ライフサイエンスコーパス: dilate

1 Adjacent intrahepatic bile ducts were dilated.

2 st, the ventricles are collapsed rather than dilated.

3 hy were identified, of which 2631 (59%) were dilated, 673 (15%) were hypertrophic, and 480 (11%) were

4 hat may represent muscles whose contractions dilate and constrict the pupil, respectively.

5 ure to dynamic visual stimuli, blood vessels dilate and the flow of blood within vessels increases to

6 Vascular tips were dilated and fused, resulting in glomeruloid vessels.

7 Debilitating heart conditions, notably dilated and hypertrophic cardiomyopathies (CMs), are ass

8 Dilated and hypertrophic cardiomyopathies are the most c

9 The chronic CAVB animals developed dilated and hypertrophied ventricles with preserved syst

10 ntial subset of cases, patients present with dilated and poorly contracting hearts without evidence o

11 who found FLNC variants among patients with dilated and restrictive cardiomyopathies, pointed to thi

12 eleis plaques, irregular venous caliber with dilated and sclerotic segments, perivenular hemorrhages,

13 ofibres; however, the resistance network was dilated and unresponsive to vasoactive agents.

14 of a conformational change that alternately dilates and constricts a collar-like intracellular entra

15 ured 9-month-old infants' brain responses to dilating and constricting pupils in the context of viewi

16 tients and families, including hypertrophic, dilated, and arrhythmogenic cardiomyopathies and inherit

17 the umbilical vein was surgically reopened, dilated, and cannulated.

18 pt knockout mice were viable but with mildly dilated aortas that had a 43% reduction in NAD(+) in the

19 pupils that constrict to ~60% of their fully dilated areas within several minutes of light exposure.

20 Here, we show in mice that CO(2)/H(+) dilates arterioles in other chemoreceptor regions, thus

21 In SBS, the remaining SB may dilate as part of intestinal adaptation.

22 from ridge to valley and because pores have dilated as the deep rock has been exhumed by erosion.

23 neuro-axonal dystrophy, including abnormally dilated axon terminals and increased amounts of synapses

24 Furthermore, dilated bile duct is the only risk factor for bile duct

25 Furthermore, dilated bile duct was the only risk factor for bile duct

26 by a primitive myxoid stroma with cystically dilated bile ducts.

27 Small, round clear spheres in vitreous on dilated biomicroscopic retinal examination.

28 ions were composed predominantly of variably dilated, branching tubular structures with pseudo-apocri

29 tions of amino acids in the open pore, which dilates by outward movements of subunits.

30 art:body weight ratio, coupled with a severe dilated cardiomyopathic phenotype resulting in death wit

31 nd how TNNT2 variants cause hypertrophic and dilated cardiomyopathies could improve heart failure ris

32 NT2 variants are a cause of hypertrophic and dilated cardiomyopathies, which promote heart failure by

33 re detected in about one-third of idiopathic dilated cardiomyopathies.

34 rtrophic cardiomyopathy (1/500 individuals), dilated cardiomyopathy (1/250) and arrhythmogenic right

35 arrhythmias (69% and 52%, respectively), and dilated cardiomyopathy (74% and 14%, respectively).

36 vestigate the frequency and genetic basis of dilated cardiomyopathy (DCM) among relatives of index pa

37 ) is an established therapy in patients with dilated cardiomyopathy (DCM) and conduction disorders.

38 in striated muscle tropomyosin are linked to dilated cardiomyopathy (DCM) and hypertrophic cardiomyop

39 ompared them with samples from patients with dilated cardiomyopathy (DCM) and inflammatory cardiomyop

40 Genotype-phenotype correlations in dilated cardiomyopathy (DCM) and, in particular, the eff

41 c disease identified an Iranian patient with dilated cardiomyopathy (DCM) as a carrier of a novel, ho

42 ABSTRACT: Dilated cardiomyopathy (DCM) can be caused by mutations

43 own to be associated with the development of dilated cardiomyopathy (DCM) in Doberman Pinchers (DPs).

44 exploring the molecular basis of congenital dilated cardiomyopathy (DCM) in genome-edited pigs homoz

45 Dilated cardiomyopathy (DCM) is a clinical diagnosis cha

46 Dilated cardiomyopathy (DCM) is a common condition, whic

47 Dilated cardiomyopathy (DCM) is a condition of abnormal

48 Dilated cardiomyopathy (DCM) is a genetically heterogene

49 Dilated cardiomyopathy (DCM) is a leading cause of morbi

50 Familial dilated cardiomyopathy (DCM) is a leading cause of sudde

51 Dilated cardiomyopathy (DCM) is a major cause of mortali

52 Dilated cardiomyopathy (DCM) is a major co-existing form

53 Dilated cardiomyopathy (DCM) is an important cause of he

54 Dilated cardiomyopathy (DCM) is an important cause of he

55 Dilated cardiomyopathy (DCM) is associated with mutation

56 Dilated cardiomyopathy (DCM) is genetically heterogeneou

57 The cause of idiopathic dilated cardiomyopathy (DCM) is unknown by definition, b

58 ix hypertrophic cardiomyopathy (HCM) and two dilated cardiomyopathy (DCM) mutants were studied by bio

59 We investigated a dilated cardiomyopathy (DCM) mutation (F764L) in human b

60 rdiomyocytes (iPSC-CMs) from patients with a dilated cardiomyopathy (DCM) mutation, troponin T (TnT)-

61 tudies reveal reduced force generation and a dilated cardiomyopathy (DCM) phenotype.

62 hythmogenic right ventricular cardiomyopathy/dilated cardiomyopathy (DCM) with an initial focus on PL

63 a frequency of 6% among Danish patients with dilated cardiomyopathy (DCM), it was the aim to investig

64 CCM), hypertrophic cardiomyopathy (HCM), and dilated cardiomyopathy (DCM).

65 cause a diverse array of diseases, including dilated cardiomyopathy (DCM).

66 s) are common in individuals with idiopathic dilated cardiomyopathy (DCM).

67 on and systolic dysfunction resembling human dilated cardiomyopathy (DCM).

68 ost frequently mutated genes associated with dilated cardiomyopathy (DCM).

69 n-truncating variants (TTNtv) commonly cause dilated cardiomyopathy (DCM).

70 common inciting stressors, a syndrome called dilated cardiomyopathy (DCM).

71 Genetic analysis is a first-tier test in dilated cardiomyopathy (DCM).

72 rlying structural heart disease consisted of dilated cardiomyopathy (DCM, 49%), arrhythmogenic right

73 chanically Assisted Circulatory Support with dilated cardiomyopathy (DCM, n=19 921), nonamyloid restr

74 loci that are significantly associated with dilated cardiomyopathy (false discovery corrected P</=0.

75 aring the survival of children with familial dilated cardiomyopathy (FDCM) to that of children with i

76 y (FDCM) to that of children with idiopathic dilated cardiomyopathy (IDCM) has produced conflicting r

77 005) and of patients with end-stage ischemic-dilated cardiomyopathy (mean difference, 0.13 [95% CI, 0

78 ular cardiomyopathy (ARVC) (n = 9 [8%]), and dilated cardiomyopathy (n = 5 [4%]).

79 hese, 15 (7%) were diagnosed antemortem with dilated cardiomyopathy (n=8) or ACM (n=7).

80 tic information in patients with nonischemic dilated cardiomyopathy (NIDCM).

81 hy, few studies exist in chronic nonischemic dilated cardiomyopathy (NIDCM).

82 atio, 1.08 [95% CI, 1.05-1.11]; P<0.001) and dilated cardiomyopathy (odds ratio, 1.04 [95% CI, 1.01-1

83 ion using RNA-sequencing in 97 patients with dilated cardiomyopathy and 108 non-diseased controls.

84 tem cells were used to examine a family with dilated cardiomyopathy and atrial and ventricular arrhyt

85 A subgroup of dilated cardiomyopathy and control subjects underwent ca

86 myocardial tissue and blood of patients with dilated cardiomyopathy and controls.

87 a pair of siblings with rapidly progressive dilated cardiomyopathy and death in early infancy.

88 CaMKII overexpression (mtCaMKII) have severe dilated cardiomyopathy and decreased ATP that causes ele

89 obal developmental delay, failure to thrive, dilated cardiomyopathy and epilepsy, ultimately leading

90 members) with ASD combined with a late-onset dilated cardiomyopathy and further characterize the cons

91 (2)(+) responsiveness in a model of familial dilated cardiomyopathy and improve cardiac function and

92 nd inflammation in patients with nonischemic dilated cardiomyopathy and inflammatory cardiomyopathy (

93 Here the authors focus on genetics of dilated cardiomyopathy and provide a roadmap for impleme

94 Patients with ischemic or dilated cardiomyopathy and reduced left ventricular ejec

95 characterized by fibrosis: human and murine dilated cardiomyopathy and repaired tetralogy of Fallot.

96 relationship of ivabradine in children with dilated cardiomyopathy and symptomatic chronic HF.

97 ubunit-encoding Kcne2 (Kcne2(CS-/-) ) causes dilated cardiomyopathy and terminal HF (median longevity

98 the titin and sarcomere variants that cause dilated cardiomyopathy and the desmosomal variants that

99 istry than in large cohorts of patients with dilated cardiomyopathy and TTNtv cardiomyopathy and not

100 Moreover, TTNtv associated with dilated cardiomyopathy are estimated to be present in 0.

101 ause an autosomal dominant inherited form of dilated cardiomyopathy associated with cardiac conductio

102 scular dystrophy (DMD) develop a progressive dilated cardiomyopathy characterized by inflammatory cel

103 of cardiovascular mortality compared with a dilated cardiomyopathy control group (odds ratio, 1.10 [

104 Patients with myocarditis or dilated cardiomyopathy develop autoantibodies to SERCA2a

105 in quality control, we used a mouse model of dilated cardiomyopathy driven by cardiac restricted over

106 cardioverter defibrillator in patients with dilated cardiomyopathy for the primary prevention of sud

107 he decedents with an antemortem diagnosis of dilated cardiomyopathy fulfilled definite 2010 Task Forc

108 opathy Precision Medicine Study is that most dilated cardiomyopathy has a genetic basis.

109 ified in association with ASD and late-onset dilated cardiomyopathy in a large, multi-generational fa

110 verse cardiovascular events in patients with dilated cardiomyopathy in a multicenter setting as part

111 and clinically relevant treatment to rescue dilated cardiomyopathy in patients with DMD.

112 isease models of myocardial infarction (MI), dilated cardiomyopathy induced via aortic banding, and s

113 Because a common finding in dilated cardiomyopathy is a reduction in the myofilament

114 Dilated cardiomyopathy is associated with increased risk

115 Heart failure due to dilated cardiomyopathy is frequently caused by myocardit

116 involved in viral RNA replication.IMPORTANCE Dilated cardiomyopathy is the most common indication for

117 ual myocardial injury whereas others develop dilated cardiomyopathy is unclear.

118 LN R14del mutation are at risk of developing dilated cardiomyopathy or arrhythmogenic right ventricul

119 tes myocardial inflammation with established dilated cardiomyopathy or hypokinetic nondilated phenoty

120 heart failure instead of the commonly taught dilated cardiomyopathy pathway.

121 with cardiac ribosome occupancy levels of 30 dilated cardiomyopathy patients demonstrates that these

122 Methods Thirty-four dilated cardiomyopathy patients, 30 ischemic cardiomyopa

123 an tissues, and upregulated in the hearts of dilated cardiomyopathy patients.

124 idence of heart failure hospitalization than dilated cardiomyopathy patients.

125 milar cardiovascular risk when compared with dilated cardiomyopathy patients.

126 arrhythmias in LVNC patients were similar to dilated cardiomyopathy patients.

127 d that the knockout strategy ameliorates the dilated cardiomyopathy phenotype in vitro.

128 The hypothesis of the Dilated Cardiomyopathy Precision Medicine Study is that

129 ere calculated in parents of 128 present-day dilated cardiomyopathy probands with TTNtv using the rev

130 ntractions are known to trigger a reversible dilated cardiomyopathy referred as arrhythmia-induced ca

131 Consecutive patients with dilated cardiomyopathy referred for cardiac magnetic res

132 ral functions involved in the development of dilated cardiomyopathy remain unclear.

133 ome occupancy in the hearts of patients with dilated cardiomyopathy suggested the same posttranscript

134 r envelope gene SYNE1 in a child with severe dilated cardiomyopathy that underwent transplant, as wel

135 -nine patients with ischemic or non-ischemic dilated cardiomyopathy undergoing prophylactic ICD impla

136 tal RNA from cardiac tissue of patients with dilated cardiomyopathy was extracted, and sequences corr

137 Ang II levels in explanted human hearts with dilated cardiomyopathy were elevated despite ACE inhibit

138 ailure medications in patients with previous dilated cardiomyopathy who were now asymptomatic, whose

139 Patients with dilated cardiomyopathy whose symptoms and cardiac functi

140 Many patients deemed to have recovered from dilated cardiomyopathy will relapse following treatment

141 iffered considerably: loss of titin leads to dilated cardiomyopathy with combined systolic and diasto

142 The primary endpoint was a relapse of dilated cardiomyopathy within 6 months, defined by a red

143 that often presents as heart failure due to dilated cardiomyopathy years after anthracycline exposur

144 earts of transplant recipients (ischemic and dilated cardiomyopathy), and from nonused donor hearts.

145 164 ischemic cardiomyopathy, 150 nonischemic dilated cardiomyopathy), the mean left ventricular eject

146 ard ratios were 1.15 (95% CI, 1.14-1.17) for dilated cardiomyopathy, 1.09 (95% CI, 1.06-1.12) for hyp

147 Of these, 481 were diagnosed with dilated cardiomyopathy, 246 had hypertrophic cardiomyopa

148 ARVC, 9 (25%) of 36; LQTS, 48 (20%) of 238; dilated cardiomyopathy, 5 (9%) of 58; and HCM, 28 (8%) o

149 this critical overlap region associated with dilated cardiomyopathy, A277V, will alter Tpm binding an

150 is patients, 11 ischemic heart disease, nine dilated cardiomyopathy, and 11 nonfailing donors.

151 ble cases by cardiac MRI, 3 were found to be dilated cardiomyopathy, and 2 were found to be end-stage

152 120 patients with ischemic, 60 patients with dilated cardiomyopathy, and 30 patients with normal LVEF

153 n of function occurs in 20% of children with dilated cardiomyopathy, and 40% die or undergo transplan

154 uctive cardiomyopathy, 131 genes/17 ncRNA in dilated cardiomyopathy, and 51 genes/5 ncRNA in ischemic

155 ertrophic obstructive cardiomyopathy, 151 in dilated cardiomyopathy, and 55 in ischemic cardiomyopath

156 ive cardiomyopathy, ischemic cardiomyopathy, dilated cardiomyopathy, and 9 control patients with nonf

157 nalysis of fibrotic scarring in non-ischemic dilated cardiomyopathy, and its relationship to electric

158 the regulation of development, ischemic and dilated cardiomyopathy, and myocardial infarction.

159 ntified in a pediatric patient with sporadic dilated cardiomyopathy, and we determined a molecular me

160 ecellularized ECM resulting from ischemic or dilated cardiomyopathy, as well as from mouse infarcted

161 in the hearts of individuals with idiopathic dilated cardiomyopathy, as well as the hearts of patient

162 are frequent causes of acute myocarditis and dilated cardiomyopathy, but an effective antiviral thera

163 gene mutations are a known cause of familial dilated cardiomyopathy, but the precise mechanisms trigg

164 age-dependent cardiac phenotypes, including dilated cardiomyopathy, cardiac conduction disturbance,

165 In pediatric patients with dilated cardiomyopathy, compared with dimension and area

166 involvement is characterized by progressive dilated cardiomyopathy, decreased fractional shortening

167 Ntv) are the most prevalent genetic cause of dilated cardiomyopathy, found in <=25% of familial cases

168 is the major cause of death and manifests as dilated cardiomyopathy, heart failure, arrhythmias, and

169 c cardiomyopathy, ischemic heart disease, or dilated cardiomyopathy, in comparison to nonfailing hear

170 an be a precursor of chronic myocarditis and dilated cardiomyopathy, leading causes of heart transpla

171 Compared with other forms of dilated cardiomyopathy, mutations in LMNA are responsibl

172 In patients with dilated cardiomyopathy, myocardial Gal-3 expression corr

173 in gene are associated with hypertrophic and dilated cardiomyopathy, respectively.

174 with a subsequent cardiomyopathy, especially dilated cardiomyopathy, starting already at mildly eleva

175 if concentric hypertrophy does progress to a dilated cardiomyopathy, such a transition would occur ov

176 s large multinational study of patients with dilated cardiomyopathy, the presence of LGE showed stron

177 se in risk with higher BMI, particularly for dilated cardiomyopathy, where a hazard ratio of 4.71 (95

178 Mutations in A-type nuclear lamins cause dilated cardiomyopathy, which is postulated to result fr

179 sed cardiac microtissue contraction, whereas dilated cardiomyopathy-associated variants decreased con

180 sed cardiac microtissue contraction, whereas dilated cardiomyopathy-associated variants decreased con

181 ult cardiac vasculature and thereby prevents dilated cardiomyopathy-like defects.

182 nd in heart tissue from patients with MI and dilated cardiomyopathy.

183 TTN) are the leading known cause of familial dilated cardiomyopathy.

184 lly induced trophic signaling and eventually dilated cardiomyopathy.

185 s with regard to monitoring for (late-onset) dilated cardiomyopathy.

186 viral proteinase 2A in cases of unexplained dilated cardiomyopathy.

187 (TTNtv) are the most common genetic cause of dilated cardiomyopathy.

188 duced myocyte complex N-glycosylation causes dilated cardiomyopathy.

189 scle cell leads to myocyte instability and a dilated cardiomyopathy.

190 ative relatives, and 9 with noncompaction or dilated cardiomyopathy.

191 to one that leads to chronic remodeling and dilated cardiomyopathy.

192 hmogenic right ventricular cardiomyopathy or dilated cardiomyopathy.

193 ation (Id cDKOs), which develops adult-onset dilated cardiomyopathy.

194 (TTNtv) are the commonest cause of heritable dilated cardiomyopathy.

195 induced hypertrophy and in human hearts with dilated cardiomyopathy.

196 ular arrhythmias in patients with idiopathic dilated cardiomyopathy.

197 t ventricle (LV) ultimately transitions to a dilated cardiomyopathy.

198 se-specific considerations were required for dilated cardiomyopathy.

199 dilation, and a fourth-degree relative with dilated cardiomyopathy.

200 understanding of the genetic architecture of dilated cardiomyopathy.

201 d on the uncharacterized lncRNA GATA6-AS1 in dilated cardiomyopathy.

202 alterations of signaling pathways leading to dilated cardiomyopathy.

203 respiratory failure, and in the older cases, dilated cardiomyopathy.

204 We find that a dilated central region has the strongest impact in thick

205 cluding wide residual networks, dropouts and dilated convolutions.

206 -myosin variants cause hypertrophic (HCM) or dilated (DCM) cardiomyopathy by disrupting sarcomere con

207 Dilated distal vessels extending to the pleura and fissu

208 Fluorescein angiography showed bilateral dilated, ectatic capillaries and late phase dye leak.

209 ay, resulting in a buildup of virions within dilated ER vesicles.IMPORTANCE In humans, symptoms of RV

210 eported an eye care visit and 21% reported a dilated examination in the past 12 months.

211 All participants underwent a dilated examination to determine the grade of DR.

212 tion, clinical information, and time to last dilated eye exam.

213 significantly associated with an increase in dilated eye examination rates within the first 2 years a

214 th incomes below 138% of the FPL receiving a dilated eye examination within the past year due to Medi

215 wer odds of reporting ever having received a dilated eye examination.

216 rty-eight percent reported ever having had a dilated eye examination.

217 atients with diabetes reported ever having a dilated eye examination; only 39.62% reported receiving

218 ecessary to increase and sustain the rate of dilated eye examinations among diabetic populations.

219 ponders who were asked about the presence of dilated eye examinations from years before and after Med

220 yndrome, and five of six individuals who had dilated eye exams had retinal pigmentary abnormalities.

221 We found that pupil size dilated following these surprising events, in the absenc

222 t learning.SIGNIFICANCE STATEMENT Pupil size dilates following increase in mental effort, surprise, o

223 42 cases (16.7%), the residual iris aperture dilated from 36.6 +/- 15.4 mm(2) preoperatively to 61.1

224 Agreement between TRI and dilated fundus examination (DFE) findings was determined

225 Dilated fundus examination and fundus photography were e

226 t a comprehensive eye examination, including dilated fundus examination and imaging.

227 ects were obtained and cross-referenced with dilated fundus examination findings with regard to DR se

228 went visual acuity (VA) testing, refraction, dilated fundus examination fluorescein angiography (FA)

229 Dilated fundus examination showed an inferior bullous RD

230 Dilated fundus examination was unremarkable.

231 urement of intraocular pressure, gonioscopy, dilated fundus examination, and fluorescein angiography

232 Avascular retina was identified on dilated fundus examination, fluorescein angiography, or

233 isual acuity, slit-lamp biomicroscopy, and a dilated fundus examination.

234 ma, emphasizing the importance of a detailed dilated fundus examination.

235 by eliminating formal visual acuity (VA) and dilated fundus examinations (DFEs) were assessed for est

236 comprehensive ocular examination, including dilated fundus photography.

237 osis, including the majority of those with a dilated funduscopic examination in the past year.

238 Dilated funduscopic examinations were performed by an op

239 sual acuity, IOP, slit lamp examination, and dilated funduscopy as well as comparative measurements b

240 d distended duodenum filled with fluid and a dilated gallbladder containing dilute bile with high bic

241 ography of the abdomen at admission revealed dilated gas-filled small-bowel loops, suggestive of obst

242 The adult mutant mice developed dilated hearts and showed significantly shorter life spa

243 hosphorylation is significantly decreased in dilated human hearts, supporting the notion that modulat

244 Indocyanine green angiography revealed dilated hyperpermeable choroidal vasculature on the nasa

245 -year follow-up and divided into categories: dilated, hypertrophic, alcohol/drug-induced, and other.

246 Pupils tend to dilate in response to surprising events, but it is not k

247 ction of cardiac arrest, the right ventricle dilated in all groups (p < 0.01 for all).

248 lumination reflects brain arousal state, and dilates in response to novel information, or surprisal.

249 lammation, basal zone hyperplasia (BZH), and dilated intercellular spaces, and the underlying process

250 An abdominal CT demonstrated a dilated intrahepatic biliary tree with left proximal int

251 xtracellular matrix (ECM) abnormalities, and dilated intrascleral veins, yet, no dilation of arteries

252 However, the right ventricle was dilated, irrespective of the cause of arrest, and diagno

253 t the rate at which the pupil constricts and dilates is matched to the temporal structure of our visu

254 ilators that increase their energy supply by dilating local blood vessels, a mechanism termed neurova

255 se with interval cardiovascular events and a dilated LV (increased LV end-diastolic volume [EDV] inde

256 as minimal, and few participants developed a dilated LV.

257 m in diameter, was inserted to the straight, dilated main duct and plastic stent(s) were inserted to

258 multilocular cysts of the pancreas and a non-dilated main pancreatic duct (<5 mm).

259 larger than 4 cm (P < 0.001), presence of a dilated main pancreatic duct of over 4 mm (P < 0.001), h

260 of 88 patients with chronic pancreatitis, a dilated main pancreatic duct, and who only recently star

261 e because of mechanical interaction with the dilated main pulmonary artery (MPA).

262 e fully convolutional network (FCN), SegNet, Dilated-Net, original U-Net, and Faster R-CNN models and

263 Transillumination revealed fully dilated, non-reactive RE pupil, clear lens and tubular r

264 diogram study, and those with a diagnosis of dilated, noncompaction, alcoholic, or toxic cardiomyopat

265 those younger than 10 years, undergo annual dilated ophthalmic examination, looking for evidence of

266 aire and underwent a single study visit with dilated ophthalmic examination, OCT angiography (OCTA),

267 intraocular pressure monitoring, slit-lamp, dilated ophthalmoscopy, and fundus examinations.

268 ntraocular pressure measurement, gonioscopy, dilated ophthalmoscopy, and standard automated perimetry

269 isease and may develop as the left ventricle dilates or remodels or as a result of leaflet tethering

270 er PD (ASA 3 OR 0.59 [0.44-0.80]), whereas a dilated pancreatic duct (>3 mm) and pancreatic ductal ad

271 Dilated peripheral vessels were present in 21/33 (63.6%)

272 abnormal lung and the presence and extent of dilated peripheral vessels) and perfusion defects on DEC

273 tation of mitochondrial cardiomyopathy, with dilated phenotype and pathologic evidence of biventricul

274 In patients with bicuspid aortic valve and dilated proximal ascending aorta, we sought to assess (1

275 valence of LMCA extrinsic compression from a dilated pulmonary artery (PA) in patients with PAH and a

276 ed pupils, eyes from A. irradians with fully dilated pupils provide approximately three times the sen

277 mined using 12x to 16x magnification through dilated pupils with ocular saccades before an injection.

278 y retention, dry mouth, lack of tears, fixed dilated pupils, and diffuse anhidrosis 7 days after a fe

279 m of "flushing out" crystals by purposefully dilating renal tubules has not previously been recognize

280 ncreased NO bioavailability that dynamically dilated resistance vessels in vivo under basal condition

281 ning for diabetic retinopathy include annual dilated retinal examinations to detect proliferative ret

282 ze with systolic dysfunction; and pattern 4, dilated RV systolic dysfunction.

283 al RV size and systolic function; pattern 2, dilated RV with preserved systolic function; pattern 3,

284 tissue included acinar cells and cystically dilated secretory ducts without islets of Langerhans.

285 uman hepatic vascular cavernoma phenotype of dilated sinusoidal capillaries with defective branching

286 7%, 32 of 37), and 54% (20 of 37) revealed a dilated sludge-filled gallbladder, suggestive of bile st

287 elevated risk of cardiomyopathy, especially dilated, starting at levels considered normal (BMI, 22.5

288 layed conductive, constricted, occluded, and dilated states, in qualitative agreement with the well-d

289 Hypoxia dilates systemic arteries, including renal arteries.

290 wear a mask, 73% wear gloves, and 45% always dilate the eyes before injection.

291 he MIGS device, and thus the more SC that it dilates, the greater the outflow facility.

292 tion, a neurovascular dysplasia resulting in dilated, thin-walled vessels that tend to rupture, incre

293 gest that "decisive" and "distilled" moments dilate time in the viewer's mind and might allow the mod

294 de, propagating, hyperpolarizing signal that dilates upstream arterioles to rapidly increase local bl

295 This disease manifested as a massively dilated urinary bladder, or megabladder, with disrupted

296 CS includes dilated vasculature, marked cardiac hypertrophy, and oth

297 ive hamartomas containing abnormal tortuous, dilated vessels prominent in the forelimbs.

298 Dilated vessels were seen in 41 cases (85%), with 38 (78

299 In contrast, CO(2) dilates vessels in other brain areas such as the amygdal

300 The right ventricle was dilated without evidence of right ventricular outflow tr