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1 ative distinction of CH3-group oxidation and dioxygenation.
2 actone from the ring fission product of 5NSA dioxygenation.
3 itrosyl hemoglobin rather than destroy it by dioxygenation.
4 ccurs most effectively via O(2)-dependent NO dioxygenation.
5 m in which peroxidative cleavage precedes AA dioxygenation.
6 were kinetically competent to participate in dioxygenation.
7 tive cleavage is not the initiating event in dioxygenation.
8 m in which peroxidative cleavage precedes AA dioxygenation.
9  of an electrochemical TEMPO-mediated allene dioxygenation.
10 o, alter the availability of O(2) for Nt-Cys dioxygenation.
11 urement of oxidase and myeloperoxidase (MPO) dioxygenation activities in whole blood.
12 g the molecular mechanisms involved in thiol dioxygenation and sets the stage for exploration of the
13                  Through N-terminal cysteine dioxygenation and the N-degron pathway, ADO regulates th
14 anism by which HbN recycles itself during NO dioxygenation and the reductase that participates in thi
15 mes are remarkable in that they catalyze two dioxygenations and two cyclizations of the native substr
16 n experiment and theory showed that rates of dioxygenation are determined by the enzymatic O2 activat
17 he oxidation of fluoranthene is initiated by dioxygenation at the C-1,2, C-2,3, and C-7,8 positions.
18             It also suggests that tryptophan dioxygenation by MPO and superoxide may occur during inf
19 isomers, 2,6-dinitrotoluene, and naphthalene dioxygenation by NBDO varied considerably, the correlati
20                             Because aromatic dioxygenation by nonheme iron dioxygenases is frequently
21 o mimic the bioinorganic chemistry of indole dioxygenation by TDO and IDO, challenging the widely acc
22 netically competent to participate in the 15-dioxygenation event.
23 aromatic ring activation (monooxygenation vs dioxygenation), (ii) the type of EDO enzymes, and (iii)
24 or biodegradation pathways initiated via (i) dioxygenation, (ii) reduction, and (iii) CH3-group oxida
25 ncomitantly, the physiological role of thiol dioxygenation in prokaryotes and eukaryotes has been stu
26 ysis of OPP's inhibition of arachidonic acid dioxygenation indicated mixed inhibition toward the ferr
27                                     Cysteine dioxygenation is catalyzed in mammalian cells by 2-amino
28                            The major site of dioxygenation is the C-2,3 dioxygenation route, which co
29                                        Thiol dioxygenation is the initial oxidation step that commits
30 ited reactivity toward nitric oxide (NO) via dioxygenation [k(NOD)(RcoM) = 6 to 8 x 10(6) M(-1)s(-1)
31 and CO binding kinetics, and steady-state NO dioxygenation kinetics.
32 om(-1) s(-1) indicate an extremely efficient dioxygenation mechanism.
33 utes a novel approach to the challenging 1,6-dioxygenation motif.
34 oboglobin models to promote the nitric oxide dioxygenation (NOD) reaction similar to oxy-hemes.
35  Proteomic analysis revealed that tryptophan dioxygenation occurs on the abundant neutrophil protein
36         Since it is unlikely that 11- and 15-dioxygenation occurs via different mechanisms, our findi
37                       A mild, metal-free 1,2-dioxygenation of 1,3-dienes using TEMPO and carboxylic a
38 poxygenase-1 (15-hLO-1), which catalyzes the dioxygenation of 1,4-cis,cis-pentadiene-containing polyu
39                          We investigated the dioxygenation of a series of nitroaromatic compounds to
40                                  Two in two: Dioxygenation of alkenyl boronic acids has been achieved
41 vity under mild conditions was validated for dioxygenation of alkynes to highly demanding labile synt
42 g biologically relevant oxidation reactions: dioxygenation of an indole derivative, and H-atom abstra
43                                              Dioxygenation of aromatic rings is frequently the initia
44 obalt-catalyzed regioselective unsymmetrical dioxygenation of gem-difluoroalkenes using phenols and m
45 -formation reaction, while also preventing a dioxygenation of its cysteinate substrate?
46 orresponding isotope effects, especially for dioxygenation of N-substituted, aromatic contaminants, a
47                                          The dioxygenation of nitric oxide by oxyheme in globin prote
48 rtmentalization of hemoglobin and subsequent dioxygenation of nitric oxide may explain the vascular c
49                   Our study illustrates that dioxygenation of nitroaromatic contaminants exhibits a l
50         The corresponding (13)C-KIEs for the dioxygenation of nitrobenzene and 2-nitrotoluene were 1.
51                                              Dioxygenation of nitrobenzene to catechol and 2-nitrotol
52 periment and theory also agreed well for the dioxygenation of nitrobenzene, which was associated with
53 os for the competing CH3-group oxidation and dioxygenation of nitrotoluenes by MnO4(-) were obtained
54 ansport and uptake processes and larger than dioxygenation of other aromatic hydrocarbons.
55  15-lipoxygenase (15-LO) participates in the dioxygenation of polyenoic fatty acids.
56 ant and mammalian lipoxygenases catalyze the dioxygenation of polyunsaturated fatty acids, which cont
57 iquitous family of enzymes that catalyze the dioxygenation of polyunsaturated fatty acids.
58 LOXs) catalyze the regio- and stereospecific dioxygenation of polyunsaturated membrane-embedded fatty
59  alpha,beta-unsaturated lactone derived from dioxygenation of pyrene at an apical ring, 2H-naphtho[2,
60 ae alkaloids, all based on the key enzymatic dioxygenation of suitable aromatic precursors.
61  the parent cysteamine-bound complexes - the dioxygenation of the chalcogen atoms was observed.
62       Similar observations were made for the dioxygenation of these substrates by 2NTDO.
63     Here, we systematically investigated the dioxygenation of two nitroaromatic compounds (nitrobenze
64 ed in the plastid envelope and catalyzed the dioxygenation of unsaturated membrane fatty acids, leadi
65 riants are created that exclusively catalyze dioxygenation or sequential monooxygenation chemistry.
66        To better define the Cygb-mediated NO dioxygenation process in vascular smooth muscle cells (S
67 19Z-hexaenoic acid (isomer IV), and a double dioxygenation product 10S,17S-dihydroxy-docosa-4Z,7Z,11E
68  observed consistently in the substrates and dioxygenation products.
69 kdown resulted in a small decrease in the NO dioxygenation rate (V(NO)), while depletion of ascorbate
70               Haptoglobin does not change NO dioxygenation rates of Hb; rather, the large size of the
71             The structure indicates that the dioxygenation reaction is initiated by a direct attack o
72 olysis occurs secondarily to the accelerated dioxygenation reaction of plasma oxyhemoglobin with endo
73  is inactivated by cell-free hemoglobin in a dioxygenation reaction that also oxidizes hemoglobin to
74       Moreover, HMS catalyzes a very similar dioxygenation reaction to that of HPPD, adding the secon
75 ially met-R state Hb that arises from the NO dioxygenation reaction.
76 with the ability to perform precise mono- or dioxygenation reactions on a variety of substrates.
77                  Alkene aminooxygenation and dioxygenation reactions that result in carbonyl products
78 Individual reduction, ligand binding, and NO dioxygenation reactions were examined at 20 degrees C, w
79 he endothelium, via accelerated nitric oxide dioxygenation reactions with free plasma hemoglobin.
80 The major site of dioxygenation is the C-2,3 dioxygenation route, which consists of 18 enzymatic step
81                          The C-1,2 and C-2,3 dioxygenation routes degrade fluoranthene via fluorene-t
82 te, thereby preventing the possible thiolate dioxygenation side reaction.
83 enantioselective alkene aminooxygenation and dioxygenation that directly provide enantioenriched 2-fo
84 ygenase (IDO), the founding members, promote dioxygenation through a two-step monooxygenation pathway
85 s the rate law for 3,5-di-tert-butylcatechol dioxygenation when one begins with Pierpont's [VO(DBSQ)(
86  86-89% of 2,4-DNT transformation was due to dioxygenation while TNT was mostly reduced and 2,6-DNT r
87 ce, this is an experimental observation of a dioxygenation with O(2) mediated by a cobalt center in a
88 sotope effects for aniline and diphenylamine dioxygenation with those from abiotic oxidation by manga
89  a framework to explain cofactor-independent dioxygenation within a protein architecture generally em