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1 egistration (central-peripheral rivalry-type diplopia).
2 deviations, degree of muscle underaction and diplopia.
3 were primarily for acute- or subacute-onset diplopia.
4 st to characterize the type and cause of the diplopia.
5 function consisting of central nystagmus and diplopia.
6 necessary but is not sufficient for CPR-type diplopia.
7 may explain subjective reports of monocular diplopia.
8 ta) at distance and a phoria at near without diplopia.
9 were subjective and objective improvement of diplopia.
10 event long-term sequelae of enophthalmos and diplopia.
11 /12.5-20/50), and 34 patients (81%) reported diplopia.
12 table suture was performed to treat vertical diplopia.
13 ure was defined as worsening or no change in diplopia.
14 ltered consciousness followed by symptomatic diplopia.
15 c correction in adults based on the cause of diplopia.
16 a translocated inferiorly, causing binocular diplopia.
17 The patient also complained of diplopia.
18 potential cause of strabismus and binocular diplopia.
19 reased risk for postoperative strabismus and diplopia.
20 disruption of central fusion, and monocular diplopia.
21 even enhanced VFV may be inadequate to avert diplopia.
22 report of fusion, until the subject reported diplopia.
23 strabismus, ophthalmoplegia, and paradoxical diplopia.
24 tal septum intact can minimize postoperative diplopia.
25 gist and oculoplastic surgeon to correct the diplopia.
26 nt for 50% of patients (36/72) with baseline diplopia.
27 associated with increased odds of developing diplopia.
28 No patient had new-onset primary gaze diplopia.
29 positions, and development of postoperative diplopia.
30 seminoma presented with vertigo, ataxia, and diplopia.
31 tients (all Knosp 3-4) experienced transient diplopia.
32 ficulties, particularly due to the impact of diplopia.
33 rs to offer minimal long-term improvement in diplopia.
34 n for papilledema, visual field defects, and diplopia.
35 CPR-type diplopia and those without CPR-type diplopia.
36 %) had CPR-type diplopia and 37 (66%) had no diplopia.
37 56 patients were excluded for other types of diplopia.
38 nics to determine the prevalence of CPR-type diplopia.
39 tion when other causes did not fully explain diplopia.
40 etermine clinical associations with CPR-type diplopia.
41 e clinical findings associated with CPR-type diplopia.
42 M, 25 had symptomatic diplopia and 25 had no diplopia.
43 CI, 38.6%-77.7%) of ED visits, primarily for diplopia.
44 Frequency, type, and cause of diplopia.
45 plopia vs patients with ERM without CPR-type diplopia.
46 an epiretinal membrane (ERM) and presenting diplopia.
47 and where other causes did not fully explain diplopia.
48 mpleted the Diplopia Questionnaire to assess diplopia.
49 ared to have central-peripheral rivalry-type diplopia), 1 (4%) optical/refractive error (monocular di
50 ntraumatic and traumatic eye conditions were diplopia (11.69%) and closed fracture of the orbital flo
51 ad a relatively high incidence of persistent diplopia (12%) and corneal edema (20%), although half of
53 , 1 (4%) optical/refractive error (monocular diplopia), 2 (8%) mixed retinal misregistration (central
54 order of binocular vision (strabismus, 2.3%; diplopia, 2.2%; amblyopia, 0.9%; and nystagmus, 0.2%).
59 sole cause (central-peripheral rivalry-type diplopia), 7 (28%) strabismus (1 of 7 initally appeared
62 onsidered in any patient with vision loss or diplopia accompanied by neurologic symptoms and in the a
75 s surgery in patients over 80 years resolves diplopia and improves binocular alignment and stereopsis
76 ptoms of tearing, lagophthalmos, ptosis, and diplopia and measured margin-to-reflex distance (MRD) 1
80 nts had a monocular suppression or alternate diplopia and suppression at sensory tests (BSGs and WFDT
82 characteristics, treatment, and outcomes of diplopia and/or strabismus following plaque brachytherap
83 egistration (central-peripheral rivalry-type diplopia) and strabismus, and for 4 (16%) diplopia cause
87 of binocular vision (strabismus, amblyopia, diplopia, and nystagmus) may have on musculoskeletal inj
89 BFM-90 reduced the recurrence of strabismus, diplopia, and proptosis, but did not correct deficits in
90 spect to proptosis, Clinical Activity Score, diplopia, and quality of life than placebo; serious adve
95 anial nerve [CN] III, IV, VI, and VII palsy; diplopia; and optic neuritis) and new diagnoses of other
96 c symptoms and signs such as vision loss and diplopia are common in patients with stroke, patients ar
98 symptoms (eg, isolated vertigo, dysarthria, diplopia) are not consistently classified as transient i
100 We defined central-peripheral rivalry-type diplopia as presenting symptomatic diplopia with evidenc
101 noptophore; CPR-type diplopia was defined as diplopia associated with evidence of retinal misregistra
105 0.05 to 1.1, P = .01) than patients without diplopia, but similar aniseikonia (Awaya new aniseikonia
106 e metamorphopsia than those without CPR-type diplopia, but there is considerable individual variabili
107 fy the efficacy of monovision correction for diplopia by measuring the functional impact on vision-sp
113 surgeons treating esotropia or exotropia for diplopia control or reconstructive goals were collected.
114 the highest success rate was for motor plus diplopia criteria (67%) and the lowest success rate was
115 defining success as either meeting motor and diplopia criteria or showing improvement in HRQOL beyond
119 opters by simultaneous prism cover test; (2) diplopia criteria, none or only rare in primary distance
120 18%) were classified as failure by motor and diplopia criteria, with 39 of 40 able to exceed Adult St
122 ifests at an older age than KSS (p = 0.003), diplopia does not correlate with disease duration (p = 0
124 type of strabismus associated with binocular diplopia due to glaucoma surgery was hypertropia (10/11
127 alysis with prominent bulbar palsies such as diplopia, dysarthria, dysphonia, and dysphagia that woul
128 frequency): hemiparesis, vertigo/dizziness, diplopia, dysarthria, nystagmus, nausea/vomiting, head p
131 tal reconstruction in an effort to alleviate diplopia, enophthalmos, orbital dystopia, and extraocula
135 icle is to report the incidence and cause of diplopia following cataract surgery and laser in-situ ke
140 st patients requiring surgery for strabismic diplopia following teprotumumab achieve good outcomes wi
145 ot statistically different, for treatment of diplopia from exotropia (64%, 95% CI 43%-80%; P = .184).
146 of dichoptic training with the computer game Diplopia Game (Vivid Vision) run in the Oculus Rift OC D
149 nd orbital blowout fractures associated with diplopia had the lowest satisfaction rates, 55 and 8%, r
153 s with binocular vision will be sensitive to diplopia in any gaze direction; in such cases, the conse
154 To report the prevalence, type, and cause of diplopia in medically and surgically treated patients wi
155 d this suggests a mechanism that can explain diplopia in patients made exotropic after surgery for es
157 omes were as follows: prevalence of CPR-type diplopia in patients with ERM seen in retinal disease cl
159 Ground-in or Fresnel prism may alleviate diplopia in relatively small vertical deviation; however
160 To determine the prevalence of CPR-type diplopia in retinal disease clinic patients with ERM and
161 e most common diagnosis in both settings was diplopia (International Classification of Diseases, Nint
164 d for LASIK procedures, the leading cause of diplopia is decompensation of pre-existing strabismus.
168 in visual field defects, visual acuity, and diplopia is typically observed after emergent applicatio
169 % CI, 0.29-0.40), intermittent or inconstant diplopia/large proptosis (0.43; 95% CI, 0.36-0.49), no d
170 arge proptosis (0.43; 95% CI, 0.36-0.49), no diplopia/large proptosis (0.46; 95% CI, 0.40-0.52), and
171 d in the most severe disease state (constant diplopia/large proptosis) with 0.30 (95% CI, 0.24-0.36),
173 S) as the cause of chronic or acute acquired diplopia may avert neurologic evaluation and imaging in
178 ts with retinal misregistration and CPR-type diplopia (minimum frequency of "sometimes" at distance a
179 ing diplopia may have 1 of several causes of diplopia, most commonly retinal misregistration (central
180 elevated intraocular pressure, tube erosion, diplopia, motility disturbances, and corneal decompensat
185 ient or persistent monocular ghost images or diplopia occurred in 10 of 178 eyes (5.6%), sometimes re
189 ia occurred in 11.2% of patients, persistent diplopia occurred in only 1.9% of patients and was treat
191 of TED, including soft tissue inflammation, diplopia, ocular motility restriction, and appearance.
192 eek 24), a diplopia response (a reduction in diplopia of >=1 grade), and the mean change in overall s
193 cture (OR, 9.1; 95% CI, 2.8-30.0; P = .002), diplopia on primary gaze (OR, 6.7; 95% CI, 1.7-25.1; P =
195 female, three male) presented with episodic diplopia or facial paresthesias with subsequent brainste
197 All patients had preoperative monocular diplopia or unstable vision attributable to the subluxat
198 loss (OR, 2.72; 95% CI, 1.25-5.75; P = .01), diplopia (OR, 3.33; 95% CI, 1.00-10.29; P = .04), headac
202 ophthalmoplegia and nystagmus, resulting in diplopia, oscillopsia, blurred visual, loss of stereopsi
203 mptoms of new onset lid swelling (p < 0.01), diplopia (p < 0.01), flashing lights (p = 0.02), or droo
204 eported in the IIG group: 2% versus 10% with diplopia (P = 0.039) and 3% versus 10% with enophthalmos
207 (7 of 31; 95% CI, 10% to 41%), with CPR-type diplopia present in 16% (5 of 31; 95% CI, 5% to 34%).
211 with TED and clinical activity score >=4, +/-diplopia/proptosis) and refined using interviews with US
212 ated right retro-orbital pain and later with diplopia, ptosis, 6th nerve and pupil-sparing partial 3r
214 as diplopia rated "never" or "rarely" on the diplopia questionnaire for reading and straight-ahead di
215 tionnaire (100-0, best to worst HRQOL) and a diplopia questionnaire in a clinical practice before pri
216 n of ascertaining patient symptoms using the Diplopia Questionnaire may be useful in these patients.
218 tically significant 58.6% improvement in the Diplopia Questionnaire score in our patients (P < .0001)
220 primary outcome: Based on the results of the Diplopia Questionnaire, 85% of patients experienced sign
221 diplopic for distance and reading, using the Diplopia Questionnaire, at an outcome examination as clo
227 ) ambulatory and 49790 (95% CI, 38318-61262) diplopia-related ED visits occurred annually; 12.3% of a
228 ambulatory setting, but approximately 16% of diplopia-related ED visits resulted in a stroke or trans
233 patients with a final CAS of 0 or 1, higher diplopia responder rate, and a larger improvement in the
234 change in proptosis (-2.82 mm vs. -0.54 mm), diplopia response (68% [19 of 28] vs. 29% [8 of 28]), an
235 al visits (from baseline through week 24), a diplopia response (a reduction in diplopia of >=1 grade)
237 on (11 for proptosis change [n = 419], 4 for diplopia response [n = 125], and 2 teprotumumab [n = 79]
239 se responders, proptosis, CAS of 0 or 1, and diplopia responses were maintained in 29 of 32 patients
242 lides at 5 and 10 degrees), and cause of any diplopia (retinal misregistration vs strabismus vs optic
243 ifference was found in proptosis (P = 0.07), diplopia score (P = 0.4), or duration of TED (P = 0.4) b
246 ivity score (CAS), proptosis, and the Gorman diplopia score were reviewed at baseline, at the end of
249 30 (95% CI, 0.24-0.36), followed by constant diplopia/small proptosis (0.34; 95% CI, 0.29-0.40), inte
250 , 0.40-0.52), and intermittent or inconstant diplopia/small proptosis (0.52; 95% CI, 0.45-0.58).
257 pen globes and open wounds of ocular adnexa, diplopia, superficial corneal and/or conjunctival injuri
258 rmance of activities at near after esotropia-diplopia surgery (odds ratio 3.0, 95% CI 1.5-6.4; P = .0
261 experienced occasional episodes of transient diplopia that developed while reading in the evening.
262 ival chemosis, pain on eye movement, minimal diplopia, the usual absence of proptosis, and general pr
265 evalence of monocular diplopia and binocular diplopia unrelated to glaucoma surgery was similar among
267 perceived visual quality and ocular injury, diplopia, visual performance, and blast exposure charact
268 fered between patients with ERM and CPR-type diplopia vs patients with ERM without CPR-type diplopia.
274 totype-frame test and synoptophore; CPR-type diplopia was defined as diplopia associated with evidenc
275 sk model for the prediction of postoperative diplopia was derived using a development dataset (70% of
281 eoperative visual acuity better than 20/400, diplopia was reported at the first postoperative visit b
283 d 350 mm(2) models had similar outcomes, but diplopia was significantly associated with the 350 mm(2)
286 visits in the United States by patients with diplopia were analyzed in this prespecified secondary an
288 The factors that predicted postoperative diplopia were: age at injury, preoperative enophthalmos,
291 achusetts in the summer with acute binocular diplopia when looking down and to the left, which starte
293 However, 10 of 12 patients never experienced diplopia when the nonfoveal face was presented to tempor
296 Our predictive model rules out postoperative diplopia with an 87.9% sensitivity and a 95.8% NPV for a
297 alry-type diplopia as presenting symptomatic diplopia with evidence of retinal misregistration, and w
298 weeks is recommended in cases of symptomatic diplopia with positive forced ductions and evidence of o
300 acture thought to be in need of repair, with diplopia within 30 degrees of primary gaze, and/or enoph