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1 ryotic organisms can be exploited to provide disease resistance.
2 ection, was targeted through RNAi to develop disease resistance.
3 8 family affects expression of NLR mRNAs and disease resistance.
4 spikelets, depicting the role of HvWRKY23 in disease resistance.
5 include cis-elements known to be involved in disease resistance.
6 e thought to have an important role in plant disease resistance.
7 inhibition of H2O2 degradation and enhanced disease resistance.
8 logous repair function may be a mechanism of disease resistance.
9 bition of host signaling networks, restoring disease resistance.
10 susceptibility to vaccine therapy, and human disease resistance.
11 eeding programs aiming to improve growth and disease resistance.
12 reduced in NOD-Idd22 mice, correlating with disease resistance.
13 growth has resulted in temperature-resilient disease resistance.
14 genome editing technologies for engineering disease resistance.
15 nsporters play a role in sugar signaling for disease resistance.
16 tissues and may therefore be involved in the disease resistance.
17 ene silencing in pathogenic fungi and confer disease resistance.
18 necdotally linked to enhanced broad-spectrum disease resistance.
19 rm interfering TALEs, or iTALEs) to overcome disease resistance.
20 prediction models and genomic selection for disease resistance.
21 of BAK1 leads to enhanced virulence, but not disease resistance.
22 rograms trying to incorporate broad-spectrum disease resistance.
23 rs autophagy in the host that coincides with disease resistance.
24 responsiveness and broad-spectrum bacterial disease resistance.
25 roles of HCT1806 and HCT4918 in Rp1-mediated disease resistance.
26 lective elimination of aggregates may confer disease resistance.
27 in expression did not compensate for loss of disease resistance.
28 nes may be enriched in functions involved in disease resistance.
29 o essential for sugars and glycerol-mediated disease resistance.
30 rly life and with implications for metabolic disease resistance.
31 (Hordeum vulgare) MORCs also are involved in disease resistance.
32 eaks, management strategies and breeding for disease resistance.
33 xity of AML biology and of the mechanisms of disease resistance.
34 sitive and eliminate the negative aspects of disease resistance.
35 carcass and meat quality, reproduction, and disease resistance.
36 nd how to generate crops with broad-spectrum disease resistance.
37 echanisms underlying MAPK functions in plant disease resistance.
38 25 is required for maintaining XA21-mediated disease resistance.
39 advanced our understanding of broad-spectrum disease resistance.
40 ogen-induced salicylic acid accumulation and disease resistance.
41 volved in antioxidation, detoxification, and disease resistance.
42 s symbiont colonization without compromising disease resistance.
43 associated with immunological variability or disease resistance.
44 ressing AtOXR2 (oeOXR2 plants) show enhanced disease resistance.
45 y compromises RPW8.1-mediated cell death and disease resistance.
46 riggering hypersensitive cell death (HR) and disease resistance.
47 e for breeding and molecular engineering for disease resistance.
48 e connections with variation in quantitative disease resistance.
49 improve marker-based selection for pancreas disease resistance.
50 es could contribute to SCA6's broad-spectrum disease resistance.
51 H(2) O(2) concentration and thereby improve disease resistance.
52 rotein kinase BWMK1, which is a regulator of disease resistance.
53 alized metabolism can contribute to improved disease resistance.
54 anism may have different breeding merits for disease resistance.
55 ediated by SA and JA, leading to compromised disease resistance.
56 volutionary context of breeding for improved disease resistance.
57 re enriched for important traits, especially disease resistance.
58 s) Pht1 family and also plays a role in rice disease resistance.
59 substitution in the CsSGR protein, and thus disease resistance.
60 th Oas1b protein confers flavivirus-specific disease resistance.
61 NLs is encoded by 2 gene families, ACTIVATED DISEASE RESISTANCE 1 (ADR1) and N REQUIREMENT GENE 1 (NR
62 lators of stress signaling, such as ENHANCED DISEASE RESISTANCE 1 (EDR1), ensure that stress response
64 bind to tobacco cytoplasmic kinase ENHANCED DISEASE RESISTANCE 1-like (EDR1-like), which triggers th
65 ly improve a range of crop traits, including disease resistance, abiotic stress tolerance, yield, nut
66 ransfer DNA insertion lines exhibit enhanced disease resistance after inoculation with virulent Pseud
67 of new technologies for engineering durable disease resistance against major pathogens of poplar and
70 SmD3b, or PRMT5 function results in enhanced disease resistance against the virulent oomycete pathoge
71 ust have the complete package of high yield, disease resistance, agronomic performance, and end-use q
74 naturally occurring performance-enhancing or disease- resistance alleles, including alteration of sin
75 ve pressure driving the local persistence of disease resistance among its wildlife hosts in endemic a
76 ically consider survival as an indicator for disease resistance (an individual's propensity to avoid
77 oligodendrocyte glycoprotein (MOG) promotes disease resistance and CD4(+) T cell deletion within the
80 ing to identify genotypes that show signs of disease resistance and leverage these genotypes in resto
81 tion mutants wrky22-1 and wrky22-2 had lower disease resistance and lower induction of innate immunit
82 l be no shortage of GE applications totackle disease resistance and other farmer and consumer priorit
83 l functions, including genes associated with disease resistance and others involved in morphological
84 r traits such as seed size and color, foliar disease resistance and others, also providing a cornerst
85 omposition is intimately connected to fungal disease resistance and outline a potential route for eng
88 ock-defense cross talk could help to improve disease resistance and productivity in economically impo
89 d its performance by predicting quantitative disease resistance and quantitative functional traits in
92 e-rich repeat protein repertoire involved in disease resistance and the characterization of Sm1(6), a
94 phological and physiological traits, such as disease resistance and yield that need to be maintained
98 scope of implementing genomics selection for disease resistance, and specifically for quantitative re
100 ggest that breeding for traits involved with disease resistance, and tolerance to cold- and heat-indu
102 ty may be functionally important in terms of disease-resistance, and that Bd prevalence and/or host s
103 ying mechanisms of how plant development and disease resistance are coordinately programed remain elu
104 fficient breeding process in which trials of disease resistance are integrated with other traits.
108 tative probenazole inducible protein), plant disease resistance as well as enzymes involved in flavon
109 ssential knowledge for understanding genetic disease resistance as well as local adaptation to changi
112 begin development of durable (long-lasting) disease resistance beyond the limits imposed by conventi
113 that the two receptors contribute equally to disease resistance both on the leaf surface and in the a
115 e altered under diet-restriction to increase disease resistance, but our findings suggest that increa
116 ic improvement of production traits, such as disease resistance, but progress is limited by the herit
117 cesses including endoreduplication and plant disease resistance, but the molecular mechanism underlyi
118 umans and animals support the development of disease resistance, but we do not understand the mechani
119 ion patterns correlate with high stress- and disease-resistance, but proximate mechanisms for this tr
120 unctions for NRT2.1 that may influence plant disease resistance by down-regulating biotic stress defe
121 er protective MHC class II molecules, afford disease resistance by engaging a naturally occurring con
122 mbers execute their function and spectrum of disease resistance by recognizing the cognate TALEs in p
123 mbers execute their function and spectrum of disease resistance by recognizing the cognate TALEs in t
124 MAPKs or MPKs), play critical roles in plant disease resistance by regulating multiple defense respon
126 ed for fast growth and families selected for disease resistance can alter their mechanisms of calcite
129 dentification of genes controlling QTL-based disease resistance contributes to breeding for cultivars
130 d high-quality diets did not differ in their disease resistance, despite differing in their body cond
132 dopsis thaliana compromises host and nonhost disease resistance due to open stomata during pathogen i
133 ad positive effects on enhancing anthracnose disease resistance during storage and also gave a residu
136 related to major agronomic traits, including disease resistance, flowering time, glucosinolate metabo
138 sistance in plants by means of an endogenous disease resistance gene from Arabidopsis thaliana named
139 redicted protein-coding genes, including 292 disease resistance gene homologs, and nine genes determi
140 n essential H3K9 histone demethylase and the disease resistance gene RECOGNITION OF PERONOSPORA PARAS
141 a TE inserted into the Arabidopsis thaliana disease resistance gene RPP7 recruited the histone mark
143 ucine-rich-repeat proteins (NLRs), the major disease-resistance gene families, has been unexplored in
144 e to pathogen attack, whereas enhancement of disease resistance generally compromises crop yield.
146 eotide-binding, leucine-rich repeat (NB-LRR) disease resistance genes by small RNAs is particularly u
151 As part of a long-term goal to define key disease resistance genes in cacao, here we use a transcr
152 his work that artificial evolution of NB-LRR disease resistance genes in crops can be enhanced by mod
153 The chitinase gene clusters and NBS-LRR disease resistance genes in this region suggest the poss
154 uggest that long-term balancing selection on disease resistance genes may have maintained ancestral h
155 e, and showed that new genes, exemplified by disease resistance genes, are preferentially located in
156 t-dominated loci were especially common from disease resistance genes, including from a large number
157 ubgenomes and use them to identify candidate disease resistance genes, to guide tetraploid transcript
161 family analysis showed a significant loss of disease-resistance genes such as those encoding NB-ARC d
164 nt progress in plant genetic engineering for disease resistance highlights future challenges and oppo
165 nd provides an important mechanism for their disease resistance; however, many aspects of the cell wa
166 tion, there has been success in breeding for disease resistance; however, much of this work and resea
167 sduction pathways in cancer progression, and disease resistance; (ii) intratumoral heterogeneity and
168 ded NLR proteins have a role in quantitative disease resistance in addition to dominant gene resistan
175 atic industrial products as well as increase disease resistance in energy and agricultural crops.
178 complex genomic architecture of quantitative disease resistance in long-generation trees, and constit
180 ested the condition-dependence of growth and disease resistance in male and female Gryllus texensis f
181 of disease incidence and severity, a lack of disease resistance in planting materials, shortages of l
185 and tbr mutants also suppress powdery mildew disease resistance in pmr6, a mutant defective in a puta
190 results also indicate that studying extreme disease resistance in the face of extensive exposure can
191 ustrate how bacterial effectors that trigger disease resistance in the host can evolve to interfere w
193 antitative trait locus (QTL) mapping for the disease resistances in Gy14 and further map-based clonin
195 causal SNP was significantly associated with disease resistances in natural and breeding populations.
196 ons for white pine blister rust quantitative disease resistance included 453 SNPs involved in wide bi
197 four DEMs regulating three DEGs involved in disease resistance, including miR156, miR172f, miR172g,
200 ization of the genetic components underlying disease resistance is a major research area in maize whi
209 ght into molecular mechanisms that determine disease resistance levels in Vitis species native to the
210 anisms controlling variation in quantitative disease resistance loci are not well understood in plant
214 mplications for the understanding of natural disease resistance mechanisms at the species level and f
217 multiple pathogens broadens the spectrum of disease resistances mediated by single plant immune rece
220 subspecies characteristics, particularly in disease resistance of indica and cold-stress tolerance o
228 d (S)NO concentrations, fully suppressed the disease resistance phenotype established from SRG3 but n
229 ne is a negative regulator of cell death and disease resistance, possibly through regulating ROS and
230 tectures, including growth, development, and disease-resistance properties, measured in a Pinus taeda
231 og of the Arabidopsis (Arabidopsis thaliana) disease resistance protein 1 protein in Triticum urartu
232 very and characterization of the Arabidopsis disease resistance protein RPS5 and its guardee PBS1, th
233 ssinosteroid signaling, peroxisome function, disease resistance, protein phosphorylation and light pe
234 itive selection and there were more diverged disease resistance proteins in the more widely distribut
235 ed families of genes, such as those encoding disease resistance proteins or transcription factors.
236 nucleotide-binding leucine-rich repeat (NLR) disease resistance proteins recognize specific pathogen
237 enzymes involved in rubber biosynthesis and disease resistance proteins that are expanded in the gen
241 ed to identify loci involved in Quantitative Disease Resistance (QDR) to different isolates of the pl
245 us vulgaris) that is associated with several disease resistance (R) genes of known function, includin
246 h is associated with increased expression of disease resistance (R) genes similar to the animal NOD1
247 cated crop species harbor multiple, diverse, disease resistance (R) genes that could be used to engin
248 have evolved a limited repertoire of NB-LRR disease resistance (R) genes to protect themselves again
250 ion of both PAMP-triggered basal defense and disease resistance (R) protein-mediated defense in Musa
252 gered immunity (ETI) is activated when plant disease resistance (R) proteins recognize the presence o
254 derived metabolites, and the accumulation of disease resistance-related secondary metabolites and dif
255 veral gene families in J. hindsii, including disease resistance-related wall-associated kinase (WAK),
258 e we provide evidence that the RCT1-mediated disease resistance requires the combined presence of the
259 ry biology, with implications for predicting disease resistance, response to environmental change, an
260 lecular pattern-triggered immunity (PTI) and disease resistance responses to different types of patho
261 Efe-AfpA may therefore be a component of the disease resistance seen in endophyte-infected strong cre
263 r examination of known micro RNAs related to disease-resistance, showed significant down-regulation o
264 suggested that MLA10-mediated cell-death and disease resistance signaling occur independently, in the
268 food security, including its roles in plant disease resistance, stress tolerance, and crop yield, an
269 es were found to be involved in quantitative disease resistance, suggesting these newly reported gene
270 nctional role(s) of occlusions in host plant disease resistance/susceptibility remains controversial.
271 evaluate the breeding value of the material, disease resistance tests and agronomical trait investiga
273 a high-quality diet had significantly poorer disease resistance than females on a low-quality diet an
275 enerally, our work shows that in addition to disease resistance, the costs of immunity vary between i
278 plants exhibit dwarfed morphology, enhanced disease resistance to bacteria and increased PAMP-trigge
279 line variation, but increases (decreases) in disease resistance to E. muscae are not consistently ass
280 expression of CRK28 in Arabidopsis increased disease resistance to P. syringae Expression of CRK28 in
282 lant resistance gene conferring quantitative disease resistance to plants against Fusarium species.
283 to leaf spontaneous cell death and enhanced disease resistance to powdery mildew via the SA-dependen
284 We found that fzl mutants showed enhanced disease resistance to the bacterial pathogen Pseudomonas
285 e known and hypothesized roles in autoimmune diseases, resistance to viruses, reproductive conditions
286 f multigene segments, using as the example a disease resistance trait of high economic importance.
287 analysis showed that certain metabolomic and disease-resistance traits are largely controlled by the
290 vely regulate RPW8.1-mediated cell death and disease resistance via suppressing RPW8.1 expression.
291 In addition, we show that ERA1 function in disease resistance was independent of its role in stomat
293 otein and non-protein coding genes for which disease resistance was the first biological annotation.
295 cell wall strengthening, stress response and disease resistance were differentially expressed in wate
296 Key genes potentially involved in cacao disease resistance were identified by transcriptomic ana
297 egions is sufficient to provide quantitative disease resistance, which is associated with genome-wide
298 essed QTL can greatly improve broad-spectrum disease resistance while having only limited and inconsi
299 whole panel identified 29 QTL for height and disease resistance with allelic variation across donors.
300 n rice enables us to engineer broad-spectrum disease resistance without compromising plant fitness in