ライフサイエンスコーパス: dilated

1 Adjacent intrahepatic bile ducts were dilated.

2 st, the ventricles are collapsed rather than dilated.

3 hy were identified, of which 2631 (59%) were dilated, 673 (15%) were hypertrophic, and 480 (11%) were

4 Vascular tips were dilated and fused, resulting in glomeruloid vessels.

5 Debilitating heart conditions, notably dilated and hypertrophic cardiomyopathies (CMs), are ass

6 Dilated and hypertrophic cardiomyopathies are the most c

7 The chronic CAVB animals developed dilated and hypertrophied ventricles with preserved syst

8 ntial subset of cases, patients present with dilated and poorly contracting hearts without evidence o

9 who found FLNC variants among patients with dilated and restrictive cardiomyopathies, pointed to thi

10 eleis plaques, irregular venous caliber with dilated and sclerotic segments, perivenular hemorrhages,

11 ofibres; however, the resistance network was dilated and unresponsive to vasoactive agents.

12 tients and families, including hypertrophic, dilated, and arrhythmogenic cardiomyopathies and inherit

13 the umbilical vein was surgically reopened, dilated, and cannulated.

14 pt knockout mice were viable but with mildly dilated aortas that had a 43% reduction in NAD(+) in the

15 ts presenting with aortic insufficiency or a dilated aortic annulus or ascending aorta were at greate

16 HODS AND Ascending aortas from patients with dilated aortopathy were immunostained for NAMPT, reveali

17 pupils that constrict to ~60% of their fully dilated areas within several minutes of light exposure.

18 from ridge to valley and because pores have dilated as the deep rock has been exhumed by erosion.

19 s associated with bicuspid aortic valves and dilated ascending aortic geometries that are not assessa

20 neuro-axonal dystrophy, including abnormally dilated axon terminals and increased amounts of synapses

21 Furthermore, dilated bile duct is the only risk factor for bile duct

22 Furthermore, dilated bile duct was the only risk factor for bile duct

23 by a primitive myxoid stroma with cystically dilated bile ducts.

24 Small, round clear spheres in vitreous on dilated biomicroscopic retinal examination.

25 ions were composed predominantly of variably dilated, branching tubular structures with pseudo-apocri

26 art:body weight ratio, coupled with a severe dilated cardiomyopathic phenotype resulting in death wit

27 nd how TNNT2 variants cause hypertrophic and dilated cardiomyopathies could improve heart failure ris

28 NT2 variants are a cause of hypertrophic and dilated cardiomyopathies, which promote heart failure by

29 re detected in about one-third of idiopathic dilated cardiomyopathies.

30 rtrophic cardiomyopathy (1/500 individuals), dilated cardiomyopathy (1/250) and arrhythmogenic right

31 arrhythmias (69% and 52%, respectively), and dilated cardiomyopathy (74% and 14%, respectively).

32 vestigate the frequency and genetic basis of dilated cardiomyopathy (DCM) among relatives of index pa

33 ) is an established therapy in patients with dilated cardiomyopathy (DCM) and conduction disorders.

34 in striated muscle tropomyosin are linked to dilated cardiomyopathy (DCM) and hypertrophic cardiomyop

35 ompared them with samples from patients with dilated cardiomyopathy (DCM) and inflammatory cardiomyop

36 Genotype-phenotype correlations in dilated cardiomyopathy (DCM) and, in particular, the eff

37 c disease identified an Iranian patient with dilated cardiomyopathy (DCM) as a carrier of a novel, ho

38 ABSTRACT: Dilated cardiomyopathy (DCM) can be caused by mutations

39 own to be associated with the development of dilated cardiomyopathy (DCM) in Doberman Pinchers (DPs).

40 exploring the molecular basis of congenital dilated cardiomyopathy (DCM) in genome-edited pigs homoz

41 Dilated cardiomyopathy (DCM) is a clinical diagnosis cha

42 Dilated cardiomyopathy (DCM) is a common condition, whic

43 Dilated cardiomyopathy (DCM) is a condition of abnormal

44 Dilated cardiomyopathy (DCM) is a genetically heterogene

45 Dilated cardiomyopathy (DCM) is a leading cause of morbi

46 Familial dilated cardiomyopathy (DCM) is a leading cause of sudde

47 Dilated cardiomyopathy (DCM) is a major cause of mortali

48 Dilated cardiomyopathy (DCM) is a major co-existing form

49 Dilated cardiomyopathy (DCM) is an important cause of he

50 Dilated cardiomyopathy (DCM) is an important cause of he

51 Dilated cardiomyopathy (DCM) is associated with mutation

52 Dilated cardiomyopathy (DCM) is genetically heterogeneou

53 The cause of idiopathic dilated cardiomyopathy (DCM) is unknown by definition, b

54 ix hypertrophic cardiomyopathy (HCM) and two dilated cardiomyopathy (DCM) mutants were studied by bio

55 We investigated a dilated cardiomyopathy (DCM) mutation (F764L) in human b

56 rdiomyocytes (iPSC-CMs) from patients with a dilated cardiomyopathy (DCM) mutation, troponin T (TnT)-

57 tudies reveal reduced force generation and a dilated cardiomyopathy (DCM) phenotype.

58 Peripartum cardiomyopathy (PPCM) and dilated cardiomyopathy (DCM) show similarities in clinic

59 hythmogenic right ventricular cardiomyopathy/dilated cardiomyopathy (DCM) with an initial focus on PL

60 a frequency of 6% among Danish patients with dilated cardiomyopathy (DCM), it was the aim to investig

61 CCM), hypertrophic cardiomyopathy (HCM), and dilated cardiomyopathy (DCM).

62 cause a diverse array of diseases, including dilated cardiomyopathy (DCM).

63 s) are common in individuals with idiopathic dilated cardiomyopathy (DCM).

64 on and systolic dysfunction resembling human dilated cardiomyopathy (DCM).

65 ost frequently mutated genes associated with dilated cardiomyopathy (DCM).

66 n-truncating variants (TTNtv) commonly cause dilated cardiomyopathy (DCM).

67 reifuss muscular dystrophy (EDMD) as well as dilated cardiomyopathy (DCM).

68 common inciting stressors, a syndrome called dilated cardiomyopathy (DCM).

69 Genetic analysis is a first-tier test in dilated cardiomyopathy (DCM).

70 rlying structural heart disease consisted of dilated cardiomyopathy (DCM, 49%), arrhythmogenic right

71 chanically Assisted Circulatory Support with dilated cardiomyopathy (DCM, n=19 921), nonamyloid restr

72 patients with aortic stenosis (AS, n=9) and dilated cardiomyopathy (DCM, n=6).

73 Inflammatory dilated cardiomyopathy (DCMi) is a major cause of heart

74 loci that are significantly associated with dilated cardiomyopathy (false discovery corrected P</=0.

75 aring the survival of children with familial dilated cardiomyopathy (FDCM) to that of children with i

76 y (FDCM) to that of children with idiopathic dilated cardiomyopathy (IDCM) has produced conflicting r

77 005) and of patients with end-stage ischemic-dilated cardiomyopathy (mean difference, 0.13 [95% CI, 0

78 ular cardiomyopathy (ARVC) (n = 9 [8%]), and dilated cardiomyopathy (n = 5 [4%]).

79 hese, 15 (7%) were diagnosed antemortem with dilated cardiomyopathy (n=8) or ACM (n=7).

80 tic information in patients with nonischemic dilated cardiomyopathy (NIDCM).

81 hy, few studies exist in chronic nonischemic dilated cardiomyopathy (NIDCM).

82 atio, 1.08 [95% CI, 1.05-1.11]; P<0.001) and dilated cardiomyopathy (odds ratio, 1.04 [95% CI, 1.01-1

83 ion using RNA-sequencing in 97 patients with dilated cardiomyopathy and 108 non-diseased controls.

84 Here, we studied a family with dilated cardiomyopathy and associated conduction system

85 tem cells were used to examine a family with dilated cardiomyopathy and atrial and ventricular arrhyt

86 we link a subset of 517 epigenetic loci with dilated cardiomyopathy and cardiac gene expression.

87 A subgroup of dilated cardiomyopathy and control subjects underwent ca

88 myocardial tissue and blood of patients with dilated cardiomyopathy and controls.

89 a pair of siblings with rapidly progressive dilated cardiomyopathy and death in early infancy.

90 CaMKII overexpression (mtCaMKII) have severe dilated cardiomyopathy and decreased ATP that causes ele

91 obal developmental delay, failure to thrive, dilated cardiomyopathy and epilepsy, ultimately leading

92 members) with ASD combined with a late-onset dilated cardiomyopathy and further characterize the cons

93 (2)(+) responsiveness in a model of familial dilated cardiomyopathy and improve cardiac function and

94 nd inflammation in patients with nonischemic dilated cardiomyopathy and inflammatory cardiomyopathy (

95 Here the authors focus on genetics of dilated cardiomyopathy and provide a roadmap for impleme

96 Patients with ischemic or dilated cardiomyopathy and reduced left ventricular ejec

97 characterized by fibrosis: human and murine dilated cardiomyopathy and repaired tetralogy of Fallot.

98 relationship of ivabradine in children with dilated cardiomyopathy and symptomatic chronic HF.

99 ubunit-encoding Kcne2 (Kcne2(CS-/-) ) causes dilated cardiomyopathy and terminal HF (median longevity

100 the titin and sarcomere variants that cause dilated cardiomyopathy and the desmosomal variants that

101 istry than in large cohorts of patients with dilated cardiomyopathy and TTNtv cardiomyopathy and not

102 Moreover, TTNtv associated with dilated cardiomyopathy are estimated to be present in 0.

103 ause an autosomal dominant inherited form of dilated cardiomyopathy associated with cardiac conductio

104 scular dystrophy (DMD) develop a progressive dilated cardiomyopathy characterized by inflammatory cel

105 of cardiovascular mortality compared with a dilated cardiomyopathy control group (odds ratio, 1.10 [

106 Patients with myocarditis or dilated cardiomyopathy develop autoantibodies to SERCA2a

107 in quality control, we used a mouse model of dilated cardiomyopathy driven by cardiac restricted over

108 cardioverter defibrillator in patients with dilated cardiomyopathy for the primary prevention of sud

109 he decedents with an antemortem diagnosis of dilated cardiomyopathy fulfilled definite 2010 Task Forc

110 opathy Precision Medicine Study is that most dilated cardiomyopathy has a genetic basis.

111 ified in association with ASD and late-onset dilated cardiomyopathy in a large, multi-generational fa

112 verse cardiovascular events in patients with dilated cardiomyopathy in a multicenter setting as part

113 and clinically relevant treatment to rescue dilated cardiomyopathy in patients with DMD.

114 isease models of myocardial infarction (MI), dilated cardiomyopathy induced via aortic banding, and s

115 Because a common finding in dilated cardiomyopathy is a reduction in the myofilament

116 Dilated cardiomyopathy is associated with increased risk

117 Heart failure due to dilated cardiomyopathy is frequently caused by myocardit

118 involved in viral RNA replication.IMPORTANCE Dilated cardiomyopathy is the most common indication for

119 ual myocardial injury whereas others develop dilated cardiomyopathy is unclear.

120 LN R14del mutation are at risk of developing dilated cardiomyopathy or arrhythmogenic right ventricul

121 tes myocardial inflammation with established dilated cardiomyopathy or hypokinetic nondilated phenoty

122 heart failure instead of the commonly taught dilated cardiomyopathy pathway.

123 with cardiac ribosome occupancy levels of 30 dilated cardiomyopathy patients demonstrates that these

124 Methods Thirty-four dilated cardiomyopathy patients, 30 ischemic cardiomyopa

125 an tissues, and upregulated in the hearts of dilated cardiomyopathy patients.

126 idence of heart failure hospitalization than dilated cardiomyopathy patients.

127 milar cardiovascular risk when compared with dilated cardiomyopathy patients.

128 arrhythmias in LVNC patients were similar to dilated cardiomyopathy patients.

129 ssion are each important determinants of the dilated cardiomyopathy phenotype and are controlled by g

130 d that the knockout strategy ameliorates the dilated cardiomyopathy phenotype in vitro.

131 The hypothesis of the Dilated Cardiomyopathy Precision Medicine Study is that

132 ere calculated in parents of 128 present-day dilated cardiomyopathy probands with TTNtv using the rev

133 ntractions are known to trigger a reversible dilated cardiomyopathy referred as arrhythmia-induced ca

134 Consecutive patients with dilated cardiomyopathy referred for cardiac magnetic res

135 ral functions involved in the development of dilated cardiomyopathy remain unclear.

136 ome occupancy in the hearts of patients with dilated cardiomyopathy suggested the same posttranscript

137 r envelope gene SYNE1 in a child with severe dilated cardiomyopathy that underwent transplant, as wel

138 -nine patients with ischemic or non-ischemic dilated cardiomyopathy undergoing prophylactic ICD impla

139 y shows that different gene mutations induce dilated cardiomyopathy via diverse cellular pathways.

140 tal RNA from cardiac tissue of patients with dilated cardiomyopathy was extracted, and sequences corr

141 Ang II levels in explanted human hearts with dilated cardiomyopathy were elevated despite ACE inhibit

142 ailure medications in patients with previous dilated cardiomyopathy who were now asymptomatic, whose

143 Patients with dilated cardiomyopathy whose symptoms and cardiac functi

144 Many patients deemed to have recovered from dilated cardiomyopathy will relapse following treatment

145 iffered considerably: loss of titin leads to dilated cardiomyopathy with combined systolic and diasto

146 in the mouse heart for the first time during dilated cardiomyopathy with heart failure.

147 The primary endpoint was a relapse of dilated cardiomyopathy within 6 months, defined by a red

148 that often presents as heart failure due to dilated cardiomyopathy years after anthracycline exposur

149 earts of transplant recipients (ischemic and dilated cardiomyopathy), and from nonused donor hearts.

150 164 ischemic cardiomyopathy, 150 nonischemic dilated cardiomyopathy), the mean left ventricular eject

151 ard ratios were 1.15 (95% CI, 1.14-1.17) for dilated cardiomyopathy, 1.09 (95% CI, 1.06-1.12) for hyp

152 Of these, 481 were diagnosed with dilated cardiomyopathy, 246 had hypertrophic cardiomyopa

153 ARVC, 9 (25%) of 36; LQTS, 48 (20%) of 238; dilated cardiomyopathy, 5 (9%) of 58; and HCM, 28 (8%) o

154 this critical overlap region associated with dilated cardiomyopathy, A277V, will alter Tpm binding an

155 is patients, 11 ischemic heart disease, nine dilated cardiomyopathy, and 11 nonfailing donors.

156 ble cases by cardiac MRI, 3 were found to be dilated cardiomyopathy, and 2 were found to be end-stage

157 120 patients with ischemic, 60 patients with dilated cardiomyopathy, and 30 patients with normal LVEF

158 n of function occurs in 20% of children with dilated cardiomyopathy, and 40% die or undergo transplan

159 uctive cardiomyopathy, 131 genes/17 ncRNA in dilated cardiomyopathy, and 51 genes/5 ncRNA in ischemic

160 ertrophic obstructive cardiomyopathy, 151 in dilated cardiomyopathy, and 55 in ischemic cardiomyopath

161 ive cardiomyopathy, ischemic cardiomyopathy, dilated cardiomyopathy, and 9 control patients with nonf

162 nalysis of fibrotic scarring in non-ischemic dilated cardiomyopathy, and its relationship to electric

163 the regulation of development, ischemic and dilated cardiomyopathy, and myocardial infarction.

164 ntified in a pediatric patient with sporadic dilated cardiomyopathy, and we determined a molecular me

165 ecellularized ECM resulting from ischemic or dilated cardiomyopathy, as well as from mouse infarcted

166 in the hearts of individuals with idiopathic dilated cardiomyopathy, as well as the hearts of patient

167 are frequent causes of acute myocarditis and dilated cardiomyopathy, but an effective antiviral thera

168 gene mutations are a known cause of familial dilated cardiomyopathy, but the precise mechanisms trigg

169 age-dependent cardiac phenotypes, including dilated cardiomyopathy, cardiac conduction disturbance,

170 In pediatric patients with dilated cardiomyopathy, compared with dimension and area

171 involvement is characterized by progressive dilated cardiomyopathy, decreased fractional shortening

172 Ntv) are the most prevalent genetic cause of dilated cardiomyopathy, found in <=25% of familial cases

173 is the major cause of death and manifests as dilated cardiomyopathy, heart failure, arrhythmias, and

174 c cardiomyopathy, ischemic heart disease, or dilated cardiomyopathy, in comparison to nonfailing hear

175 an be a precursor of chronic myocarditis and dilated cardiomyopathy, leading causes of heart transpla

176 Compared with other forms of dilated cardiomyopathy, mutations in LMNA are responsibl

177 In patients with dilated cardiomyopathy, myocardial Gal-3 expression corr

178 in gene are associated with hypertrophic and dilated cardiomyopathy, respectively.

179 with a subsequent cardiomyopathy, especially dilated cardiomyopathy, starting already at mildly eleva

180 if concentric hypertrophy does progress to a dilated cardiomyopathy, such a transition would occur ov

181 ted from a patient suffering from idiopathic dilated cardiomyopathy, suggesting that such mutant viru

182 s large multinational study of patients with dilated cardiomyopathy, the presence of LGE showed stron

183 se in risk with higher BMI, particularly for dilated cardiomyopathy, where a hazard ratio of 4.71 (95

184 Mutations in A-type nuclear lamins cause dilated cardiomyopathy, which is postulated to result fr

185 sed cardiac microtissue contraction, whereas dilated cardiomyopathy-associated variants decreased con

186 sed cardiac microtissue contraction, whereas dilated cardiomyopathy-associated variants decreased con

187 ult cardiac vasculature and thereby prevents dilated cardiomyopathy-like defects.

188 TTN) are the leading known cause of familial dilated cardiomyopathy.

189 lly induced trophic signaling and eventually dilated cardiomyopathy.

190 s with regard to monitoring for (late-onset) dilated cardiomyopathy.

191 viral proteinase 2A in cases of unexplained dilated cardiomyopathy.

192 (TTNtv) are the most common genetic cause of dilated cardiomyopathy.

193 duced myocyte complex N-glycosylation causes dilated cardiomyopathy.

194 scle cell leads to myocyte instability and a dilated cardiomyopathy.

195 ative relatives, and 9 with noncompaction or dilated cardiomyopathy.

196 hmogenic right ventricular cardiomyopathy or dilated cardiomyopathy.

197 to one that leads to chronic remodeling and dilated cardiomyopathy.

198 (TTNtv) are the commonest cause of heritable dilated cardiomyopathy.

199 ation (Id cDKOs), which develops adult-onset dilated cardiomyopathy.

200 induced hypertrophy and in human hearts with dilated cardiomyopathy.

201 se-specific considerations were required for dilated cardiomyopathy.

202 ular arrhythmias in patients with idiopathic dilated cardiomyopathy.

203 t ventricle (LV) ultimately transitions to a dilated cardiomyopathy.

204 g heart rate of children with chronic HF and dilated cardiomyopathy.

205 dilation, and a fourth-degree relative with dilated cardiomyopathy.

206 understanding of the genetic architecture of dilated cardiomyopathy.

207 d on the uncharacterized lncRNA GATA6-AS1 in dilated cardiomyopathy.

208 alterations of signaling pathways leading to dilated cardiomyopathy.

209 respiratory failure, and in the older cases, dilated cardiomyopathy.

210 nd in heart tissue from patients with MI and dilated cardiomyopathy.

211 We find that a dilated central region has the strongest impact in thick

212 er medical record based on their most recent dilated comprehensive eye examination by a primary eye c

213 roduce a network architecture based on using dilated convolutions to capture features at different im

214 cluding wide residual networks, dropouts and dilated convolutions.

215 -myosin variants cause hypertrophic (HCM) or dilated (DCM) cardiomyopathy by disrupting sarcomere con

216 Dilated distal vessels extending to the pleura and fissu

217 The right ventricle was dilated during resuscitation from cardiac arrest caused

218 Fluorescein angiography showed bilateral dilated, ectatic capillaries and late phase dye leak.

219 ay, resulting in a buildup of virions within dilated ER vesicles.IMPORTANCE In humans, symptoms of RV

220 eported an eye care visit and 21% reported a dilated examination in the past 12 months.

221 All participants underwent a dilated examination to determine the grade of DR.

222 tion, clinical information, and time to last dilated eye exam.

223 significantly associated with an increase in dilated eye examination rates within the first 2 years a

224 th incomes below 138% of the FPL receiving a dilated eye examination within the past year due to Medi

225 wer odds of reporting ever having received a dilated eye examination.

226 rty-eight percent reported ever having had a dilated eye examination.

227 atients with diabetes reported ever having a dilated eye examination; only 39.62% reported receiving

228 ecessary to increase and sustain the rate of dilated eye examinations among diabetic populations.

229 ponders who were asked about the presence of dilated eye examinations from years before and after Med

230 yndrome, and five of six individuals who had dilated eye exams had retinal pigmentary abnormalities.

231 We found that pupil size dilated following these surprising events, in the absenc

232 42 cases (16.7%), the residual iris aperture dilated from 36.6 +/- 15.4 mm(2) preoperatively to 61.1

233 Agreement between TRI and dilated fundus examination (DFE) findings was determined

234 Dilated fundus examination and fundus photography were e

235 t a comprehensive eye examination, including dilated fundus examination and imaging.

236 ects were obtained and cross-referenced with dilated fundus examination findings with regard to DR se

237 went visual acuity (VA) testing, refraction, dilated fundus examination fluorescein angiography (FA)

238 Dilated fundus examination showed an inferior bullous RD

239 Dilated fundus examination was unremarkable.

240 urement of intraocular pressure, gonioscopy, dilated fundus examination, and fluorescein angiography

241 Avascular retina was identified on dilated fundus examination, fluorescein angiography, or

242 isual acuity, slit-lamp biomicroscopy, and a dilated fundus examination.

243 ma, emphasizing the importance of a detailed dilated fundus examination.

244 distance and near visual acuity evaluation; dilated fundus examination; OCT with 12 x 6-mm thickness

245 by eliminating formal visual acuity (VA) and dilated fundus examinations (DFEs) were assessed for est

246 comprehensive ocular examination, including dilated fundus photography.

247 osis, including the majority of those with a dilated funduscopic examination in the past year.

248 Dilated funduscopic examinations were performed by an op

249 sual acuity, IOP, slit lamp examination, and dilated funduscopy as well as comparative measurements b

250 d distended duodenum filled with fluid and a dilated gallbladder containing dilute bile with high bic

251 ography of the abdomen at admission revealed dilated gas-filled small-bowel loops, suggestive of obst

252 The adult mutant mice developed dilated hearts and showed significantly shorter life spa

253 hosphorylation is significantly decreased in dilated human hearts, supporting the notion that modulat

254 Indocyanine green angiography revealed dilated hyperpermeable choroidal vasculature on the nasa

255 -year follow-up and divided into categories: dilated, hypertrophic, alcohol/drug-induced, and other.

256 ction of cardiac arrest, the right ventricle dilated in all groups (p < 0.01 for all).

257 lammation, basal zone hyperplasia (BZH), and dilated intercellular spaces, and the underlying process

258 An abdominal CT demonstrated a dilated intrahepatic biliary tree with left proximal int

259 xtracellular matrix (ECM) abnormalities, and dilated intrascleral veins, yet, no dilation of arteries

260 However, the right ventricle was dilated, irrespective of the cause of arrest, and diagno

261 se with interval cardiovascular events and a dilated LV (increased LV end-diastolic volume [EDV] inde

262 as minimal, and few participants developed a dilated LV.

263 m in diameter, was inserted to the straight, dilated main duct and plastic stent(s) were inserted to

264 multilocular cysts of the pancreas and a non-dilated main pancreatic duct (<5 mm).

265 larger than 4 cm (P < 0.001), presence of a dilated main pancreatic duct of over 4 mm (P < 0.001), h

266 of 88 patients with chronic pancreatitis, a dilated main pancreatic duct, and who only recently star

267 e because of mechanical interaction with the dilated main pulmonary artery (MPA).

268 e fully convolutional network (FCN), SegNet, Dilated-Net, original U-Net, and Faster R-CNN models and

269 Transillumination revealed fully dilated, non-reactive RE pupil, clear lens and tubular r

270 diogram study, and those with a diagnosis of dilated, noncompaction, alcoholic, or toxic cardiomyopat

271 those younger than 10 years, undergo annual dilated ophthalmic examination, looking for evidence of

272 aire and underwent a single study visit with dilated ophthalmic examination, OCT angiography (OCTA),

273 intraocular pressure monitoring, slit-lamp, dilated ophthalmoscopy, and fundus examinations.

274 ntraocular pressure measurement, gonioscopy, dilated ophthalmoscopy, and standard automated perimetry

275 er PD (ASA 3 OR 0.59 [0.44-0.80]), whereas a dilated pancreatic duct (>3 mm) and pancreatic ductal ad

276 Dilated peripheral vessels were present in 21/33 (63.6%)

277 abnormal lung and the presence and extent of dilated peripheral vessels) and perfusion defects on DEC

278 tation of mitochondrial cardiomyopathy, with dilated phenotype and pathologic evidence of biventricul

279 In patients with bicuspid aortic valve and dilated proximal ascending aorta, we sought to assess (1

280 valence of LMCA extrinsic compression from a dilated pulmonary artery (PA) in patients with PAH and a

281 ed pupils, eyes from A. irradians with fully dilated pupils provide approximately three times the sen

282 mined using 12x to 16x magnification through dilated pupils with ocular saccades before an injection.

283 y retention, dry mouth, lack of tears, fixed dilated pupils, and diffuse anhidrosis 7 days after a fe

284 ncreased NO bioavailability that dynamically dilated resistance vessels in vivo under basal condition

285 ning for diabetic retinopathy include annual dilated retinal examinations to detect proliferative ret

286 ze with systolic dysfunction; and pattern 4, dilated RV systolic dysfunction.

287 al RV size and systolic function; pattern 2, dilated RV with preserved systolic function; pattern 3,

288 tissue included acinar cells and cystically dilated secretory ducts without islets of Langerhans.

289 uman hepatic vascular cavernoma phenotype of dilated sinusoidal capillaries with defective branching

290 7%, 32 of 37), and 54% (20 of 37) revealed a dilated sludge-filled gallbladder, suggestive of bile st

291 elevated risk of cardiomyopathy, especially dilated, starting at levels considered normal (BMI, 22.5

292 layed conductive, constricted, occluded, and dilated states, in qualitative agreement with the well-d

293 Participants underwent dilated stereo-digital fundus photography graded accordi

294 tion, a neurovascular dysplasia resulting in dilated, thin-walled vessels that tend to rupture, incre

295 lso revealed a left dysplastic kidney with a dilated, tortuous ureter.

296 This disease manifested as a massively dilated urinary bladder, or megabladder, with disrupted

297 CS includes dilated vasculature, marked cardiac hypertrophy, and oth

298 ive hamartomas containing abnormal tortuous, dilated vessels prominent in the forelimbs.

299 Dilated vessels were seen in 41 cases (85%), with 38 (78

300 The right ventricle was dilated without evidence of right ventricular outflow tr