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1 ocytes may be caused by increased mechanical distending and shear forces and/or impaired adhesion to
2 urgically prepare a mouse for CRD, construct distending balloons, distend the colon, and accumulate a
4 uit allowed independent control of pressures distending carotid, aortic and coronary baroreceptors.
5 nnective tissue compartments accountable for distending digital pulp under normal circumstances and s
6 nd NO synthesis, intermittent compressive or distending forces applied to ex vivo nonbeating hearts w
8 of the H. pullorum strains induced cellular distending phenotype, actin cytoskeleton remodeling, and
9 ce, and enhanced left ventricular transmural distending pressure (all P <0.01), with no increase in p
14 iffness and vasodilatation, as a function of distending pressure derived by photo-plethysmographic vo
15 the relationship between Zrs and continuous distending pressure during an increasing/decreasing cont
16 ography-derived lung volume, high continuous distending pressure had adverse cardiopulmonary effects.
17 l; 3) to evaluate how the optimal continuous distending pressure identified by Zrs relates to the poi
19 cy ventilation was initiated at a continuous distending pressure of 10 cm H2O and incrementally incre
22 lung mechanics and improving the continuous distending pressure optimization during high-frequency o
23 tion limb, identifying an optimal continuous distending pressure that was, on average, 1.1 +/- 1.7 cm
24 minates throughout gestation and generates a distending pressure to stretch the lung and stimulate gr
27 e during an increasing/decreasing continuous distending pressure trial; 3) to evaluate how the optima
29 ary arterioles (diameter, 90.0+/-3.4 microm; distending pressure, 20 mm Hg) preconstricted by 30% to
30 Provided transpulmonary pressure is the lung-distending pressure, and that chest wall elastance may v
31 ation relies on the generation of a constant distending pressure, small tidal volumes and rapid respi
35 nts with high Ers are at risk of excess lung-distending pressures and may derive greater clinical ben
36 rtic compliance over a wide range of passive distending pressures, and to study pharmacologically ind
41 reased activity following termination of the distending stimulus whereas Type II neurons typically ha
42 ction to the perfusion circuit and pressures distending the aortic arch, carotid sinus and coronary a
43 blind-ending vessels from the ventral aorta, distending the arteries and precipitating fusion with an
46 etemcomitans strains that express cytolethal distending toxin (Cdt) are associated with localized agg
47 eptible mouse strains, produces a cytolethal distending toxin (CDT) consisting of CdtA, CdtB, and Cdt
48 paticus expresses a member of the cytolethal distending toxin (CDT) family of bacterial cytotoxins.
51 ents, we recently showed that the cytolethal distending toxin (Cdt) from the periodontal pathogen Agg
52 tion was reduced by flagellum and cytolethal distending toxin (CDT) gene mutants, treatment of the su
53 the role of the virulence factor cytolethal distending toxin (CDT) in the pathogenesis of this organ
58 inobacillus actinomycetemcomitans cytolethal distending toxin (Cdt) is a potent immunotoxin that indu
59 egatibacter actinomycetemcomitans cytolethal distending toxin (Cdt) is dependent upon the integrity o
60 egatibacter actinomycetemcomitans cytolethal distending toxin (Cdt) is dependent upon the integrity o
69 inobacillus actinomycetemcomitans cytolethal distending toxin (Cdt) results in dose-dependent G2 arre
70 B polypeptide of Escherichia coli cytolethal distending toxin (CDT) shares significant pattern-specif
71 Campylobacter jejuni encodes a cytolethal distending toxin (CDT) that causes cells to arrest in th
72 actinomycetemcomitans produces a cytolethal distending toxin (Cdt) that inhibits the proliferation o
73 hilus ducreyi expresses a soluble cytolethal distending toxin (CDT) that is encoded by the cdtABC gen
74 hilus ducreyi expresses a soluble cytolethal distending toxin (CDT) that kills HeLa, HEp-2, and other
76 erichia coli strains that produce cytolethal distending toxin (CDT) were analyzed for their virulence
80 Many H. ducreyi strains express cytolethal distending toxin (CDT), and recombinant CDT causes apopt
85 ential virulence factors, such as cytolethal distending toxin (CDT), in vivo are poorly understood.
86 regions in the leukotoxin (lkt), cytolethal distending toxin (cdt), major fimbrial subunit (flp-1),
88 ia have evolved a toxin, known as cytolethal distending toxin (CDT), that has the ability to control
89 code a multisubunit toxin, termed cytolethal distending toxin (CDT), that induces cell cycle arrest,
90 r jejuni produces a toxin, called cytolethal distending toxin (CDT), which causes direct DNA damage l
91 ny bacterial pathogens encode the cytolethal distending toxin (CDT), which causes host cells to arres
99 ; (b) the newly discovered toxin, cytolethal distending toxin (CDT); and (c) a secreted chaperonin 60
100 suppressive factor encoded by the cytolethal distending toxin (cdt)B gene, which is homologous to a f
102 act homologs of the gene encoding cytolethal distending toxin (cdtB), which interrupts the eukaryotic
103 notably diarrhea-associated cdtB (cytolethal distending toxin [46%]) and urinary tract infection-asso
108 shed, but the association between cytolethal distending toxin and disease is incompletely understood.
109 the evolution of two toxin genes-cytolethal distending toxin B (cdtB) and apoptosis inducing protein
110 rcially available version of anti-cytolethal distending toxin B and anti-vinculin antibodies, and tes
111 ys residue, a CdtB homologue from cytolethal distending toxin can form a functional complex with ArtA
112 ylobacter coli, the production of cytolethal distending toxin correlated positively (P < 0.0001) only
114 tion of the H. hepaticus-specific cytolethal distending toxin gene and showed similar animal coloniza
116 ted the role of LuxS in motility, cytolethal distending toxin production, agglutination, and intestin
117 cretion system needle protein, or cytolethal distending toxin revealed a direct correlation between t
118 encoding toxins of animal cells, cytolethal distending toxin subunit B (cdtB) and apoptosis-inducing
120 ), gafD (G fimbriae), cnf1, cdtB (cytolethal distending toxin), cvaC (colicin V), and ibeA (invasion
122 ed genes needed for the action of cytolethal distending toxin, including a cell-surface protein that
123 nostic assay: hemolysin E (HlyE), cytolethal distending toxin, S. Typhi lipopolysaccharide (LPS), and
124 ogue of the active subunit of the cytolethal distending toxin, which causes DNA damage leading to cel
126 verall the pathotypes, apart from cytolethal distending toxin-producing E. coli, were recovered both
130 hich is homologous to a family of cytolethal distending toxins (Cdt) expressed by several gram-negati
131 hich is homologous to a family of cytolethal distending toxins (Cdt) expressed by several Gram-negati