortic dilatation and other structural cardiac abnormalities we

71 lateral ureteral obstruction-induced tubular dilatation and proliferation, preserved Klf4, and suppre

72 We found that the CSD-induced brief dilatation and prolonged constriction of pial arteries,

73 The vessels had no dilatation and rarely had post-cannulation bleeding.

74 te the association between coronary arterial dilatation and retinal microvasculature in a pilot setti

75 ntify and select relevant studies of balloon dilatation and stenting for aortic coarctation based on

76 ess and comparative effectiveness of balloon dilatation and stenting for aortic coarctation.

77 ent of the left ventricle (LV) cause annular dilatation and tethering of the mitral valve leaflets, t

78 s based on pancreatic calcifications, ductal dilatation, and atrophy visualized by imaging with compu

79 w H(2)S(n) generation, impaired flow-induced dilatation, and failure to detect beta3 integrin S-sulfh

80 (44.1-88.9%) of patients undergoing balloon dilatation, and in 99.5% (97.5-100.0%) and 93.8% (88.5-9

81 n, left ventricular hypertrophy, left atrial dilatation, and interstitial fibrosis.

82 ed to a significant reduction in LV mass, LV dilatation, and neurohormonal activation, and it preserv

83 rease in fractional shortening, hypertrophy, dilatation, and premature death.

84 those with early evidence of coronary artery dilatation, and those with extreme abnormalities in labo

85 thological end point: irreversible bronchial dilatation arrived at through diverse etiologies.

86 Heat therapy improved endothelium-dependent dilatation, arterial stiffness, intima media thickness a

87 Using pupillary dilatation as a measure of central noradrenergic signall

88 chieving </=20 mm Hg were lower with balloon dilatation as compared with stenting (odds ratio, 0.105

89 hermore, capillaries contribute to metabolic dilatation as they dilate arterioles directly upstream i

90 ricuspid regurgitation and tricuspid annular dilatation, as well as with appreciation of the high ris

91 function (via brachial artery flow-mediated dilatation) at sea level (344 m) and high altitude (3800

92 function (via brachial artery flow-mediated dilatation) at sea level (344 m) and high altitude (3800

93 metabolic endpoints [including flow-mediated dilatation, augmentation index, lipoprotein status (by n

94 icular remodeling, greater right ventricular dilatation (base, 34+/-7 versus 31+/-6 and 30+/-6 mm, P=

95 n independent determinant for early brachial dilatation (beta = -0.286, p = 0.013).

96 e FBA, there was no longer any difference in dilatation between old and young arteries.

97 t regulator of acute vessel permeability and dilatation but also provide evidence that antagonizing P

98 uced right ventricular pressure increase and dilatation, but left ventricular end-diastolic volume im

99 In addition, pore dilatation by a mutation in the pore-lining region alter

100 y between the peri-brachial fat and brachial dilatation can be translated into novel clinical tools t

101 lized to assess the influence of ET-1 on the dilatation capacity of vascular smooth muscle cells (sod

102 russide, suggesting that ET-1 diminishes the dilatation capacity of vascular smooth muscle cells.

103 tive axon density) and endothelium-dependent dilatation (carbachol) of the MCA were not different bet

104 VPA significantly ( P<0.05) reduced atrial dilatation, cardiomyocyte enlargement, atrial fibrosis,

105 t reactions of PBG were noted, including PBG dilatation, cell proliferation, and maturation.

106 er in children with RPD at the FAS and later dilatation (cHR 25.13, 95% CI 13.26-47.64, p < 0.001) an

107 hildren without RPD at the FAS who had later dilatation (cHR 62.06, 95% CI 41.10-93.71, p < 0.001) th

108 (-1) m(-2), P < 0.02), and improved arterial dilatation Deltabrachial artery flow-mediated dilatation

109 ilatation Deltabrachial artery flow-mediated dilatation/Deltadilation response to glyceryl nitrate (9

110 Anal dilatation did not improve outcome(s).

111 ess, which increases secondary to arteriolar dilatation downstream.

112 tion in SC size and a loss of physiologic SC dilatation during accommodative effort, which may reflec

113 children at risk of developing severe aortic dilatation during their pediatric follow-up is still cha

114 n 59%, smooth stenosis in 19%, and segmental dilatation/ectasia in 56%.

115 smooth stenosis, diffuse or focal; segmental dilatation/ectasia; and tortuosity.

116 Endothelium-dependent dilatation (ED) is abnormal in patients with SLE, and en

117 Endothelium-dependent dilatation (EDD), assessed by the maximal dilatation to

118 dysfunction (impaired endothelium-dependent dilatation, EDD) and aortic stiffening (increased aortic

119 Numerically more patients undergoing balloon dilatation experienced severe complications during admis

120 cified lesions in which noncompliant balloon dilatation failed (n=22 lesions), and (group C) tertiary

121 velop an age-dependent phenotype with atrial dilatation, fibrosis, and atrial fibrillation.

122 function as assessed by using flow-mediated dilatation (FMD) and arterial compliance as assessed by

123 tion (n = 82) was assessed by flow-meditated dilatation (FMD) at baseline and 6 mo.

124 ED was evaluated by means of flow-mediated dilatation (FMD) of the brachial artery.

125 e (WSR) stimulus or an altered flow-mediated dilatation (FMD) response to the WSR stimulus.

126 e (WSR) stimulus or an altered flow-mediated dilatation (FMD) response.

127 on assessed by brachial artery flow-mediated dilatation (FMD) was measured before, immediately follow

128 ssed the prognostic utility of flow-mediated dilatation (FMD), a marker of vascular reactivity, which

129 Flow-mediated dilatation (FMD), FGF-23, serum lipid, hsCRP levels, BMI

130 ntral pulse pressure (cPP) and flow-mediated dilatation (FMD).

131 tion in the brachial artery by flow-mediated dilatation (FMD).

132 ned with the use of ultrasound flow-mediated dilatation (FMD).

133 to post-exercise reductions in flow-mediated dilatation (FMD).

134 ngle passive limb movement and flow-mediated dilatation (FMD).

135 would increase brachial artery flow-mediated dilatation (FMD).

136 To evaluate the degree of brachial dilatation, follow-up US was performed at 1 month after

137 Sinusoidal dilatation found in the absence of an impaired sinusoida

138 eeks of heat therapy increased flow-mediated dilatation from 5.6 +/- 0.3 to 10.9 +/- 1.0% (P < 0.01)

139 Mechanisms mediating the spread of dilatation from local to remote sites have been well stu

140 Patients with a brachial dilatation greater than median level showed a 1.8-times

141 olar changes, including periarteriolar space dilatation, haemosiderin deposition and inflammation, ar

142 of arteriolosclerosis, periarteriolar space dilatation, haemosiderin leakage, microinfarcts, and mic

143 ins) improve cutaneous endothelium-dependent dilatation; however, whether statin therapy alters skin

144 stress-induced cardiac fibrosis and chamber dilatation, improving systolic and diastolic functions.

145 r geometric changes were associated with LAD dilatation in 11 children with new onset of KD.

146 holecystic stranding was seen in 19, biliary dilatation in 12, liver infiltration in 13 and fat in 7

147 effects of irbesartan on the rate of aortic dilatation in children and adults with Marfan syndrome.

148 iated with a reduction in the rate of aortic dilatation in children and young adults with Marfan synd

149 study was to identify risk factors of aortic dilatation in children with BAV.

150 prognostic significance of small bowel (SB) dilatation in children with short bowel syndrome (SBS).

151 copy to monitor intracellular pH and luminal dilatation in enteroids under basal and regulated condit

152 ed increase in brachial artery flow-mediated dilatation in humans The increase in flow-mediated dilat

153 fetal anomaly scan (FAS) and/or evidence of dilatation in later investigations, adjusting for other

154 Among children with RPD at the FAS but no dilatation in later pregnancy or postpartum, we did not

155 ospital admissions when there was persistent dilatation in later pregnancy or postpartum.

156 iency specifically induces right ventricular dilatation in mouse embryos at embryonic day 16.5.

157 ransduction in vitro as well as flow-induced dilatation in murine mesenteric arteries.

158 Src inhibition did not increase dilatation in old arteries treated with the VE-cadherin

159 ries but did not affect the already impaired dilatation in old arteries.

160 adherens junctions and increased endothelial dilatation in old, but not young, arteries.

161 icant change in diameter of P7 arteries, and dilatation in P21 arteries.

162 kage, anastomotic stricture, and anastomotic dilatation in patients with lower thoracic esophageal ca

163 was demonstrated to be protective against RV dilatation in patients with repaired tetralogy of Fallot

164 independent determinants for ascending aorta dilatation in pediatric patients.

165 y blocked by fremanezumab, it did not induce dilatation in pial arteries, pial veins, or dural veins.

166 By measuring pupillary dilatation in response to these stimuli-and simulating t

167 Left atrial dilatation in the population is more common in black and

168 cellular matrix may help prevent ventricular dilatation in the pressure-overloaded RV.

169 lem with the leaflets) or secondary (chamber dilatation in the setting of cardiomyopathy).

170 n fibronectin (FN) contributes to functional dilatation in these arterioles.

171 -blocking antibody; FBA) reduced endothelial dilatation in young arteries but did not affect the alre

172 Saracatinib did not affect dilatation in young arteries.

173 perivascular nerve stimulation (PNS) evoked dilatation in Young but not Old MAs while dilatations to

174 green angiography revealed chroidal vascular dilatations in both eyes in the late phase.

175 rotid arteries and aortae, and flow-mediated dilatations in third-order mesenteric resistance arterie

176 Relative amplitudes of dilatation increased with contraction duration and with

177 Pore dilatation increases the bicarbonate permeability (P HC

178 Here, we provide evidence that pore dilatation increases the bicarbonate permeability (P HC

179 or AI, there is significant ascending aorta dilatation independent of valve morphology.

180 EC-denuded arterioles failed to produce any dilatation indicating that endothelium was required for

181 ion in his general condition, showed jejunal dilatation, intestinal intramural gas, portomesenteric v

182 Left ventricular (LV) dilatation is a key step in transition to heart failure

183 greater total heart volume caused by atrial dilatation, leading to elevated filling pressures throug

184 lower left ventricular [LV] mass, reduced LV dilatation, less LV sphericity) versus the control group

185 wall, cholelithiasis, infiltration, biliary dilatation, lymph nodes, complications.

186 LA dilatation may be mediated by blood pressure control and

187 The dynamic increase in P HC O3/ Cl by pore dilatation may have many physiological and pathophysiolo

188 arker of the LV ejection according to the LV dilatation may predict postoperative LVD and outcome aft

189 tion in humans The increase in flow-mediated dilatation occurred in the face of an unaltered shear st

190 No complications related to aortic dilatation occurred in this cohort.

191 elevated shear stress secondary to metabolic dilatation of arterioles.

192 aortic left renal vein with gross aneurysmal dilatation of both pre- and retro-aortic part of the ren

193 ged constriction of pial arteries, prolonged dilatation of dural arteries and PPE are all unaffected

194 ough CGRP infusion gave rise to the expected dilatation of dural arteries, which was effectively bloc

195 component (P = 0.014), main pancreatic duct dilatation of more than 5 mm (P < 0.001), and jaundice (

196 triction of stenosed epicardial segments and dilatation of normal segments, with trends toward revers

197 whereas the brief constriction and prolonged dilatation of pial veins are affected.

198 mmation, hepatocellular swelling, steatosis, dilatation of portal lymphatics, and periductal fibrosis

199 n-mydriatic camera at a medical clinic, with dilatation of pupil of those who have ungradable images,

200 Abdominal aortic aneurysm (AAA) is a local dilatation of the abdominal aortic vessel wall and is am

201 ct in the penile urethra and associated mild dilatation of the anterior urethra ending in a smooth bu

202 ion (14.2% versus 6.7%, P<0.001) and diffuse dilatation of the aortic root and ascending aorta (16.2%

203 stal to the sinotubular junction, or diffuse dilatation of the aortic root and ascending aorta.

204 ) is caused by the progressive weakening and dilatation of the aortic wall and can lead to aortic dis

205 f Valsalva or sinotubular junction, isolated dilatation of the ascending aorta distal to the sinotubu

206 lly, aged Npr2(+/-);Ldlr(-/-) mice developed dilatation of the ascending aortic, with greater aneurys

207 Flow-mediated dilatation of the brachial artery increased in the inter

208 phenotypes are expressed in the kidney with dilatation of the collecting ducts, systemic hypertensio

209 metastasis by inducing lymphangiogenesis and dilatation of the lymphatic vasculature, facilitating tu

210 Dilatation of the neoaortic root was common (76%), and r

211 tionship between the diameter and aneurysmal dilatation of the paraumbilical vein (PUV) and the prese

212 fic differences in patients with and without dilatation of the pulmonal trunk.

213 Men had more frequently isolated dilatation of the sinus of Valsalva or sinotubular junct

214 opathy configuration was defined as isolated dilatation of the sinus of Valsalva or sinotubular junct

215 ynamic profiles predispose these patients to dilatation of the thoracic aorta, which is generally sil

216 In severe cases dilatation of the ureter, renal pelvis, and calyces migh

217 via alphaARs; with advanced age, attenuated dilatation of upstream branches will restrict muscle blo

218 egrity give rise to varying degrees of local dilatations of the thoracic aorta, with enlargement typi

219 g if age >64 years), intrapulmonary vascular dilatation on CE, and absence of lung disease.

220 The impact of dilatation on parenteral nutrition (PN) dependence and s

221 The optimal cutoff for optic nerve sheath dilatation on ultrasonography was 5.0 mm.

222 hallenge the paradigm that right ventricular dilatation on ultrasound during cardiopulmonary resuscit

223 ght patients (14%) developed coronary artery dilatation or aneurysm.

224 on the likelihood of subjects having cardiac dilatation or hypertrophy according to standard cardiac

225 nd there is a risk of overdiagnosing cardiac dilatation or hypertrophy in a proportion of active, hea

226 GRP contributes to the induction of arterial dilatation or PPE by CSD in female rats, and how these e

227 ve before or after the induction of arterial dilatation or PPE by CSD, the inability of fremanezumab

228 66; 95% CI 0.86-3.23; P = 0.1339), stricture dilatation (OR 1.90; 95% CI 0.16-3.88; P = 0.0767), and

229 lability is associated with ascending aortic dilatation, outflow tract malrotation, overriding aorta,

230 in sensitivity (P = 0.033) and flow-mediated dilatation (P < 0.001), while aortic pulse wave velocity

231 tomosis more frequently required anastomotic dilatation (P = 0.02).

232 ificant oversizing group underwent less post-dilatation (P=0.002) but achieved greater stent expansio

233 essness, sleep disturbance, cyanosis, venous dilatation, paresthesia, headache, and tinnitus) in the

234 Right ventricular (RV) dilatation persisted at follow-up in 92% of participants

235 the natural history of the RPD (whether the dilatation persists in later pregnancy or postpartum) or

236 ed pronephric-outlet obstruction and cloacal dilatation, phenocopying human congenital LUTO.

237 SB dilatation predicts prolonged PN duration and decreased

238 Dilatation pressure and balloon diameter at the highest

239 d not affect the percentage of flow-mediated dilatation (primary endpoint) or other measures of vascu

240 The local arteriolar dilatation produced by contraction of skeletal muscle is

241 endent of the severity of LV dysfunction, LV dilatation, pulmonary hypertension, severity of tricuspi

242 vascular function measured as flow-mediated dilatation (R = -0.3, P < 0.01) or endothelial injury ma

243 siderable flame-generated enstrophy, and the dilatation rate and baroclinic torque contributions to t

244 BRZ regimes, with diminishing influences of dilatation rate and baroclinic torque.

245 However, losartan reduced only aortic root dilatation rate in haploinsufficient patients (no losart

246 s, and topologies existing for all values of dilatation rate remain significant contributors.

247 ly, flow topologies associated with positive dilatation rate values, contribute significantly to the

248 Ascending aorta dilatation rate was significantly increased in patients

249 The strongest predictors of an increased dilatation rate were severe aortic stenosis, moderate an

250 ing BAV exhibited a very slow proximal aorta dilatation rate.

251 ersion, heat therapy increased flow-mediated dilatation, reduced arterial stiffness, reduced mean art

252 leocecal valve, both estimates of maximal SB dilatation remained significant independent predictors f

253 known whether mild-to-moderate renal pelvis dilatation (RPD) identified at 18-20 weeks gestation is

254 Whereas women exhibited RV cavity dilatation (RV end-diastolic volume, +1.0 mL per BMI poi

255 to increased afterload and left ventricular dilatation secondary to volume overload.

256 Interestingly, the early brachial dilatation showed significant correlations with diabetes

257 1.989; 95% CI: 1.403 to 2.818), lack of pre-dilatation (SHR: 1.485; 95% CI: 1.065 to 2.069), and tre

258 5 stenting (423 participants) and 12 balloon dilatation studies (361 participants), including patient

259 stenting (3397 participants) and 62 balloon dilatation studies (4331 participants).

260 ndependently associated with ascending aorta dilatation, suggesting that hemodynamic factors influenc

261 ervation revealed endoplasmic reticulum (ER) dilatation, suggestive of ER stress, and smaller insulin

262 Cardiac dilatation suggests clinically important changes with bo

263 se, characterised by internal carotid artery dilatation, terminal segment stenosis and absent basal c

264 and they had a significantly lower brachial dilatation than patients with successful AVF during earl

265 Acetylcholine caused endothelium-dependent dilatation that was decreased in old compared to young a

266 threshold of aortic root or ascending aortic dilatation that would justify surgical intervention in p

267 bosis (treated 2 weeks after pPCI by balloon dilatation-this patient stopped all medications after pP

268 nsequently, that drugs that prevent vascular dilatation through different molecular pathways may have

269 (function-blocking antibody, FBA) inhibited dilatation to acetylcholine in young, but not old, arter

270 unction [area-under-the-curve carotid artery dilatation to acetylcholine in young: 345 +/- 16 AU vs.

271 Venular dilatation to acetylcholine was blunted in OZR vs. LZR d

272 nt dilatation (EDD), assessed by the maximal dilatation to acetylcholine, was approximately 40% lower

273 MAs were lost in Old MAs along with impaired dilatation to calcitonin gene-related peptide (CGRP).

274 dothelial denudation reduced the efficacy of dilatation to CGRP by approximately 30% in Old MAs yet i

275 Endothelial denudation attenuated dilatation to CGRP in Old MAs yet enhanced dilatation to

276 d dilatation to CGRP in Old MAs yet enhanced dilatation to CGRP in Young MAs while abolishing all dil

277 cking this signalling sequence decreased the dilatation to skeletal muscle contraction, indicating th

278 acy approximately 15% in Young MAs while all dilatations to ACh were abolished.

279 ed dilatation in Young but not Old MAs while dilatations to ACh were not different between age groups

280 on to CGRP in Young MAs while abolishing all dilatations to ACh.

281 tudied using acetylcholine (ACh), but remote dilatations to contraction of skeletal muscle fibres als

282 OP experts graded 84 images showing vascular dilatation, tortuosity, or both and 251 images showing n

283 capture fundus images before and after pupil dilatation, using a hand-held non-mydriatic (Visuscout 1

284 A-BRS when overexpanded 1.3 mm above nominal dilatation values ( approximately 48%) and lower number

285 plex flow profiles are associated with aorta dilatation, ventricle remodeling, aneurysms, and develop

286 thetized rats, EFS failed to stimulate major dilatation via sensory-motor nerves but induced sympathe

287 The mean rate of aortic root dilatation was 0.53 mm per year (95% CI 0.39 to 0.67) in

288 By multivariable analysis, neoaortic root dilatation was associated with worse neoaortic valve reg

289 69-6.75, p = 0.185), except when the initial dilatation was bilateral (cHR 4.77, 95% CI 1.17-19.47, p

290 or hypoxic pregnant mice to dilate and this dilatation was partially reversed by the NOS inhibitor l

291 Aortic dilatation was present in 29% of the patients, and 26% h

292 After junctional disruption with the FBA, dilatation was similar in young and old arteries.

293 Potassium-induced dilatations were unaffected by inhibitors of TRPV4, IK a

294 P was <12 mm Hg in 11 patients (44%) with LA dilatation, whereas PCWP was <25 mm Hg in 1 patient (4%)

295 e dysfunction and progressive proximal aorta dilatation, which can lead to aortic dissection.

296 itor, in Marfan syndrome might reduce aortic dilatation, which is associated with dissection and rupt

297 Neoaortic root dilatation, which is present in most patients and progre

298 sociation of early optimal brachial arterial dilatation with a successful AVF maturation and assessed

299 lial cytoplasmatic vacuolization and luminal dilatation with flattening of the epithelium.

300 be effective because this entity has annular dilatation without leaflet deformation.

301 MIS-C group (4%) manifested coronary artery dilatation (z score = 3.15) in the acute phase, showing